BibTeX

@misc{6f36f0acfdac1c222a61919e07ffc189c68e49b4,
  author = {C. E. Landhuis and R. Poulton and D. Welch and R. Hancox},
  title = {Childhood Sleep Time and Long-Term Risk for Obesity: A 32-Year Prospective Birth Cohort Study},
  journal = {Pediatrics},
  year = {2008},
  volume = {122},
  pages = {955 - 960},
  doi = {10.1542/peds.2007-3521},
  abstract = {OBJECTIVE. Associations between short sleep duration and increased BMI have been found in children and adults. However, it is not known whether short sleep time during childhood has long-term consequences. We assessed the association between sleep time in childhood and adult BMI in a birth cohort. METHODS. Study members were a general-population birth cohort of 1037 participants (502 female) who were born in Dunedin, New Zealand, between April 1972 and March 1973. Parental reports of bedtimes and rising times collected at ages 5, 7, 9, and 11 years were used to estimate childhood sleep time. Linear regression analysis was used to analyze the association between childhood sleep time and BMI measured at 32 years of age. RESULTS. Shorter childhood sleep times were significantly associated with higher adult BMI values. This association remained after adjustment for adult sleep time and the potential confounding effects of early childhood BMI, childhood socioeconomic status, parental BMIs, child and adult television viewing, adult physical activity, and adult smoking. In logistic regression analyses, more sleep time during childhood was associated with lower odds of obesity at 32 years of age. This association was significant after adjustment for multiple potential confounding factors. CONCLUSIONS. These findings suggest that sleep restriction in childhood increases the long-term risk for obesity. Ensuring that children get adequate sleep may be a useful strategy for stemming the current obesity epidemic.}
}

RIS

TY  - JOUR
AU  - C. E. Landhuis
AU  - R. Poulton
AU  - D. Welch
AU  - R. Hancox
T1  - Childhood Sleep Time and Long-Term Risk for Obesity: A 32-Year Prospective Birth Cohort Study
JO  - Pediatrics
PY  - 2008
VL  - 122
SP  - 955 - 960
EP  - 955 - 960
DO  - 10.1542/peds.2007-3521
AB  - OBJECTIVE. Associations between short sleep duration and increased BMI have been found in children and adults. However, it is not known whether short sleep time during childhood has long-term consequences. We assessed the association between sleep time in childhood and adult BMI in a birth cohort. METHODS. Study members were a general-population birth cohort of 1037 participants (502 female) who were born in Dunedin, New Zealand, between April 1972 and March 1973. Parental reports of bedtimes and rising times collected at ages 5, 7, 9, and 11 years were used to estimate childhood sleep time. Linear regression analysis was used to analyze the association between childhood sleep time and BMI measured at 32 years of age. RESULTS. Shorter childhood sleep times were significantly associated with higher adult BMI values. This association remained after adjustment for adult sleep time and the potential confounding effects of early childhood BMI, childhood socioeconomic status, parental BMIs, child and adult television viewing, adult physical activity, and adult smoking. In logistic regression analyses, more sleep time during childhood was associated with lower odds of obesity at 32 years of age. This association was significant after adjustment for multiple potential confounding factors. CONCLUSIONS. These findings suggest that sleep restriction in childhood increases the long-term risk for obesity. Ensuring that children get adequate sleep may be a useful strategy for stemming the current obesity epidemic.
ER  - 

BibTeX

@misc{a2561ff2ca104e38b739ae569c00f9dac48524e7,
  author = {Emily K. Snell and E. Adam and G. Duncan},
  title = {Sleep and the body mass index and overweight status of children and adolescents.},
  journal = {Child development},
  year = {2007},
  volume = {78 1},
  pages = {
          309-23
        },
  doi = {10.1111/J.1467-8624.2007.00999.X},
  abstract = {Associations between sleep and the body mass index (BMI) and overweight status of children and adolescents were estimated using longitudinal data from a nationally representative sample of 2,281 children aged 3-12 years at baseline. Controlling for baseline BMI, children who slept less, went to bed later, or got up earlier at the time of the first assessment had higher BMIs 5 years later and were more likely to be overweight. Child age moderated the relationship between bedtime and BMI. In addition, the study reports nationally representative data on the sleep habits of American children aged 3-18 years. This study underscores the likely importance of sleep on children's physical health and suggests that sleep is important for understanding childhood weight problems.}
}

RIS

TY  - JOUR
AU  - Emily K. Snell
AU  - E. Adam
AU  - G. Duncan
T1  - Sleep and the body mass index and overweight status of children and adolescents.
JO  - Child development
PY  - 2007
VL  - 78 1
SP  - 
          309-23
        
EP  - 
          309-23
        
DO  - 10.1111/J.1467-8624.2007.00999.X
AB  - Associations between sleep and the body mass index (BMI) and overweight status of children and adolescents were estimated using longitudinal data from a nationally representative sample of 2,281 children aged 3-12 years at baseline. Controlling for baseline BMI, children who slept less, went to bed later, or got up earlier at the time of the first assessment had higher BMIs 5 years later and were more likely to be overweight. Child age moderated the relationship between bedtime and BMI. In addition, the study reports nationally representative data on the sleep habits of American children aged 3-18 years. This study underscores the likely importance of sleep on children's physical health and suggests that sleep is important for understanding childhood weight problems.
ER  - 

BibTeX

@misc{25151293bfc6a7f2feafc3fb5d655bc744a69775,
  author = {J. Bell and F. Zimmerman},
  title = {Shortened nighttime sleep duration in early life and subsequent childhood obesity.},
  journal = {Archives of pediatrics & adolescent medicine},
  year = {2010},
  volume = {164 9},
  pages = {
          840-5
        },
  doi = {10.1001/archpediatrics.2010.143},
  abstract = {OBJECTIVE
To test associations between daytime and nighttime sleep duration and subsequent obesity in children and adolescents.


DESIGN
Prospective cohort.


SETTING
Panel Survey of Income Dynamics Child Development Supplements (1997 and 2002) from US children.


PARTICIPANTS
Subjects aged 0 to 13 years (n = 1930) at baseline (1997).


MAIN EXPOSURES
Binary indicators of short daytime and nighttime sleep duration (<25th percentile of age-normalized sleep scores) at baseline.


MAIN OUTCOME MEASURES
Body mass index at follow-up (2002) was converted to age- and sex-specific z scores and trichotomized (normal weight, overweight, obese) using established cut points. Ordered logistic regression was used to model body mass index classification as a function of short daytime and nighttime sleep at baseline and follow-up, and important covariates included socioeconomic status, parents' body mass index, and, for children older than 4 years, body mass index at baseline.


RESULTS
For younger children (aged 0-4 years at baseline), short duration of nighttime sleep at baseline was strongly associated with increased risk of subsequent overweight or obesity (odds ratio = 1.80; 95% confidence interval, 1.16-2.80). For older children (aged 5-13 years), baseline sleep was not associated with subsequent weight status; however, contemporaneous sleep was inversely associated. Daytime sleep had little effect on subsequent obesity in either group.


CONCLUSIONS
Shortened sleep duration in early life is a modifiable risk factor with important implications for obesity prevention and treatment. Insufficient nighttime sleep among infants and preschool-aged children may be a lasting risk factor for subsequent obesity. Napping does not appear to be a substitute for nighttime sleep in terms of obesity prevention.}
}

RIS

TY  - JOUR
AU  - J. Bell
AU  - F. Zimmerman
T1  - Shortened nighttime sleep duration in early life and subsequent childhood obesity.
JO  - Archives of pediatrics & adolescent medicine
PY  - 2010
VL  - 164 9
SP  - 
          840-5
        
EP  - 
          840-5
        
DO  - 10.1001/archpediatrics.2010.143
AB  - OBJECTIVE
To test associations between daytime and nighttime sleep duration and subsequent obesity in children and adolescents.


DESIGN
Prospective cohort.


SETTING
Panel Survey of Income Dynamics Child Development Supplements (1997 and 2002) from US children.


PARTICIPANTS
Subjects aged 0 to 13 years (n = 1930) at baseline (1997).


MAIN EXPOSURES
Binary indicators of short daytime and nighttime sleep duration (<25th percentile of age-normalized sleep scores) at baseline.


MAIN OUTCOME MEASURES
Body mass index at follow-up (2002) was converted to age- and sex-specific z scores and trichotomized (normal weight, overweight, obese) using established cut points. Ordered logistic regression was used to model body mass index classification as a function of short daytime and nighttime sleep at baseline and follow-up, and important covariates included socioeconomic status, parents' body mass index, and, for children older than 4 years, body mass index at baseline.


RESULTS
For younger children (aged 0-4 years at baseline), short duration of nighttime sleep at baseline was strongly associated with increased risk of subsequent overweight or obesity (odds ratio = 1.80; 95% confidence interval, 1.16-2.80). For older children (aged 5-13 years), baseline sleep was not associated with subsequent weight status; however, contemporaneous sleep was inversely associated. Daytime sleep had little effect on subsequent obesity in either group.


CONCLUSIONS
Shortened sleep duration in early life is a modifiable risk factor with important implications for obesity prevention and treatment. Insufficient nighttime sleep among infants and preschool-aged children may be a lasting risk factor for subsequent obesity. Napping does not appear to be a substitute for nighttime sleep in terms of obesity prevention.
ER  - 

BibTeX

@misc{b5e8b451d29e23668b2dc73fd9009a7cf9380939,
  author = {James E. Gangwisch and D. Malaspina and B. Boden-Albala and S. Heymsfield},
  title = {Inadequate sleep as a risk factor for obesity: analyses of the NHANES I.},
  journal = {Sleep},
  year = {2005},
  volume = {28 10},
  pages = {
          1289-96
        },
  doi = {10.1093/SLEEP/28.10.1289},
  abstract = {STUDY OBJECTIVES
Sleep deprivation has been hypothesized to contribute toward obesity by decreasing leptin, increasing ghrelin, and compromising insulin sensitivity. This study examines cross-sectional and longitudinal data from a large United States sample to determine whether sleep duration is associated with obesity and weight gain.


DESIGN
Longitudinal analyses of the 1982-1984, 1987, and 1992 NHANES I Followup Studies and cross-sectional analysis of the 1982-1984 study.


SETTING
Probability sample of the civilian noninstitutionalized population of the United States.


PARTICIPANTS
Sample sizes of 9,588 for the cross-sectional analyses, 8,073 for the 1987, and 6,981 for the 1992 longitudinal analyses.


MEASUREMENTS AND RESULTS
Measured weight in 1982-1984 and self-reported weights in 1987 and 1992. Subjects between the ages of 32 and 49 years with self-reported sleep durations at baseline less than 7 hours had higher average body mass indexes and were more likely to be obese than subjects with sleep durations of 7 hours. Sleep durations over 7 hours were not consistently associated with either an increased or decreased likelihood of obesity in the cross-sectional and longitudinal results. Each additional hour of sleep at baseline was negatively associated with change in body mass index over the follow-up period, but this association was small and statistically insignificant.


CONCLUSIONS
These findings support the hypothesis that sleep duration is associated with obesity in a large longitudinally monitored United States sample. These observations support earlier experimental sleep studies and provide a basis for future studies on weight control interventions that increase the quantity and quality of sleep.}
}

RIS

TY  - JOUR
AU  - James E. Gangwisch
AU  - D. Malaspina
AU  - B. Boden-Albala
AU  - S. Heymsfield
T1  - Inadequate sleep as a risk factor for obesity: analyses of the NHANES I.
JO  - Sleep
PY  - 2005
VL  - 28 10
SP  - 
          1289-96
        
EP  - 
          1289-96
        
DO  - 10.1093/SLEEP/28.10.1289
AB  - STUDY OBJECTIVES
Sleep deprivation has been hypothesized to contribute toward obesity by decreasing leptin, increasing ghrelin, and compromising insulin sensitivity. This study examines cross-sectional and longitudinal data from a large United States sample to determine whether sleep duration is associated with obesity and weight gain.


DESIGN
Longitudinal analyses of the 1982-1984, 1987, and 1992 NHANES I Followup Studies and cross-sectional analysis of the 1982-1984 study.


SETTING
Probability sample of the civilian noninstitutionalized population of the United States.


PARTICIPANTS
Sample sizes of 9,588 for the cross-sectional analyses, 8,073 for the 1987, and 6,981 for the 1992 longitudinal analyses.


MEASUREMENTS AND RESULTS
Measured weight in 1982-1984 and self-reported weights in 1987 and 1992. Subjects between the ages of 32 and 49 years with self-reported sleep durations at baseline less than 7 hours had higher average body mass indexes and were more likely to be obese than subjects with sleep durations of 7 hours. Sleep durations over 7 hours were not consistently associated with either an increased or decreased likelihood of obesity in the cross-sectional and longitudinal results. Each additional hour of sleep at baseline was negatively associated with change in body mass index over the follow-up period, but this association was small and statistically insignificant.


CONCLUSIONS
These findings support the hypothesis that sleep duration is associated with obesity in a large longitudinally monitored United States sample. These observations support earlier experimental sleep studies and provide a basis for future studies on weight control interventions that increase the quantity and quality of sleep.
ER  - 

BibTeX

@misc{12492b9b48a1dd790a143244604ab04ce2ba12c6,
  author = {J. Chaput and J. Despres and C. Bouchard and A. Tremblay},
  title = {The association between sleep duration and weight gain in adults: a 6-year prospective study from the Quebec Family Study.},
  journal = {Sleep},
  year = {2008},
  volume = {31 4},
  pages = {
          517-23
        },
  doi = {10.1093/SLEEP/31.4.517},
  abstract = {STUDY OBJECTIVE
To investigate the relationship between sleep duration and subsequent body weight and fat gain.


DESIGN
Six-year longitudinal study.


SETTING
Community setting.


PARTICIPANTS
Two hundred seventy-six adults aged 21 to 64 years from the Quebec Family Study. More than half of the sample is drawn from families with at least 1 parent and 1 offspring with a body mass index of 32 kg/m2 or higher.


MEASUREMENTS AND RESULTS
Body composition measurements and self-reported sleep duration were determined. Changes in adiposity indices were compared between short- (5-6 hours), average- (7-8 hours), and long- (9-10 hours) duration sleeper groups. After adjustment for age, sex, and baseline body mass index, short-duration sleepers gained 1.98 kg (95% confidence interval: 1.16-2.82) more and long-duration sleepers gained 1.58 kg (95% CI: 1.02-2.56) more than did average-duration sleepers over 6 years. Short- and long-duration sleepers were 35% and 25% more likely to experience a 5-kg weight gain, respectively, as compared with average-duration sleepers over 6 years. The risk of developing obesity was elevated for short- and long-duration sleepers as compared with average-duration sleepers, with 27% and 21% increases in risk, respectively. These associations remained significant after inclusion of important covariates and were not affected by adjustment for energy intake and physical activity participation.


CONCLUSIONS
This study provides evidence that both short and long sleeping times predict an increased risk of future body weight and fat gain in adults. Hence, these results emphasize the need to add sleep duration to the panel of determinants that contribute to weight gain and obesity.}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - J. Despres
AU  - C. Bouchard
AU  - A. Tremblay
T1  - The association between sleep duration and weight gain in adults: a 6-year prospective study from the Quebec Family Study.
JO  - Sleep
PY  - 2008
VL  - 31 4
SP  - 
          517-23
        
EP  - 
          517-23
        
DO  - 10.1093/SLEEP/31.4.517
AB  - STUDY OBJECTIVE
To investigate the relationship between sleep duration and subsequent body weight and fat gain.


DESIGN
Six-year longitudinal study.


SETTING
Community setting.


PARTICIPANTS
Two hundred seventy-six adults aged 21 to 64 years from the Quebec Family Study. More than half of the sample is drawn from families with at least 1 parent and 1 offspring with a body mass index of 32 kg/m2 or higher.


MEASUREMENTS AND RESULTS
Body composition measurements and self-reported sleep duration were determined. Changes in adiposity indices were compared between short- (5-6 hours), average- (7-8 hours), and long- (9-10 hours) duration sleeper groups. After adjustment for age, sex, and baseline body mass index, short-duration sleepers gained 1.98 kg (95% confidence interval: 1.16-2.82) more and long-duration sleepers gained 1.58 kg (95% CI: 1.02-2.56) more than did average-duration sleepers over 6 years. Short- and long-duration sleepers were 35% and 25% more likely to experience a 5-kg weight gain, respectively, as compared with average-duration sleepers over 6 years. The risk of developing obesity was elevated for short- and long-duration sleepers as compared with average-duration sleepers, with 27% and 21% increases in risk, respectively. These associations remained significant after inclusion of important covariates and were not affected by adjustment for energy intake and physical activity participation.


CONCLUSIONS
This study provides evidence that both short and long sleeping times predict an increased risk of future body weight and fat gain in adults. Hence, these results emphasize the need to add sleep duration to the panel of determinants that contribute to weight gain and obesity.
ER  - 

BibTeX

@misc{b74142d60523a0cead8a5343af3155cb65ebfa3e,
  author = {J. Theorell-Haglöw and C. Janson and C. Berne and E. Lindberg},
  title = {Sleep duration and obesity in women - A 10 year prospective study},
  journal = {European Respiratory Journal},
  year = {2012},
  volume = {40},
  pages = {1887},
  abstract = {Background: Obesity is highly related to obstructive sleep apnea syndrome. Research has shown that one potential cause of obesity may be short sleep duration. Aim: The aim was to assess how sleep duration and obesity is related over a 10 year period, in a population-based sample of women.

Methods: A total of 5,003 non-pregnant women (response rate 80 %) >=30 years, answered a 10-year follow-up questionnaire. The questionnaire included questions on sleep duration, weight, height, waist circumference, snoring and life style factors. Regression analysis was performed to analyze independent associations between sleep duration and measures of obesity.

Results: In the whole population 31% (n=2,127) of the women had increased their weight by at least 15 kg, 37% (n=2,549) had a BMI>=30 kg/m2 and 52% were centrally obese (waist circumference ≥88cm). Both short (<6h) (OR=1.38; 95%CI 1.06-1.80) and long sleep duration (≥9h) (1.86; 1.32-2.62) showed to be risk factors for general obesity (BMI≥30 kg/m2) after controlling for confounders. When dividing the women by age both short and long sleep duration were risk factors in the younger age group (age at baseline <40 years). In women above age 40 years at baseline only long sleep duration remained as a risk factor for general obesity after controlling for confounders. In addition, short sleep duration was shown as a risk factor for central obesity after adjustments (1.46; 1.002-2.14) in younger women, whereas long sleep duration was a risk factor in women age ≥40 years (1.86; 1.17-2.96).

Conclusion: Both short and long sleeping women had a greater risk of general obesity compared with normal sleepers (6-9h). Young short sleepers were also more at risk for central obesity.}
}

RIS

TY  - JOUR
AU  - J. Theorell-Haglöw
AU  - C. Janson
AU  - C. Berne
AU  - E. Lindberg
T1  - Sleep duration and obesity in women - A 10 year prospective study
JO  - European Respiratory Journal
PY  - 2012
VL  - 40
SP  - 1887
EP  - 1887
AB  - Background: Obesity is highly related to obstructive sleep apnea syndrome. Research has shown that one potential cause of obesity may be short sleep duration. Aim: The aim was to assess how sleep duration and obesity is related over a 10 year period, in a population-based sample of women.

Methods: A total of 5,003 non-pregnant women (response rate 80 %) >=30 years, answered a 10-year follow-up questionnaire. The questionnaire included questions on sleep duration, weight, height, waist circumference, snoring and life style factors. Regression analysis was performed to analyze independent associations between sleep duration and measures of obesity.

Results: In the whole population 31% (n=2,127) of the women had increased their weight by at least 15 kg, 37% (n=2,549) had a BMI>=30 kg/m2 and 52% were centrally obese (waist circumference ≥88cm). Both short (<6h) (OR=1.38; 95%CI 1.06-1.80) and long sleep duration (≥9h) (1.86; 1.32-2.62) showed to be risk factors for general obesity (BMI≥30 kg/m2) after controlling for confounders. When dividing the women by age both short and long sleep duration were risk factors in the younger age group (age at baseline <40 years). In women above age 40 years at baseline only long sleep duration remained as a risk factor for general obesity after controlling for confounders. In addition, short sleep duration was shown as a risk factor for central obesity after adjustments (1.46; 1.002-2.14) in younger women, whereas long sleep duration was a risk factor in women age ≥40 years (1.86; 1.17-2.96).

Conclusion: Both short and long sleeping women had a greater risk of general obesity compared with normal sleepers (6-9h). Young short sleepers were also more at risk for central obesity.
ER  - 

BibTeX

@misc{6ed1657c6202011debbd11f8a14edafb041dddb2,
  author = {J. Chaput},
  title = {Short sleep duration promoting overconsumption of food: A reward-driven eating behavior?},
  journal = {Sleep},
  year = {2010},
  volume = {33 9},
  pages = {
          1135-6
        },
  doi = {10.1093/SLEEP/33.9.1135},
  abstract = {THE PROOF-OF-CONCEPT FOR SHORT SLEEP DURATION AS A POSSIBLE CAUSE OF OBESITY IS GROWING.1,2 PROSPECTIVE COHORT STUDIES HAVE consistently shown that short sleep duration is associated with weight gain and incidence of obesity in children and adults. Intervention studies have provided a mechanistic explanation for the short sleep-obesity connection (i.e., that sleep restriction impacts hormonal regulation of appetite). Ongoing research is trying to determine whether sleep extension in sleep deprived obese individuals can influence appetite control and/or reduce body fat.3 Given the high prevalence of chronic sleep restriction, studies aiming to elucidate the possible causal association between short sleep duration and obesity are of utmost importance from a public health standpoint. 
 
The potential mechanisms by which short sleep duration may influence body weight are under scrutiny. In order to produce weight gain, reduced sleep must either increase energy intake and/or reduce energy expenditure. Decreased energy expenditure as an explanation for why short sleep duration is associated with weight gain requires further research. The increased fatigue and tiredness associated with sleeping too little generally decreases people's motivation to follow exercise regimens. In particular, recent results have shown that short-term sleep restriction (2 nights of 4 h in bed) was accompanied by a decrease in daytime spontaneous physical activity in healthy men.4 Interestingly, the reduction in overall physical activity was explained by a shift toward less intense activities under free-living conditions. Reductions in involuntary physical activity (also called non-exercise activity thermogenesis, or NEAT) may be another mechanism by which lack of sleep influences energy expenditure. However, more research using objective physical activity monitoring to track NEAT levels in relation to lack of sleep is needed in order to improve our understanding of the contribution of this energy expenditure component. 
 
On the other side of the energy balance equation, increased energy intake appears to be the most plausible explanation as to why short sleepers have a higher risk of becoming obese. Firstly, short sleep duration might increase the risk of weight gain by preventing the restoration of a hormonal profile facilitating appetite control (homeostatic regulation of feeding). Indeed, lack of sleep has been reported to decrease leptin levels, increase ghrelin levels, alter glucose homeostasis, and activate the orexin system.5 Secondly, lack of sleep could also lead to weight gain and obesity by increasing the time available for eating and by making the maintenance of a healthy lifestyle more difficult. In an environment where food is highly palatable and readily available, caloric intake may be directly proportional to the time spent awake, especially if most of waking hours are spent in sedentary activities, such as watching television, where snacking is common. This concept is supported by recent data showing that recurrent bedtime restriction under free-living conditions did not down-regulate the satiety hormone leptin nor up-regulate the appetite-stimulating hormone ghrelin, but increased intake of calories from snacks, which suggests hedonic rather than homeostatic mediators of the shift in food selection.6 Likewise, results from another study have shown that habitual short sleepers (average of 6 h/night) ate more often (i.e., > 3 meals/day, with more frequent nibbling) than did long sleepers.7 Hence, the hedonic value of food intake might play a crucial role in the short sleep-obesity link. 
 
The cross-sectional study of Weiss and colleagues in this issue of SLEEP provides further evidence to support the theory of the hedonic value of food intake associated with short sleep duration.8 Indeed, the authors observe that shorter sleep duration in adolescents was associated with a relatively higher caloric intake derived from fat and a lower caloric intake from carbohydrates. Additionally, shorter sleep duration was associated with a 2-fold increased risk of consuming ≥ 475 kcal/day from snacks in unadjusted analyses. As highlighted by the authors, increased snacking and consumption of high energy foods may reflect effects of short sleep duration on stress responses and reward seeking behaviors. Strengths of this study include (i) the use of an objective measure of sleep duration (wrist actigraphy); (ii) the quantitative measures of macronutrient intake (multiple-pass 24-h food recalls); and (iii) the free-living environment. 
 
An important target of future studies should be the non-homeostatic, reward-driven feeding behavior that could be affected by short sleep duration. Because bedtime restriction inevitably results in more extended exposure to readily available, energy-dense, palatable foods, the regulation of energy balance can be easily compromised in such circumstances. In particular, the advent of functional magnetic resonance imaging is likely to be instrumental in documenting food-related reward activation in the brain after sleep restriction.9 Interestingly, a recent study in adolescents reports that impaired sleep was associated with less reactivity of reward-related brain systems, suggesting that more exciting rewards are required to create the same level of neural activation.10 Because the orexin system is a critical regulator of sleep/wake states, and of feeding behavior and reward processes, it is a potentially important therapeutic target for the treatment of obesity.11 
 
Recent research and the mounting body of evidence regarding the health-related effects of short sleep duration remind us that sleep is not a waste of time and that voluntary sleep restriction can impact our health in various ways. Furthermore, the study of appetite control in our modern world reminds us that our quest for reward and pleasure is not finely tuned to our biology.12,13 With modern sedentary activities (e.g., television viewing, video game playing, cognitive working, listening to music), promoting overconsumption of food “in the absence of hunger,”14 a better understanding of these environmental stimuli is needed if we are to develop behavioral strategies to cope with them effectively.}
}

RIS

TY  - JOUR
AU  - J. Chaput
T1  - Short sleep duration promoting overconsumption of food: A reward-driven eating behavior?
JO  - Sleep
PY  - 2010
VL  - 33 9
SP  - 
          1135-6
        
EP  - 
          1135-6
        
DO  - 10.1093/SLEEP/33.9.1135
AB  - THE PROOF-OF-CONCEPT FOR SHORT SLEEP DURATION AS A POSSIBLE CAUSE OF OBESITY IS GROWING.1,2 PROSPECTIVE COHORT STUDIES HAVE consistently shown that short sleep duration is associated with weight gain and incidence of obesity in children and adults. Intervention studies have provided a mechanistic explanation for the short sleep-obesity connection (i.e., that sleep restriction impacts hormonal regulation of appetite). Ongoing research is trying to determine whether sleep extension in sleep deprived obese individuals can influence appetite control and/or reduce body fat.3 Given the high prevalence of chronic sleep restriction, studies aiming to elucidate the possible causal association between short sleep duration and obesity are of utmost importance from a public health standpoint. 
 
The potential mechanisms by which short sleep duration may influence body weight are under scrutiny. In order to produce weight gain, reduced sleep must either increase energy intake and/or reduce energy expenditure. Decreased energy expenditure as an explanation for why short sleep duration is associated with weight gain requires further research. The increased fatigue and tiredness associated with sleeping too little generally decreases people's motivation to follow exercise regimens. In particular, recent results have shown that short-term sleep restriction (2 nights of 4 h in bed) was accompanied by a decrease in daytime spontaneous physical activity in healthy men.4 Interestingly, the reduction in overall physical activity was explained by a shift toward less intense activities under free-living conditions. Reductions in involuntary physical activity (also called non-exercise activity thermogenesis, or NEAT) may be another mechanism by which lack of sleep influences energy expenditure. However, more research using objective physical activity monitoring to track NEAT levels in relation to lack of sleep is needed in order to improve our understanding of the contribution of this energy expenditure component. 
 
On the other side of the energy balance equation, increased energy intake appears to be the most plausible explanation as to why short sleepers have a higher risk of becoming obese. Firstly, short sleep duration might increase the risk of weight gain by preventing the restoration of a hormonal profile facilitating appetite control (homeostatic regulation of feeding). Indeed, lack of sleep has been reported to decrease leptin levels, increase ghrelin levels, alter glucose homeostasis, and activate the orexin system.5 Secondly, lack of sleep could also lead to weight gain and obesity by increasing the time available for eating and by making the maintenance of a healthy lifestyle more difficult. In an environment where food is highly palatable and readily available, caloric intake may be directly proportional to the time spent awake, especially if most of waking hours are spent in sedentary activities, such as watching television, where snacking is common. This concept is supported by recent data showing that recurrent bedtime restriction under free-living conditions did not down-regulate the satiety hormone leptin nor up-regulate the appetite-stimulating hormone ghrelin, but increased intake of calories from snacks, which suggests hedonic rather than homeostatic mediators of the shift in food selection.6 Likewise, results from another study have shown that habitual short sleepers (average of 6 h/night) ate more often (i.e., > 3 meals/day, with more frequent nibbling) than did long sleepers.7 Hence, the hedonic value of food intake might play a crucial role in the short sleep-obesity link. 
 
The cross-sectional study of Weiss and colleagues in this issue of SLEEP provides further evidence to support the theory of the hedonic value of food intake associated with short sleep duration.8 Indeed, the authors observe that shorter sleep duration in adolescents was associated with a relatively higher caloric intake derived from fat and a lower caloric intake from carbohydrates. Additionally, shorter sleep duration was associated with a 2-fold increased risk of consuming ≥ 475 kcal/day from snacks in unadjusted analyses. As highlighted by the authors, increased snacking and consumption of high energy foods may reflect effects of short sleep duration on stress responses and reward seeking behaviors. Strengths of this study include (i) the use of an objective measure of sleep duration (wrist actigraphy); (ii) the quantitative measures of macronutrient intake (multiple-pass 24-h food recalls); and (iii) the free-living environment. 
 
An important target of future studies should be the non-homeostatic, reward-driven feeding behavior that could be affected by short sleep duration. Because bedtime restriction inevitably results in more extended exposure to readily available, energy-dense, palatable foods, the regulation of energy balance can be easily compromised in such circumstances. In particular, the advent of functional magnetic resonance imaging is likely to be instrumental in documenting food-related reward activation in the brain after sleep restriction.9 Interestingly, a recent study in adolescents reports that impaired sleep was associated with less reactivity of reward-related brain systems, suggesting that more exciting rewards are required to create the same level of neural activation.10 Because the orexin system is a critical regulator of sleep/wake states, and of feeding behavior and reward processes, it is a potentially important therapeutic target for the treatment of obesity.11 
 
Recent research and the mounting body of evidence regarding the health-related effects of short sleep duration remind us that sleep is not a waste of time and that voluntary sleep restriction can impact our health in various ways. Furthermore, the study of appetite control in our modern world reminds us that our quest for reward and pleasure is not finely tuned to our biology.12,13 With modern sedentary activities (e.g., television viewing, video game playing, cognitive working, listening to music), promoting overconsumption of food “in the absence of hunger,”14 a better understanding of these environmental stimuli is needed if we are to develop behavioral strategies to cope with them effectively.
ER  - 

BibTeX

@misc{0e70b32529dc68c4fe0f2a586b737db79a93f331,
  author = {S. Stranges and F. Cappuccio and N. Kandala and Michelle A. Miller and F. Taggart and M. Kumari and J. Ferrie and M. Shipley and E. Brunner and M. Marmot},
  title = {Cross-sectional versus prospective associations of sleep duration with changes in relative weight and body fat distribution: the Whitehall II Study.},
  journal = {American journal of epidemiology},
  year = {2008},
  volume = {167 3},
  pages = {
          321-9
        },
  doi = {10.1093/AJE/KWM302},
  abstract = {A cross-sectional relation between short sleep and obesity has not been confirmed prospectively. The authors examined the relation between sleep duration and changes in body mass index and waist circumference using the Whitehall II Study, a prospective cohort of 10,308 white-collar British civil servants aged 35-55 years in 1985-1988. Data were gathered in 1997-1999 and 2003-2004. Sleep duration and other covariates were assessed. Changes in body mass index and waist circumference were assessed between the two phases. The incidence of obesity (body mass index: > or =30 kg/m(2)) was assessed among nonobese participants at baseline. In cross-sectional analyses (n = 5,021), there were significant, inverse associations (p < 0.001) between duration of sleep and both body mass index and waist circumference. Compared with 7 hours of sleep, a short duration of sleep (< or =5 hours) was associated with higher body mass index (beta = 0.82 units, 95% confidence interval (CI): 0.38, 1.26) and waist circumference (beta = 1.88 cm, 95% CI: 0.64, 3.12), as well as an increased risk of obesity (odds ratio(adjusted) = 1.65, 95% CI: 1.22, 2.24). In prospective analyses, a short duration of sleep was not associated with significant changes in body mass index (beta = -0.06, 95% CI: -0.26, 0.14) or waist circumference (beta = 0.44, 95% CI: -0.23, 1.12), nor with the incidence of obesity (odds ratio(adjusted) = 1.05, 95% CI: 0.60, 1.82). There is no temporal relation between short duration of sleep and future changes in measures of body weight and central adiposity.}
}

RIS

TY  - JOUR
AU  - S. Stranges
AU  - F. Cappuccio
AU  - N. Kandala
AU  - Michelle A. Miller
AU  - F. Taggart
AU  - M. Kumari
AU  - J. Ferrie
AU  - M. Shipley
AU  - E. Brunner
AU  - M. Marmot
T1  - Cross-sectional versus prospective associations of sleep duration with changes in relative weight and body fat distribution: the Whitehall II Study.
JO  - American journal of epidemiology
PY  - 2008
VL  - 167 3
SP  - 
          321-9
        
EP  - 
          321-9
        
DO  - 10.1093/AJE/KWM302
AB  - A cross-sectional relation between short sleep and obesity has not been confirmed prospectively. The authors examined the relation between sleep duration and changes in body mass index and waist circumference using the Whitehall II Study, a prospective cohort of 10,308 white-collar British civil servants aged 35-55 years in 1985-1988. Data were gathered in 1997-1999 and 2003-2004. Sleep duration and other covariates were assessed. Changes in body mass index and waist circumference were assessed between the two phases. The incidence of obesity (body mass index: > or =30 kg/m(2)) was assessed among nonobese participants at baseline. In cross-sectional analyses (n = 5,021), there were significant, inverse associations (p < 0.001) between duration of sleep and both body mass index and waist circumference. Compared with 7 hours of sleep, a short duration of sleep (< or =5 hours) was associated with higher body mass index (beta = 0.82 units, 95% confidence interval (CI): 0.38, 1.26) and waist circumference (beta = 1.88 cm, 95% CI: 0.64, 3.12), as well as an increased risk of obesity (odds ratio(adjusted) = 1.65, 95% CI: 1.22, 2.24). In prospective analyses, a short duration of sleep was not associated with significant changes in body mass index (beta = -0.06, 95% CI: -0.26, 0.14) or waist circumference (beta = 0.44, 95% CI: -0.23, 1.12), nor with the incidence of obesity (odds ratio(adjusted) = 1.05, 95% CI: 0.60, 1.82). There is no temporal relation between short duration of sleep and future changes in measures of body weight and central adiposity.
ER  - 

BibTeX

@misc{1c09859b15737efe143bb249442b01937327b68c,
  author = {C. Nishiura and H. Hashimoto},
  title = {A 4-Year Study of the Association between Short Sleep Duration and Change in Body Mass Index in Japanese Male Workers},
  journal = {Journal of Epidemiology},
  year = {2010},
  volume = {20},
  pages = {385 - 390},
  doi = {10.2188/jea.JE20100019},
  abstract = {Background Studies of Western populations have shown an inconsistent longitudinal association between short sleep duration and change in body mass index (BMI); a recent Japanese cohort study reported a significant association in men, but over a 1-year period. The aim of this longitudinal study was to examine whether this association was robust over a 4-year interval in Japanese men. Methods A total of 3803 middle-aged Japanese male white-collar workers (mean age 47.8 years, mean BMI 23.9 kg/m2) in Tokyo, Japan, were included in this study from 1994–1995 (baseline) to 1998–1999 (follow-up). Height and weight were objectively measured at annual health checkups, and other data, including sleep duration, were collected using a structured interview. We used linear regression models to estimate change in BMI, after adjustment for covariates. The reference category for sleep duration was set to 7 hours, to conform with previous studies. Results As compared with participants sleeping 7 hours, those sleeping 5 hours or less had a significantly higher BMI at baseline (beta coefficient: 0.34 kg/m2, 95% confidence interval (CI): 0.03, 0.65) and gained 0.15 kg/m2 in BMI over 4 years (95% CI: 0.03, 0.27), after adjustment for age, baseline BMI, lifestyle behavior, and medication. Conclusions The longitudinal association between short sleep duration at baseline and relative increase in BMI was significant in Japanese male workers over a 4-year interval.}
}

RIS

TY  - JOUR
AU  - C. Nishiura
AU  - H. Hashimoto
T1  - A 4-Year Study of the Association between Short Sleep Duration and Change in Body Mass Index in Japanese Male Workers
JO  - Journal of Epidemiology
PY  - 2010
VL  - 20
SP  - 385 - 390
EP  - 385 - 390
DO  - 10.2188/jea.JE20100019
AB  - Background Studies of Western populations have shown an inconsistent longitudinal association between short sleep duration and change in body mass index (BMI); a recent Japanese cohort study reported a significant association in men, but over a 1-year period. The aim of this longitudinal study was to examine whether this association was robust over a 4-year interval in Japanese men. Methods A total of 3803 middle-aged Japanese male white-collar workers (mean age 47.8 years, mean BMI 23.9 kg/m2) in Tokyo, Japan, were included in this study from 1994–1995 (baseline) to 1998–1999 (follow-up). Height and weight were objectively measured at annual health checkups, and other data, including sleep duration, were collected using a structured interview. We used linear regression models to estimate change in BMI, after adjustment for covariates. The reference category for sleep duration was set to 7 hours, to conform with previous studies. Results As compared with participants sleeping 7 hours, those sleeping 5 hours or less had a significantly higher BMI at baseline (beta coefficient: 0.34 kg/m2, 95% confidence interval (CI): 0.03, 0.65) and gained 0.15 kg/m2 in BMI over 4 years (95% CI: 0.03, 0.27), after adjustment for age, baseline BMI, lifestyle behavior, and medication. Conclusions The longitudinal association between short sleep duration at baseline and relative increase in BMI was significant in Japanese male workers over a 4-year interval.
ER  - 

BibTeX

@misc{e0c88a6882735e0c32b7b701d00dd375992d3c16,
  author = {Bradley M. Appelhans and I. Janssen and J. Cursio and K. Matthews and M. Hall and E. Gold and J. Burns and H. Kravitz},
  title = {Sleep duration and weight change in midlife women: The SWAN Sleep Study},
  journal = {Obesity (Silver Spring, Md.)},
  year = {2012},
  volume = {21},
  pages = {77 - 84},
  doi = {10.1002/oby.20251},
  abstract = {Short sleep duration has been associated with higher current BMI and subsequent weight gain. However, most prior longitudinal studies are limited by reliance on self‐reported sleep duration, and none accounted for the potential confounding effect of sleep‐disordered breathing. The associations of sleep duration with current BMI and BMI change were examined among 310 midlife women in the Study of Women's Health Across the Nation (SWAN) Sleep Study (2003‐2005).}
}

RIS

TY  - JOUR
AU  - Bradley M. Appelhans
AU  - I. Janssen
AU  - J. Cursio
AU  - K. Matthews
AU  - M. Hall
AU  - E. Gold
AU  - J. Burns
AU  - H. Kravitz
T1  - Sleep duration and weight change in midlife women: The SWAN Sleep Study
JO  - Obesity (Silver Spring, Md.)
PY  - 2012
VL  - 21
SP  - 77 - 84
EP  - 77 - 84
DO  - 10.1002/oby.20251
AB  - Short sleep duration has been associated with higher current BMI and subsequent weight gain. However, most prior longitudinal studies are limited by reliance on self‐reported sleep duration, and none accounted for the potential confounding effect of sleep‐disordered breathing. The associations of sleep duration with current BMI and BMI change were examined among 310 midlife women in the Study of Women's Health Across the Nation (SWAN) Sleep Study (2003‐2005).
ER  - 

BibTeX

@misc{b3bb6062390c289e78299662840f5ec3829c3b41,
  author = {C. Magee and Jeong Kyu Lee},
  title = {Short sleep duration is associated with increased BMI in adolescents over time},
  journal = {Evidence-Based Medicine},
  year = {2013},
  volume = {19},
  pages = {e5 - e5},
  doi = {10.1136/eb-2013-101436},
  abstract = {Commentary on: Mitchell JA, Rodriguez D, Schmitz KH, et al. Sleep duration and adolescent obesity. Pediatrics 2013;131:e1428–34.[OpenUrl][1][Abstract/FREE Full Text][2]

There is growing interest surrounding short sleep duration as a potential contributor to obesity.1 Most research has been cross-sectional and hence the nature of the temporal associations between sleep duration and indices of body composition is less clear. This is an issue because the association between sleep duration and obesity could be bidirectional.2 More well-designed longitudinal studies are therefore needed to examine the relationships between sleep duration and obesity, especially in adolescents where data are particularly limited.

This study investigated whether sleep duration was associated with changes in body mass index (BMI) over time in 1390 adolescents recruited from four schools in the …

 [1]: {openurl}?query=rft.jtitle%253DPediatrics%26rft_id%253Dinfo%253Adoi%252F10.1542%252Fpeds.2012-2368%26rft_id%253Dinfo%253Apmid%252F23569090%26rft.genre%253Darticle%26rft_val_fmt%253Dinfo%253Aofi%252Ffmt%253Akev%253Amtx%253Ajournal%26ctx_ver%253DZ39.88-2004%26url_ver%253DZ39.88-2004%26url_ctx_fmt%253Dinfo%253Aofi%252Ffmt%253Akev%253Amtx%253Actx
 [2]: /lookup/ijlink?linkType=ABST&journalCode=pediatrics&resid=131/5/e1428&atom=%2Febmed%2F19%2F1%2Fe5.atom}
}

RIS

TY  - JOUR
AU  - C. Magee
AU  - Jeong Kyu Lee
T1  - Short sleep duration is associated with increased BMI in adolescents over time
JO  - Evidence-Based Medicine
PY  - 2013
VL  - 19
SP  - e5 - e5
EP  - e5 - e5
DO  - 10.1136/eb-2013-101436
AB  - Commentary on: Mitchell JA, Rodriguez D, Schmitz KH, et al. Sleep duration and adolescent obesity. Pediatrics 2013;131:e1428–34.[OpenUrl][1][Abstract/FREE Full Text][2]

There is growing interest surrounding short sleep duration as a potential contributor to obesity.1 Most research has been cross-sectional and hence the nature of the temporal associations between sleep duration and indices of body composition is less clear. This is an issue because the association between sleep duration and obesity could be bidirectional.2 More well-designed longitudinal studies are therefore needed to examine the relationships between sleep duration and obesity, especially in adolescents where data are particularly limited.

This study investigated whether sleep duration was associated with changes in body mass index (BMI) over time in 1390 adolescents recruited from four schools in the …

 [1]: {openurl}?query=rft.jtitle%253DPediatrics%26rft_id%253Dinfo%253Adoi%252F10.1542%252Fpeds.2012-2368%26rft_id%253Dinfo%253Apmid%252F23569090%26rft.genre%253Darticle%26rft_val_fmt%253Dinfo%253Aofi%252Ffmt%253Akev%253Amtx%253Ajournal%26ctx_ver%253DZ39.88-2004%26url_ver%253DZ39.88-2004%26url_ctx_fmt%253Dinfo%253Aofi%252Ffmt%253Akev%253Amtx%253Actx
 [2]: /lookup/ijlink?linkType=ABST&journalCode=pediatrics&resid=131/5/e1428&atom=%2Febmed%2F19%2F1%2Fe5.atom
ER  - 

BibTeX

@misc{29ce19df3bec5c124b0e94299664e1ef93be4750,
  author = {Catalina Ramírez-Contreras and Alicia Santamaría-Orleans and M. Izquierdo-Pulido and M. F. Zerón-Rugerio},
  title = {Sleep dimensions are associated with obesity, poor diet quality and eating behaviors in school-aged children},
  journal = {Frontiers in Nutrition},
  year = {2022},
  volume = {9},
  doi = {10.3389/fnut.2022.959503},
  abstract = {Objectives The aim of this cross-sectional study was to investigate the association between sleep dimensions (duration, patterns, and disturbances) with body mass index (BMI), diet quality, and eating behaviors in school-aged children. Additionally, we aimed to investigate whether obesogenic eating behaviors (higher food responsiveness, lower satiety responsiveness, and less slowness in eating) and poor diet quality could mediate the potential association between sleep and obesity in school-aged children. Materials and methods For all participants (n = 588 children, age 5–12 years; 51% girls) we evaluated: sleep dimensions, BMI, diet quality, eating behaviors (food responsiveness, satiety responsiveness and slowness in eating). Linear regression models were used to test associations between exposure and outcome variables. Additionally, path analysis was conducted to test whether eating behaviors mediated the relationship between sleep and obesity. Results Shorter sleep duration (β = −0.722, p = 0.009) and greater sleep disturbances (β = 0.031, p = 0.012) were significantly associated with BMI. Additionally, we observed that diet quality was significantly associated with sleep duration (β = 0.430, p = 0.004), the midpoint of sleep (β = −0.927, p < 0.001), and sleep disturbances (β = −0.029, p < 0.001). Among other findings, greater sleep disturbances were associated with food responsiveness (β = 0.017, p < 0.001), satiety responsiveness (β = 0.015, p < 0.001), and slowness in eating (β = 0.012, p < 0.001). Importantly, food responsiveness was found as significant mediator of the relationship between sleep and BMI (R = 0.427, R2 = 0.182, p < 0.001). Conclusions Late sleep patterns, short sleep duration, and greater sleep disturbances are significantly related with what and how school-aged children eat. Importantly, poor diet quality was significantly related to all three sleep dimensions, while eating behaviors had a significant relationship with greater sleep disturbances. These findings may be relevant to the development of behavioral targets to prevent childhood obesity, including sleep hygiene guidelines as a strategy to improve children's eating habits, as well as their BMI.}
}

RIS

TY  - JOUR
AU  - Catalina Ramírez-Contreras
AU  - Alicia Santamaría-Orleans
AU  - M. Izquierdo-Pulido
AU  - M. F. Zerón-Rugerio
T1  - Sleep dimensions are associated with obesity, poor diet quality and eating behaviors in school-aged children
JO  - Frontiers in Nutrition
PY  - 2022
VL  - 9
DO  - 10.3389/fnut.2022.959503
AB  - Objectives The aim of this cross-sectional study was to investigate the association between sleep dimensions (duration, patterns, and disturbances) with body mass index (BMI), diet quality, and eating behaviors in school-aged children. Additionally, we aimed to investigate whether obesogenic eating behaviors (higher food responsiveness, lower satiety responsiveness, and less slowness in eating) and poor diet quality could mediate the potential association between sleep and obesity in school-aged children. Materials and methods For all participants (n = 588 children, age 5–12 years; 51% girls) we evaluated: sleep dimensions, BMI, diet quality, eating behaviors (food responsiveness, satiety responsiveness and slowness in eating). Linear regression models were used to test associations between exposure and outcome variables. Additionally, path analysis was conducted to test whether eating behaviors mediated the relationship between sleep and obesity. Results Shorter sleep duration (β = −0.722, p = 0.009) and greater sleep disturbances (β = 0.031, p = 0.012) were significantly associated with BMI. Additionally, we observed that diet quality was significantly associated with sleep duration (β = 0.430, p = 0.004), the midpoint of sleep (β = −0.927, p < 0.001), and sleep disturbances (β = −0.029, p < 0.001). Among other findings, greater sleep disturbances were associated with food responsiveness (β = 0.017, p < 0.001), satiety responsiveness (β = 0.015, p < 0.001), and slowness in eating (β = 0.012, p < 0.001). Importantly, food responsiveness was found as significant mediator of the relationship between sleep and BMI (R = 0.427, R2 = 0.182, p < 0.001). Conclusions Late sleep patterns, short sleep duration, and greater sleep disturbances are significantly related with what and how school-aged children eat. Importantly, poor diet quality was significantly related to all three sleep dimensions, while eating behaviors had a significant relationship with greater sleep disturbances. These findings may be relevant to the development of behavioral targets to prevent childhood obesity, including sleep hygiene guidelines as a strategy to improve children's eating habits, as well as their BMI.
ER  - 

BibTeX

@misc{c2075cc819456cc164030590157aba09f83c7c3d,
  author = {C. Magee and P. Caputi and D. Iverson},
  title = {The Longitudinal Relationship Between Sleep Duration and Body Mass Index in Children: A Growth Mixture Modeling Approach},
  journal = {Journal of Developmental & Behavioral Pediatrics},
  year = {2013},
  volume = {34},
  pages = {165–173},
  doi = {10.1097/DBP.0b013e318289aa51.},
  abstract = {Objective: A growing number of studies indicate that shorter sleep durations could contribute to obesity in children. The objective of this article was to further examine the longitudinal relationship between sleep duration and body mass index (BMI) in children by using a growth mixture modeling approach. Method: This article used prospective data from the Longitudinal Study of Australian Children. Participants included 1079 children aged 4 to 5 years (2004) followed up until age 10 to 11 years (2010). Growth mixture modeling was performed to examine the longitudinal association between sleep duration and body mass index within distinct body mass index trajectories. Results: The results indicated 3 distinct body mass index trajectories: healthy weight, early onset obesity, and later onset obesity. Longitudinal inverse associations were evident between sleep duration and body mass index in the Early Onset Trajectory. There were some associations between sleep duration and body mass index in the other trajectories. Conclusions: This article provides further insight into the longitudinal relationship between sleep duration and body mass index in children. In particular, the results indicate that shorter sleep durations are primarily associated with body mass index in children with early onset obesity.}
}

RIS

TY  - JOUR
AU  - C. Magee
AU  - P. Caputi
AU  - D. Iverson
T1  - The Longitudinal Relationship Between Sleep Duration and Body Mass Index in Children: A Growth Mixture Modeling Approach
JO  - Journal of Developmental & Behavioral Pediatrics
PY  - 2013
VL  - 34
SP  - 165–173
EP  - 165–173
DO  - 10.1097/DBP.0b013e318289aa51.
AB  - Objective: A growing number of studies indicate that shorter sleep durations could contribute to obesity in children. The objective of this article was to further examine the longitudinal relationship between sleep duration and body mass index (BMI) in children by using a growth mixture modeling approach. Method: This article used prospective data from the Longitudinal Study of Australian Children. Participants included 1079 children aged 4 to 5 years (2004) followed up until age 10 to 11 years (2010). Growth mixture modeling was performed to examine the longitudinal association between sleep duration and body mass index within distinct body mass index trajectories. Results: The results indicated 3 distinct body mass index trajectories: healthy weight, early onset obesity, and later onset obesity. Longitudinal inverse associations were evident between sleep duration and body mass index in the Early Onset Trajectory. There were some associations between sleep duration and body mass index in the other trajectories. Conclusions: This article provides further insight into the longitudinal relationship between sleep duration and body mass index in children. In particular, the results indicate that shorter sleep durations are primarily associated with body mass index in children with early onset obesity.
ER  - 

BibTeX

@misc{8d34e1e736d2c931a8241d26386cd250b9aaadcf,
  author = {Mayumi Watanabe and Hiroshi Kikuchi and Katsutoshi Tanaka and Masaya Takahashi},
  title = {Association of short sleep duration with weight gain and obesity at 1-year follow-up: a large-scale prospective study.},
  journal = {Sleep},
  year = {2010},
  volume = {33 2},
  pages = {
          161-7
        },
  doi = {10.1093/SLEEP/33.2.161},
  abstract = {STUDY OBJECTIVES
To investigate the association between short sleep duration and elevated body mass index (BMI) and obesity in a large sample of Japanese adults over a short period.


DESIGN
Prospective design with baseline in 2006 and 1-year follow-up.


SETTING
Workplaces of an electric power company in Japan.


PARTICIPANTS
35,247 company employees (31,477 men, 3,770 women) distributed throughout Japan.


MEASUREMENTS AND RESULTS
Measured weight and height and self-reported sleep duration were obtained at annual health checkup in 2006 and 2007. Weight change was defined as the difference in body mass index (BMI) between the baseline and 1 year later. Relative to the reference category (sleep duration 7-8 h), short sleep duration (< 5 and 5-6 h) and long sleep duration > or = 9 h were associated with an increased risk of weight gain among men after adjustment for covariates. Of the non-obese (BMI < 25) men at baseline, 5.8% became obese (BMI > or = 25) 1 year later. Higher incidence of obesity was observed among the groups with shorter sleep duration. Adjusted odds ratios for the development of obesity were 1.91 (95% CI 1.36, 2.67) and 1.50 (95% CI 1.24, 1.80) in men who slept < 5 and 5-6 h, respectively. No significant association between sleep duration and weight gain or obesity was found for women.


CONCLUSIONS
Short sleep duration was associated with weight gain and the development of obesity over 1 year in men, but not in women.}
}

RIS

TY  - JOUR
AU  - Mayumi Watanabe
AU  - Hiroshi Kikuchi
AU  - Katsutoshi Tanaka
AU  - Masaya Takahashi
T1  - Association of short sleep duration with weight gain and obesity at 1-year follow-up: a large-scale prospective study.
JO  - Sleep
PY  - 2010
VL  - 33 2
SP  - 
          161-7
        
EP  - 
          161-7
        
DO  - 10.1093/SLEEP/33.2.161
AB  - STUDY OBJECTIVES
To investigate the association between short sleep duration and elevated body mass index (BMI) and obesity in a large sample of Japanese adults over a short period.


DESIGN
Prospective design with baseline in 2006 and 1-year follow-up.


SETTING
Workplaces of an electric power company in Japan.


PARTICIPANTS
35,247 company employees (31,477 men, 3,770 women) distributed throughout Japan.


MEASUREMENTS AND RESULTS
Measured weight and height and self-reported sleep duration were obtained at annual health checkup in 2006 and 2007. Weight change was defined as the difference in body mass index (BMI) between the baseline and 1 year later. Relative to the reference category (sleep duration 7-8 h), short sleep duration (< 5 and 5-6 h) and long sleep duration > or = 9 h were associated with an increased risk of weight gain among men after adjustment for covariates. Of the non-obese (BMI < 25) men at baseline, 5.8% became obese (BMI > or = 25) 1 year later. Higher incidence of obesity was observed among the groups with shorter sleep duration. Adjusted odds ratios for the development of obesity were 1.91 (95% CI 1.36, 2.67) and 1.50 (95% CI 1.24, 1.80) in men who slept < 5 and 5-6 h, respectively. No significant association between sleep duration and weight gain or obesity was found for women.


CONCLUSIONS
Short sleep duration was associated with weight gain and the development of obesity over 1 year in men, but not in women.
ER  - 

BibTeX

@misc{8cf284ff3a3b735c23eeb60e3f9e012d4ed5551e,
  author = {G. Hasler and Daniel J Buysse and R. Klaghofer and A. Gamma and V. Ajdacic and D. Eich and W. Rössler and J. Angst},
  title = {The association between short sleep duration and obesity in young adults: a 13-year prospective study.},
  journal = {Sleep},
  year = {2004},
  volume = {27 4},
  pages = {
          661-6
        },
  doi = {10.1093/SLEEP/27.4.661},
  abstract = {STUDY OBJECTIVES
Obesity has become a major health problem with increasing prevalence. Given the limited availability of effective treatment of weight problems, the identification of potentially modifiable risk factors may lead to preventive approaches to obesity. The objective of this study was to test the hypothesis that short sleep duration is associated with obesity and weight gain during young adulthood.


DESIGN
Prospective single-age cohort study of young adults. Information was derived from 4 interviews when participants were ages 27, 29, 34, and 40 years.


SETTING
Community setting.


PARTICIPANTS
496 young adults.


MEASUREMENTS AND RESULTS
Trained health professionals administered a semistructured interview for psychiatric and medical conditions and health habits. This study showed an association between short sleep duration and obesity (at age 27 years, odds ratio: 7.4, 95% confidence interval: 1.3-43.1) and a negative association between sleep duration and body mass index in young adults. These associations persisted after controlling for a variety of potentially confounding variables, including family history of weight problems, levels of physical activity, and demographic variables. Associations between sleep duration and obesity diminished after age 34 years. There was a trend (P = .08) for average change rate of weight gain to be negatively associated with average change rate of sleep duration.


CONCLUSIONS
Because sleep duration is a potentially modifiable risk factor, these findings might have important clinical implications for the prevention and treatment of obesity.}
}

RIS

TY  - JOUR
AU  - G. Hasler
AU  - Daniel J Buysse
AU  - R. Klaghofer
AU  - A. Gamma
AU  - V. Ajdacic
AU  - D. Eich
AU  - W. Rössler
AU  - J. Angst
T1  - The association between short sleep duration and obesity in young adults: a 13-year prospective study.
JO  - Sleep
PY  - 2004
VL  - 27 4
SP  - 
          661-6
        
EP  - 
          661-6
        
DO  - 10.1093/SLEEP/27.4.661
AB  - STUDY OBJECTIVES
Obesity has become a major health problem with increasing prevalence. Given the limited availability of effective treatment of weight problems, the identification of potentially modifiable risk factors may lead to preventive approaches to obesity. The objective of this study was to test the hypothesis that short sleep duration is associated with obesity and weight gain during young adulthood.


DESIGN
Prospective single-age cohort study of young adults. Information was derived from 4 interviews when participants were ages 27, 29, 34, and 40 years.


SETTING
Community setting.


PARTICIPANTS
496 young adults.


MEASUREMENTS AND RESULTS
Trained health professionals administered a semistructured interview for psychiatric and medical conditions and health habits. This study showed an association between short sleep duration and obesity (at age 27 years, odds ratio: 7.4, 95% confidence interval: 1.3-43.1) and a negative association between sleep duration and body mass index in young adults. These associations persisted after controlling for a variety of potentially confounding variables, including family history of weight problems, levels of physical activity, and demographic variables. Associations between sleep duration and obesity diminished after age 34 years. There was a trend (P = .08) for average change rate of weight gain to be negatively associated with average change rate of sleep duration.


CONCLUSIONS
Because sleep duration is a potentially modifiable risk factor, these findings might have important clinical implications for the prevention and treatment of obesity.
ER  - 

BibTeX

@misc{221ad5bb9ab8167b5beeeb188f391fc8ec360708,
  author = {V. Seegers and D. Petit and B. Falissard and F. Vitaro and R. Tremblay and J. Montplaisir and E. Touchette},
  title = {Short sleep duration and body mass index: a prospective longitudinal study in preadolescence.},
  journal = {American journal of epidemiology},
  year = {2011},
  volume = {173 6},
  pages = {
          621-9
        },
  doi = {10.1093/aje/kwq389},
  abstract = {Short sleep duration is associated with incidence of overweight and obesity in preadolescent children. The authors performed regression analyses on data from the Quebec Longitudinal Study of Kindergarten Children (1986-1987), a prospective cohort study comprising 1,916 preadolescent children in Canada. The aim was to assess associations between time spent in bed and body mass index reported by mothers after adjusting for numerous confounding factors, such as pubertal status. Time-in-bed and body mass index trajectories were computed using a semiparametric model mixture. Time-in-bed trajectories were classified as short (15% of the preadolescents), 10.5-hour (68%), and 11-hour (17%) sleep-duration trajectories, decreasing over time. Body mass index trajectories were classified as normal weight (68% of the preadolescents), overweight (27%), and obese (5%). The short sleep trajectory was associated with an increased odds ratio of being in the overweight body mass index trajectory (odds ratio (OR)=1.55, 95% confidence interval (CI): 1.39, 1.71) or in the obese body mass index trajectory (OR=3.26, 95% CI: 3.20, 3.29) compared with the 11-hour trajectory. One hour less of sleep per night at 10 years of age was associated with an increased odds ratio of being overweight (OR=1.51, 95% CI: 1.28, 1.76) or obese (OR=2.07; 95% CI: 1.51, 2.84) at 13 years of age.}
}

RIS

TY  - JOUR
AU  - V. Seegers
AU  - D. Petit
AU  - B. Falissard
AU  - F. Vitaro
AU  - R. Tremblay
AU  - J. Montplaisir
AU  - E. Touchette
T1  - Short sleep duration and body mass index: a prospective longitudinal study in preadolescence.
JO  - American journal of epidemiology
PY  - 2011
VL  - 173 6
SP  - 
          621-9
        
EP  - 
          621-9
        
DO  - 10.1093/aje/kwq389
AB  - Short sleep duration is associated with incidence of overweight and obesity in preadolescent children. The authors performed regression analyses on data from the Quebec Longitudinal Study of Kindergarten Children (1986-1987), a prospective cohort study comprising 1,916 preadolescent children in Canada. The aim was to assess associations between time spent in bed and body mass index reported by mothers after adjusting for numerous confounding factors, such as pubertal status. Time-in-bed and body mass index trajectories were computed using a semiparametric model mixture. Time-in-bed trajectories were classified as short (15% of the preadolescents), 10.5-hour (68%), and 11-hour (17%) sleep-duration trajectories, decreasing over time. Body mass index trajectories were classified as normal weight (68% of the preadolescents), overweight (27%), and obese (5%). The short sleep trajectory was associated with an increased odds ratio of being in the overweight body mass index trajectory (odds ratio (OR)=1.55, 95% confidence interval (CI): 1.39, 1.71) or in the obese body mass index trajectory (OR=3.26, 95% CI: 3.20, 3.29) compared with the 11-hour trajectory. One hour less of sleep per night at 10 years of age was associated with an increased odds ratio of being overweight (OR=1.51, 95% CI: 1.28, 1.76) or obese (OR=2.07; 95% CI: 1.51, 2.84) at 13 years of age.
ER  - 

BibTeX

@misc{bad040c9b88c59a0e9971f480c6d1a4630dab84c,
  author = {B. Morrissey and L. Orellana and S. Allender and Claudia Strugnell},
  title = {The Sleep-Obesity Nexus: Assessment of Multiple Sleep Dimensions and Weight Status Among Victorian Primary School Children},
  journal = {Nature and Science of Sleep},
  year = {2022},
  volume = {14},
  pages = {581 - 591},
  doi = {10.2147/NSS.S352357},
  abstract = {Purpose To examine the association between sleep and weight status across multiple dimensions of sleep (duration, efficiency, quality and timing) and assess the cumulative influence of these dimensions on the overall sleep-obesity association. Participants and Methods Cross-sectional data from 2253 students aged 8.8 to 13.5 years participating in two monitoring studies across regional Victoria was analyzed. Students were invited to have measures of height and weight taken and to complete a self-report electronic questionnaire on demographic characteristics and health behaviors. Logistic regression models were used to assess association between sleep dimensions and BMI z-scores. Results Beyond sleep duration, poor perceived sleep quality, delayed sleep initiation, later bed times and sleep-wake timing, all significantly increased the odds of overweight/obesity (OR 1.47, 95% CI: 1.07–2.01; OR 1.25, 95% CI: 1.02–1.55; and OR 1.70. 95% CI: 1.28–2.28, respectively). Additionally, a cumulative effect of having multiple poorly scored sleep dimensions was found, where four or more poorly scored sleep dimensions more than doubled the odds of overweight/obesity among children (OR 2.25, 95% CI: 1.41–3.58). Conclusion This study highlights the importance of measuring and considering multiple dimensions of sleep, along with the individual and additive influence of the sleep dimension on the sleep-obesity nexus.}
}

RIS

TY  - JOUR
AU  - B. Morrissey
AU  - L. Orellana
AU  - S. Allender
AU  - Claudia Strugnell
T1  - The Sleep-Obesity Nexus: Assessment of Multiple Sleep Dimensions and Weight Status Among Victorian Primary School Children
JO  - Nature and Science of Sleep
PY  - 2022
VL  - 14
SP  - 581 - 591
EP  - 581 - 591
DO  - 10.2147/NSS.S352357
AB  - Purpose To examine the association between sleep and weight status across multiple dimensions of sleep (duration, efficiency, quality and timing) and assess the cumulative influence of these dimensions on the overall sleep-obesity association. Participants and Methods Cross-sectional data from 2253 students aged 8.8 to 13.5 years participating in two monitoring studies across regional Victoria was analyzed. Students were invited to have measures of height and weight taken and to complete a self-report electronic questionnaire on demographic characteristics and health behaviors. Logistic regression models were used to assess association between sleep dimensions and BMI z-scores. Results Beyond sleep duration, poor perceived sleep quality, delayed sleep initiation, later bed times and sleep-wake timing, all significantly increased the odds of overweight/obesity (OR 1.47, 95% CI: 1.07–2.01; OR 1.25, 95% CI: 1.02–1.55; and OR 1.70. 95% CI: 1.28–2.28, respectively). Additionally, a cumulative effect of having multiple poorly scored sleep dimensions was found, where four or more poorly scored sleep dimensions more than doubled the odds of overweight/obesity among children (OR 2.25, 95% CI: 1.41–3.58). Conclusion This study highlights the importance of measuring and considering multiple dimensions of sleep, along with the individual and additive influence of the sleep dimension on the sleep-obesity nexus.
ER  - 

BibTeX

@misc{a069d1188e56ba02fa4d9583021552fd5a2bb701,
  author = {P. Collings},
  title = {Independent associations of sleep timing, duration and quality with adiposity and weight status in a national sample of adolescents: The UK Millennium Cohort Study},
  journal = {Journal of Sleep Research},
  year = {2021},
  volume = {31},
  doi = {10.1111/jsr.13436},
  abstract = {Short sleep appears to elevate obesity risk in youth; however, sleep is a multidimensional construct, and few studies have investigated parameters beyond duration. The objective of this study was to investigate if sleep onset time, duration, latency and night waking frequency are independently associated with adiposity and weight status in UK adolescents. This was a cross‐sectional observational study of 10,619, 13–15 years olds. Adjusted linear and logistic regressions were used to investigate associations of self‐reported sleep characteristics with adiposity markers (body mass index z‐score and percent body fat) and weight status. Compared with a sleep onset before 10pm, later sleep timing was associated with higher adiposity and higher likelihood of overweight and obesity in boys (after midnight, odds ratio [95% confidence interval]: 1.76 [1.19–2.60]) and girls (between 11pm and 11:59pm: 1.36 [1.17–1.65]). Sleeping ≤ 8 hr, compared with > 9–10 hr, was associated with higher odds of overweight and obesity in both sexes (boys: 1.80 [1.38–2.35]; girls: 1.38 [1.06–1.79]), and so too was sleeping > 10 hr in girls (1.31 [1.06–1.62]), indicating evidence for a U‐shaped association. Also in girls, compared to a sleep latency of 16‐30 min, sleep latencies ≥ 46 min were associated with higher adiposity (46–60 min, beta coefficient [95% confidence interval], percent body fat: 1.47 [0.57–2.36]) and higher likelihood of overweight and obesity (46–60 min: 1.39 [1.05–1.83]), and often as opposed to never waking in the night was associated with higher adiposity (body mass index z‐score: 0.24 [0.08–0.41]; percent body fat: 1.44 [0.44–2.44]). Sleep duration and timing in both sexes, and sleep quality in girls, appear to be independently associated with adiposity and weight status in adolescence, and may be important targets for obesity prevention.}
}

RIS

TY  - JOUR
AU  - P. Collings
T1  - Independent associations of sleep timing, duration and quality with adiposity and weight status in a national sample of adolescents: The UK Millennium Cohort Study
JO  - Journal of Sleep Research
PY  - 2021
VL  - 31
DO  - 10.1111/jsr.13436
AB  - Short sleep appears to elevate obesity risk in youth; however, sleep is a multidimensional construct, and few studies have investigated parameters beyond duration. The objective of this study was to investigate if sleep onset time, duration, latency and night waking frequency are independently associated with adiposity and weight status in UK adolescents. This was a cross‐sectional observational study of 10,619, 13–15 years olds. Adjusted linear and logistic regressions were used to investigate associations of self‐reported sleep characteristics with adiposity markers (body mass index z‐score and percent body fat) and weight status. Compared with a sleep onset before 10pm, later sleep timing was associated with higher adiposity and higher likelihood of overweight and obesity in boys (after midnight, odds ratio [95% confidence interval]: 1.76 [1.19–2.60]) and girls (between 11pm and 11:59pm: 1.36 [1.17–1.65]). Sleeping ≤ 8 hr, compared with > 9–10 hr, was associated with higher odds of overweight and obesity in both sexes (boys: 1.80 [1.38–2.35]; girls: 1.38 [1.06–1.79]), and so too was sleeping > 10 hr in girls (1.31 [1.06–1.62]), indicating evidence for a U‐shaped association. Also in girls, compared to a sleep latency of 16‐30 min, sleep latencies ≥ 46 min were associated with higher adiposity (46–60 min, beta coefficient [95% confidence interval], percent body fat: 1.47 [0.57–2.36]) and higher likelihood of overweight and obesity (46–60 min: 1.39 [1.05–1.83]), and often as opposed to never waking in the night was associated with higher adiposity (body mass index z‐score: 0.24 [0.08–0.41]; percent body fat: 1.44 [0.44–2.44]). Sleep duration and timing in both sexes, and sleep quality in girls, appear to be independently associated with adiposity and weight status in adolescence, and may be important targets for obesity prevention.
ER  - 

BibTeX

@misc{69890695e1af264ff376e0112ca6aa85f87d313e,
  author = {A. Calvin and R. Carter and T. Adachi and P. Macedo and F. Albuquerque and C. van der Walt and J. Bukartyk and Diane E. Davison and J. Levine and V. Somers},
  title = {Effects of experimental sleep restriction on caloric intake and activity energy expenditure.},
  journal = {Chest},
  year = {2013},
  volume = {144 1},
  pages = {
          79-86
        },
  doi = {10.1378/chest.12-2829},
  abstract = {BACKGROUND
Epidemiologic studies link short sleep duration to obesity and weight gain. Insufficient sleep appears to alter circulating levels of the hormones leptin and ghrelin, which may promote appetite, although the effects of sleep restriction on caloric intake and energy expenditure are unclear. We sought to determine the effect of 8 days/8 nights of sleep restriction on caloric intake, activity energy expenditure, and circulating levels of leptin and ghrelin.


METHODS
We conducted a randomized study of usual sleep vs a sleep restriction of two-thirds of normal sleep time for 8 days/8 nights in a hospital-based clinical research unit. The main outcomes were caloric intake, activity energy expenditure, and circulating levels of leptin and ghrelin.


RESULTS
Caloric intake in the sleep-restricted group increased by +559 kcal/d (SD, 706 kcal/d, P=.006) and decreased in the control group by -118 kcal/d (SD, 386 kcal/d, P=.51) for a net change of +677 kcal/d (95% CI, 148-1,206 kcal/d; P=.014). Sleep restriction was not associated with changes in activity energy expenditure (P=.62). No change was seen in levels of leptin (P=.27) or ghrelin (P=.21).


CONCLUSIONS
Sleep restriction was associated with an increase in caloric consumption with no change in activity energy expenditure or leptin and ghrelin concentrations. Increased caloric intake without any accompanying increase in energy expenditure may contribute to obesity in people who are exposed to long-term sleep restriction.


TRIAL REGISTRATION
ClinicalTrials.gov; No.: NCT01334788; URL: www.clinicaltrials.gov.}
}

RIS

TY  - JOUR
AU  - A. Calvin
AU  - R. Carter
AU  - T. Adachi
AU  - P. Macedo
AU  - F. Albuquerque
AU  - C. van der Walt
AU  - J. Bukartyk
AU  - Diane E. Davison
AU  - J. Levine
AU  - V. Somers
T1  - Effects of experimental sleep restriction on caloric intake and activity energy expenditure.
JO  - Chest
PY  - 2013
VL  - 144 1
SP  - 
          79-86
        
EP  - 
          79-86
        
DO  - 10.1378/chest.12-2829
AB  - BACKGROUND
Epidemiologic studies link short sleep duration to obesity and weight gain. Insufficient sleep appears to alter circulating levels of the hormones leptin and ghrelin, which may promote appetite, although the effects of sleep restriction on caloric intake and energy expenditure are unclear. We sought to determine the effect of 8 days/8 nights of sleep restriction on caloric intake, activity energy expenditure, and circulating levels of leptin and ghrelin.


METHODS
We conducted a randomized study of usual sleep vs a sleep restriction of two-thirds of normal sleep time for 8 days/8 nights in a hospital-based clinical research unit. The main outcomes were caloric intake, activity energy expenditure, and circulating levels of leptin and ghrelin.


RESULTS
Caloric intake in the sleep-restricted group increased by +559 kcal/d (SD, 706 kcal/d, P=.006) and decreased in the control group by -118 kcal/d (SD, 386 kcal/d, P=.51) for a net change of +677 kcal/d (95% CI, 148-1,206 kcal/d; P=.014). Sleep restriction was not associated with changes in activity energy expenditure (P=.62). No change was seen in levels of leptin (P=.27) or ghrelin (P=.21).


CONCLUSIONS
Sleep restriction was associated with an increase in caloric consumption with no change in activity energy expenditure or leptin and ghrelin concentrations. Increased caloric intake without any accompanying increase in energy expenditure may contribute to obesity in people who are exposed to long-term sleep restriction.


TRIAL REGISTRATION
ClinicalTrials.gov; No.: NCT01334788; URL: www.clinicaltrials.gov.
ER  - 

BibTeX

@misc{8184758f227b67a6f4470cfb3455df0506f815ba,
  author = {D. Buscemi and A. Kumar and K. Nugent},
  title = {351 THE ASSOCIATION BETWEEN SLEEP TIME AND BODY MASS INDEX IN CLINIC PATIENTS.},
  journal = {Journal of Investigative Medicine},
  year = {2006},
  volume = {54},
  pages = {S319 - S319},
  doi = {10.2310/6650.2005.X0008.350},
  abstract = {Background Obesity has become a major health problem in the United States. The identification of potentially modifiable risk factors should lead to better treatment of this problem. Epidemiological studies have shown an association between shorter sleep times and increased body mass index (BMI). We used a survey instrument in our clinic patients to evaluate the association between sleep time and BMI and to identify potential confounding factors. Methods We obtained a convenience sample of consecutive patients in internal medicine resident clinics. Two hundred patients agreed to participate and four refused. Patients were given a structured questionnaire asking about sleeping, eating, and lifestyle habits. We used descriptive statistics, logistic regression, and multivariate linear regression for data analysis. Unstable sleep was defined as having greater than a 2 hr range in to bed times or greater than a 2 hr range in wake-up times, or naps of length greater than 1 hr. Results The final sample had 199 patients (74 men and 125 women). The median age was 54 with a range of 18 to 89. The mean BMI was 28.8 in men and 29.1 in women (p < .05 by t-test). Forty-one percent of the patients had a BMI $ 30. The mean sleep time was 7.7 hr in men and 8.0 in women (p > .05 by t-test). BMI was higher in younger patients (18-49) than older patients (> 50) (p < .05). The average BMI was higher in working patients than nonworking patients, and these working patients had a shorter sleep time (p < .05). Patients with unstable sleep patterns had a higher BMI (p > .05). Logistic regression demonstrated that the probability of obesity (BMI $ 30) increased in women (odds ratio [OR]: 2.7),in young patients (18-49 yrs, OR: 4.9), with short sleep periods (first quartile: 3-6.9 hrs, OR: 2.82), with dieting (OR: 7.03), with drinking alcohol (OR: 4.11), in diabetics (OR: 2.39), in hypertensive patients (OR: 2.39), and in sleep apnea (OR: 8.77). Subgroup analysis with logistic regression demonstrated that short sleep periods increased the probability of obesity in males and in stable sleepers but not in women or in unstable sleepers. Sleep period had a U-shaped effect in young patients (18-49) but not older patients. Conclusion Adult BMI depends on genetic background, environmental influences, and acquired diseases. In this clinic population, shorter sleep time (3-6.9 hrs), age (18-49), and female gender increased the probability of obesity. Our subgroup analysis suggests that weight loss programs should focus on young males with short sleep times (less than 7 hours). In addition, our results suggest that sleep research studies need to separate patients with stable sleep patterns from patients with unstable patterns.}
}

RIS

TY  - JOUR
AU  - D. Buscemi
AU  - A. Kumar
AU  - K. Nugent
T1  - 351 THE ASSOCIATION BETWEEN SLEEP TIME AND BODY MASS INDEX IN CLINIC PATIENTS.
JO  - Journal of Investigative Medicine
PY  - 2006
VL  - 54
SP  - S319 - S319
EP  - S319 - S319
DO  - 10.2310/6650.2005.X0008.350
AB  - Background Obesity has become a major health problem in the United States. The identification of potentially modifiable risk factors should lead to better treatment of this problem. Epidemiological studies have shown an association between shorter sleep times and increased body mass index (BMI). We used a survey instrument in our clinic patients to evaluate the association between sleep time and BMI and to identify potential confounding factors. Methods We obtained a convenience sample of consecutive patients in internal medicine resident clinics. Two hundred patients agreed to participate and four refused. Patients were given a structured questionnaire asking about sleeping, eating, and lifestyle habits. We used descriptive statistics, logistic regression, and multivariate linear regression for data analysis. Unstable sleep was defined as having greater than a 2 hr range in to bed times or greater than a 2 hr range in wake-up times, or naps of length greater than 1 hr. Results The final sample had 199 patients (74 men and 125 women). The median age was 54 with a range of 18 to 89. The mean BMI was 28.8 in men and 29.1 in women (p < .05 by t-test). Forty-one percent of the patients had a BMI $ 30. The mean sleep time was 7.7 hr in men and 8.0 in women (p > .05 by t-test). BMI was higher in younger patients (18-49) than older patients (> 50) (p < .05). The average BMI was higher in working patients than nonworking patients, and these working patients had a shorter sleep time (p < .05). Patients with unstable sleep patterns had a higher BMI (p > .05). Logistic regression demonstrated that the probability of obesity (BMI $ 30) increased in women (odds ratio [OR]: 2.7),in young patients (18-49 yrs, OR: 4.9), with short sleep periods (first quartile: 3-6.9 hrs, OR: 2.82), with dieting (OR: 7.03), with drinking alcohol (OR: 4.11), in diabetics (OR: 2.39), in hypertensive patients (OR: 2.39), and in sleep apnea (OR: 8.77). Subgroup analysis with logistic regression demonstrated that short sleep periods increased the probability of obesity in males and in stable sleepers but not in women or in unstable sleepers. Sleep period had a U-shaped effect in young patients (18-49) but not older patients. Conclusion Adult BMI depends on genetic background, environmental influences, and acquired diseases. In this clinic population, shorter sleep time (3-6.9 hrs), age (18-49), and female gender increased the probability of obesity. Our subgroup analysis suggests that weight loss programs should focus on young males with short sleep times (less than 7 hours). In addition, our results suggest that sleep research studies need to separate patients with stable sleep patterns from patients with unstable patterns.
ER  - 

BibTeX

@misc{d0908cee12666e7410e7320e7cb71b28b5a596b4,
  author = {G. Nam and Kyungdo Han and Do Hoon Kim and Jee Hyun Lee and Won Hee Seo},
  title = {Sleep duration is associated with body fat and muscle mass and waist‐to‐height ratio beyond conventional obesity parameters in Korean adolescent boys},
  journal = {Journal of Sleep Research},
  year = {2017},
  volume = {26},
  doi = {10.1111/jsr.12502},
  abstract = {While evidence has supported a strong association between sleep duration and obesity globally, results from studies of children and adolescents have been conflicting, and information about a sex‐specific association has been limited. This study aimed to investigate the association of sleep duration with various parameters of obesity among South Korean adolescents. This population‐based, cross‐sectional study analysed the data obtained from the Korea National Health and Nutrition Examination Surveys (KNHANES) 2009 and 2010. Data of 990 adolescents were analysed. Sleep duration was based on a self‐reported questionnaire. Body mass index (BMI), waist circumference (WC), waist‐to‐height ratio (WHtR), body fat percentage (BFP) and skeletal muscle index (SMI, appendicular skeletal muscle mass as a percentage of body weight) were assessed as parameters of obesity. Mean sleep duration in boys was associated inversely with BMI, WC, WHtR and BFP and positively with SMI. Proportions of the highest quartile of BMI, WC, WHtR and BFP and the lowest quartile of SMI increased significantly with increased sleep duration only in boys. Also, in boys, decreased sleep duration was associated significantly with the increased risk of the highest quartile of BMI, WC, WHtR and BFP and the lowest quartile of SMI, even after adjusting for confounding factors. However, in girls, there was no significant association between sleep duration and obesity parameters except WC. Periodic assessment of sleep duration in relation to body fat or muscle mass in male adolescents may be considered, especially in those who are at risk for obesity or related disorders.}
}

RIS

TY  - JOUR
AU  - G. Nam
AU  - Kyungdo Han
AU  - Do Hoon Kim
AU  - Jee Hyun Lee
AU  - Won Hee Seo
T1  - Sleep duration is associated with body fat and muscle mass and waist‐to‐height ratio beyond conventional obesity parameters in Korean adolescent boys
JO  - Journal of Sleep Research
PY  - 2017
VL  - 26
DO  - 10.1111/jsr.12502
AB  - While evidence has supported a strong association between sleep duration and obesity globally, results from studies of children and adolescents have been conflicting, and information about a sex‐specific association has been limited. This study aimed to investigate the association of sleep duration with various parameters of obesity among South Korean adolescents. This population‐based, cross‐sectional study analysed the data obtained from the Korea National Health and Nutrition Examination Surveys (KNHANES) 2009 and 2010. Data of 990 adolescents were analysed. Sleep duration was based on a self‐reported questionnaire. Body mass index (BMI), waist circumference (WC), waist‐to‐height ratio (WHtR), body fat percentage (BFP) and skeletal muscle index (SMI, appendicular skeletal muscle mass as a percentage of body weight) were assessed as parameters of obesity. Mean sleep duration in boys was associated inversely with BMI, WC, WHtR and BFP and positively with SMI. Proportions of the highest quartile of BMI, WC, WHtR and BFP and the lowest quartile of SMI increased significantly with increased sleep duration only in boys. Also, in boys, decreased sleep duration was associated significantly with the increased risk of the highest quartile of BMI, WC, WHtR and BFP and the lowest quartile of SMI, even after adjusting for confounding factors. However, in girls, there was no significant association between sleep duration and obesity parameters except WC. Periodic assessment of sleep duration in relation to body fat or muscle mass in male adolescents may be considered, especially in those who are at risk for obesity or related disorders.
ER  - 

BibTeX

@misc{48becfb359e1d598bf9f4cc7b2e8e62128afad44,
  author = {Y. Danielsen and S. Pallesen and K. Stormark and I. Nordhus and B. Bjorvatn},
  title = {The relationship between school day sleep duration and body mass index in Norwegian children (aged 10-12).},
  journal = {International journal of pediatric obesity : IJPO : an official journal of the International Association for the Study of Obesity},
  year = {2010},
  volume = {5 3},
  pages = {
          214-20
        },
  doi = {10.3109/17477160903473739},
  abstract = {OBJECTIVE
To examine the relationship between sleep duration and body mass index (BMI) in Norwegian children (aged 10-12).


METHODS
Children and parents of a population of primary school children (N=9 430) were invited to complete a questionnaire containing questions about usual bedtimes, wake-up times, self and parent evaluations of pubertal maturation, parental education and economic status, and parent-reported height and weight of the child. Sixty per cent of parents and children participated. BMI was available for 4 158 children, 44% of the original sample.


RESULTS
A U-shaped relationship was found between sleep duration and age- and gender-adjusted BMI. Shorter and longer sleep durations were significantly related to high BMI. The percentage of obesity and overweight was higher in the shortest sleep duration group compared with intermediate sleep durations. Crude logistic regression analyses showed a significant association between early pubertal maturation and both obesity and overweight. Socio-economic status was significantly related to overweight. Short sleep duration showed a significant association with obesity but not with overweight. In an adjusted logistic regression model, short sleep duration was still significantly associated with obesity when controlling for physical maturation and socio-economic status.


CONCLUSIONS
High BMI was associated with short and long self and parent-reported duration of sleep. The percentage of obesity/overweight was higher in the shortest sleep duration group than for intermediate sleep durations. Short sleep duration was significantly associated with obesity but not with overweight. Adjusting for physical maturation and socio-economic status, short sleep duration still showed a significant association with obesity.}
}

RIS

TY  - JOUR
AU  - Y. Danielsen
AU  - S. Pallesen
AU  - K. Stormark
AU  - I. Nordhus
AU  - B. Bjorvatn
T1  - The relationship between school day sleep duration and body mass index in Norwegian children (aged 10-12).
JO  - International journal of pediatric obesity : IJPO : an official journal of the International Association for the Study of Obesity
PY  - 2010
VL  - 5 3
SP  - 
          214-20
        
EP  - 
          214-20
        
DO  - 10.3109/17477160903473739
AB  - OBJECTIVE
To examine the relationship between sleep duration and body mass index (BMI) in Norwegian children (aged 10-12).


METHODS
Children and parents of a population of primary school children (N=9 430) were invited to complete a questionnaire containing questions about usual bedtimes, wake-up times, self and parent evaluations of pubertal maturation, parental education and economic status, and parent-reported height and weight of the child. Sixty per cent of parents and children participated. BMI was available for 4 158 children, 44% of the original sample.


RESULTS
A U-shaped relationship was found between sleep duration and age- and gender-adjusted BMI. Shorter and longer sleep durations were significantly related to high BMI. The percentage of obesity and overweight was higher in the shortest sleep duration group compared with intermediate sleep durations. Crude logistic regression analyses showed a significant association between early pubertal maturation and both obesity and overweight. Socio-economic status was significantly related to overweight. Short sleep duration showed a significant association with obesity but not with overweight. In an adjusted logistic regression model, short sleep duration was still significantly associated with obesity when controlling for physical maturation and socio-economic status.


CONCLUSIONS
High BMI was associated with short and long self and parent-reported duration of sleep. The percentage of obesity/overweight was higher in the shortest sleep duration group than for intermediate sleep durations. Short sleep duration was significantly associated with obesity but not with overweight. Adjusting for physical maturation and socio-economic status, short sleep duration still showed a significant association with obesity.
ER  - 

BibTeX

@misc{8e982910fb3f4213a833809dc97f4533dddac25a,
  author = {J. Chaput and M. Lambert and K. Gray‐donald and J. McGrath and M. Tremblay and J. O'Loughlin and A. Tremblay},
  title = {Short sleep duration is independently associated with overweight and obesity in Quebec children.},
  journal = {Canadian journal of public health = Revue canadienne de sante publique},
  year = {2011},
  volume = {102 5},
  pages = {
          369-74
        },
  doi = {10.17269/CJPH.102.2749},
  abstract = {OBJECTIVE
To investigate the association of sleep duration with adiposity and to determine if caloric intake and physical activity mediate this relationship.


METHODS
The Quebec Adiposity and Lifestyle Investigation in Youth (QUALITY) study is an ongoing longitudinal investigation of Caucasian children with at least one obese biological parent. Children (n = 550) with an average age of 9.6 years (SD = 0.9) who provided complete data at baseline were included in the cross-sectional analyses. Objective measures of adiposity (BMI Z-score, waist circumference, percent body fat measured by dual-energy X-ray absorptiometry), sleep duration and physical activity (accelerometer over 7 days), and diet (24-hour food recalls) were collected. Children were categorized into 4 groups according to sleep duration: < 10 hours, 10-10.9 hours, 11-11.9 hours, and > or = 12 hours of sleep per night.


RESULTS
We observed a U-shaped relationship between sleep duration and all adiposity indices. None of energy intake, snacking, screen time or physical activity intensity differed significantly between sleep categories. After adjusting for age, sex, Tanner stage, highest educational level of the parents, total annual family income, and parental BMI, only short-duration sleepers (< 10 hours) had an increased odds of overweight/obesity (OR 2.08, 95% CI 1.16-3.67). Addition of total energy intake and physical activity to the model did not change the association substantially (OR 2.05, 95% CI 1.15-3.63).


CONCLUSION
The present study provides evidence that short sleep duration is a risk factor for overweight and obesity in children, independent of potential covariates. These results further emphasize the need to add sleep duration to the determinants of obesity.}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - M. Lambert
AU  - K. Gray‐donald
AU  - J. McGrath
AU  - M. Tremblay
AU  - J. O'Loughlin
AU  - A. Tremblay
T1  - Short sleep duration is independently associated with overweight and obesity in Quebec children.
JO  - Canadian journal of public health = Revue canadienne de sante publique
PY  - 2011
VL  - 102 5
SP  - 
          369-74
        
EP  - 
          369-74
        
DO  - 10.17269/CJPH.102.2749
AB  - OBJECTIVE
To investigate the association of sleep duration with adiposity and to determine if caloric intake and physical activity mediate this relationship.


METHODS
The Quebec Adiposity and Lifestyle Investigation in Youth (QUALITY) study is an ongoing longitudinal investigation of Caucasian children with at least one obese biological parent. Children (n = 550) with an average age of 9.6 years (SD = 0.9) who provided complete data at baseline were included in the cross-sectional analyses. Objective measures of adiposity (BMI Z-score, waist circumference, percent body fat measured by dual-energy X-ray absorptiometry), sleep duration and physical activity (accelerometer over 7 days), and diet (24-hour food recalls) were collected. Children were categorized into 4 groups according to sleep duration: < 10 hours, 10-10.9 hours, 11-11.9 hours, and > or = 12 hours of sleep per night.


RESULTS
We observed a U-shaped relationship between sleep duration and all adiposity indices. None of energy intake, snacking, screen time or physical activity intensity differed significantly between sleep categories. After adjusting for age, sex, Tanner stage, highest educational level of the parents, total annual family income, and parental BMI, only short-duration sleepers (< 10 hours) had an increased odds of overweight/obesity (OR 2.08, 95% CI 1.16-3.67). Addition of total energy intake and physical activity to the model did not change the association substantially (OR 2.05, 95% CI 1.15-3.63).


CONCLUSION
The present study provides evidence that short sleep duration is a risk factor for overweight and obesity in children, independent of potential covariates. These results further emphasize the need to add sleep duration to the determinants of obesity.
ER  - 

BibTeX

@misc{aebabc8655092396610a943e1dd68c2043fd8d4b,
  author = {Chihiro 千尋 Nishiura 西浦 and Hideki 英樹 Hashimoto 橋本},
  title = {Sleep Duration and Weight Gain: Reconsideration by Panel Data Analysis},
  journal = {Journal of Epidemiology},
  year = {2014},
  volume = {24},
  pages = {404 - 409},
  doi = {10.2188/jea.JE20140012},
  abstract = {Background Inconsistent findings in previous studies of the association between sleep duration and changes in body mass index (BMI) may be attributed to misclassification of sleep duration fluctuations over time and unmeasured confounders such as genetic factors. The aim of the present study was to overcome these failings by using repeated measurements and panel data analysis to examine the sleep-BMI association. Methods Panel data were derived by secondary use of the data from mandatory health checkups at a Japanese gas company. Male non-shift workers aged 19–39 years in 2007 were annually followed until 2010 (n = 1687, 6748 records). BMI was objectively measured, and sleep duration was self-reported. Results Compared with 7-hour sleepers, panel analysis with the population-averaged model showed a significant increase in BMI among 5-hour (0.11 kg/m2, P = 0.001), 6-hour (0.07 kg/m2, P = 0.038), and ≥8-hour (0.19 kg/m2, P = 0.009) sleepers. On the other hand, after adjustment for unobserved time-invariant confounders using the fixed-effects model, the magnitude of the association was considerably attenuated and no longer significant (5-hour, 0.07 kg/m2, P = 0.168; 6-hour, 0.02 kg/m2, P = 0.631; ≥8-hour sleepers, 0.06 kg/m2, P = 0.460). Conclusions The longitudinal association between sleep duration and changes in BMI may be upwardly biased by unobserved time-invariant confounders rather than misclassified sleep duration. The net effect of sleep duration on weight gain may therefore be less than previously believed.}
}

RIS

TY  - JOUR
AU  - Chihiro 千尋 Nishiura 西浦
AU  - Hideki 英樹 Hashimoto 橋本
T1  - Sleep Duration and Weight Gain: Reconsideration by Panel Data Analysis
JO  - Journal of Epidemiology
PY  - 2014
VL  - 24
SP  - 404 - 409
EP  - 404 - 409
DO  - 10.2188/jea.JE20140012
AB  - Background Inconsistent findings in previous studies of the association between sleep duration and changes in body mass index (BMI) may be attributed to misclassification of sleep duration fluctuations over time and unmeasured confounders such as genetic factors. The aim of the present study was to overcome these failings by using repeated measurements and panel data analysis to examine the sleep-BMI association. Methods Panel data were derived by secondary use of the data from mandatory health checkups at a Japanese gas company. Male non-shift workers aged 19–39 years in 2007 were annually followed until 2010 (n = 1687, 6748 records). BMI was objectively measured, and sleep duration was self-reported. Results Compared with 7-hour sleepers, panel analysis with the population-averaged model showed a significant increase in BMI among 5-hour (0.11 kg/m2, P = 0.001), 6-hour (0.07 kg/m2, P = 0.038), and ≥8-hour (0.19 kg/m2, P = 0.009) sleepers. On the other hand, after adjustment for unobserved time-invariant confounders using the fixed-effects model, the magnitude of the association was considerably attenuated and no longer significant (5-hour, 0.07 kg/m2, P = 0.168; 6-hour, 0.02 kg/m2, P = 0.631; ≥8-hour sleepers, 0.06 kg/m2, P = 0.460). Conclusions The longitudinal association between sleep duration and changes in BMI may be upwardly biased by unobserved time-invariant confounders rather than misclassified sleep duration. The net effect of sleep duration on weight gain may therefore be less than previously believed.
ER  - 

BibTeX

@misc{546a63c016b98e1f35cf9a63fda2df238969c0ce,
  author = {Boram Lee and S. Hong and Sang Ik Cho and S. Kim},
  title = {Analysis of the Relationship between Sleep Duration and Body Mass Index among Korean Adolescents Using Data from the 2013-2015 Korea National Health and Nutrition Examination Survey},
  journal = {Korean Journal of Family Medicine},
  year = {2020},
  volume = {41},
  pages = {346 - 351},
  doi = {10.4082/kjfm.18.0143},
  abstract = {Background Obesity in adolescence can continue up to adulthood and lead to various complications. There has been a continuous increase in the obesity incidence among adolescents, which is an emerging social issue. Studies have reported that decreased sleep duration in adolescents can have negative effects on health, leading to obesity. Therefore, in the present study, we aimed to investigate the relationship between sleep duration and body mass index among South Korean middle and high school students aged 12–18 years. Methods The participants of the Sixth National Health and Nutrition Survey conducted during 2013–2015 were screened. We selected 1,177 adolescents aged between 12 and 18 years who were enrolled in middle and high school or on leave of absence and had available data for sleep duration and body mass index (BMI) in the survey. Analyses of variance and covariance were performed after adjusting for confounding variables, including age, gender, waist circumference, and drinking experience, that showed significant correlations in univariate logistic regression. Results The analysis of covariance revealed that an increase in sleep duration led to a corresponding decrease in the mean BMI among all students and middle school students. Conclusion Significant negative correlations were confirmed between sleep duration and BMI in all students and middle school students.}
}

RIS

TY  - JOUR
AU  - Boram Lee
AU  - S. Hong
AU  - Sang Ik Cho
AU  - S. Kim
T1  - Analysis of the Relationship between Sleep Duration and Body Mass Index among Korean Adolescents Using Data from the 2013-2015 Korea National Health and Nutrition Examination Survey
JO  - Korean Journal of Family Medicine
PY  - 2020
VL  - 41
SP  - 346 - 351
EP  - 346 - 351
DO  - 10.4082/kjfm.18.0143
AB  - Background Obesity in adolescence can continue up to adulthood and lead to various complications. There has been a continuous increase in the obesity incidence among adolescents, which is an emerging social issue. Studies have reported that decreased sleep duration in adolescents can have negative effects on health, leading to obesity. Therefore, in the present study, we aimed to investigate the relationship between sleep duration and body mass index among South Korean middle and high school students aged 12–18 years. Methods The participants of the Sixth National Health and Nutrition Survey conducted during 2013–2015 were screened. We selected 1,177 adolescents aged between 12 and 18 years who were enrolled in middle and high school or on leave of absence and had available data for sleep duration and body mass index (BMI) in the survey. Analyses of variance and covariance were performed after adjusting for confounding variables, including age, gender, waist circumference, and drinking experience, that showed significant correlations in univariate logistic regression. Results The analysis of covariance revealed that an increase in sleep duration led to a corresponding decrease in the mean BMI among all students and middle school students. Conclusion Significant negative correlations were confirmed between sleep duration and BMI in all students and middle school students.
ER  - 

BibTeX

@misc{bb373d3cb00dd9f8be8a610977d95b6a956693d5,
  author = {Jing Fan and Caicui Ding and Weiyan Gong and Fan Yuan and Yan Zhang and Ganyu Feng and Chao Song and Ailing Liu},
  title = {Association of Sleep Duration and Overweight/Obesity among Children in China},
  journal = {International Journal of Environmental Research and Public Health},
  year = {2020},
  volume = {17},
  doi = {10.3390/ijerph17061962},
  abstract = {To investigate the association of sleep duration with overweight and obesity among children aged 6 to 17 years in China, 2010–2012 data from the China National Nutrition and Health Surveillance (CNHHS) were analyzed. A total of 35,414 children were recruited in the survey. Body mass index (BMI) was converted into three categories: normal weight, overweight and obesity. In multinomial logistic regression model, sleep duration was divided into four groups: very short, short, recommended and long. In restricted cubic splines (RCS), sleep duration was examined as a continuous variable in relation to overweight and obesity. In the very short and short groups, sleep duration was a risk factor for obesity after adjusting for the potential impacts of age, gender, residence, family income, leisure sedentary behavior (SB) and leisure exercise, with OR (Odds Ratio) = 3.01 (95% CI (confidence interval): 2.19–4.15) and OR = 1.24 (95% CI: 1.14–1.35), respectively. The adjusted OR of overweight for short sleep duration relative to a recommended sleep duration was 1.17(95% CI: 1.09–1.26). No significant associations of very short sleep with overweight, of long sleep duration with overweight and obesity were found. The RCS curves between sleep duration and overweight and obesity were both inverted J-shaped. To conclude, the shorter the sleep duration, the higher the risk of overweight and obesity in children. Increasing sleep duration would have a positive effect on reducing overweight and obesity rates in Chinese children.}
}

RIS

TY  - JOUR
AU  - Jing Fan
AU  - Caicui Ding
AU  - Weiyan Gong
AU  - Fan Yuan
AU  - Yan Zhang
AU  - Ganyu Feng
AU  - Chao Song
AU  - Ailing Liu
T1  - Association of Sleep Duration and Overweight/Obesity among Children in China
JO  - International Journal of Environmental Research and Public Health
PY  - 2020
VL  - 17
DO  - 10.3390/ijerph17061962
AB  - To investigate the association of sleep duration with overweight and obesity among children aged 6 to 17 years in China, 2010–2012 data from the China National Nutrition and Health Surveillance (CNHHS) were analyzed. A total of 35,414 children were recruited in the survey. Body mass index (BMI) was converted into three categories: normal weight, overweight and obesity. In multinomial logistic regression model, sleep duration was divided into four groups: very short, short, recommended and long. In restricted cubic splines (RCS), sleep duration was examined as a continuous variable in relation to overweight and obesity. In the very short and short groups, sleep duration was a risk factor for obesity after adjusting for the potential impacts of age, gender, residence, family income, leisure sedentary behavior (SB) and leisure exercise, with OR (Odds Ratio) = 3.01 (95% CI (confidence interval): 2.19–4.15) and OR = 1.24 (95% CI: 1.14–1.35), respectively. The adjusted OR of overweight for short sleep duration relative to a recommended sleep duration was 1.17(95% CI: 1.09–1.26). No significant associations of very short sleep with overweight, of long sleep duration with overweight and obesity were found. The RCS curves between sleep duration and overweight and obesity were both inverted J-shaped. To conclude, the shorter the sleep duration, the higher the risk of overweight and obesity in children. Increasing sleep duration would have a positive effect on reducing overweight and obesity rates in Chinese children.
ER  - 

BibTeX

@misc{79f0eacc0956f56ccd7b8b7cf2a2328b7f6d6c19,
  author = {C. Magee and P. Caputi and D. Iverson},
  title = {Is Sleep Duration Associated With Obesity in Older Australian Adults?},
  journal = {Journal of Aging and Health},
  year = {2010},
  volume = {22},
  pages = {1235 - 1255},
  doi = {10.1177/0898264310372780},
  abstract = {Objectives: To investigate the relationship between sleep duration and obesity in older Australian adults. Method: Self-reported health data were collected through the 45 and Up Study. Multinomial logistic regression models were used to test the relationship between sleep duration and body mass index, controlling for a range of demographic and health-related variables. Results: The sample included 45,325 Australian adults aged 55 to 95 years. The regression models demonstrated that short and long sleep were associated with obesity in 55- to 64-year-olds but not in those aged 65 years and above. Discussion: The present results suggest that the relationships between sleep duration and obesity previously reported in young and middle-aged adults are not evident in older adults. The absence of these relationships could reflect a combination of age-related factors that impact on sleep and body composition.}
}

RIS

TY  - JOUR
AU  - C. Magee
AU  - P. Caputi
AU  - D. Iverson
T1  - Is Sleep Duration Associated With Obesity in Older Australian Adults?
JO  - Journal of Aging and Health
PY  - 2010
VL  - 22
SP  - 1235 - 1255
EP  - 1235 - 1255
DO  - 10.1177/0898264310372780
AB  - Objectives: To investigate the relationship between sleep duration and obesity in older Australian adults. Method: Self-reported health data were collected through the 45 and Up Study. Multinomial logistic regression models were used to test the relationship between sleep duration and body mass index, controlling for a range of demographic and health-related variables. Results: The sample included 45,325 Australian adults aged 55 to 95 years. The regression models demonstrated that short and long sleep were associated with obesity in 55- to 64-year-olds but not in those aged 65 years and above. Discussion: The present results suggest that the relationships between sleep duration and obesity previously reported in young and middle-aged adults are not evident in older adults. The absence of these relationships could reflect a combination of age-related factors that impact on sleep and body composition.
ER  - 

BibTeX

@misc{87fdf1e4e85e2ce2c4a65316e71c0450e190f19f,
  author = {N. Häusler and R. Heinzer and J. Haba-Rubio and P. Marques-Vidal},
  title = {Does sleep affect weight gain? Assessing subjective sleep and polysomnography measures in a population-based cohort study (CoLaus/HypnoLaus).},
  journal = {Sleep},
  year = {2019},
  volume = {42 6},
  doi = {10.1093/sleep/zsz077},
  abstract = {STUDY OBJECTIVES
Although several studies have linked short and long sleep duration to weight gain, mixed results exist. Contrarily, few studies associated objectively measured sleep characteristics with weight gain. We investigated the association between several sleep characteristics measured by questionnaire and polysomnography with prospective weight gain in a population-based, middle-aged cohort.


METHODS
Three samples were analyzed: sample 1 (n = 2551, 47.3% men, 56.9 ± 10.3 years) had data for subjective sleep characteristics, sample 2 (n = 1422, 49.4% men, 57.6 ± 10.4 years) had objective sleep assessment (polysomnography), and sample 3 consisting of 1259 subjects included in both samples. Multivariable logistic regressions were performed to assess the relationship between sleep characteristics and ≥5 kg weight gain during a median follow-up of 5.3 years.


RESULTS
In both study samples, 12% of the subjects gained ≥5 kg during follow-up. Multivariable analyses showed poor subjective sleep quality (as assessed by Pittsburgh Sleep Quality Index: odds ratio [95% confidence interval] = 1.54 [1.19 to 1.99]), percentage of sleep spent in stage 2 (1.32 [1.10 to 1.58]), and less than 90% oxygen saturation (SpO2 < 90) (1.23 [1.07 to 1.41]); moderate/severe Oxygen Desaturation Index (1.70 [1.01 to 2.85]) and autonomic arousal duration (1.22 [1.02 to 1.45]) were related to ≥5 kg weight gain. Only poor subjective sleep quality was robustly associated with weight gain in all sensitivity analyses, except in female subsamples.


CONCLUSIONS
Poor subjective sleep quality, and to some extent moderate to severe oxygen desaturation, but no other sleep characteristics, were robustly associated with weight gain. Future studies should confirm the relationship between sleep quality and weight gain, assess sex differences, and investigate underlying mechanisms.}
}

RIS

TY  - JOUR
AU  - N. Häusler
AU  - R. Heinzer
AU  - J. Haba-Rubio
AU  - P. Marques-Vidal
T1  - Does sleep affect weight gain? Assessing subjective sleep and polysomnography measures in a population-based cohort study (CoLaus/HypnoLaus).
JO  - Sleep
PY  - 2019
VL  - 42 6
DO  - 10.1093/sleep/zsz077
AB  - STUDY OBJECTIVES
Although several studies have linked short and long sleep duration to weight gain, mixed results exist. Contrarily, few studies associated objectively measured sleep characteristics with weight gain. We investigated the association between several sleep characteristics measured by questionnaire and polysomnography with prospective weight gain in a population-based, middle-aged cohort.


METHODS
Three samples were analyzed: sample 1 (n = 2551, 47.3% men, 56.9 ± 10.3 years) had data for subjective sleep characteristics, sample 2 (n = 1422, 49.4% men, 57.6 ± 10.4 years) had objective sleep assessment (polysomnography), and sample 3 consisting of 1259 subjects included in both samples. Multivariable logistic regressions were performed to assess the relationship between sleep characteristics and ≥5 kg weight gain during a median follow-up of 5.3 years.


RESULTS
In both study samples, 12% of the subjects gained ≥5 kg during follow-up. Multivariable analyses showed poor subjective sleep quality (as assessed by Pittsburgh Sleep Quality Index: odds ratio [95% confidence interval] = 1.54 [1.19 to 1.99]), percentage of sleep spent in stage 2 (1.32 [1.10 to 1.58]), and less than 90% oxygen saturation (SpO2 < 90) (1.23 [1.07 to 1.41]); moderate/severe Oxygen Desaturation Index (1.70 [1.01 to 2.85]) and autonomic arousal duration (1.22 [1.02 to 1.45]) were related to ≥5 kg weight gain. Only poor subjective sleep quality was robustly associated with weight gain in all sensitivity analyses, except in female subsamples.


CONCLUSIONS
Poor subjective sleep quality, and to some extent moderate to severe oxygen desaturation, but no other sleep characteristics, were robustly associated with weight gain. Future studies should confirm the relationship between sleep quality and weight gain, assess sex differences, and investigate underlying mechanisms.
ER  - 

BibTeX

@misc{679dcb7dc68af2d82b65fce0354baf48a7ebc8dd,
  author = {J. Araújo and M. Severo and E. Ramos},
  title = {Sleep Duration and Adiposity During Adolescence},
  journal = {Pediatrics},
  year = {2012},
  volume = {130},
  pages = {e1146 - e1154},
  doi = {10.1542/peds.2011-1116},
  abstract = {BACKGROUND AND OBJECTIVE: The association between sleep and obesity has been described in different age groups. However, there are not sufficient data to clarify the inconsistent results reported in adolescents. Our objective was to study the associations between sleep duration and adiposity at 13 and at 17 years of age, with both cross-sectional and longitudinal approaches. METHODS: We evaluated, as part of an urban population-based cohort (EPITeen), 1171 adolescents at both 13 and 17 years of age. Sleep duration was estimated by self-reported bedtimes and wake-up times. Age- and gender-specific BMI z scores were calculated based on Centers for Disease Control and Prevention references. Body fat percentage (BF%) was assessed by bioelectrical impedance. Regression coefficients (β) and respective 95% confidence intervals (CIs) were used to estimate the association between sleep and BMI z scores and BF%. Additionally, a cross-lagged analysis was performed to investigate the causal relations. RESULTS: In the cross-sectional analysis, at 13 years, sleep duration was inversely associated with BMI z score only in boys (β = −0.155, 95% CI: −0.267 to −0.043); at 17 years, a positive association was found among girls but was only significant for BF% (β = 0.510, 95% CI: 0.061–0.958). In the longitudinal approach, sleep duration at age 13 was inversely associated with BMI z score (β = −0.123, 95% CI: −0.233 to −0.012) and BF% (β = −0.731, 95% CI: −1.380 to −0.081) at 17 years only in boys. These significant associations disappeared after adjustment for adiposity at 13 years. These results were corroborated by those from cross-lagged analysis. CONCLUSIONS: Our results showed an effect of sleep duration in adiposity at younger ages of adolescence and suggested gender differences in this association.}
}

RIS

TY  - JOUR
AU  - J. Araújo
AU  - M. Severo
AU  - E. Ramos
T1  - Sleep Duration and Adiposity During Adolescence
JO  - Pediatrics
PY  - 2012
VL  - 130
SP  - e1146 - e1154
EP  - e1146 - e1154
DO  - 10.1542/peds.2011-1116
AB  - BACKGROUND AND OBJECTIVE: The association between sleep and obesity has been described in different age groups. However, there are not sufficient data to clarify the inconsistent results reported in adolescents. Our objective was to study the associations between sleep duration and adiposity at 13 and at 17 years of age, with both cross-sectional and longitudinal approaches. METHODS: We evaluated, as part of an urban population-based cohort (EPITeen), 1171 adolescents at both 13 and 17 years of age. Sleep duration was estimated by self-reported bedtimes and wake-up times. Age- and gender-specific BMI z scores were calculated based on Centers for Disease Control and Prevention references. Body fat percentage (BF%) was assessed by bioelectrical impedance. Regression coefficients (β) and respective 95% confidence intervals (CIs) were used to estimate the association between sleep and BMI z scores and BF%. Additionally, a cross-lagged analysis was performed to investigate the causal relations. RESULTS: In the cross-sectional analysis, at 13 years, sleep duration was inversely associated with BMI z score only in boys (β = −0.155, 95% CI: −0.267 to −0.043); at 17 years, a positive association was found among girls but was only significant for BF% (β = 0.510, 95% CI: 0.061–0.958). In the longitudinal approach, sleep duration at age 13 was inversely associated with BMI z score (β = −0.123, 95% CI: −0.233 to −0.012) and BF% (β = −0.731, 95% CI: −1.380 to −0.081) at 17 years only in boys. These significant associations disappeared after adjustment for adiposity at 13 years. These results were corroborated by those from cross-lagged analysis. CONCLUSIONS: Our results showed an effect of sleep duration in adiposity at younger ages of adolescence and suggested gender differences in this association.
ER  - 

BibTeX

@misc{8b7be86553333cef77551c2d5a4e08a7dc46722c,
  author = {Liuqianzi Yan and Xiao Rong Chen and Bo Chen and Ya-min Bai and Jian Hong Li and X. Zhang and Z. Dong and Hao Wang and S. Mi and Wen-hua Zhao},
  title = {Gender-specific Association of Sleep Duration with Body Mass Index, Waist Circumference, and Body Fat in Chinese Adults.},
  journal = {Biomedical and environmental sciences : BES},
  year = {2017},
  volume = {30 3},
  pages = {
          157-169
        },
  doi = {10.3967/bes2017.023},
  abstract = {OBJECTIVE
To examine the association between habitual sleep duration and obesity among Chinese adults.


METHODS
The association of sleep duration and obesity was investigated among 7,094 community-dwelling Chinese adults. Sleep duration was self-reported. In this study, obesity was defined as follows: body mass index (BMI) ⋝ 28 kg/m2, waist circumference (WC) ⋝ 85 cm in men and ⋝ 80 cm in women, and percent body fat (%BF) ⋝ 25 in men and ⋝ 35 in women. Logistic and quantile regressions were employed to examine relationships of interest.


RESULTS
Overall, 6.42% of the participants reported short sleep durations (< 6 h/d) while 14.71% reported long (⋝ 9 h/d) sleep durations. Long sleepers (⋝ 9 h/d) represented a greater frequency of women with obesity [odds ratio (OR): 1.30; 95% confidence interval (CI), 1.02-1.67] and high body fat (1.43, 1.04-1.96) than those who slept 7-8 h/d. An association between long sleep times and higher BMI estimations was found across the 10th-75th percentile of the BMI distribution. Among men, long sleepers (⋝ 9 h/d) presented lower risks of developing abdominal obesity compared with individuals who slept 7-8 h/d (OR = 0.79, 95% CI: 0.44-0.99).


CONCLUSION
Our study suggests that long sleep durations are associated with general obesity in Chinese women but reduced waist circumferences in men. Confirmatory studies are needed to determine the heterogeneous association of sleep time and obesity by gender.}
}

RIS

TY  - JOUR
AU  - Liuqianzi Yan
AU  - Xiao Rong Chen
AU  - Bo Chen
AU  - Ya-min Bai
AU  - Jian Hong Li
AU  - X. Zhang
AU  - Z. Dong
AU  - Hao Wang
AU  - S. Mi
AU  - Wen-hua Zhao
T1  - Gender-specific Association of Sleep Duration with Body Mass Index, Waist Circumference, and Body Fat in Chinese Adults.
JO  - Biomedical and environmental sciences : BES
PY  - 2017
VL  - 30 3
SP  - 
          157-169
        
EP  - 
          157-169
        
DO  - 10.3967/bes2017.023
AB  - OBJECTIVE
To examine the association between habitual sleep duration and obesity among Chinese adults.


METHODS
The association of sleep duration and obesity was investigated among 7,094 community-dwelling Chinese adults. Sleep duration was self-reported. In this study, obesity was defined as follows: body mass index (BMI) ⋝ 28 kg/m2, waist circumference (WC) ⋝ 85 cm in men and ⋝ 80 cm in women, and percent body fat (%BF) ⋝ 25 in men and ⋝ 35 in women. Logistic and quantile regressions were employed to examine relationships of interest.


RESULTS
Overall, 6.42% of the participants reported short sleep durations (< 6 h/d) while 14.71% reported long (⋝ 9 h/d) sleep durations. Long sleepers (⋝ 9 h/d) represented a greater frequency of women with obesity [odds ratio (OR): 1.30; 95% confidence interval (CI), 1.02-1.67] and high body fat (1.43, 1.04-1.96) than those who slept 7-8 h/d. An association between long sleep times and higher BMI estimations was found across the 10th-75th percentile of the BMI distribution. Among men, long sleepers (⋝ 9 h/d) presented lower risks of developing abdominal obesity compared with individuals who slept 7-8 h/d (OR = 0.79, 95% CI: 0.44-0.99).


CONCLUSION
Our study suggests that long sleep durations are associated with general obesity in Chinese women but reduced waist circumferences in men. Confirmatory studies are needed to determine the heterogeneous association of sleep time and obesity by gender.
ER  - 

BibTeX

@misc{c35696077e94c9d7b8eab1de9e72d06f43dd38e0,
  author = {J. Chaput and L. Klingenberg and A. Sjödin},
  title = {Do all sedentary activities lead to weight gain: sleep does not},
  journal = {Current Opinion in Clinical Nutrition and Metabolic Care},
  year = {2010},
  volume = {13},
  pages = {601–607},
  doi = {10.1097/MCO.0b013e32833ef30e},
  abstract = {Purpose of reviewTo discuss the benefits of having a good night's sleep for body weight stability. Recent findingsExperimental studies have shown that short-term partial sleep restriction decreases glucose tolerance, increases sympathetic tone, elevates cortisol concentrations, decreases the satiety hormone leptin, increases the appetite-stimulating hormone ghrelin, and increases hunger and appetite. Short sleep duration might increase the risk of becoming obese, because it does not allow the recovery of a hormonal profile facilitating appetite control. Lack of sleep could also lead to weight gain and obesity by increasing the time available for eating and by making the maintenance of a healthy lifestyle more difficult. Furthermore, the increased fatigue and tiredness associated with sleeping too little could lessen one's resolve to follow exercise regimens. SummaryShort sleep duration appears to be a novel and independent risk factor for obesity. With the growing prevalence of chronic sleep restriction, any causal association between reduced sleep and obesity would have substantial importance from a public health standpoint. Future research is needed to determine whether sleep extension in sleep-deprived obese individuals will influence appetite control and/or reduce the amount of body fat.}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - L. Klingenberg
AU  - A. Sjödin
T1  - Do all sedentary activities lead to weight gain: sleep does not
JO  - Current Opinion in Clinical Nutrition and Metabolic Care
PY  - 2010
VL  - 13
SP  - 601–607
EP  - 601–607
DO  - 10.1097/MCO.0b013e32833ef30e
AB  - Purpose of reviewTo discuss the benefits of having a good night's sleep for body weight stability. Recent findingsExperimental studies have shown that short-term partial sleep restriction decreases glucose tolerance, increases sympathetic tone, elevates cortisol concentrations, decreases the satiety hormone leptin, increases the appetite-stimulating hormone ghrelin, and increases hunger and appetite. Short sleep duration might increase the risk of becoming obese, because it does not allow the recovery of a hormonal profile facilitating appetite control. Lack of sleep could also lead to weight gain and obesity by increasing the time available for eating and by making the maintenance of a healthy lifestyle more difficult. Furthermore, the increased fatigue and tiredness associated with sleeping too little could lessen one's resolve to follow exercise regimens. SummaryShort sleep duration appears to be a novel and independent risk factor for obesity. With the growing prevalence of chronic sleep restriction, any causal association between reduced sleep and obesity would have substantial importance from a public health standpoint. Future research is needed to determine whether sleep extension in sleep-deprived obese individuals will influence appetite control and/or reduce the amount of body fat.
ER  - 

BibTeX

@misc{7d3a457d32899ac957cd1501141b275ac498857e,
  author = {P. Lyytikäinen and O. Rahkonen and E. Lahelma and T. Lallukka},
  title = {Association of sleep duration with weight and weight gain: a prospective follow‐up study},
  journal = {Journal of Sleep Research},
  year = {2011},
  volume = {20},
  doi = {10.1111/j.1365-2869.2010.00903.x},
  abstract = {The objective of this study was to examine the associations of sleep duration with subsequent weight and major weight gain in women and men during a follow‐up period of 5–7 years. The data were derived from the Helsinki Health Study cohort mail questionnaire surveys among 40–60‐year‐old employees of the City of Helsinki, Finland. At baseline in 2000–2002, 8960 people responded to the survey (80% women, response rate 67%). The follow‐up survey was conducted in 2007 among all respondents to the baseline survey (n = 7332, response rate 83%). Sleep duration (5 h or less up to 10 h or more) and weight and weight gain of at least 5 kg were based on self‐reports. Analyses of variance and logistic regression analyses were used to examine the associations between sleep duration and weight, as well as major weight gain. Short sleep duration was associated with major weight gain [odds ratio (OR) 1.52; 95% confidence interval (CI) 1.08, 2.14] during the follow‐up. Adjusting for several covariates had only minor effects on that association. Long sleep duration was associated with major weight gain after adjusting for age (OR 1.35; 95% CI 1.00–1.81). No associations were found among men. Sleep duration was associated with major weight gain among middle‐aged employed women. Short sleep may be a risk factor for subsequent weight gain.}
}

RIS

TY  - JOUR
AU  - P. Lyytikäinen
AU  - O. Rahkonen
AU  - E. Lahelma
AU  - T. Lallukka
T1  - Association of sleep duration with weight and weight gain: a prospective follow‐up study
JO  - Journal of Sleep Research
PY  - 2011
VL  - 20
DO  - 10.1111/j.1365-2869.2010.00903.x
AB  - The objective of this study was to examine the associations of sleep duration with subsequent weight and major weight gain in women and men during a follow‐up period of 5–7 years. The data were derived from the Helsinki Health Study cohort mail questionnaire surveys among 40–60‐year‐old employees of the City of Helsinki, Finland. At baseline in 2000–2002, 8960 people responded to the survey (80% women, response rate 67%). The follow‐up survey was conducted in 2007 among all respondents to the baseline survey (n = 7332, response rate 83%). Sleep duration (5 h or less up to 10 h or more) and weight and weight gain of at least 5 kg were based on self‐reports. Analyses of variance and logistic regression analyses were used to examine the associations between sleep duration and weight, as well as major weight gain. Short sleep duration was associated with major weight gain [odds ratio (OR) 1.52; 95% confidence interval (CI) 1.08, 2.14] during the follow‐up. Adjusting for several covariates had only minor effects on that association. Long sleep duration was associated with major weight gain after adjusting for age (OR 1.35; 95% CI 1.00–1.81). No associations were found among men. Sleep duration was associated with major weight gain among middle‐aged employed women. Short sleep may be a risk factor for subsequent weight gain.
ER  - 

BibTeX

@misc{c043291c01af6444681624ada609cd56ab0de29b,
  author = {A. A. Schäfer and M. Domingues and D. Dahly and Fernanda de Oliveira Meller and H. Gonçalves and F. Wehrmeister and M. Assunção},
  title = {Sleep Duration Trajectories and Body Composition in Adolescents: Prospective Birth Cohort Study},
  journal = {PLoS ONE},
  year = {2016},
  volume = {11},
  doi = {10.1371/journal.pone.0152348},
  abstract = {We aimed to estimate the association between sleep duration trajectories and body composition in adolescents. We used data from participants of the 1993 Pelotas (Brazil) Birth Cohort Study who were later followed up at age 18 years (response rate of 81.3%). At the time, 3974 adolescents had complete data on body composition, which was assessed by air displacement plethysmography. Sleep duration was self-reported by participants at ages 11 and 18 years. Analyses were sex-stratified. The mean sleep duration at 11 years was 9.7 (SD 1.4) and 8.4 (SD 1.9) at 18 years. Sleep duration was dichotomized as inadequate (<8 hours/day) or adequate (≥8 hours/day). Mean body mass, fat mass, and fat-free mass indices at 18 years were 23.4 kg/m2 (SD 4.5), 6.1 kg/m2 (SD 3.9) and 17.3 kg/m2 (SD 2.5), respectively. Girls who reported inadequate sleep duration at 11 years of age, but adequate sleep duration at 18, on average experienced an increase in body mass index (β = 0.39 z-scores; 95% CI 0.13, 0.65), fat mass index (β = 0.30 z-scores; 95% CI 0.07, 0.53), and fat-free mass index (β = 0.24 z-scores; 95% CI 0.08, 0.39) compared to those who had adequate sleep duration at both time points. The results suggest that changes in sleep duration across adolescence may impact body composition in later adolescence and that this may differ by sex.}
}

RIS

TY  - JOUR
AU  - A. A. Schäfer
AU  - M. Domingues
AU  - D. Dahly
AU  - Fernanda de Oliveira Meller
AU  - H. Gonçalves
AU  - F. Wehrmeister
AU  - M. Assunção
T1  - Sleep Duration Trajectories and Body Composition in Adolescents: Prospective Birth Cohort Study
JO  - PLoS ONE
PY  - 2016
VL  - 11
DO  - 10.1371/journal.pone.0152348
AB  - We aimed to estimate the association between sleep duration trajectories and body composition in adolescents. We used data from participants of the 1993 Pelotas (Brazil) Birth Cohort Study who were later followed up at age 18 years (response rate of 81.3%). At the time, 3974 adolescents had complete data on body composition, which was assessed by air displacement plethysmography. Sleep duration was self-reported by participants at ages 11 and 18 years. Analyses were sex-stratified. The mean sleep duration at 11 years was 9.7 (SD 1.4) and 8.4 (SD 1.9) at 18 years. Sleep duration was dichotomized as inadequate (<8 hours/day) or adequate (≥8 hours/day). Mean body mass, fat mass, and fat-free mass indices at 18 years were 23.4 kg/m2 (SD 4.5), 6.1 kg/m2 (SD 3.9) and 17.3 kg/m2 (SD 2.5), respectively. Girls who reported inadequate sleep duration at 11 years of age, but adequate sleep duration at 18, on average experienced an increase in body mass index (β = 0.39 z-scores; 95% CI 0.13, 0.65), fat mass index (β = 0.30 z-scores; 95% CI 0.07, 0.53), and fat-free mass index (β = 0.24 z-scores; 95% CI 0.08, 0.39) compared to those who had adequate sleep duration at both time points. The results suggest that changes in sleep duration across adolescence may impact body composition in later adolescence and that this may differ by sex.
ER  - 

BibTeX

@misc{57a1e28ecafbd12a3cc54540a422d8733073477b,
  author = {Seung-Ho Han and Jae-Yong Yee and J. Pyo},
  title = {Impact of Short Sleep Duration on the Incidence of Obesity and Overweight among Children and Adolescents},
  journal = {Medicina},
  year = {2022},
  volume = {58},
  doi = {10.3390/medicina58081037},
  abstract = {Background and Objectives: The prevalence of obesity among children is increasing and is highlighting many problems. Lack of sleep is common among children and adolescents. Although several studies have investigated sleep duration and overweight and obesity from a sex perspective, data regarding age and sex effects remain limited and inconclusive. This study aimed to evaluate the risk(s) for overweight or obesity according to sleep duration among children and adolescents; to evaluate the effect of short sleep duration on the incidence of overweight/obesity among children and adolescents; and to evaluate sex differences in the risk of overweight or obesity with shorter sleep durations. Materials and Methods: The PubMed database was searched for relevant studies published up to June 30, 2021. Odds ratios for obesity/overweight were estimated for short compared with long sleep duration. Subgroup analysis based on sleep duration, sex, and study location was also performed. Results: The estimated odds ratio for combined obesity and overweight was 1.171 (95% confidence interval (CI) 1.092–1.256) according to short sleep duration. Obesity/overweight with short sleep duration was significantly prevalent in the <6 and 6–10 years’ subgroups (odds ratio 1.226 (95% CI 1.083–1.387) and 1.341 (95% CI 1.175–1.530), respectively). Among boys, short sleep duration was significantly correlated with a high occurrence of obesity/overweight (odds ratio 1.294 (95% CI 1.153–1.452)); no such correlation was found among girls. Conclusions: Short sleep duration may increase risk of obesity among children and adolescents, especially those <6 and 6–10 years of age. In the subgroup analysis, the incidence of obesity/overweight for short sleep time revealed significant results among Asians and boys.}
}

RIS

TY  - JOUR
AU  - Seung-Ho Han
AU  - Jae-Yong Yee
AU  - J. Pyo
T1  - Impact of Short Sleep Duration on the Incidence of Obesity and Overweight among Children and Adolescents
JO  - Medicina
PY  - 2022
VL  - 58
DO  - 10.3390/medicina58081037
AB  - Background and Objectives: The prevalence of obesity among children is increasing and is highlighting many problems. Lack of sleep is common among children and adolescents. Although several studies have investigated sleep duration and overweight and obesity from a sex perspective, data regarding age and sex effects remain limited and inconclusive. This study aimed to evaluate the risk(s) for overweight or obesity according to sleep duration among children and adolescents; to evaluate the effect of short sleep duration on the incidence of overweight/obesity among children and adolescents; and to evaluate sex differences in the risk of overweight or obesity with shorter sleep durations. Materials and Methods: The PubMed database was searched for relevant studies published up to June 30, 2021. Odds ratios for obesity/overweight were estimated for short compared with long sleep duration. Subgroup analysis based on sleep duration, sex, and study location was also performed. Results: The estimated odds ratio for combined obesity and overweight was 1.171 (95% confidence interval (CI) 1.092–1.256) according to short sleep duration. Obesity/overweight with short sleep duration was significantly prevalent in the <6 and 6–10 years’ subgroups (odds ratio 1.226 (95% CI 1.083–1.387) and 1.341 (95% CI 1.175–1.530), respectively). Among boys, short sleep duration was significantly correlated with a high occurrence of obesity/overweight (odds ratio 1.294 (95% CI 1.153–1.452)); no such correlation was found among girls. Conclusions: Short sleep duration may increase risk of obesity among children and adolescents, especially those <6 and 6–10 years of age. In the subgroup analysis, the incidence of obesity/overweight for short sleep time revealed significant results among Asians and boys.
ER  - 

BibTeX

@misc{3afd225f0962a9766c3fa23c50461214fa85b4cd,
  author = {E. Osondu and E. Aneni and Maribeth Rouseff and Sankalp Das and D. Parris and Henry Guzman and Thinh H. Tran and A. Younus and R. Ahmad and S. Ali and M. Latif and T. Feldman and E. Veledar and K. Nasir},
  title = {Abstract P304: The Association of Sleep Duration and Severe Obesity in a Working Population: The Baptist Health South Florida Employee Study},
  journal = {Circulation},
  year = {2016},
  volume = {133},
  abstract = {Background: Although studies have described an inverse relationship between sleep duration and obesity, there is limited knowledge about the association between sleep duration and severe obesity in...}
}

RIS

TY  - JOUR
AU  - E. Osondu
AU  - E. Aneni
AU  - Maribeth Rouseff
AU  - Sankalp Das
AU  - D. Parris
AU  - Henry Guzman
AU  - Thinh H. Tran
AU  - A. Younus
AU  - R. Ahmad
AU  - S. Ali
AU  - M. Latif
AU  - T. Feldman
AU  - E. Veledar
AU  - K. Nasir
T1  - Abstract P304: The Association of Sleep Duration and Severe Obesity in a Working Population: The Baptist Health South Florida Employee Study
JO  - Circulation
PY  - 2016
VL  - 133
AB  - Background: Although studies have described an inverse relationship between sleep duration and obesity, there is limited knowledge about the association between sleep duration and severe obesity in...
ER  - 

BibTeX

@misc{f24d81ea827ce56944ab8062df3eddbafe469140,
  author = {V. Yiengprugsawan and C. Banwell and S. Seubsman and A. Sleigh},
  title = {Short sleep and obesity in a large national cohort of Thai adults},
  journal = {BMJ Open},
  year = {2012},
  volume = {2},
  doi = {10.1136/bmjopen-2011-000561},
  abstract = {Objective To investigate the relationship between short sleep and obesity among Thai adults. Design Both 4-year longitudinal and cross-sectional analyses of a large national cohort. Setting Thai adults residing nationwide from 2005 to 2009. Participants Cohort members were enrolled as distance learners at Sukhothai Thammathirat Open University (N=87 134 in 2005 and 60 569 at 2009 follow-up). At 2005 baseline, 95% were between 20 and 49 years of age. Measures Self-reported sleep duration was categorised as <6, 6, 7, 8 and ≥9 h. For all analyses (2005 and 2009 cross-sectional and 2005–2009 longitudinal), we used multinomial logistic regression models to assess the effect of sleep duration on abnormal body size (underweight, overweight-at-risk, obese). Results were adjusted for an array of relevant covariates. Results At the last cohort follow-up in 2009, cross-sectional associations linked short sleep (<6 h) and obesity: adjusted ORs (AOR) =1.49, 95% CIs 1.32 to 1.68 for women and AOR=1.36, 95% CI 1.21 to 1.52 for men. The earlier cross-sectional baseline results in 2005 were quite similar. Longitudinal analysis (2005–2009) of 4-year incremental weight gain (5 to <10%, 10 to <20% and 20%+) strongly supported the short sleep–obesity relationship (significant AORs of 1.10, 1.30 and 1.69, respectively). Conclusions The results are internally consistent (2005 and 2009) and longitudinally confirmatory of a short sleep effect on obesity among Thai adults. Further research is needed to elucidate causal mechanisms underlying the sleep–obesity relationship.}
}

RIS

TY  - JOUR
AU  - V. Yiengprugsawan
AU  - C. Banwell
AU  - S. Seubsman
AU  - A. Sleigh
T1  - Short sleep and obesity in a large national cohort of Thai adults
JO  - BMJ Open
PY  - 2012
VL  - 2
DO  - 10.1136/bmjopen-2011-000561
AB  - Objective To investigate the relationship between short sleep and obesity among Thai adults. Design Both 4-year longitudinal and cross-sectional analyses of a large national cohort. Setting Thai adults residing nationwide from 2005 to 2009. Participants Cohort members were enrolled as distance learners at Sukhothai Thammathirat Open University (N=87 134 in 2005 and 60 569 at 2009 follow-up). At 2005 baseline, 95% were between 20 and 49 years of age. Measures Self-reported sleep duration was categorised as <6, 6, 7, 8 and ≥9 h. For all analyses (2005 and 2009 cross-sectional and 2005–2009 longitudinal), we used multinomial logistic regression models to assess the effect of sleep duration on abnormal body size (underweight, overweight-at-risk, obese). Results were adjusted for an array of relevant covariates. Results At the last cohort follow-up in 2009, cross-sectional associations linked short sleep (<6 h) and obesity: adjusted ORs (AOR) =1.49, 95% CIs 1.32 to 1.68 for women and AOR=1.36, 95% CI 1.21 to 1.52 for men. The earlier cross-sectional baseline results in 2005 were quite similar. Longitudinal analysis (2005–2009) of 4-year incremental weight gain (5 to <10%, 10 to <20% and 20%+) strongly supported the short sleep–obesity relationship (significant AORs of 1.10, 1.30 and 1.69, respectively). Conclusions The results are internally consistent (2005 and 2009) and longitudinally confirmatory of a short sleep effect on obesity among Thai adults. Further research is needed to elucidate causal mechanisms underlying the sleep–obesity relationship.
ER  - 

BibTeX

@misc{a0adcfac8f877813737720c91345997e4c73b775,
  author = {Y. Li and R. Tao and D. Gao and B. Wen and B. Dong and Y. Song and Z. Zou and J. Ma},
  title = {[A study on the relationship between insufficient sleep and obesity among children and adolescents in China].},
  journal = {Zhonghua liu xing bing xue za zhi = Zhonghua liuxingbingxue zazhi},
  year = {2020},
  volume = {41 6},
  pages = {
          845-849
        },
  doi = {10.3760/cma.j.cn112338-20190711-00514},
  abstract = {Objective: To analyze the relationship between insufficient sleep and obesity or central obesity in Chinese Han children and adolescents aged 9-18 years. Methods: A total of 172 710 students who participated in the 2014 Chinese National Survey on Students Constitution and Health with complete data of sleep duration and physical examination, were selected as study subjects. Insufficient sleep was defined, according to the amount of sleep for pediatric populations recommended by the American Academy of Sleep Medicine. Obesity and central obesity of children and adolescents were judged by experts from the Group of China Obesity Task Force and Health Industry Standards in China. Differences between groups were compared by using the t test or χ(2) test. Logistic regression method was applied to assess the relationship between insufficient sleep and obesity or central obesity. Results: In 2014, numbers of students with insufficient sleep, obesity and central obesity among Chinese Han children and adolescents aged 9-18 years appeared as 133 410 (77.2%), 11 145 (6.5%), and 12 603 (17.8%), respectively. Among the students with insufficient sleep, 8 358 (6.3%) were with obesity and 12 244 (17.9%) were with central obesity. The prevalence of obesity and central obesity among boys with insufficient sleep was higher than that in girls. Pupils with insufficient sleep showed the highest prevalence of obesity and central obesity. After controlling for potential confounders, the risk of obesity appeared an increase of 14.5% (OR=1.145, 95%CI: 1.092-1.200) and the risk of central obesity increased by 12.7% (OR=1.127, 95%CI: 1.078-1.178) in students with insufficient sleep, when compared with those with adequate sleep. Compared with those whose daily sleep duration was less than 6 hours, the ones who slept 7-10 hours per day showed significantly reduction on the risk of obesity and central obesity in students. Conclusions: Insufficient sleep significantly increase the risk of obesity and central obesity in children and adolescents while adequate sleep of 7-10 hours per day would reduce the risk of obesity and central obesity in students.}
}

RIS

TY  - JOUR
AU  - Y. Li
AU  - R. Tao
AU  - D. Gao
AU  - B. Wen
AU  - B. Dong
AU  - Y. Song
AU  - Z. Zou
AU  - J. Ma
T1  - [A study on the relationship between insufficient sleep and obesity among children and adolescents in China].
JO  - Zhonghua liu xing bing xue za zhi = Zhonghua liuxingbingxue zazhi
PY  - 2020
VL  - 41 6
SP  - 
          845-849
        
EP  - 
          845-849
        
DO  - 10.3760/cma.j.cn112338-20190711-00514
AB  - Objective: To analyze the relationship between insufficient sleep and obesity or central obesity in Chinese Han children and adolescents aged 9-18 years. Methods: A total of 172 710 students who participated in the 2014 Chinese National Survey on Students Constitution and Health with complete data of sleep duration and physical examination, were selected as study subjects. Insufficient sleep was defined, according to the amount of sleep for pediatric populations recommended by the American Academy of Sleep Medicine. Obesity and central obesity of children and adolescents were judged by experts from the Group of China Obesity Task Force and Health Industry Standards in China. Differences between groups were compared by using the t test or χ(2) test. Logistic regression method was applied to assess the relationship between insufficient sleep and obesity or central obesity. Results: In 2014, numbers of students with insufficient sleep, obesity and central obesity among Chinese Han children and adolescents aged 9-18 years appeared as 133 410 (77.2%), 11 145 (6.5%), and 12 603 (17.8%), respectively. Among the students with insufficient sleep, 8 358 (6.3%) were with obesity and 12 244 (17.9%) were with central obesity. The prevalence of obesity and central obesity among boys with insufficient sleep was higher than that in girls. Pupils with insufficient sleep showed the highest prevalence of obesity and central obesity. After controlling for potential confounders, the risk of obesity appeared an increase of 14.5% (OR=1.145, 95%CI: 1.092-1.200) and the risk of central obesity increased by 12.7% (OR=1.127, 95%CI: 1.078-1.178) in students with insufficient sleep, when compared with those with adequate sleep. Compared with those whose daily sleep duration was less than 6 hours, the ones who slept 7-10 hours per day showed significantly reduction on the risk of obesity and central obesity in students. Conclusions: Insufficient sleep significantly increase the risk of obesity and central obesity in children and adolescents while adequate sleep of 7-10 hours per day would reduce the risk of obesity and central obesity in students.
ER  - 

BibTeX

@misc{c1f980276c29b3f1bb5efff9c451e75fde1ec7d2,
  author = {Amy R Goetz and Ishita Jindal and J. Moreno and M. Puyau and A. Adolph and S. Musaad and N. Butte and F. Bacha},
  title = {The Role of Sleep and Eating Patterns in Adiposity Gain among Preschool-Aged Children.},
  journal = {The American journal of clinical nutrition},
  year = {2022},
  doi = {10.1093/ajcn/nqac197},
  abstract = {BACKGROUND
Short sleep duration is related to risk for obesity in preschool children. However, the underlying mechanism(s) are not clear.


OBJECTIVE
We evaluated the relationship between sleep characteristics with body composition, energetics and weight-regulating behaviors in preschool-aged children; and the longitudinal associations between children's sleep and eating patterns with body composition at 1-year follow-up.


METHODS
Data were drawn from a longitudinal study of 118 children aged 3-5 years. Sleep (duration, midpoint, regularity) and physical activity (PA) were measured by accelerometry over 6 consecutive days; total energy expenditure (TEE) using the doubly-labeled water method; body composition (fat mass, fat-free mass, and %body fat) by dual energy x-ray absorptiometry; and dietary intake (energy intake, timing) using two 24-h recalls. Multivariable regression was used to estimate interindividual associations of sleep parameters with body composition, PA, TEE and dietary outcomes; and to examine the relationship between sleep and dietary behaviors with body composition one year later.


RESULTS
Cross-sectionally, later sleep midpoint was associated with greater fat mass (0.33; 95% CI: 0.05, 0.60) and %body fat (0.92; 95% CI: 0.15, 1.70). Later sleep midpoint was associated with delayed morning (0.51; 95% CI: 0.28, 0.74) and evening meal times (0.41; 95% CI: 0.29, 0.53), higher nighttime (45.6; 95% CI: 19.7, 71.4), and lower morning (-44.8; 95% CI: -72.0, -17.6) energy intake. Longitudinally, shorter sleep duration (-0.02; 95% CI: -0.03, -0.00) and later meal timing (0.83; 95% CI: 0.24, 1.42) were associated with higher %body fat 1 year later.


CONCLUSIONS
Shorter sleep duration and later meal timing are associated with adiposity gain in preschoolers.}
}

RIS

TY  - JOUR
AU  - Amy R Goetz
AU  - Ishita Jindal
AU  - J. Moreno
AU  - M. Puyau
AU  - A. Adolph
AU  - S. Musaad
AU  - N. Butte
AU  - F. Bacha
T1  - The Role of Sleep and Eating Patterns in Adiposity Gain among Preschool-Aged Children.
JO  - The American journal of clinical nutrition
PY  - 2022
DO  - 10.1093/ajcn/nqac197
AB  - BACKGROUND
Short sleep duration is related to risk for obesity in preschool children. However, the underlying mechanism(s) are not clear.


OBJECTIVE
We evaluated the relationship between sleep characteristics with body composition, energetics and weight-regulating behaviors in preschool-aged children; and the longitudinal associations between children's sleep and eating patterns with body composition at 1-year follow-up.


METHODS
Data were drawn from a longitudinal study of 118 children aged 3-5 years. Sleep (duration, midpoint, regularity) and physical activity (PA) were measured by accelerometry over 6 consecutive days; total energy expenditure (TEE) using the doubly-labeled water method; body composition (fat mass, fat-free mass, and %body fat) by dual energy x-ray absorptiometry; and dietary intake (energy intake, timing) using two 24-h recalls. Multivariable regression was used to estimate interindividual associations of sleep parameters with body composition, PA, TEE and dietary outcomes; and to examine the relationship between sleep and dietary behaviors with body composition one year later.


RESULTS
Cross-sectionally, later sleep midpoint was associated with greater fat mass (0.33; 95% CI: 0.05, 0.60) and %body fat (0.92; 95% CI: 0.15, 1.70). Later sleep midpoint was associated with delayed morning (0.51; 95% CI: 0.28, 0.74) and evening meal times (0.41; 95% CI: 0.29, 0.53), higher nighttime (45.6; 95% CI: 19.7, 71.4), and lower morning (-44.8; 95% CI: -72.0, -17.6) energy intake. Longitudinally, shorter sleep duration (-0.02; 95% CI: -0.03, -0.00) and later meal timing (0.83; 95% CI: 0.24, 1.42) were associated with higher %body fat 1 year later.


CONCLUSIONS
Shorter sleep duration and later meal timing are associated with adiposity gain in preschoolers.
ER  - 

BibTeX

@misc{83f744100cd9ad5ebba9d418330622b4750ebce0,
  author = {Anna Delahunt and M. Conway and C. McDonnell and Sharleen L. O Reilly and Linda O Keeffe and P. Kearney and J. Mehegan and F. Mcauliffe},
  title = {Sleep duration and eating behaviours are associated with body composition in 5-year-old children: findings from the ROLO longitudinal birth cohort study},
  journal = {British Journal of Nutrition},
  year = {2021},
  volume = {127},
  pages = {1750 - 1760},
  doi = {10.1017/S0007114521002725},
  abstract = {Abstract Inadequate sleep and poor eating behaviours are associated with higher risk of childhood overweight and obesity. Less is known about the influence sleep has on eating behaviours and consequently body composition. Furthermore, whether associations differ in boys and girls has not been investigated extensively. We investigate associations between sleep, eating behaviours and body composition in cross-sectional analysis of 5-year-old children. Weight, height, BMI, mid upper arm circumference (MUAC), abdominal circumference (AC) and skinfold measurements were obtained. Maternal reported information on child’s eating behaviour and sleep habits were collected using validated questionnaires. Multiple linear regression examined associations between sleep, eating behaviours and body composition. Sleep duration was negatively associated with BMI, with 1-h greater sleep duration associated with 0·24 kg/m2 (B = 0·24, CI −0·42, −0·03, P = 0·026) lower BMI and 0·21 cm lower (B = –0·21, CI −0·41, −0·02, P = 0·035) MUAC. When stratified by sex, girls showed stronger inverse associations between sleep duration (h) and BMI (kg/m2) (B = –0·32; CI −0·60, −0·04, P = 0·024), MUAC (cm) (B = –0·29; CI −0·58, 0·000, P = 0·05) and AC (cm) (B = –1·10; CI −1·85, −0·21, P = 0·014) than boys. Positive associations for ‘Enjoys Food’ and ‘Food Responsiveness’ with BMI, MUAC and AC were observed in girls only. Inverse associations between sleep duration and ‘Emotional Undereating’ and ‘Food Fussiness’ were observed in both sexes, although stronger in boys. Sleep duration did not mediate the relationship between eating behaviours and BMI. Further exploration is required to understand how sleep impacts eating behaviours and consequently body composition and how sex influences this relationship.}
}

RIS

TY  - JOUR
AU  - Anna Delahunt
AU  - M. Conway
AU  - C. McDonnell
AU  - Sharleen L. O Reilly
AU  - Linda O Keeffe
AU  - P. Kearney
AU  - J. Mehegan
AU  - F. Mcauliffe
T1  - Sleep duration and eating behaviours are associated with body composition in 5-year-old children: findings from the ROLO longitudinal birth cohort study
JO  - British Journal of Nutrition
PY  - 2021
VL  - 127
SP  - 1750 - 1760
EP  - 1750 - 1760
DO  - 10.1017/S0007114521002725
AB  - Abstract Inadequate sleep and poor eating behaviours are associated with higher risk of childhood overweight and obesity. Less is known about the influence sleep has on eating behaviours and consequently body composition. Furthermore, whether associations differ in boys and girls has not been investigated extensively. We investigate associations between sleep, eating behaviours and body composition in cross-sectional analysis of 5-year-old children. Weight, height, BMI, mid upper arm circumference (MUAC), abdominal circumference (AC) and skinfold measurements were obtained. Maternal reported information on child’s eating behaviour and sleep habits were collected using validated questionnaires. Multiple linear regression examined associations between sleep, eating behaviours and body composition. Sleep duration was negatively associated with BMI, with 1-h greater sleep duration associated with 0·24 kg/m2 (B = 0·24, CI −0·42, −0·03, P = 0·026) lower BMI and 0·21 cm lower (B = –0·21, CI −0·41, −0·02, P = 0·035) MUAC. When stratified by sex, girls showed stronger inverse associations between sleep duration (h) and BMI (kg/m2) (B = –0·32; CI −0·60, −0·04, P = 0·024), MUAC (cm) (B = –0·29; CI −0·58, 0·000, P = 0·05) and AC (cm) (B = –1·10; CI −1·85, −0·21, P = 0·014) than boys. Positive associations for ‘Enjoys Food’ and ‘Food Responsiveness’ with BMI, MUAC and AC were observed in girls only. Inverse associations between sleep duration and ‘Emotional Undereating’ and ‘Food Fussiness’ were observed in both sexes, although stronger in boys. Sleep duration did not mediate the relationship between eating behaviours and BMI. Further exploration is required to understand how sleep impacts eating behaviours and consequently body composition and how sex influences this relationship.
ER  - 

BibTeX

@misc{8bc05c4573435c485458f2b151de9eaf3917b299,
  author = {J. Najafian and N. Mohammadifard and Z. Siadat and G. Sadri and M. Ramazani and Fatemeh Nouri},
  title = {Association between Sleep Duration and Body Mass Index and Waist Circumference},
  journal = {Iranian Journal of Medical Sciences},
  year = {2010},
  volume = {35},
  pages = {140-144},
  abstract = {Insufficient sleep may lead to adverse cardio-metabolic effects by influencing body weight, blood pressure, and glucose tolerance. We aimed to assess the relation between sleep duration and indices of obesity including body mass index (BMI) and waist circumference in a sample population in central Iran. We selected a sub-sample of 1770 individuals from Isfahan Healthy Heart Program. The data regarding the sleep duration was collected by using a validated questionnaire. The relation of sleep duration with BMI and waist circumference was examined by using categorical logistic regression with adjustment for sex, BMI, and daily calorie intake. Sleep duration time less than 5 hours/day compared with 7-8 hours/day increased the odds ratio for abdominal obesity in people aged under 60 years [OR=2.49 (95%CI 1.40-4.43)]. In individuals aged under 60 years, this relation was significant for both men, [OR=2.64 (95%CI 1.166.02]) and women [OR=2.38 (95% CI 1.05-5.39)]. In addition, in women, sleep time > 9 hours was negatively related to waist circumference. Sleep duration time less than 5 hours per day increased odds ratio of overweight only in women [OR=1.75 (95% CI 1.07-2.85)]. Sleep duration time under 5 hours in people aged less than 60 years was positively associated with waist circumference. In women, sleep duration time over 9 hours was negatively associated with waist circumference. Only in women, sleep time under 5 hours /night increased BMI. Short sleep duration was associated with abdominal obesity and this was independent of its relation to BMI. Iran J Med Sci 2010; 35(2): 140-144.}
}

RIS

TY  - JOUR
AU  - J. Najafian
AU  - N. Mohammadifard
AU  - Z. Siadat
AU  - G. Sadri
AU  - M. Ramazani
AU  - Fatemeh Nouri
T1  - Association between Sleep Duration and Body Mass Index and Waist Circumference
JO  - Iranian Journal of Medical Sciences
PY  - 2010
VL  - 35
SP  - 140-144
EP  - 140-144
AB  - Insufficient sleep may lead to adverse cardio-metabolic effects by influencing body weight, blood pressure, and glucose tolerance. We aimed to assess the relation between sleep duration and indices of obesity including body mass index (BMI) and waist circumference in a sample population in central Iran. We selected a sub-sample of 1770 individuals from Isfahan Healthy Heart Program. The data regarding the sleep duration was collected by using a validated questionnaire. The relation of sleep duration with BMI and waist circumference was examined by using categorical logistic regression with adjustment for sex, BMI, and daily calorie intake. Sleep duration time less than 5 hours/day compared with 7-8 hours/day increased the odds ratio for abdominal obesity in people aged under 60 years [OR=2.49 (95%CI 1.40-4.43)]. In individuals aged under 60 years, this relation was significant for both men, [OR=2.64 (95%CI 1.166.02]) and women [OR=2.38 (95% CI 1.05-5.39)]. In addition, in women, sleep time > 9 hours was negatively related to waist circumference. Sleep duration time less than 5 hours per day increased odds ratio of overweight only in women [OR=1.75 (95% CI 1.07-2.85)]. Sleep duration time under 5 hours in people aged less than 60 years was positively associated with waist circumference. In women, sleep duration time over 9 hours was negatively associated with waist circumference. Only in women, sleep time under 5 hours /night increased BMI. Short sleep duration was associated with abdominal obesity and this was independent of its relation to BMI. Iran J Med Sci 2010; 35(2): 140-144.
ER  - 

BibTeX

@misc{aa14bd624dffe86caa66cc358a728e393720ae00,
  author = {C. Padez and I. Mourão and P. Moreira and Vítor Rosado},
  title = {Original Research Article Long Sleep Duration and Childhood Overweight/Obesity and Body Fat},
  journal = {},
  year = {2009},
  volume = {},
  abstract = {To assess the association between short sleep duration and overweight/obesity and body fat (BF) and to identify correlates of short sleep duration in a sample of Portuguese children. A cross-sectional study of children 7-9 years (n 5 4511) was performed between October 2002 and June 2003. Weight, height, and skinfolds were measured, and parents filled out a questionnaire about family characteristics as well as sleep duration. The prevalence of overweight/obe- sity and BF (%) both decreased by long sleep duration. After adjusted for confounders, the odds ratio (OR) for overweight/ obesity and sleep duration were as follows: reference >11 h/d; 10-11 h/d, OR: 1.3; confidence interval (CI):1.26, 1.33; 9-10 h/d, OR: 1.16; CI: 1.13, 1.19; and <9 h, OR: 3.22; CI: 3.11, 3.32. Children whose parents' had a low educational level slept less time during each night than children whose parents' had a higher educational level; children who spent more time watching television slept less time than those who watched less television, and those children engaged in physical activity slept more time each night than sedentary children. Our results showed an inverse relationship between long sleep dura- tion and overweight/obesity prevalence as well as with body fat, and these findings are important because sleep duration is a potentially modifiable risk factor that could be important to consider in the prevention and treatment of childhood obesity. Am. J. Hum. Biol. 21:371-376, 2009. ' 2009 Wiley-Liss, Inc.}
}

RIS

TY  - JOUR
AU  - C. Padez
AU  - I. Mourão
AU  - P. Moreira
AU  - Vítor Rosado
T1  - Original Research Article Long Sleep Duration and Childhood Overweight/Obesity and Body Fat
PY  - 2009
AB  - To assess the association between short sleep duration and overweight/obesity and body fat (BF) and to identify correlates of short sleep duration in a sample of Portuguese children. A cross-sectional study of children 7-9 years (n 5 4511) was performed between October 2002 and June 2003. Weight, height, and skinfolds were measured, and parents filled out a questionnaire about family characteristics as well as sleep duration. The prevalence of overweight/obe- sity and BF (%) both decreased by long sleep duration. After adjusted for confounders, the odds ratio (OR) for overweight/ obesity and sleep duration were as follows: reference >11 h/d; 10-11 h/d, OR: 1.3; confidence interval (CI):1.26, 1.33; 9-10 h/d, OR: 1.16; CI: 1.13, 1.19; and <9 h, OR: 3.22; CI: 3.11, 3.32. Children whose parents' had a low educational level slept less time during each night than children whose parents' had a higher educational level; children who spent more time watching television slept less time than those who watched less television, and those children engaged in physical activity slept more time each night than sedentary children. Our results showed an inverse relationship between long sleep dura- tion and overweight/obesity prevalence as well as with body fat, and these findings are important because sleep duration is a potentially modifiable risk factor that could be important to consider in the prevention and treatment of childhood obesity. Am. J. Hum. Biol. 21:371-376, 2009. ' 2009 Wiley-Liss, Inc.
ER  - 

BibTeX

@misc{30b48d2d78650f4753ac20cf3c5d530bd4066e54,
  author = {J. Chaput and A. Tremblay},
  title = {Does short sleep duration favor abdominal adiposity in children?},
  journal = {International journal of pediatric obesity : IJPO : an official journal of the International Association for the Study of Obesity},
  year = {2007},
  volume = {2 3},
  pages = {
          188-91
        },
  doi = {10.1080/17477160701306144},
  abstract = {The main aim of this cross-sectional study was to determine whether the increased body mass index (BMI) characterizing short-duration sleeping children is related to an increased predisposition to abdominal adiposity. A total of 422 children (211 boys and 211 girls) involved in the "Québec en Forme" Project were tested for body weight, height, waist circumference, and sleep duration. As there was no gender interaction with the other factors, a partial regression of waist circumference on hours of sleep was performed for both genders combined, adjusting for age, sex, BMI, parental obesity, parental education, total annual family income, frequency of taking breakfast, watching television, playing videogames, computer use, and frequency of practicing sports activities outside of school. Sleep duration had an independent effect on waist circumference, with the correlation between these variables remaining significant after adjustment for BMI and the several other covariates (r=- 0.17, p<0.001). In conclusion, these results suggest that short sleep duration favors abdominal adiposity in children. This finding is of particular concern since abdominal obesity is an important feature of the metabolic syndrome.}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - A. Tremblay
T1  - Does short sleep duration favor abdominal adiposity in children?
JO  - International journal of pediatric obesity : IJPO : an official journal of the International Association for the Study of Obesity
PY  - 2007
VL  - 2 3
SP  - 
          188-91
        
EP  - 
          188-91
        
DO  - 10.1080/17477160701306144
AB  - The main aim of this cross-sectional study was to determine whether the increased body mass index (BMI) characterizing short-duration sleeping children is related to an increased predisposition to abdominal adiposity. A total of 422 children (211 boys and 211 girls) involved in the "Québec en Forme" Project were tested for body weight, height, waist circumference, and sleep duration. As there was no gender interaction with the other factors, a partial regression of waist circumference on hours of sleep was performed for both genders combined, adjusting for age, sex, BMI, parental obesity, parental education, total annual family income, frequency of taking breakfast, watching television, playing videogames, computer use, and frequency of practicing sports activities outside of school. Sleep duration had an independent effect on waist circumference, with the correlation between these variables remaining significant after adjustment for BMI and the several other covariates (r=- 0.17, p<0.001). In conclusion, these results suggest that short sleep duration favors abdominal adiposity in children. This finding is of particular concern since abdominal obesity is an important feature of the metabolic syndrome.
ER  - 

BibTeX

@misc{27384fb5837ebd4db0b8e88193b8ee9b6afb2248,
  author = {Theresa Pizinger and B. Aggarwal and M. St-Onge},
  title = {Sleep Extension in Short Sleepers: An Evaluation of Feasibility and Effectiveness for Weight Management and Cardiometabolic Disease Prevention},
  journal = {Frontiers in Endocrinology},
  year = {2018},
  volume = {9},
  doi = {10.3389/fendo.2018.00392},
  abstract = {Sleep duration has become increasingly recognized as an important influencer of health. Epidemiologic and observational studies have shown associations between short sleep duration and increased risk for chronic cardiometabolic disorders, including obesity, type 2 diabetes, and cardiovascular disease. These associations have led to investigations into the potential causal pathways through which short sleep may increase risk for these disorders. Clinical intervention studies have demonstrated that restricting sleep in normal sleepers has adverse health effects, including insulin resistance, and increased blood pressure. The totality of evidence points to negative health effects of short sleep and the recognition of sleep as a lifestyle behavior that may be targeted for disease prevention. It is well established that consistent, adequate sleep is associated with the lowest risk of obesity and cardiometabolic disorders. Yet, it is unclear whether increasing sleep in short sleepers can improve health. In today's society, it is common for individuals to deprive themselves of sleep during the work week, with the intent to sleep longer during the weekend, or have “catch-up sleep.” Studies that have examined the health effects of extended sleep, post-sleep restriction, revealed some improvements in health outcomes. However, it is uncertain whether the improvements observed with catch-up sleep are sufficient to reverse the negative health effects of constant sleep restriction. Few intervention studies have been undertaken to determine whether extending sleep, long-term, in short sleepers is feasible and whether it can reduce the disease risk burden associated with short sleep duration. The purpose of this review is to highlight these studies and evaluate information related to the impact of sleep extension on risk factors for chronic cardiometabolic disorders. We discuss limitations of current research, including variability in participant characteristics and the extent to which sleep behaviors are modified and monitored. Although the evidence-base for benefits of sleep extension is still in the early stages, studies to date indicate that prolonging sleep, in short sleepers, may improve cardiometabolic risk. Finally, our review calls attention to areas that require further study and for larger scale studies of behavior modification to establish the health effects of sleep extension in short sleepers.}
}

RIS

TY  - JOUR
AU  - Theresa Pizinger
AU  - B. Aggarwal
AU  - M. St-Onge
T1  - Sleep Extension in Short Sleepers: An Evaluation of Feasibility and Effectiveness for Weight Management and Cardiometabolic Disease Prevention
JO  - Frontiers in Endocrinology
PY  - 2018
VL  - 9
DO  - 10.3389/fendo.2018.00392
AB  - Sleep duration has become increasingly recognized as an important influencer of health. Epidemiologic and observational studies have shown associations between short sleep duration and increased risk for chronic cardiometabolic disorders, including obesity, type 2 diabetes, and cardiovascular disease. These associations have led to investigations into the potential causal pathways through which short sleep may increase risk for these disorders. Clinical intervention studies have demonstrated that restricting sleep in normal sleepers has adverse health effects, including insulin resistance, and increased blood pressure. The totality of evidence points to negative health effects of short sleep and the recognition of sleep as a lifestyle behavior that may be targeted for disease prevention. It is well established that consistent, adequate sleep is associated with the lowest risk of obesity and cardiometabolic disorders. Yet, it is unclear whether increasing sleep in short sleepers can improve health. In today's society, it is common for individuals to deprive themselves of sleep during the work week, with the intent to sleep longer during the weekend, or have “catch-up sleep.” Studies that have examined the health effects of extended sleep, post-sleep restriction, revealed some improvements in health outcomes. However, it is uncertain whether the improvements observed with catch-up sleep are sufficient to reverse the negative health effects of constant sleep restriction. Few intervention studies have been undertaken to determine whether extending sleep, long-term, in short sleepers is feasible and whether it can reduce the disease risk burden associated with short sleep duration. The purpose of this review is to highlight these studies and evaluate information related to the impact of sleep extension on risk factors for chronic cardiometabolic disorders. We discuss limitations of current research, including variability in participant characteristics and the extent to which sleep behaviors are modified and monitored. Although the evidence-base for benefits of sleep extension is still in the early stages, studies to date indicate that prolonging sleep, in short sleepers, may improve cardiometabolic risk. Finally, our review calls attention to areas that require further study and for larger scale studies of behavior modification to establish the health effects of sleep extension in short sleepers.
ER  - 

BibTeX

@misc{5b3c1792c6c3429be711d6bb6d90458dd03d5c1b,
  author = {Keun-Hyok Cho and Eun-Hee Cho and J. Hur and Dayeon Shin},
  title = {Association of Sleep Duration and Obesity According to Gender and Age in Korean Adults: Results from the Korea National Health and Nutrition Examination Survey 2007–2015},
  journal = {Journal of Korean Medical Science},
  year = {2018},
  volume = {33},
  doi = {10.3346/jkms.2018.33.e345},
  abstract = {Background This study aimed to investigate associations between self-reported sleep duration and general and abdominal obesity in Korean adults stratified according to gender and age. Methods Data from 41,805 adults, 18–110 years of age, collected by the Korea National Health and Nutrition Examination Survey (KNHANES) in 2007 and 2015, were analyzed. Multivariable logistic regression was used to calculate adjusted odds ratios (AORs) and 95% confidence intervals (CIs) for obesity and abdominal obesity by sleep duration after controlling for sociodemographic and lifestyle variables. Results Among individuals 30–49 years of age, there was an increased AOR for obesity only for sleep duration ≤ 5 hour/day compared with sleep duration 6 to 8 hour/day, both in men (OR, 1.25; 95% CI, 1.02–1.54) and women (OR, 1.56; 95% CI, 1.29–1.90), after controlling for covariates. Regarding women, there was increased AOR for abdominal obesity for sleep duration ≤ 5 hour/day (OR, 1.45; 95% CI, 1.18–1.78) and ≥ 9 hour/day (OR, 1.38; 95% CI, 1.09–1.76) compared with sleep duration 6 to 8 hour/day. However, for elderly individuals (≥ 65 years), there was a negative association between sleep duration ≤ 5 hour/day and obesity, but not with abdominal obesity, in both men and women. Conclusion This study demonstrated a significant association between sleep duration and obesity, which varied according to gender and age.}
}

RIS

TY  - JOUR
AU  - Keun-Hyok Cho
AU  - Eun-Hee Cho
AU  - J. Hur
AU  - Dayeon Shin
T1  - Association of Sleep Duration and Obesity According to Gender and Age in Korean Adults: Results from the Korea National Health and Nutrition Examination Survey 2007–2015
JO  - Journal of Korean Medical Science
PY  - 2018
VL  - 33
DO  - 10.3346/jkms.2018.33.e345
AB  - Background This study aimed to investigate associations between self-reported sleep duration and general and abdominal obesity in Korean adults stratified according to gender and age. Methods Data from 41,805 adults, 18–110 years of age, collected by the Korea National Health and Nutrition Examination Survey (KNHANES) in 2007 and 2015, were analyzed. Multivariable logistic regression was used to calculate adjusted odds ratios (AORs) and 95% confidence intervals (CIs) for obesity and abdominal obesity by sleep duration after controlling for sociodemographic and lifestyle variables. Results Among individuals 30–49 years of age, there was an increased AOR for obesity only for sleep duration ≤ 5 hour/day compared with sleep duration 6 to 8 hour/day, both in men (OR, 1.25; 95% CI, 1.02–1.54) and women (OR, 1.56; 95% CI, 1.29–1.90), after controlling for covariates. Regarding women, there was increased AOR for abdominal obesity for sleep duration ≤ 5 hour/day (OR, 1.45; 95% CI, 1.18–1.78) and ≥ 9 hour/day (OR, 1.38; 95% CI, 1.09–1.76) compared with sleep duration 6 to 8 hour/day. However, for elderly individuals (≥ 65 years), there was a negative association between sleep duration ≤ 5 hour/day and obesity, but not with abdominal obesity, in both men and women. Conclusion This study demonstrated a significant association between sleep duration and obesity, which varied according to gender and age.
ER  - 

BibTeX

@misc{0231bdb1eba5255488a638a1dd9bf07f7bfc3362,
  author = {Gong Ben-xin},
  title = {The link between sleep duration and obesity in children},
  journal = {The Journal of clinical Pediatrics},
  year = {2012},
  volume = {},
  abstract = {Objective To investigate the link between sleep duration and obesity in children.Methods A total of 1 145 children aged 7 to 12 years were selected by stratified sampling.Height and weight were measured.Body mass index(BMI)was calculated.The parents and children were surveyed with questionnaire.Results The prevalence of obesity was 3.8% in all children.2.0% in children with sleep duration ≥ 10 h,5.0% in children with 8-10 sleep duration,and 10.0% in children with ≤ 8 h sleep duration.The differences were statistically significant(P 0.001).Conclusions Short sleep duration is associated with obesity in children and adequate sleep duration among children is important in prevention of obesity.}
}

RIS

TY  - JOUR
AU  - Gong Ben-xin
T1  - The link between sleep duration and obesity in children
JO  - The Journal of clinical Pediatrics
PY  - 2012
AB  - Objective To investigate the link between sleep duration and obesity in children.Methods A total of 1 145 children aged 7 to 12 years were selected by stratified sampling.Height and weight were measured.Body mass index(BMI)was calculated.The parents and children were surveyed with questionnaire.Results The prevalence of obesity was 3.8% in all children.2.0% in children with sleep duration ≥ 10 h,5.0% in children with 8-10 sleep duration,and 10.0% in children with ≤ 8 h sleep duration.The differences were statistically significant(P 0.001).Conclusions Short sleep duration is associated with obesity in children and adequate sleep duration among children is important in prevention of obesity.
ER  - 

BibTeX

@misc{64272dd0d8c9590ae3218a27050b39d152a1a6f1,
  author = {Mie M. Andersen and T. Laurberg and Anne-Louise Bjerregaard and A. Sandbæk and S. Brage and D. Vistisen and J. S. Quist and J. Bruun and D. Witte},
  title = {The association between sleep duration and detailed measures of obesity: A cross sectional analysis in the ADDITION‐PRO study},
  journal = {Obesity Science & Practice},
  year = {2022},
  volume = {9},
  pages = {226 - 234},
  doi = {10.1002/osp4.640},
  abstract = {Sleep duration is associated with BMI and waist circumference. However, less is known about whether sleep duration affects different measurements of obesity differently.}
}

RIS

TY  - JOUR
AU  - Mie M. Andersen
AU  - T. Laurberg
AU  - Anne-Louise Bjerregaard
AU  - A. Sandbæk
AU  - S. Brage
AU  - D. Vistisen
AU  - J. S. Quist
AU  - J. Bruun
AU  - D. Witte
T1  - The association between sleep duration and detailed measures of obesity: A cross sectional analysis in the ADDITION‐PRO study
JO  - Obesity Science & Practice
PY  - 2022
VL  - 9
SP  - 226 - 234
EP  - 226 - 234
DO  - 10.1002/osp4.640
AB  - Sleep duration is associated with BMI and waist circumference. However, less is known about whether sleep duration affects different measurements of obesity differently.
ER  - 

BibTeX

@misc{b165ae2481a175ad52824530afbcfe7c1447e36e,
  author = {Dana R. Schreiber and Natalie D Dautovich},
  title = {Sleep Duration as an Indirect Link Between Sleep Timing and Weight in Midlife Women.},
  journal = {Journal of women's health},
  year = {2019},
  doi = {10.1089/jwh.2018.7088},
  abstract = {Objective: Midlife women are at risk of obesity. Poor sleep outcomes including inadequate sleep duration and variable sleep timing are risk factors for obesity, but there is a lack of understanding on how specific sleep constructs uniquely or concurrently are associated with weight outcomes in this population. This study examined the unique association of sleep timing with weight outcomes and how sleep timing works in conjunction with sleep duration to influence weight in midlife women. Materials and Methods: An archival analysis was performed using the Midlife in the United States-II study (MIDUS-II). The sample consisted of 132 midlife women (40-64; M = 52.9, standard deviation = 6.94). Sleep timing (mean sleep time, variability) and duration were measured through actigraphy and daily sleep diaries. Weight was assessed using body mass index (BMI) and waist circumference measures. Results: PROCESS mediation and moderation analyses assessed direct, indirect, and moderating pathways. Sleep duration emerged as an indirect link between sleep timing (mean and variability) and weight outcomes (95% CI = 0.0001-0.0123; 95% CI = 0.0007-0.0378; 95% CI = 0.0079-0.1006). Sleep timing (95% CI = -0.0144 to 0.0076; 95% CI = -0.0358 to 0.0219) and sleep time variability (95% CI = -0.0124 to 0.0438; 95% CI = -0.0533 to 0.0939) were not directly associated with BMI or waist circumference. Also, sleep timing and duration did not interact to influence weight outcomes. Conclusions: Sleep duration, rather than sleep timing, is associated with weight outcomes, and is an indirect link in the sleep timing and weight outcomes association. Future work is needed to further disentangle the impact of sleep on weight in midlife women using prospective studies, implementing daily assessments of sleep behavior.}
}

RIS

TY  - JOUR
AU  - Dana R. Schreiber
AU  - Natalie D Dautovich
T1  - Sleep Duration as an Indirect Link Between Sleep Timing and Weight in Midlife Women.
JO  - Journal of women's health
PY  - 2019
DO  - 10.1089/jwh.2018.7088
AB  - Objective: Midlife women are at risk of obesity. Poor sleep outcomes including inadequate sleep duration and variable sleep timing are risk factors for obesity, but there is a lack of understanding on how specific sleep constructs uniquely or concurrently are associated with weight outcomes in this population. This study examined the unique association of sleep timing with weight outcomes and how sleep timing works in conjunction with sleep duration to influence weight in midlife women. Materials and Methods: An archival analysis was performed using the Midlife in the United States-II study (MIDUS-II). The sample consisted of 132 midlife women (40-64; M = 52.9, standard deviation = 6.94). Sleep timing (mean sleep time, variability) and duration were measured through actigraphy and daily sleep diaries. Weight was assessed using body mass index (BMI) and waist circumference measures. Results: PROCESS mediation and moderation analyses assessed direct, indirect, and moderating pathways. Sleep duration emerged as an indirect link between sleep timing (mean and variability) and weight outcomes (95% CI = 0.0001-0.0123; 95% CI = 0.0007-0.0378; 95% CI = 0.0079-0.1006). Sleep timing (95% CI = -0.0144 to 0.0076; 95% CI = -0.0358 to 0.0219) and sleep time variability (95% CI = -0.0124 to 0.0438; 95% CI = -0.0533 to 0.0939) were not directly associated with BMI or waist circumference. Also, sleep timing and duration did not interact to influence weight outcomes. Conclusions: Sleep duration, rather than sleep timing, is associated with weight outcomes, and is an indirect link in the sleep timing and weight outcomes association. Future work is needed to further disentangle the impact of sleep on weight in midlife women using prospective studies, implementing daily assessments of sleep behavior.
ER  - 

BibTeX

@misc{4fe54b6a819f959f111a9081e8f48c3d543e9cac,
  author = {S. Patel},
  title = {' s report Title : Short sleep duration and obesity among Australian children Version : 1 Date : 22 June 2010},
  abstract = {1. The authors seek to assess the cross-sectional association between parental report of sleep duration and adiposity using an age-adjusted obesity threshold in a cohort of children from South Australia. They postulate there may be an age by sleep interaction such that the effect of short sleep on obesity risk wanes with age above 10. However, their analysis presents results stratified by age 5-12 and age 13-15 rather than the a priori hypothesis of comparing age 5-10 and 10-15 strata. The a priori hypothesis should be tested and presented in Table 2. If the authors then wish to demonstrate the results at another breakpoint, that is fine but it should be done in the setting of clearly describing to the reader that this was done post-hoc and as such, the results should be viewed as exploratory.}
}

RIS

TY  - JOUR
AU  - S. Patel
T1  - ' s report Title : Short sleep duration and obesity among Australian children Version : 1 Date : 22 June 2010
AB  - 1. The authors seek to assess the cross-sectional association between parental report of sleep duration and adiposity using an age-adjusted obesity threshold in a cohort of children from South Australia. They postulate there may be an age by sleep interaction such that the effect of short sleep on obesity risk wanes with age above 10. However, their analysis presents results stratified by age 5-12 and age 13-15 rather than the a priori hypothesis of comparing age 5-10 and 10-15 strata. The a priori hypothesis should be tested and presented in Table 2. If the authors then wish to demonstrate the results at another breakpoint, that is fine but it should be done in the setting of clearly describing to the reader that this was done post-hoc and as such, the results should be viewed as exploratory.
ER  - 

BibTeX

@misc{e2dc829b567f32e4e37d0f6a8ccecbc4dbf9db58,
  author = {Y. Park and Won‐Chul Lee and H. Yim and Young‐Moon Park},
  title = {[The association between sleep and obesity in Korean adults].},
  journal = {Journal of preventive medicine and public health = Yebang Uihakhoe chi},
  year = {2007},
  volume = {40 6},
  pages = {
          454-60
        },
  doi = {10.3961/JPMPH.2007.40.6.454},
  abstract = {OBJECTIVES
Obesity is currently an epidemic in Korea, and sleep duration is thought to be one of the risk factors for obesity. The objective of this study was to test the hypothesis that short sleep duration is associated with obesity in Korean adults.


METHODS
The data from the 2001 Korean National Health and Nutrition Survey were used, and 6,174 subjects aged 18 approximately 80 years were included in the analysis. Sleep duration was measured using information obtained from self-reported questionnaires. Obesity, the main outcome variable, was measured according to body mass index. Multiple regression modeling was used to adjust for potential confounding variables.


RESULTS
The study results revealed a negative association between sleep duration and body mass index among Korean adults. These associations persisted after controlling for the potential confounding variables.


CONCLUSIONS
These findings support the hypothesis that sleep duration is associated with obesity in Korean adults. In addition, these observations support earlier experimental sleep studies and provide a basis for future studies on weight control intervention by increasing the amount of sleep.}
}

RIS

TY  - JOUR
AU  - Y. Park
AU  - Won‐Chul Lee
AU  - H. Yim
AU  - Young‐Moon Park
T1  - [The association between sleep and obesity in Korean adults].
JO  - Journal of preventive medicine and public health = Yebang Uihakhoe chi
PY  - 2007
VL  - 40 6
SP  - 
          454-60
        
EP  - 
          454-60
        
DO  - 10.3961/JPMPH.2007.40.6.454
AB  - OBJECTIVES
Obesity is currently an epidemic in Korea, and sleep duration is thought to be one of the risk factors for obesity. The objective of this study was to test the hypothesis that short sleep duration is associated with obesity in Korean adults.


METHODS
The data from the 2001 Korean National Health and Nutrition Survey were used, and 6,174 subjects aged 18 approximately 80 years were included in the analysis. Sleep duration was measured using information obtained from self-reported questionnaires. Obesity, the main outcome variable, was measured according to body mass index. Multiple regression modeling was used to adjust for potential confounding variables.


RESULTS
The study results revealed a negative association between sleep duration and body mass index among Korean adults. These associations persisted after controlling for the potential confounding variables.


CONCLUSIONS
These findings support the hypothesis that sleep duration is associated with obesity in Korean adults. In addition, these observations support earlier experimental sleep studies and provide a basis for future studies on weight control intervention by increasing the amount of sleep.
ER  - 

BibTeX

@misc{e7d85b69dec38045b73cc7580700ecfe21b399f3,
  author = {E. Cauter and K. Knutson},
  title = {The ‘ Sleep Debt Study ’ : 6 days with 4-h bedtimes},
  year = {2008},
  abstract = {Sleep is an important modulator of neuroendocrine function and glucose metabolism in children as well as in adults. In recent years, sleep curtailment has become a hallmark of modern society with both children and adults having shorter bedtimes than a few decades ago. This trend for shorter sleep duration has developed over the same time period as the dramatic increase in the prevalence of obesity. There is rapidly accumulating evidence from both laboratory and epidemiological studies to indicate that chronic partial sleep loss may increase the risk of obesity and weight gain. The present article reviews laboratory evidence indicating that sleep curtailment in young adults results in a constellation of metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, elevated sympathovagal balance, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. We also review crosssectional epidemiological studies associating short sleep with increased body mass index and prospective epidemiological studies that have shown an increased risk of weight gain and obesity in children and young adults who are short sleepers. Altogether, the evidence points to a possible role of decreased sleep duration in the current epidemic of obesity.}
}

RIS

TY  - JOUR
AU  - E. Cauter
AU  - K. Knutson
T1  - The ‘ Sleep Debt Study ’ : 6 days with 4-h bedtimes
PY  - 2008
AB  - Sleep is an important modulator of neuroendocrine function and glucose metabolism in children as well as in adults. In recent years, sleep curtailment has become a hallmark of modern society with both children and adults having shorter bedtimes than a few decades ago. This trend for shorter sleep duration has developed over the same time period as the dramatic increase in the prevalence of obesity. There is rapidly accumulating evidence from both laboratory and epidemiological studies to indicate that chronic partial sleep loss may increase the risk of obesity and weight gain. The present article reviews laboratory evidence indicating that sleep curtailment in young adults results in a constellation of metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, elevated sympathovagal balance, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. We also review crosssectional epidemiological studies associating short sleep with increased body mass index and prospective epidemiological studies that have shown an increased risk of weight gain and obesity in children and young adults who are short sleepers. Altogether, the evidence points to a possible role of decreased sleep duration in the current epidemic of obesity.
ER  - 

BibTeX

@misc{fe699a7024c380f2c3953f9c6d434f837b3f72ca,
  author = {Bin Zhang and Y. Hao and Jiang-Ning Zhou and F. Jia and Xue-li Li and Yi Tang and Hui-rong Zheng},
  title = {The association between sleep patterns and overweight/obesity in Chinese children: a cross-sectional study},
  journal = {Neuropsychiatric Disease and Treatment},
  year = {2015},
  volume = {11},
  pages = {2209 - 2216},
  doi = {10.2147/NDT.S90838},
  abstract = {Objective This study evaluated the association between sleep patterns and the risk of being overweight/obese in Chinese children. Methods A total of 3,086 children (1,608 boys and 1,478 girls) between 7 and 14 years of age and studying in primary schools were recruited as eligible study participants in this study. We collected the information about children regarding sleep patterns, body height and weight, insomnia, healthy status, time allocation of daily activities, and demographic characteristics using a parental-reported questionnaire. Results Overweight/obese children were younger, predominantly male, and more prone to have suffered from illness in the past 12 months compared to normal-weight peers. They were also less prone to compensate for sleep deficits during weekends (47.6% vs 39.1%; χ2=11.637, P<0.001) and holidays (52.0% vs 42.0%; χ2=16.057, P<0.001). Sleep duration on weekdays did not affect the risk of being overweight/obese. The adjusted odds ratios for overweight/obesity (noncompensated) group using the compensated group as a reference were 1.197 (95% confidence interval [CI]: 1.004–1.493) during weekends and 1.309 (95% CI: 1.052–1.630) during holidays. Conclusion Compensation for sleep deficits on non-weekdays may ameliorate the risk of being overweight/obese in Chinese children. Moreover, no significant association between the risk of being overweight/obese and sleep duration on weekdays was demonstrated in the current study, which may be due to pervasive sleep insufficiency on weekdays in Chinese children.}
}

RIS

TY  - JOUR
AU  - Bin Zhang
AU  - Y. Hao
AU  - Jiang-Ning Zhou
AU  - F. Jia
AU  - Xue-li Li
AU  - Yi Tang
AU  - Hui-rong Zheng
T1  - The association between sleep patterns and overweight/obesity in Chinese children: a cross-sectional study
JO  - Neuropsychiatric Disease and Treatment
PY  - 2015
VL  - 11
SP  - 2209 - 2216
EP  - 2209 - 2216
DO  - 10.2147/NDT.S90838
AB  - Objective This study evaluated the association between sleep patterns and the risk of being overweight/obese in Chinese children. Methods A total of 3,086 children (1,608 boys and 1,478 girls) between 7 and 14 years of age and studying in primary schools were recruited as eligible study participants in this study. We collected the information about children regarding sleep patterns, body height and weight, insomnia, healthy status, time allocation of daily activities, and demographic characteristics using a parental-reported questionnaire. Results Overweight/obese children were younger, predominantly male, and more prone to have suffered from illness in the past 12 months compared to normal-weight peers. They were also less prone to compensate for sleep deficits during weekends (47.6% vs 39.1%; χ2=11.637, P<0.001) and holidays (52.0% vs 42.0%; χ2=16.057, P<0.001). Sleep duration on weekdays did not affect the risk of being overweight/obese. The adjusted odds ratios for overweight/obesity (noncompensated) group using the compensated group as a reference were 1.197 (95% confidence interval [CI]: 1.004–1.493) during weekends and 1.309 (95% CI: 1.052–1.630) during holidays. Conclusion Compensation for sleep deficits on non-weekdays may ameliorate the risk of being overweight/obese in Chinese children. Moreover, no significant association between the risk of being overweight/obese and sleep duration on weekdays was demonstrated in the current study, which may be due to pervasive sleep insufficiency on weekdays in Chinese children.
ER  - 

BibTeX

@misc{14646cbc8004b0e1c408c59382742be4e8fb2975,
  author = {S. Verhoef and S. Camps and H. Gonnissen and K. Westerterp and M. Westerterp-Plantenga},
  title = {Concomitant changes in sleep duration and body weight and body composition during weight loss and 3-mo weight maintenance.},
  journal = {The American journal of clinical nutrition},
  year = {2013},
  volume = {98 1},
  pages = {
          25-31
        },
  doi = {10.3945/ajcn.112.054650},
  abstract = {BACKGROUND
An inverse relation between sleep duration and body mass index (BMI) has been shown.


OBJECTIVE
We assessed the relation between changes in sleep duration and changes in body weight and body composition during weight loss.


DESIGN
A total of 98 healthy subjects (25 men), aged 20-50 y and with BMI (in kg/m(2)) from 28 to 35, followed a 2-mo very-low-energy diet that was followed by a 10-mo period of weight maintenance. Body weight, body composition (measured by using deuterium dilution and air-displacement plethysmography), eating behavior (measured by using a 3-factor eating questionnaire), physical activity (measured by using the validated Baecke's questionnaire), and sleep (estimated by using a questionnaire with the Epworth Sleepiness Scale) were assessed before and immediately after weight loss and 3- and 10-mo follow-ups.


RESULTS
The average weight loss was 10% after 2 mo of dieting and 9% and 6% after 3- and 10-mo follow-ups, respectively. Daytime sleepiness and time to fall asleep decreased during weight loss. Short (≤7 h) and average (>7 to <9 h) sleepers increased their sleep duration, whereas sleep duration in long sleepers (≥9 h) did not change significantly during weight loss. This change in sleep duration was concomitantly negatively correlated with the change in BMI during weight loss and after the 3-mo follow-up and with the change in fat mass after the 3-mo follow-up.


CONCLUSIONS
Sleep duration benefits from weight loss or vice versa. Successful weight loss, loss of body fat, and 3-mo weight maintenance in short and average sleepers are underscored by an increase in sleep duration or vice versa. This trial was registered at clinicaltrials.gov as NCT01015508.}
}

RIS

TY  - JOUR
AU  - S. Verhoef
AU  - S. Camps
AU  - H. Gonnissen
AU  - K. Westerterp
AU  - M. Westerterp-Plantenga
T1  - Concomitant changes in sleep duration and body weight and body composition during weight loss and 3-mo weight maintenance.
JO  - The American journal of clinical nutrition
PY  - 2013
VL  - 98 1
SP  - 
          25-31
        
EP  - 
          25-31
        
DO  - 10.3945/ajcn.112.054650
AB  - BACKGROUND
An inverse relation between sleep duration and body mass index (BMI) has been shown.


OBJECTIVE
We assessed the relation between changes in sleep duration and changes in body weight and body composition during weight loss.


DESIGN
A total of 98 healthy subjects (25 men), aged 20-50 y and with BMI (in kg/m(2)) from 28 to 35, followed a 2-mo very-low-energy diet that was followed by a 10-mo period of weight maintenance. Body weight, body composition (measured by using deuterium dilution and air-displacement plethysmography), eating behavior (measured by using a 3-factor eating questionnaire), physical activity (measured by using the validated Baecke's questionnaire), and sleep (estimated by using a questionnaire with the Epworth Sleepiness Scale) were assessed before and immediately after weight loss and 3- and 10-mo follow-ups.


RESULTS
The average weight loss was 10% after 2 mo of dieting and 9% and 6% after 3- and 10-mo follow-ups, respectively. Daytime sleepiness and time to fall asleep decreased during weight loss. Short (≤7 h) and average (>7 to <9 h) sleepers increased their sleep duration, whereas sleep duration in long sleepers (≥9 h) did not change significantly during weight loss. This change in sleep duration was concomitantly negatively correlated with the change in BMI during weight loss and after the 3-mo follow-up and with the change in fat mass after the 3-mo follow-up.


CONCLUSIONS
Sleep duration benefits from weight loss or vice versa. Successful weight loss, loss of body fat, and 3-mo weight maintenance in short and average sleepers are underscored by an increase in sleep duration or vice versa. This trial was registered at clinicaltrials.gov as NCT01015508.
ER  - 

BibTeX

@misc{66304ffac2c88ed4f51a7dbe91ebe84b9894f51c,
  author = {Guobo Li and Shichao Wei and Fuhao Zheng and Xingyan Xu and Yingying Cai and Siying Wu},
  title = {Association between Sleep Duration and Overweight/Obesity risk among middle-school students: a cross-sectional study in Fuzhou, China},
  year = {2020},
  doi = {10.21203/rs.3.rs-30129/v1},
  abstract = {
 Background: This study was designed to investigate the prevalence of sleep deprivation and explore the association between sleep duration and overweight or obesity in adolescents from middle school in Fuzhou, China.Methods: Questionaires focusing on sleep duration and overweight or obesity related factors were collected. A generalized linear hybrid model was used to evaluate the effects of sleep duration on overweight or obesity in school students.Results: The overall rates of overweight and obesity were 12.1% and 7.1%, respectively. The prevalence of sleep deprivation among students was 82.8%. The majority of high school students (92.5%) suffered from insufficient sleep. Compared with male children with a sleep duration of over 8 hrs a day, the odds ratios (95% CI) of overweight/obesity for those with a sleep duration of less than 6 h or 6 - 8 h, were 1.63 (1.25-2.13) and 1.06 (0.88-1.27). After adjusting social and demographic status, mental health and lifestyle factors, the odds ratios of female children were 1.38(0.99-1.93) and 1.04 (0.79-1.35), respectively.Conclusions: A large number of adolescents suffered from insufficient sleep. Sleep duration was negatively correlated with overweight or obesity among male children.}
}

RIS

TY  - JOUR
AU  - Guobo Li
AU  - Shichao Wei
AU  - Fuhao Zheng
AU  - Xingyan Xu
AU  - Yingying Cai
AU  - Siying Wu
T1  - Association between Sleep Duration and Overweight/Obesity risk among middle-school students: a cross-sectional study in Fuzhou, China
PY  - 2020
DO  - 10.21203/rs.3.rs-30129/v1
AB  - 
 Background: This study was designed to investigate the prevalence of sleep deprivation and explore the association between sleep duration and overweight or obesity in adolescents from middle school in Fuzhou, China.Methods: Questionaires focusing on sleep duration and overweight or obesity related factors were collected. A generalized linear hybrid model was used to evaluate the effects of sleep duration on overweight or obesity in school students.Results: The overall rates of overweight and obesity were 12.1% and 7.1%, respectively. The prevalence of sleep deprivation among students was 82.8%. The majority of high school students (92.5%) suffered from insufficient sleep. Compared with male children with a sleep duration of over 8 hrs a day, the odds ratios (95% CI) of overweight/obesity for those with a sleep duration of less than 6 h or 6 - 8 h, were 1.63 (1.25-2.13) and 1.06 (0.88-1.27). After adjusting social and demographic status, mental health and lifestyle factors, the odds ratios of female children were 1.38(0.99-1.93) and 1.04 (0.79-1.35), respectively.Conclusions: A large number of adolescents suffered from insufficient sleep. Sleep duration was negatively correlated with overweight or obesity among male children.
ER  - 

BibTeX

@misc{ec16bfdda0fc325069b45a5a48387e38ff6fc29e,
  author = {J. Chaput and J. Despres and C. Bouchard and A. Tremblay},
  title = {The association between short sleep duration and weight gain is dependent on disinhibited eating behavior in adults.},
  journal = {Sleep},
  year = {2011},
  volume = {34 10},
  pages = {
          1291-7
        },
  doi = {10.5665/SLEEP.1264},
  abstract = {STUDY OBJECTIVE
To investigate whether the relationship between short sleep duration and subsequent body weight gain is influenced by disinhibited eating behavior.


DESIGN
Six-year longitudinal study.


SETTING
Community setting.


PARTICIPANTS
Two hundred seventy-six adults aged 21 to 64 years from the Quebec Family Study.


MEASUREMENTS AND RESULTS
Body composition measurements, self-reported sleep duration, and disinhibition eating behavior trait (Three-Factor Eating Questionnaire) were determined at both baseline and after 6 years. For each sleep-duration group (short- [≤6 h] average, [7-8 h], and long- [≥9 h] duration sleepers), differences in weight gain and waist circumference were tested by comparing the lowest (score ≤ 3) versus the highest (score ≥ 6) disinhibition eating behavior tertiles using analysis of covariance, with adjustment for potential confounding factors. Individuals having both short sleep duration and high disinhibition eating behavior were more likely to gain weight and increase their abdominal circumference over time (P<0.05); however, short-duration sleepers having a low disinhibition eating behavior trait were not more likely to increase their adiposity indicators than were average-duration sleepers. Over the 6-year follow-up period, the incidence of overweight/obesity for short-duration sleepers with a high disinhibition eating behavior trait was 2.5 times more frequent than for short-duration sleepers with a low disinhibition eating behavior trait. Energy intake was significantly higher in short-duration sleepers with a high disinhibition eating behavior trait (P<0.05 versus all other groups).


CONCLUSIONS
We observed that having a high disinhibition eating behavior trait significantly increased the risk of overeating and gaining weight in adults characterized by short sleep duration. This observation is novel and might explain the interindividual differences in weight gain associated with short sleep duration.}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - J. Despres
AU  - C. Bouchard
AU  - A. Tremblay
T1  - The association between short sleep duration and weight gain is dependent on disinhibited eating behavior in adults.
JO  - Sleep
PY  - 2011
VL  - 34 10
SP  - 
          1291-7
        
EP  - 
          1291-7
        
DO  - 10.5665/SLEEP.1264
AB  - STUDY OBJECTIVE
To investigate whether the relationship between short sleep duration and subsequent body weight gain is influenced by disinhibited eating behavior.


DESIGN
Six-year longitudinal study.


SETTING
Community setting.


PARTICIPANTS
Two hundred seventy-six adults aged 21 to 64 years from the Quebec Family Study.


MEASUREMENTS AND RESULTS
Body composition measurements, self-reported sleep duration, and disinhibition eating behavior trait (Three-Factor Eating Questionnaire) were determined at both baseline and after 6 years. For each sleep-duration group (short- [≤6 h] average, [7-8 h], and long- [≥9 h] duration sleepers), differences in weight gain and waist circumference were tested by comparing the lowest (score ≤ 3) versus the highest (score ≥ 6) disinhibition eating behavior tertiles using analysis of covariance, with adjustment for potential confounding factors. Individuals having both short sleep duration and high disinhibition eating behavior were more likely to gain weight and increase their abdominal circumference over time (P<0.05); however, short-duration sleepers having a low disinhibition eating behavior trait were not more likely to increase their adiposity indicators than were average-duration sleepers. Over the 6-year follow-up period, the incidence of overweight/obesity for short-duration sleepers with a high disinhibition eating behavior trait was 2.5 times more frequent than for short-duration sleepers with a low disinhibition eating behavior trait. Energy intake was significantly higher in short-duration sleepers with a high disinhibition eating behavior trait (P<0.05 versus all other groups).


CONCLUSIONS
We observed that having a high disinhibition eating behavior trait significantly increased the risk of overeating and gaining weight in adults characterized by short sleep duration. This observation is novel and might explain the interindividual differences in weight gain associated with short sleep duration.
ER  - 

BibTeX

@misc{a1c828e45bf15a1e7d00a30749641eb7801722fd,
  author = {Eva Wendt and Elisabeth Pernerstorfer and J. Möseneder and Gabriele Karner and S. Pölten},
  title = {Association between sleep duration and body-mass-index in 10-to 14-year-old Austrians},
  year = {2013},
  abstract = {For many years the focus of research has been on the risk factors which contribute to the development of obesity. A combination of genetic determination, social factors and lifestyle factors are presumed to be responsible [3]. The role sleep duration plays in this context is one of the factors that has been investigated. Observations have shown that as the prevalence of obesity increases, the sleeping period decreases [4–6]. Numerous cross-sectional studies related to sleep duration and body mass index (BMI) in adults, as well as in children (particularly those under 10 years of age) demonstrate that insufficient sleep duration is associated with overweight and obesity. There appears to be a stronger tendency towards obesity in the event of shortened sleep duration for boys than girls [5, 7]. There are also potential indications that insufficient sleep duration in childhood has an effect on later body composition [8].}
}

RIS

TY  - JOUR
AU  - Eva Wendt
AU  - Elisabeth Pernerstorfer
AU  - J. Möseneder
AU  - Gabriele Karner
AU  - S. Pölten
T1  - Association between sleep duration and body-mass-index in 10-to 14-year-old Austrians
PY  - 2013
AB  - For many years the focus of research has been on the risk factors which contribute to the development of obesity. A combination of genetic determination, social factors and lifestyle factors are presumed to be responsible [3]. The role sleep duration plays in this context is one of the factors that has been investigated. Observations have shown that as the prevalence of obesity increases, the sleeping period decreases [4–6]. Numerous cross-sectional studies related to sleep duration and body mass index (BMI) in adults, as well as in children (particularly those under 10 years of age) demonstrate that insufficient sleep duration is associated with overweight and obesity. There appears to be a stronger tendency towards obesity in the event of shortened sleep duration for boys than girls [5, 7]. There are also potential indications that insufficient sleep duration in childhood has an effect on later body composition [8].
ER  - 

BibTeX

@misc{88d7fc611124fc74ab5606da872306ecfe37246c,
  author = {Y. Do},
  title = {Working paper ser ies on health and demographic change in the Asia-Pacific The Effect of Sleep Duration on Body Weight in Adolescents : Evidence from a Natural Experiment},
  year = {2014},
  abstract = {Despite a large number of observational studies consistently reporting the association between shorter sleep duration and higher body weight, causality has yet to be established at a population level. This study aims to estimate the population-level causal effect of sleep duration on adolescent body weight, using an instrumental variable (IV) approach that exploits a unique natural experiment in the context of South Korea’s highly competitive secondary education. In March 2011, amid growing concerns over the negative consequences of late-night tuition at private tutoring institutes (hagwon), authorities in 3 of the 16 administrative regions in South Korea decreed adjusting the closing hours of hagwon to 10 p.m. This policy change caused a substantial and plausibly exogenous variation in the sleep duration of the “marginal student,” whose sleep duration is most likely to be affected by the policy. The IV estimation results on a sample of general high school 10and 11-graders in the 2009−2012 Korea Youth Risk Behavior Web-based Survey show that a 1-hour increase in sleep duration led to a 0.56 kg/m reduction in body mass index, or a 4.3 percentage-point decrease in overweight/obesity. Short sleep duration among adolescents may be an important contributor to increased body weight at the population level. JEL Classification Codes: I12; I18; I28}
}

RIS

TY  - JOUR
AU  - Y. Do
T1  - Working paper ser ies on health and demographic change in the Asia-Pacific The Effect of Sleep Duration on Body Weight in Adolescents : Evidence from a Natural Experiment
PY  - 2014
AB  - Despite a large number of observational studies consistently reporting the association between shorter sleep duration and higher body weight, causality has yet to be established at a population level. This study aims to estimate the population-level causal effect of sleep duration on adolescent body weight, using an instrumental variable (IV) approach that exploits a unique natural experiment in the context of South Korea’s highly competitive secondary education. In March 2011, amid growing concerns over the negative consequences of late-night tuition at private tutoring institutes (hagwon), authorities in 3 of the 16 administrative regions in South Korea decreed adjusting the closing hours of hagwon to 10 p.m. This policy change caused a substantial and plausibly exogenous variation in the sleep duration of the “marginal student,” whose sleep duration is most likely to be affected by the policy. The IV estimation results on a sample of general high school 10and 11-graders in the 2009−2012 Korea Youth Risk Behavior Web-based Survey show that a 1-hour increase in sleep duration led to a 0.56 kg/m reduction in body mass index, or a 4.3 percentage-point decrease in overweight/obesity. Short sleep duration among adolescents may be an important contributor to increased body weight at the population level. JEL Classification Codes: I12; I18; I28
ER  - 

BibTeX

@misc{a6ee1f85d4e0a416597a68e8ded874fc1e98c65e,
  author = {R. Chamorro and S. Durán and S. Reyes and R. Ponce and Cecilia Algarín and P. Peirano},
  title = {La reducción del sueño como factor de riesgo para obesidad},
  journal = {Revista Medica De Chile},
  year = {2011},
  volume = {139},
  pages = {932-940},
  doi = {10.4067/S0034-98872011000700017},
  abstract = {Nocturnal sleep patterns may be a contributing factor for the epidemic of obesity. Epidemiologic ana experimental studies have reported that sleep restriction is an independent risk factor for weight gain and obesity. Moreover, sleep restriction is significantly associated with incidence and prevalence of obesity and several non-transmissible chronic diseases. Experimental sleep restriction is related to altered plasma leptin and ghrelin concentrations. Both hormones are directly related to appetite and satiety mechanisms. Also, a higher activity of the orexin/hypocretin system has been reported, as well as changes in glucose metabolism and autonomic nervous system. Some studies indicate that these endocrine changes could be associated with a higher diurnal food intake and preference for energy- dense foods. All these changes could result in a positive energy balance, leading to weight gain and a higher obesity risk in the long-term. The present article summarizes the epidemiologic and experimental evidence related to sleep deprivation and higher obesity risk. The possible mechanisms are highlighted.}
}

RIS

TY  - JOUR
AU  - R. Chamorro
AU  - S. Durán
AU  - S. Reyes
AU  - R. Ponce
AU  - Cecilia Algarín
AU  - P. Peirano
T1  - La reducción del sueño como factor de riesgo para obesidad
JO  - Revista Medica De Chile
PY  - 2011
VL  - 139
SP  - 932-940
EP  - 932-940
DO  - 10.4067/S0034-98872011000700017
AB  - Nocturnal sleep patterns may be a contributing factor for the epidemic of obesity. Epidemiologic ana experimental studies have reported that sleep restriction is an independent risk factor for weight gain and obesity. Moreover, sleep restriction is significantly associated with incidence and prevalence of obesity and several non-transmissible chronic diseases. Experimental sleep restriction is related to altered plasma leptin and ghrelin concentrations. Both hormones are directly related to appetite and satiety mechanisms. Also, a higher activity of the orexin/hypocretin system has been reported, as well as changes in glucose metabolism and autonomic nervous system. Some studies indicate that these endocrine changes could be associated with a higher diurnal food intake and preference for energy- dense foods. All these changes could result in a positive energy balance, leading to weight gain and a higher obesity risk in the long-term. The present article summarizes the epidemiologic and experimental evidence related to sleep deprivation and higher obesity risk. The possible mechanisms are highlighted.
ER  - 

BibTeX

@misc{9abeaa61ea098d1d7ee4225901bb1bea164fe9a5,
  author = {Michelle A. Miller and Marlot Kruisbrink and J. Wallace and Andrew O’Keeffe and Sam Valint and Chen Ji and F. Cappuccio},
  title = {Abstract MP090: Sleep Duration Predict Incident Obesity in Childhood and Adolescence: Meta-analysis of Prospective Studies},
  journal = {Circulation},
  year = {2017},
  doi = {10.1161/circ.135.suppl_1.mp090},
  abstract = {
 Objectives:
 To assess the longitudinal evidence of a relationship between duration of sleep and both incidence of overweight and/or obesity and changes in body mass index in infants, children and adolescents.
 
 
 Methods:
 We performed a systematic search of publications using MEDLINE (1966-2016), EMBASE (from 1980), Web of Science (from 1945), the Cochrane Library and manual searches without language restrictions. Studies were included if they were prospective, had a follow up >1 year, had measured duration of sleep at baseline and assessed either incident cases of overweight and/or obesity or changes in body mass index prospectively. Relative risks (RR), regression coefficients (beta) and 95% C.I. were extracted and pooled using a random effect model. Sensitivity analyses and meta-regressions were performed, heterogeneity and publication bias were also assessed.
 
 
 Results:
 Overall, 41 studies provided 56 independent cohort samples, including 98,130 boys and girls aged 0-18 years (follow-up 1 to 27 years). Sleep duration was assessed by questionnaire, either to parents or to children, actigraphy or polysomnography. Body mass index (BMI) was measured in all participants. In the pooled analyses, short duration of sleep was associated with a greater risk of becoming overweight or obese (RR: 1.57; 95% CI 1.34, 1.85; p<10
 -5
 ; n=59,471). There was significant heterogeneity (I
 2
 =92%, p<0.001), accounted for by three studies (I
 2
 =3% after their removal) and evidence of publication bias (p=0.007). The effect size increased with age (infants 1.40 [1.19, 1.65]; early childhood 1.53 [1.33, 1.76]; middle childhood 2.23 [2.18, 2.27]; adolescence 1.30 [1.11, 1.53]; p for heterogeneity <10
 -5
 ). The effect was not associated with average length of follow-up (p=0.923) or quality (p=0.475). BMI decreased for every hour of increased sleep (pooled beta: -0.01; -0.03, 0.00 kg/m
 2
 per h, p=0.032; n=16,225).
 
 
 Conclusion:
 Short duration of sleep is a significant predictor of the development of overweight, obesity and weight gain from birth to adolescence.
}
}

RIS

TY  - JOUR
AU  - Michelle A. Miller
AU  - Marlot Kruisbrink
AU  - J. Wallace
AU  - Andrew O’Keeffe
AU  - Sam Valint
AU  - Chen Ji
AU  - F. Cappuccio
T1  - Abstract MP090: Sleep Duration Predict Incident Obesity in Childhood and Adolescence: Meta-analysis of Prospective Studies
JO  - Circulation
PY  - 2017
DO  - 10.1161/circ.135.suppl_1.mp090
AB  - 
 Objectives:
 To assess the longitudinal evidence of a relationship between duration of sleep and both incidence of overweight and/or obesity and changes in body mass index in infants, children and adolescents.
 
 
 Methods:
 We performed a systematic search of publications using MEDLINE (1966-2016), EMBASE (from 1980), Web of Science (from 1945), the Cochrane Library and manual searches without language restrictions. Studies were included if they were prospective, had a follow up >1 year, had measured duration of sleep at baseline and assessed either incident cases of overweight and/or obesity or changes in body mass index prospectively. Relative risks (RR), regression coefficients (beta) and 95% C.I. were extracted and pooled using a random effect model. Sensitivity analyses and meta-regressions were performed, heterogeneity and publication bias were also assessed.
 
 
 Results:
 Overall, 41 studies provided 56 independent cohort samples, including 98,130 boys and girls aged 0-18 years (follow-up 1 to 27 years). Sleep duration was assessed by questionnaire, either to parents or to children, actigraphy or polysomnography. Body mass index (BMI) was measured in all participants. In the pooled analyses, short duration of sleep was associated with a greater risk of becoming overweight or obese (RR: 1.57; 95% CI 1.34, 1.85; p<10
 -5
 ; n=59,471). There was significant heterogeneity (I
 2
 =92%, p<0.001), accounted for by three studies (I
 2
 =3% after their removal) and evidence of publication bias (p=0.007). The effect size increased with age (infants 1.40 [1.19, 1.65]; early childhood 1.53 [1.33, 1.76]; middle childhood 2.23 [2.18, 2.27]; adolescence 1.30 [1.11, 1.53]; p for heterogeneity <10
 -5
 ). The effect was not associated with average length of follow-up (p=0.923) or quality (p=0.475). BMI decreased for every hour of increased sleep (pooled beta: -0.01; -0.03, 0.00 kg/m
 2
 per h, p=0.032; n=16,225).
 
 
 Conclusion:
 Short duration of sleep is a significant predictor of the development of overweight, obesity and weight gain from birth to adolescence.

ER  - 

BibTeX

@misc{c8ff41ff9bc1a1d0f8cecb3d0a7f11af3c5f8278,
  author = {R. Chamorro and S. Durán and S. Reyes and Rosemarie Ponce and C. Algarín and P. Peirano},
  title = {[Sleep deprivation as a risk factor for obesity].},
  journal = {Revista medica de Chile},
  year = {2011},
  volume = {139 7},
  pages = {
          932-40
        },
  abstract = {Nocturnal sleep patterns may be a contributing factor for the epidemic of obesity. Epidemiologic ana experimental studies have reported that sleep restriction is an independent risk factor for weight gain and obesity. Moreover, sleep restriction is significantly associated with incidence and prevalence of obesity and several non-transmissible chronic diseases. Experimental sleep restriction is related to altered plasma leptin and ghrelin concentrations. Both hormones are directly related to appetite and satiety mechanisms. Also, a higher activity of the orexin/hypocretin system has been reported, as well as changes in glucose metabolism and autonomic nervous system. Some studies indicate that these endocrine changes could be associated with a higher diurnal food intake and preference for energy- dense foods. All these changes could result in a positive energy balance, leading to weight gain and a higher obesity risk in the long-term. The present article summarizes the epidemiologic and experimental evidence related to sleep deprivation and higher obesity risk. The possible mechanisms are highlighted.}
}

RIS

TY  - JOUR
AU  - R. Chamorro
AU  - S. Durán
AU  - S. Reyes
AU  - Rosemarie Ponce
AU  - C. Algarín
AU  - P. Peirano
T1  - [Sleep deprivation as a risk factor for obesity].
JO  - Revista medica de Chile
PY  - 2011
VL  - 139 7
SP  - 
          932-40
        
EP  - 
          932-40
        
AB  - Nocturnal sleep patterns may be a contributing factor for the epidemic of obesity. Epidemiologic ana experimental studies have reported that sleep restriction is an independent risk factor for weight gain and obesity. Moreover, sleep restriction is significantly associated with incidence and prevalence of obesity and several non-transmissible chronic diseases. Experimental sleep restriction is related to altered plasma leptin and ghrelin concentrations. Both hormones are directly related to appetite and satiety mechanisms. Also, a higher activity of the orexin/hypocretin system has been reported, as well as changes in glucose metabolism and autonomic nervous system. Some studies indicate that these endocrine changes could be associated with a higher diurnal food intake and preference for energy- dense foods. All these changes could result in a positive energy balance, leading to weight gain and a higher obesity risk in the long-term. The present article summarizes the epidemiologic and experimental evidence related to sleep deprivation and higher obesity risk. The possible mechanisms are highlighted.
ER  - 

BibTeX

@misc{d3ee7129f31d9548796be694425788c677988ddc,
  author = {E. Touchette and D. Petit and R. Tremblay and M. Boivin and B. Falissard and C. Genolini and J. Montplaisir},
  title = {Associations between sleep duration patterns and overweight/obesity at age 6.},
  journal = {Sleep},
  year = {2008},
  volume = {31 11},
  pages = {
          1507-14
        },
  doi = {10.1093/SLEEP/31.11.1507},
  abstract = {OBJECTIVE
To investigate whether longitudinal sleep duration patterns during early childhood is a risk factor of overweight or obesity at school entry while controlling for a variety of obesogenic environmental factors.


DESIGN, SETTING, AND PARTICIPANTS
This is a prospective cohort study (March-December 1998 to December 2004) of a representative sample of infants born in 1997-1998 in the Canadian province of Quebec. Body mass index (BMI) was measured at ages 2.5 and 6 years. Sleep duration was reported yearly from 2.5 to 6 years of age by their mothers. Prenatal, postnatal (5 and 29 months), and lifestyle (6 y) potentially confounding factors for excess weight were assessed by interviews, questionnaires and hospital records. A group-based semiparametric mixture model was used to estimate developmental patterns of sleep duration. The relationship between sleep duration patterns and BMI was tested using multivariate logistic regression models to control for potentially confounding factors on 1138 children.


RESULTS
Four sleep duration patterns were identified: short persistent (5.2%), short increasing (4.7%), 10-hour persistent (50.7%), and 11-hour persistent (39.4%). After controlling for potentially confounding factors, the risk for overweight or obesity was almost 4.2 times higher for short persistent sleepers (odds ratio [OR], 4.2; 95% confidence interval [CI], 1.6 to 11.1; P = 0.003) than for 11-hour persistent sleepers.


CONCLUSIONS
Persistently short sleep duration (<10 h) during early childhood significantly increases the risk of excess weight or obesity in childhood, and appears to be independent of other obesogenic factors.}
}

RIS

TY  - JOUR
AU  - E. Touchette
AU  - D. Petit
AU  - R. Tremblay
AU  - M. Boivin
AU  - B. Falissard
AU  - C. Genolini
AU  - J. Montplaisir
T1  - Associations between sleep duration patterns and overweight/obesity at age 6.
JO  - Sleep
PY  - 2008
VL  - 31 11
SP  - 
          1507-14
        
EP  - 
          1507-14
        
DO  - 10.1093/SLEEP/31.11.1507
AB  - OBJECTIVE
To investigate whether longitudinal sleep duration patterns during early childhood is a risk factor of overweight or obesity at school entry while controlling for a variety of obesogenic environmental factors.


DESIGN, SETTING, AND PARTICIPANTS
This is a prospective cohort study (March-December 1998 to December 2004) of a representative sample of infants born in 1997-1998 in the Canadian province of Quebec. Body mass index (BMI) was measured at ages 2.5 and 6 years. Sleep duration was reported yearly from 2.5 to 6 years of age by their mothers. Prenatal, postnatal (5 and 29 months), and lifestyle (6 y) potentially confounding factors for excess weight were assessed by interviews, questionnaires and hospital records. A group-based semiparametric mixture model was used to estimate developmental patterns of sleep duration. The relationship between sleep duration patterns and BMI was tested using multivariate logistic regression models to control for potentially confounding factors on 1138 children.


RESULTS
Four sleep duration patterns were identified: short persistent (5.2%), short increasing (4.7%), 10-hour persistent (50.7%), and 11-hour persistent (39.4%). After controlling for potentially confounding factors, the risk for overweight or obesity was almost 4.2 times higher for short persistent sleepers (odds ratio [OR], 4.2; 95% confidence interval [CI], 1.6 to 11.1; P = 0.003) than for 11-hour persistent sleepers.


CONCLUSIONS
Persistently short sleep duration (<10 h) during early childhood significantly increases the risk of excess weight or obesity in childhood, and appears to be independent of other obesogenic factors.
ER  - 

BibTeX

@misc{862f488c22e246a3232e56e3eaf3a86b58e8556b,
  author = {Evangelia I Papatriantafyllou and D. Efthymiou and Evangelos Zoumbaneas and C. Popescu and E. Vassilopoulou},
  title = {Sleep Deprivation: Effects on Weight Loss and Weight Loss Maintenance},
  journal = {Nutrients},
  year = {2022},
  volume = {14},
  doi = {10.3390/nu14081549},
  abstract = {This narrative review presents the findings from intervention studies on the effects of sleep deprivation on eating habits, metabolic rate, and the hormones regulating metabolism, and discusses their relevance to weight loss efforts. Disturbed sleeping patterns lead to increased energy intake, partly from excessive snacking, mainly on foods high in fat and carbohydrates. The studies focused mainly on the effects of sleep duration, but also of sleep quality, on dietary intake during weight loss trials, and on weight loss maintenance. It is important to explore sleep routines that could enhance the efforts of obese and overweight people to lose weight, maintain their weight loss, and improve their overall health.}
}

RIS

TY  - JOUR
AU  - Evangelia I Papatriantafyllou
AU  - D. Efthymiou
AU  - Evangelos Zoumbaneas
AU  - C. Popescu
AU  - E. Vassilopoulou
T1  - Sleep Deprivation: Effects on Weight Loss and Weight Loss Maintenance
JO  - Nutrients
PY  - 2022
VL  - 14
DO  - 10.3390/nu14081549
AB  - This narrative review presents the findings from intervention studies on the effects of sleep deprivation on eating habits, metabolic rate, and the hormones regulating metabolism, and discusses their relevance to weight loss efforts. Disturbed sleeping patterns lead to increased energy intake, partly from excessive snacking, mainly on foods high in fat and carbohydrates. The studies focused mainly on the effects of sleep duration, but also of sleep quality, on dietary intake during weight loss trials, and on weight loss maintenance. It is important to explore sleep routines that could enhance the efforts of obese and overweight people to lose weight, maintain their weight loss, and improve their overall health.
ER  - 

BibTeX

@misc{09e7b4aa4fbdbe226fcbb387b74d735f02e18fc6,
  author = {Nurul Ramadhayani and Feranita Utama},
  title = {The Relationship Between Sleep Duration and Obesity in Children and Adolescents in Asia: A Meta-Analysis},
  journal = {Jurnal Ilmu Kesehatan Masyarakat},
  year = {2022},
  doi = {10.26553/jikm.2022.13.2.261-277},
  abstract = {In recent years, short sleep duration has been proposed and received attention as a modifiable factor of obesity. Observational studies on the relationship between sleep duration and obesity in children and adolescents in the Asian region have been carried out. This study aims to determine the relationship between short sleep duration and obesity in children and adolescents in Asia. This study used a meta-analysis method. Search for articles using the Pubmed, Proquest, Science direct and Google Scholar databases. Article search is limited to research in English, publications from 2000 to 2020 and free full text articles. Data is processed using the Revman 5.3 application. There were 12 research articles that were systematically reviewed from 1,281 identified articles. Ten of these articles were included in the meta-analysis. The results of the study proved that there was a significant relationship between short sleep duration and obesity in children and adolescents with a pooled OR of 1.15 (95% CI: 1.04-1.28), and an analysis of the age subgroup also showed the same results as the pooled OR. 1.17 (95% CI: 1.02-1.35) in the age group before 3-4 years and combined OR 1.29 (95% CI: 1.14-1.48) in children aged 6-12 years. Avoiding short sleep duration can be considered as one way to reduce the risk of obesity in children and adolescents.}
}

RIS

TY  - JOUR
AU  - Nurul Ramadhayani
AU  - Feranita Utama
T1  - The Relationship Between Sleep Duration and Obesity in Children and Adolescents in Asia: A Meta-Analysis
JO  - Jurnal Ilmu Kesehatan Masyarakat
PY  - 2022
DO  - 10.26553/jikm.2022.13.2.261-277
AB  - In recent years, short sleep duration has been proposed and received attention as a modifiable factor of obesity. Observational studies on the relationship between sleep duration and obesity in children and adolescents in the Asian region have been carried out. This study aims to determine the relationship between short sleep duration and obesity in children and adolescents in Asia. This study used a meta-analysis method. Search for articles using the Pubmed, Proquest, Science direct and Google Scholar databases. Article search is limited to research in English, publications from 2000 to 2020 and free full text articles. Data is processed using the Revman 5.3 application. There were 12 research articles that were systematically reviewed from 1,281 identified articles. Ten of these articles were included in the meta-analysis. The results of the study proved that there was a significant relationship between short sleep duration and obesity in children and adolescents with a pooled OR of 1.15 (95% CI: 1.04-1.28), and an analysis of the age subgroup also showed the same results as the pooled OR. 1.17 (95% CI: 1.02-1.35) in the age group before 3-4 years and combined OR 1.29 (95% CI: 1.14-1.48) in children aged 6-12 years. Avoiding short sleep duration can be considered as one way to reduce the risk of obesity in children and adolescents.
ER  - 

BibTeX

@misc{0c6fbb0cea3daaa8224c22781c65a0d62f888091,
  author = {H. Thind and S. Davies and Terri Lewis and Dorothy W Pekmezi and Retta R. Evans and M. Baskin},
  title = {Does Short Sleep Lead to Obesity Among Children and Adolescents? Current Understanding and Implications},
  journal = {American Journal of Lifestyle Medicine},
  year = {2015},
  volume = {9},
  pages = {428 - 437},
  doi = {10.1177/1559827614533911},
  abstract = {Childhood obesity continues to be a major public health concern in the United States. This work reviews the current understanding of the relationship between sleep duration and obesity among children and adolescents. A systematic search was conducted for papers published between January 2000 and July 2013 using keywords: (sleep) and (overweight or obesity or obese or body mass index or BMI or adiposity or body fat or fat) and (children or child or youth or teen or pediatric or adolescent or paediatric or childhood or adolescence or boy or girl). Reference lists of relevant articles and reviews or meta-analysis articles were checked to identify additional studies. Only empirical work and longitudinal studies that focused on children and adolescents were included in this review. The search identified 22 longitudinal studies. The majority of the reviewed studies support the presence of an inverse relationship between sleep duration and obesity. However, in some studies the relationship was not significant in adjusted analyses. Differences as a function of age and gender were also noted. Despite more than a decade of research, the debate on the association between sleep duration and obesity continues. Further research with repeated assessments, valid objective measures, and better control of potential confounding variables is warranted.}
}

RIS

TY  - JOUR
AU  - H. Thind
AU  - S. Davies
AU  - Terri Lewis
AU  - Dorothy W Pekmezi
AU  - Retta R. Evans
AU  - M. Baskin
T1  - Does Short Sleep Lead to Obesity Among Children and Adolescents? Current Understanding and Implications
JO  - American Journal of Lifestyle Medicine
PY  - 2015
VL  - 9
SP  - 428 - 437
EP  - 428 - 437
DO  - 10.1177/1559827614533911
AB  - Childhood obesity continues to be a major public health concern in the United States. This work reviews the current understanding of the relationship between sleep duration and obesity among children and adolescents. A systematic search was conducted for papers published between January 2000 and July 2013 using keywords: (sleep) and (overweight or obesity or obese or body mass index or BMI or adiposity or body fat or fat) and (children or child or youth or teen or pediatric or adolescent or paediatric or childhood or adolescence or boy or girl). Reference lists of relevant articles and reviews or meta-analysis articles were checked to identify additional studies. Only empirical work and longitudinal studies that focused on children and adolescents were included in this review. The search identified 22 longitudinal studies. The majority of the reviewed studies support the presence of an inverse relationship between sleep duration and obesity. However, in some studies the relationship was not significant in adjusted analyses. Differences as a function of age and gender were also noted. Despite more than a decade of research, the debate on the association between sleep duration and obesity continues. Further research with repeated assessments, valid objective measures, and better control of potential confounding variables is warranted.
ER  - 

BibTeX

@misc{8bfe7d42c232e4d9b6592d06d4dd23e4d333e36b,
  author = {E. van Cauter and K. Knutson},
  title = {Sleep and the epidemic of obesity in children and adults},
  journal = {European Journal of Endocrinology},
  year = {2008},
  volume = {159},
  pages = {S59 - S66},
  doi = {10.1530/EJE-08-0298},
  abstract = {Sleep is an important modulator of neuroendocrine function and glucose metabolism in children as well as in adults. In recent years, sleep curtailment has become a hallmark of modern society with both children and adults having shorter bedtimes than a few decades ago. This trend for shorter sleep duration has developed over the same time period as the dramatic increase in the prevalence of obesity. There is rapidly accumulating evidence from both laboratory and epidemiological studies to indicate that chronic partial sleep loss may increase the risk of obesity and weight gain. The present article reviews laboratory evidence indicating that sleep curtailment in young adults results in a constellation of metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, elevated sympathovagal balance, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. We also review cross-sectional epidemiological studies associating short sleep with increased body mass index and prospective epidemiological studies that have shown an increased risk of weight gain and obesity in children and young adults who are short sleepers. Altogether, the evidence points to a possible role of decreased sleep duration in the current epidemic of obesity.}
}

RIS

TY  - JOUR
AU  - E. van Cauter
AU  - K. Knutson
T1  - Sleep and the epidemic of obesity in children and adults
JO  - European Journal of Endocrinology
PY  - 2008
VL  - 159
SP  - S59 - S66
EP  - S59 - S66
DO  - 10.1530/EJE-08-0298
AB  - Sleep is an important modulator of neuroendocrine function and glucose metabolism in children as well as in adults. In recent years, sleep curtailment has become a hallmark of modern society with both children and adults having shorter bedtimes than a few decades ago. This trend for shorter sleep duration has developed over the same time period as the dramatic increase in the prevalence of obesity. There is rapidly accumulating evidence from both laboratory and epidemiological studies to indicate that chronic partial sleep loss may increase the risk of obesity and weight gain. The present article reviews laboratory evidence indicating that sleep curtailment in young adults results in a constellation of metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, elevated sympathovagal balance, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. We also review cross-sectional epidemiological studies associating short sleep with increased body mass index and prospective epidemiological studies that have shown an increased risk of weight gain and obesity in children and young adults who are short sleepers. Altogether, the evidence points to a possible role of decreased sleep duration in the current epidemic of obesity.
ER  - 

BibTeX

@misc{497ac87f15eb8bdbdec2ced07dcd24c09af09719,
  author = {Zlata Piskáčková and M. Forejt and Lucie Martykánová},
  title = {[The relationship between sleep and obesity: current perspective].},
  journal = {Casopis lekaru ceskych},
  year = {2012},
  volume = {151 6},
  pages = {
          279-85
        },
  abstract = {Disruption of circadian rhythms negatively affects regulation of metabolism and energy homeostasis. Disrupted metabolism in response to disrupted biological rhythms might lead, together with genetic background, to obesity and to other health complications. Results of epidemiologic surveys are consistent with mechanistic theory showing the interconnection between the biological rhythms, sleep and metabolism. Epidemiologic surveys confirm that sleep duration of less than 6 hours increases significantly the risk of obesity. Systematic reviews of epidemiologic surveys examining association of sleep and obesity refer to large heterogeneity in involved subjects, methodological approaches of measuring obesity and sleep, and confounders. Design of study plays also essential role in interpretation and definition of causal relationship. Reduced sleep duration in relation to obesity is in the literature discussed from different points of view: 1. as a possible primary cause of obesity, 2. as a result of comorbidities resulting from obesity and 3. as an accompanied part of the third factor contributing to obesity (e.g. long working hours, chronic emotional stress, overusing of media). Causal relationship between sleep and obesity is not yet fully elucidated, however the association is supposed to be bidirectional. The article gives an overview of current knowledge concerning the influence of sleep on the development of obesity and points to the critical points of current research.}
}

RIS

TY  - JOUR
AU  - Zlata Piskáčková
AU  - M. Forejt
AU  - Lucie Martykánová
T1  - [The relationship between sleep and obesity: current perspective].
JO  - Casopis lekaru ceskych
PY  - 2012
VL  - 151 6
SP  - 
          279-85
        
EP  - 
          279-85
        
AB  - Disruption of circadian rhythms negatively affects regulation of metabolism and energy homeostasis. Disrupted metabolism in response to disrupted biological rhythms might lead, together with genetic background, to obesity and to other health complications. Results of epidemiologic surveys are consistent with mechanistic theory showing the interconnection between the biological rhythms, sleep and metabolism. Epidemiologic surveys confirm that sleep duration of less than 6 hours increases significantly the risk of obesity. Systematic reviews of epidemiologic surveys examining association of sleep and obesity refer to large heterogeneity in involved subjects, methodological approaches of measuring obesity and sleep, and confounders. Design of study plays also essential role in interpretation and definition of causal relationship. Reduced sleep duration in relation to obesity is in the literature discussed from different points of view: 1. as a possible primary cause of obesity, 2. as a result of comorbidities resulting from obesity and 3. as an accompanied part of the third factor contributing to obesity (e.g. long working hours, chronic emotional stress, overusing of media). Causal relationship between sleep and obesity is not yet fully elucidated, however the association is supposed to be bidirectional. The article gives an overview of current knowledge concerning the influence of sleep on the development of obesity and points to the critical points of current research.
ER  - 

BibTeX

@misc{d9d49a011b81508437cab01b41ecca53b9da8aa0,
  author = {Shuang Zhang and Lian Li and Yubei Huang and Kexin Chen},
  title = {[Meta-analysis of prospective cohort studies about sleep duration and risk of weight gain and obesity in adults].},
  journal = {Zhonghua liu xing bing xue za zhi = Zhonghua liuxingbingxue zazhi},
  year = {2015},
  volume = {36 5},
  pages = {
          519-25
        },
  doi = {10.3760/CMA.J.ISSN.0254–6450.2015.05.023},
  abstract = {OBJECTIVE
This meta-analysis was conducted to quantitatively estimate the associations between sleep duration and weight gain or obesity in adults according to the literature retrieval results of related prospective cohort studies published before October 2014.


METHODS
The literature retrieval was conducted by using PubMed, Embase, Cochrane Library and Chinese databases, including CNKI, VIP and Wan Fang. The pooled relative risk (RR) and 95% confidence intervals (CI) were estimated, and the tests of the publication bias and the heterogeneity were also performed.


RESULTS
Sixteen literatures met the inclusion criteria were selected for analysis. In 285,452 adults surveyed in these studies, both short sleep duration and long sleep duration significantly increased the risk of weight gain (RR=1.26, 95% CI: 1.12-1.42; RR=1.12, 95% CI: 1.04-1.20), and short sleep duration also increased the risk of obesity (RR=1.35, 95% CI: 1.22-1.50, P<0.001), but long sleep duration was not associated with obesity. In subgroup analysis, the associations were stronger in the studies with higher quality and using <6 h and ≥8 h as the criteria to identify short and long sleep durations.


CONCLUSION
The meta-analysis indicated that both short and long sleep durations were associated with weight gain, and short sleep duration could also increase the risk of obesity. Therefore, public health efforts in promoting sufficient sleep may be important in the prevention of obesity.}
}

RIS

TY  - JOUR
AU  - Shuang Zhang
AU  - Lian Li
AU  - Yubei Huang
AU  - Kexin Chen
T1  - [Meta-analysis of prospective cohort studies about sleep duration and risk of weight gain and obesity in adults].
JO  - Zhonghua liu xing bing xue za zhi = Zhonghua liuxingbingxue zazhi
PY  - 2015
VL  - 36 5
SP  - 
          519-25
        
EP  - 
          519-25
        
DO  - 10.3760/CMA.J.ISSN.0254–6450.2015.05.023
AB  - OBJECTIVE
This meta-analysis was conducted to quantitatively estimate the associations between sleep duration and weight gain or obesity in adults according to the literature retrieval results of related prospective cohort studies published before October 2014.


METHODS
The literature retrieval was conducted by using PubMed, Embase, Cochrane Library and Chinese databases, including CNKI, VIP and Wan Fang. The pooled relative risk (RR) and 95% confidence intervals (CI) were estimated, and the tests of the publication bias and the heterogeneity were also performed.


RESULTS
Sixteen literatures met the inclusion criteria were selected for analysis. In 285,452 adults surveyed in these studies, both short sleep duration and long sleep duration significantly increased the risk of weight gain (RR=1.26, 95% CI: 1.12-1.42; RR=1.12, 95% CI: 1.04-1.20), and short sleep duration also increased the risk of obesity (RR=1.35, 95% CI: 1.22-1.50, P<0.001), but long sleep duration was not associated with obesity. In subgroup analysis, the associations were stronger in the studies with higher quality and using <6 h and ≥8 h as the criteria to identify short and long sleep durations.


CONCLUSION
The meta-analysis indicated that both short and long sleep durations were associated with weight gain, and short sleep duration could also increase the risk of obesity. Therefore, public health efforts in promoting sufficient sleep may be important in the prevention of obesity.
ER  - 

BibTeX

@misc{ffa4aa6106365b8bd7aa23946022eacb2001f366,
  author = {Xiaopeng Ji and Lauren B. Covington and F. Patterson and M. Ji and Janeese A. Brownlow},
  title = {Associations between sleep and overweight/obesity in adolescents vary by race/ethnicity and socioeconomic status.},
  journal = {Journal of advanced nursing},
  year = {2022},
  doi = {10.1111/jan.15513},
  abstract = {AIM
To examine the interaction between sleep and social determinants of health (SDOH) [race/ethnicity and socioeconomic status (SES)] on overweight/obesity in adolescents.


DESIGN
Cross-sectional.


METHODS
We conducted a secondary analysis using the 2017-2018 National Survey of Children's Health data. We included adolescents (10-17 years old) who had sleep and body mass index (BMI) data available (n = 24,337) in analyses (samples with BMI <5th percentile excluded). Parents reported children's sleep duration and regularity. High BMI (≥85th percentile) for age defines overweight/obesity. We selected SDOH (race/ethnicity, family income, primary caregiver education and neighbourhood condition) and covariates (age, sex, smoking, exercise and depression) using a hierarchical model-building approach. Accounting for complex survey design, logistic regression estimated the interaction between sleep and SDOH.


RESULTS
There were significant interactions between sleep duration and SDOH. The association between increasing sleep and decreasing odds of overweight/obesity only showed in the following subgroups: White, family income ≥400% federal poverty level (FPL) or primary caregiver' education ≥ high school. Compared with these subgroups, Hispanic adolescents and adolescents whose family income was below 100% FPL and whose caregiver education was below high school had weakened and reversed associations. Sleep regularity was not associated with overweight/obesity.


CONCLUSIONS
Increasing sleep duration was associated with a decreased risk of overweight/obesity, but the association was not present in adolescents from racial/ethnic minority groups (i.e. Hispanic) and those with low SES.


IMPACT
The study findings suggest that associations between sleep and overweight/obesity vary by race and SES. Identification of additional mechanisms for obesity is needed for racial/ethnic minority groups and those from families with low SES. Also, the complexity of these relationships underscores the importance of community-based needs assessment in the design of targeted and meaningful interventions to address complex health conditions such as poor sleep and obesity.}
}

RIS

TY  - JOUR
AU  - Xiaopeng Ji
AU  - Lauren B. Covington
AU  - F. Patterson
AU  - M. Ji
AU  - Janeese A. Brownlow
T1  - Associations between sleep and overweight/obesity in adolescents vary by race/ethnicity and socioeconomic status.
JO  - Journal of advanced nursing
PY  - 2022
DO  - 10.1111/jan.15513
AB  - AIM
To examine the interaction between sleep and social determinants of health (SDOH) [race/ethnicity and socioeconomic status (SES)] on overweight/obesity in adolescents.


DESIGN
Cross-sectional.


METHODS
We conducted a secondary analysis using the 2017-2018 National Survey of Children's Health data. We included adolescents (10-17 years old) who had sleep and body mass index (BMI) data available (n = 24,337) in analyses (samples with BMI <5th percentile excluded). Parents reported children's sleep duration and regularity. High BMI (≥85th percentile) for age defines overweight/obesity. We selected SDOH (race/ethnicity, family income, primary caregiver education and neighbourhood condition) and covariates (age, sex, smoking, exercise and depression) using a hierarchical model-building approach. Accounting for complex survey design, logistic regression estimated the interaction between sleep and SDOH.


RESULTS
There were significant interactions between sleep duration and SDOH. The association between increasing sleep and decreasing odds of overweight/obesity only showed in the following subgroups: White, family income ≥400% federal poverty level (FPL) or primary caregiver' education ≥ high school. Compared with these subgroups, Hispanic adolescents and adolescents whose family income was below 100% FPL and whose caregiver education was below high school had weakened and reversed associations. Sleep regularity was not associated with overweight/obesity.


CONCLUSIONS
Increasing sleep duration was associated with a decreased risk of overweight/obesity, but the association was not present in adolescents from racial/ethnic minority groups (i.e. Hispanic) and those with low SES.


IMPACT
The study findings suggest that associations between sleep and overweight/obesity vary by race and SES. Identification of additional mechanisms for obesity is needed for racial/ethnic minority groups and those from families with low SES. Also, the complexity of these relationships underscores the importance of community-based needs assessment in the design of targeted and meaningful interventions to address complex health conditions such as poor sleep and obesity.
ER  - 

BibTeX

@misc{ffddf782752df1369fd99d4d92c1f62ebface79c,
  author = {J. Broussard and S. Klein},
  title = {Insufficient sleep and obesity: cause or consequence},
  journal = {Obesity (Silver Spring, Md.)},
  year = {2022},
  volume = {30},
  pages = {1914 - 1916},
  doi = {10.1002/oby.23539},
  abstract = {Sleep is an ancient and evolutionarily conserved biological process that, when disturbed, increases the risk for a variety of diseases in people, including diabetes, cardiovascular disease, kidney disease, and cancer. Although results from epidemiological studies support the link between insufficient sleep and an increased risk of obesity, the directionality of this link is unknown. Results from short‐term controlled clinical studies, conducted almost exclusively in people who are normal weight, demonstrate that sleep restriction increases hunger, appetite, energy intake, and body weight. However, the authors are not aware of any studies that have evaluated the effect of more than 3 weeks of experimental sleep restriction on obesity risk factors, and few studies have been conducted in people with preexisting obesity. This Perspective reviews the link between insufficient sleep and obesity risk and the potential therapeutic effects of sleep extension in people with chronic sleep insufficiency.}
}

RIS

TY  - JOUR
AU  - J. Broussard
AU  - S. Klein
T1  - Insufficient sleep and obesity: cause or consequence
JO  - Obesity (Silver Spring, Md.)
PY  - 2022
VL  - 30
SP  - 1914 - 1916
EP  - 1914 - 1916
DO  - 10.1002/oby.23539
AB  - Sleep is an ancient and evolutionarily conserved biological process that, when disturbed, increases the risk for a variety of diseases in people, including diabetes, cardiovascular disease, kidney disease, and cancer. Although results from epidemiological studies support the link between insufficient sleep and an increased risk of obesity, the directionality of this link is unknown. Results from short‐term controlled clinical studies, conducted almost exclusively in people who are normal weight, demonstrate that sleep restriction increases hunger, appetite, energy intake, and body weight. However, the authors are not aware of any studies that have evaluated the effect of more than 3 weeks of experimental sleep restriction on obesity risk factors, and few studies have been conducted in people with preexisting obesity. This Perspective reviews the link between insufficient sleep and obesity risk and the potential therapeutic effects of sleep extension in people with chronic sleep insufficiency.
ER  - 

BibTeX

@misc{8021ae9695dd21cb08d0e58198baee8e5513b112,
  author = {P. L. Capers and Aaron D. Fobian and K. Kaiser and Rohit Borah and David B. Allison},
  title = {A systematic review and meta‐analysis of randomized controlled trials of the impact of sleep duration on adiposity and components of energy balance},
  journal = {Obesity Reviews},
  year = {2015},
  volume = {16},
  doi = {10.1111/obr.12296},
  abstract = {Recent epidemiological and ecological trends in humans indicate a possible causal relationship between sleep duration and energy balance. We aimed to find experimental evidence that has tested this relationship between sleep duration and measures of body composition, food intake or biomarkers related to food intake. We conducted a systematic literature review using six databases throughout 7 August 2014. We sought reports of randomized controlled trials where sleep duration was manipulated and measured outcomes were body weight or other body composition metrics, food intake, and/or biomarkers related to eating. We found 18 unique studies meeting all criteria: eight studies with an outcome of body weight (4 – increased sleep, 4 – reduced sleep); four studies on food intake; four studies of sleep restriction on total energy expenditure and three of respiratory quotient; and four studies on leptin and/or ghrelin. Few controlled experimental studies have addressed the question of the effect of sleep on body weight/composition and eating. The available experimental literature suggests that sleep restriction increases food intake and total energy expenditure with inconsistent effects on integrated energy balance as operationalized by weight change. Future controlled trials that examine the impact of increased sleep on body weight/energy balance factors are warranted.}
}

RIS

TY  - JOUR
AU  - P. L. Capers
AU  - Aaron D. Fobian
AU  - K. Kaiser
AU  - Rohit Borah
AU  - David B. Allison
T1  - A systematic review and meta‐analysis of randomized controlled trials of the impact of sleep duration on adiposity and components of energy balance
JO  - Obesity Reviews
PY  - 2015
VL  - 16
DO  - 10.1111/obr.12296
AB  - Recent epidemiological and ecological trends in humans indicate a possible causal relationship between sleep duration and energy balance. We aimed to find experimental evidence that has tested this relationship between sleep duration and measures of body composition, food intake or biomarkers related to food intake. We conducted a systematic literature review using six databases throughout 7 August 2014. We sought reports of randomized controlled trials where sleep duration was manipulated and measured outcomes were body weight or other body composition metrics, food intake, and/or biomarkers related to eating. We found 18 unique studies meeting all criteria: eight studies with an outcome of body weight (4 – increased sleep, 4 – reduced sleep); four studies on food intake; four studies of sleep restriction on total energy expenditure and three of respiratory quotient; and four studies on leptin and/or ghrelin. Few controlled experimental studies have addressed the question of the effect of sleep on body weight/composition and eating. The available experimental literature suggests that sleep restriction increases food intake and total energy expenditure with inconsistent effects on integrated energy balance as operationalized by weight change. Future controlled trials that examine the impact of increased sleep on body weight/energy balance factors are warranted.
ER  - 

BibTeX

@misc{a160b27126e227341c51e557f6b3c96a3f41a6ba,
  author = {Ji-Eun Lee},
  title = {Sleep and Obesity in Childhood},
  journal = {Journal of Obesity & Metabolic Syndrome},
  year = {2017},
  volume = {26},
  pages = {84 - 85},
  doi = {10.7570/JOMES.2017.26.2.84},
  abstract = {Sleep deprivation in adults has been recognized as one of the key risk factors for the development of complications related to obesity and metabolic syndrome. Indeed, numerous observational studies have demonstrated an inverse association between the duration of sleep time and obesity in adulthood.1,2 Many studies have indicated that childhood obesity is also related to short sleep duration3, although there have been a number of dissenting reports in this regard.4 However, most systematic reviews have suggested that there is a relationship between short sleep time and the high prevalence of obesity in childhood.5,6 For instance, a large meta-analysis of 11 longitudinal studies showed that the risk of obesity in a pediatric group was 2.15 times higher in children with shortened sleep duration compared to children without short sleep duration.7 Appropriate sleep in childhood is known to have a lasting beneficial impact into adulthood, although the role of sleep during childhood is quite complicated. This is due to the various changes in sleep time duration and sleep behavior associated with both changes in growth and changes in lifestyle during childhood; thus, short sleep durations are predicted to affect different lifecycle stages in different ways. The adolescent period is associated with a higher risk of obesity compared to the pediatric period. Further, obese adolescents with short sleep durations are more likely to be associated with biological and lifestyle-related changes.6 Sleep curtailment can also lead to reduced physical activity, daytime sleepiness, and increased food intake.8 Sleep deprivation in childhood may therefore be considered to be a significant predictor of obesity and behavioral sleep interventions can provide one way to reduce the risk of obesity. In Korea, 75.3% of adolescents have reported experiencing sleep deprivation, and the rate of sleep curtailment has been shown to gradually increase with age.9 The prevalence of overweight risk has been reported to be higher in a group that had less than eight hours of sleep compared to a group who had more than eight hours of sleep.10 Energy homeostasis is the result of a combination of biological factors (i.e., circadian rhythms and sleep-wake homeostasis) and environmental factors (i.e., diet and physical activity). In the past few years, numerous studies have been conducted on the relationship between sleep-wake cycle regulation and metabolic function11 and recent studies have also been conducted on the sleep dimensions associated with childhood obesity. Beyond sleep duration, sleeping timing patterns can also serve to increase obesity risk.12 Although shifted sleep schedules and evening chronotypes have recently been found to be linked to adiposity in adults, related studies in children are remain rare. One study found that Korean middle school students who experienced socalled “social jet lag” tended to be more obese than elementary}
}

RIS

TY  - JOUR
AU  - Ji-Eun Lee
T1  - Sleep and Obesity in Childhood
JO  - Journal of Obesity & Metabolic Syndrome
PY  - 2017
VL  - 26
SP  - 84 - 85
EP  - 84 - 85
DO  - 10.7570/JOMES.2017.26.2.84
AB  - Sleep deprivation in adults has been recognized as one of the key risk factors for the development of complications related to obesity and metabolic syndrome. Indeed, numerous observational studies have demonstrated an inverse association between the duration of sleep time and obesity in adulthood.1,2 Many studies have indicated that childhood obesity is also related to short sleep duration3, although there have been a number of dissenting reports in this regard.4 However, most systematic reviews have suggested that there is a relationship between short sleep time and the high prevalence of obesity in childhood.5,6 For instance, a large meta-analysis of 11 longitudinal studies showed that the risk of obesity in a pediatric group was 2.15 times higher in children with shortened sleep duration compared to children without short sleep duration.7 Appropriate sleep in childhood is known to have a lasting beneficial impact into adulthood, although the role of sleep during childhood is quite complicated. This is due to the various changes in sleep time duration and sleep behavior associated with both changes in growth and changes in lifestyle during childhood; thus, short sleep durations are predicted to affect different lifecycle stages in different ways. The adolescent period is associated with a higher risk of obesity compared to the pediatric period. Further, obese adolescents with short sleep durations are more likely to be associated with biological and lifestyle-related changes.6 Sleep curtailment can also lead to reduced physical activity, daytime sleepiness, and increased food intake.8 Sleep deprivation in childhood may therefore be considered to be a significant predictor of obesity and behavioral sleep interventions can provide one way to reduce the risk of obesity. In Korea, 75.3% of adolescents have reported experiencing sleep deprivation, and the rate of sleep curtailment has been shown to gradually increase with age.9 The prevalence of overweight risk has been reported to be higher in a group that had less than eight hours of sleep compared to a group who had more than eight hours of sleep.10 Energy homeostasis is the result of a combination of biological factors (i.e., circadian rhythms and sleep-wake homeostasis) and environmental factors (i.e., diet and physical activity). In the past few years, numerous studies have been conducted on the relationship between sleep-wake cycle regulation and metabolic function11 and recent studies have also been conducted on the sleep dimensions associated with childhood obesity. Beyond sleep duration, sleeping timing patterns can also serve to increase obesity risk.12 Although shifted sleep schedules and evening chronotypes have recently been found to be linked to adiposity in adults, related studies in children are remain rare. One study found that Korean middle school students who experienced socalled “social jet lag” tended to be more obese than elementary
ER  - 

BibTeX

@misc{2df2837d58ec2e468fbe4c03072e8b5fefef1437,
  author = {L. Brondel and D. Davenne},
  title = {Partial sleep deprivation and food intake in men},
  journal = {},
  year = {2013},
  volume = {},
  pages = {90-103},
  doi = {10.3920/978-90-8686-763-9_6},
  abstract = {Does lack of sleep lead to obesity? Many studies show that in Western countries the duration of sleep has decreased in recent years. Epidemiological approaches have shown a link between short sleep duration and increased body mass index, but these studies did not identify that this link is causal and which mechanisms are involved. Experimental studies may help in that matter. Some of the metabolic effects of sleep deprivation in humans and in animals are reviewed. Then the results of a recent paper are given, which show that food intake is increased by acute sleep deprivation in humans and discussed in regards with the different hypothesis about the mechanisms involved. Finally, the increase of food intake during restricted sleep might be an archaic phylogenetic phenomenon which is still present and active in humans. This phenomenon could be the same as the seasonal variations in sleep and food intake according to the length of day and night in animals and could be mediated by melatonin secretion.}
}

RIS

TY  - JOUR
AU  - L. Brondel
AU  - D. Davenne
T1  - Partial sleep deprivation and food intake in men
PY  - 2013
SP  - 90-103
EP  - 90-103
DO  - 10.3920/978-90-8686-763-9_6
AB  - Does lack of sleep lead to obesity? Many studies show that in Western countries the duration of sleep has decreased in recent years. Epidemiological approaches have shown a link between short sleep duration and increased body mass index, but these studies did not identify that this link is causal and which mechanisms are involved. Experimental studies may help in that matter. Some of the metabolic effects of sleep deprivation in humans and in animals are reviewed. Then the results of a recent paper are given, which show that food intake is increased by acute sleep deprivation in humans and discussed in regards with the different hypothesis about the mechanisms involved. Finally, the increase of food intake during restricted sleep might be an archaic phylogenetic phenomenon which is still present and active in humans. This phenomenon could be the same as the seasonal variations in sleep and food intake according to the length of day and night in animals and could be mediated by melatonin secretion.
ER  - 

BibTeX

@misc{f3903301f267e4840e7c4dfe8229d1bab346abaa,
  author = {Peter Prayogo Hsieh and Florencia Christina Sindhu and Made Ratna Dewi and Gusti Lanang Sidiartha},
  title = {Hubungan antara durasi tidur dengan kejadian obesitas pada anak: sebuah tinjauan sistematis},
  journal = {Intisari Sains Medis},
  year = {2023},
  doi = {10.15562/ism.v14i1.1303},
  abstract = {Background: Obesity in children is a serious health problem with a rapidly increasing global prevalence. Children with obesity are at high risk of developing several metabolic diseases as adults. Studies show that short sleep duration is a risk factor for obesity. Short sleep duration will affect the diurnal rhythm and disrupt homeostasis and energy metabolism. This systematic review aims to determine the relationship between sleep duration and obesity in children.
Methods: A systematic literature review was carried out by literature searching on PubMed and Google Scholar using the PRISMA diagram and based on the eligibility criteria in selecting literature. The study was limited to the last five years (2017-2021). Studies that met the criteria were included in the analysis.
Results: There were 17 studies included in the analysis with cross sectional studies, prospective cohorts and clinical trials study designs regarding the relationship between sleep duration and obesity in children. All studies show a strong association between sleep duration and obesity in children and adolescents. The underlying mechanism is short sleep duration affects the balance of hormones controlling hunger and appetite so that appetite increases and causes excess energy intake, resulting in obesity. Children with short sleep duration are 2-4 times more prone to obesity compared to children with normal sleep duration.
Conclusion: There is a significant relationship between sleep duration and obesity in children and adolescents. Adequate sleep duration is recommended to prevent obesity and the risk of metabolic and cardiovascular diseases.}
}

RIS

TY  - JOUR
AU  - Peter Prayogo Hsieh
AU  - Florencia Christina Sindhu
AU  - Made Ratna Dewi
AU  - Gusti Lanang Sidiartha
T1  - Hubungan antara durasi tidur dengan kejadian obesitas pada anak: sebuah tinjauan sistematis
JO  - Intisari Sains Medis
PY  - 2023
DO  - 10.15562/ism.v14i1.1303
AB  - Background: Obesity in children is a serious health problem with a rapidly increasing global prevalence. Children with obesity are at high risk of developing several metabolic diseases as adults. Studies show that short sleep duration is a risk factor for obesity. Short sleep duration will affect the diurnal rhythm and disrupt homeostasis and energy metabolism. This systematic review aims to determine the relationship between sleep duration and obesity in children.
Methods: A systematic literature review was carried out by literature searching on PubMed and Google Scholar using the PRISMA diagram and based on the eligibility criteria in selecting literature. The study was limited to the last five years (2017-2021). Studies that met the criteria were included in the analysis.
Results: There were 17 studies included in the analysis with cross sectional studies, prospective cohorts and clinical trials study designs regarding the relationship between sleep duration and obesity in children. All studies show a strong association between sleep duration and obesity in children and adolescents. The underlying mechanism is short sleep duration affects the balance of hormones controlling hunger and appetite so that appetite increases and causes excess energy intake, resulting in obesity. Children with short sleep duration are 2-4 times more prone to obesity compared to children with normal sleep duration.
Conclusion: There is a significant relationship between sleep duration and obesity in children and adolescents. Adequate sleep duration is recommended to prevent obesity and the risk of metabolic and cardiovascular diseases.
ER  - 

BibTeX

@misc{0af301b3a7cef99ff1aac356183192e58d397ecd,
  author = {A. Kwon and Youngha Choi and Sujin Kim and Kyungchul Song and Junghwan Suh and H. Chae and Ho-Seong Kim},
  title = {Characteristic Sleep Patterns and Associated Obesity in Adolescents},
  journal = {Life},
  year = {2022},
  volume = {12},
  doi = {10.3390/life12091316},
  abstract = {Poor sleep adversely affects health and may cause obesity. Poor sleep includes short sleep duration, low quality of sleep, and sleep discrepancy. Although most studies have focused on the association between sleep duration and obesity, poor sleep is a significant risk factor for obesity. Adolescents have characteristic sleep patterns which correspond to poor sleep. Adolescents sleep late due to various biological and psychosocial factors; also, they wake up early to be on time for school. This causes them to sleep less. To make up for this sleep debt, adolescents sleep more on non-school days, which causes sleep discrepancies. Therefore, since adolescents have characteristic sleep patterns, an in-depth investigation is needed to identify whether poor sleep is a risk for obesity. This article presents an overview of the characteristic sleep patterns of adolescents, and reviews studies on the association of each sleep pattern with obesity.}
}

RIS

TY  - JOUR
AU  - A. Kwon
AU  - Youngha Choi
AU  - Sujin Kim
AU  - Kyungchul Song
AU  - Junghwan Suh
AU  - H. Chae
AU  - Ho-Seong Kim
T1  - Characteristic Sleep Patterns and Associated Obesity in Adolescents
JO  - Life
PY  - 2022
VL  - 12
DO  - 10.3390/life12091316
AB  - Poor sleep adversely affects health and may cause obesity. Poor sleep includes short sleep duration, low quality of sleep, and sleep discrepancy. Although most studies have focused on the association between sleep duration and obesity, poor sleep is a significant risk factor for obesity. Adolescents have characteristic sleep patterns which correspond to poor sleep. Adolescents sleep late due to various biological and psychosocial factors; also, they wake up early to be on time for school. This causes them to sleep less. To make up for this sleep debt, adolescents sleep more on non-school days, which causes sleep discrepancies. Therefore, since adolescents have characteristic sleep patterns, an in-depth investigation is needed to identify whether poor sleep is a risk for obesity. This article presents an overview of the characteristic sleep patterns of adolescents, and reviews studies on the association of each sleep pattern with obesity.
ER  - 

BibTeX

@misc{5240e5964026c9cd8e593760e36bde7998bb259c,
  author = {J. Martínez-Gómez and J. Fernández-Alvira and A. de Cos-Gandoy and P. Bodega and M. de Miguel and A. Tresserra-Rimbau and Emily P. Laveriano-Santos and V. Carral and I. Carvajal and R. Estruch and R. Lamuela-Raventós and G. Santos-Beneit and V. Fuster and R. Fernández-Jiménez},
  title = {Sleep duration and its association with adiposity markers in adolescence: a cross-sectional and longitudinal study.},
  journal = {European journal of preventive cardiology},
  year = {2023},
  doi = {10.1093/eurjpc/zwad137},
  abstract = {BACKGROUND
Large studies linking adolescents' objectively measured sleep duration and adiposity markers are lacking.


AIM
To characterize sleep duration and its cross-sectional and longitudinal associations with adiposity markers in adolescence.


METHODS
Seven-day accelerometry was performed in a cohort of adolescents enrolled in the SI! Program for Secondary Schools trial in Spain at approximately age 12 (1216 adolescents, 49.6% girls), 14 (1026 adolescents, 51.3% girls), and 16 (872 adolescents, 51.7% girls) years. Participants were classified as very short sleepers (VSS; <7 h), short sleepers (SS; 7 to <8 h), or recommended-time sleepers (RTS; 8 to 10 h). Adjusted associations between sleep duration and adiposity markers were analyzed using generalized linear and Poisson models.


RESULTS
At ∼12 years, 33.7% of adolescents met sleep recommendations, and this percentage decreased with advancing age (22.6% at ∼14 and 18.7% at ∼16 years). Compared with RTS, overweight/obesity prevalence ratios (PR) at ∼12, 14, and 16 years among SS were 1.19 (95%CI 1.09-1.30), 1.41 (95%CI 1.34-1.48), and 0.99 (95%CI 0.77-1.26) and among VSS were 1.30 (95%CI 1.28-1.32), 1.93 (95%CI 1.41-2.64), and 1.32 (95%CI 1.26-1.37). Compared with adolescents who always met sleep recommendations, the prevalence of overweight/obesity was ∼5 times higher in those never meeting recommendations or meeting them only once. Similar trends were observed for waist-to-height ratio (p=0.010) and fat-mass index (p=0.024).


CONCLUSION
Most adolescents did not meet sleep recommendations. Shorter sleep duration was independently associated with unfavorable adiposity markers, and such adverse impact was cumulative. Health promotion programs should emphasize the importance of good sleep habits.}
}

RIS

TY  - JOUR
AU  - J. Martínez-Gómez
AU  - J. Fernández-Alvira
AU  - A. de Cos-Gandoy
AU  - P. Bodega
AU  - M. de Miguel
AU  - A. Tresserra-Rimbau
AU  - Emily P. Laveriano-Santos
AU  - V. Carral
AU  - I. Carvajal
AU  - R. Estruch
AU  - R. Lamuela-Raventós
AU  - G. Santos-Beneit
AU  - V. Fuster
AU  - R. Fernández-Jiménez
T1  - Sleep duration and its association with adiposity markers in adolescence: a cross-sectional and longitudinal study.
JO  - European journal of preventive cardiology
PY  - 2023
DO  - 10.1093/eurjpc/zwad137
AB  - BACKGROUND
Large studies linking adolescents' objectively measured sleep duration and adiposity markers are lacking.


AIM
To characterize sleep duration and its cross-sectional and longitudinal associations with adiposity markers in adolescence.


METHODS
Seven-day accelerometry was performed in a cohort of adolescents enrolled in the SI! Program for Secondary Schools trial in Spain at approximately age 12 (1216 adolescents, 49.6% girls), 14 (1026 adolescents, 51.3% girls), and 16 (872 adolescents, 51.7% girls) years. Participants were classified as very short sleepers (VSS; <7 h), short sleepers (SS; 7 to <8 h), or recommended-time sleepers (RTS; 8 to 10 h). Adjusted associations between sleep duration and adiposity markers were analyzed using generalized linear and Poisson models.


RESULTS
At ∼12 years, 33.7% of adolescents met sleep recommendations, and this percentage decreased with advancing age (22.6% at ∼14 and 18.7% at ∼16 years). Compared with RTS, overweight/obesity prevalence ratios (PR) at ∼12, 14, and 16 years among SS were 1.19 (95%CI 1.09-1.30), 1.41 (95%CI 1.34-1.48), and 0.99 (95%CI 0.77-1.26) and among VSS were 1.30 (95%CI 1.28-1.32), 1.93 (95%CI 1.41-2.64), and 1.32 (95%CI 1.26-1.37). Compared with adolescents who always met sleep recommendations, the prevalence of overweight/obesity was ∼5 times higher in those never meeting recommendations or meeting them only once. Similar trends were observed for waist-to-height ratio (p=0.010) and fat-mass index (p=0.024).


CONCLUSION
Most adolescents did not meet sleep recommendations. Shorter sleep duration was independently associated with unfavorable adiposity markers, and such adverse impact was cumulative. Health promotion programs should emphasize the importance of good sleep habits.
ER  - 

BibTeX

@misc{cd9408610f496850ed46fb5e14d5a447a7098faf,
  author = {Jonathan A. Mitchell and D. Rodriguez and K. Schmitz and J. Audrain-McGovern},
  title = {Sleep Duration and Adolescent Obesity},
  journal = {Pediatrics},
  year = {2013},
  volume = {131},
  pages = {e1428 - e1434},
  doi = {10.1542/peds.2012-2368},
  abstract = {OBJECTIVES: Short sleep has been associated with adolescent obesity. Most studies used a cross-sectional design and modeled BMI categories. We sought to determine if sleep duration was associated with BMI distribution changes from age 14 to 18. METHODS: Adolescents were recruited from suburban high schools in Philadelphia when entering ninth grade (n = 1390) and were followed-up every 6 months through 12th grade. Height and weight were self-reported, and BMIs were calculated (kg/m2). Hours of sleep were self-reported. Quantile regression was used to model the 10th, 25th, 50th, 75th, and 90th BMI percentiles as dependent variables; study wave and sleep were the main predictors. RESULTS: BMI increased from age 14 to 18, with the largest increase observed at the 90th BMI percentile. Each additional hour of sleep was associated with decreases in BMI at the 10th (–0.04; 95% confidence interval [CI]: –0.11, 0.03), 25th (–0.12; 95% CI: –0.20, –0.04), 50th (–0.15; 95% CI: –0.24, –0.06), 75th (–0.25; 95% CI: –0.38, –0.12), and 90th (–0.27; 95% CI: -0.45, -0.09) BMI percentiles. The strength of the association was stronger at the upper tail of the BMI distribution. Increasing sleep from 7.5 to 10.0 hours per day at age 18 predicted a reduction in the proportion of adolescents >25 kg/m2 by 4%. CONCLUSIONS: More sleep was associated with nonuniform changes in BMI distribution from age 14 to 18. Increasing sleep among adolescents, especially those in the upper half of the BMI distribution, may help prevent overweight and obesity.}
}

RIS

TY  - JOUR
AU  - Jonathan A. Mitchell
AU  - D. Rodriguez
AU  - K. Schmitz
AU  - J. Audrain-McGovern
T1  - Sleep Duration and Adolescent Obesity
JO  - Pediatrics
PY  - 2013
VL  - 131
SP  - e1428 - e1434
EP  - e1428 - e1434
DO  - 10.1542/peds.2012-2368
AB  - OBJECTIVES: Short sleep has been associated with adolescent obesity. Most studies used a cross-sectional design and modeled BMI categories. We sought to determine if sleep duration was associated with BMI distribution changes from age 14 to 18. METHODS: Adolescents were recruited from suburban high schools in Philadelphia when entering ninth grade (n = 1390) and were followed-up every 6 months through 12th grade. Height and weight were self-reported, and BMIs were calculated (kg/m2). Hours of sleep were self-reported. Quantile regression was used to model the 10th, 25th, 50th, 75th, and 90th BMI percentiles as dependent variables; study wave and sleep were the main predictors. RESULTS: BMI increased from age 14 to 18, with the largest increase observed at the 90th BMI percentile. Each additional hour of sleep was associated with decreases in BMI at the 10th (–0.04; 95% confidence interval [CI]: –0.11, 0.03), 25th (–0.12; 95% CI: –0.20, –0.04), 50th (–0.15; 95% CI: –0.24, –0.06), 75th (–0.25; 95% CI: –0.38, –0.12), and 90th (–0.27; 95% CI: -0.45, -0.09) BMI percentiles. The strength of the association was stronger at the upper tail of the BMI distribution. Increasing sleep from 7.5 to 10.0 hours per day at age 18 predicted a reduction in the proportion of adolescents >25 kg/m2 by 4%. CONCLUSIONS: More sleep was associated with nonuniform changes in BMI distribution from age 14 to 18. Increasing sleep among adolescents, especially those in the upper half of the BMI distribution, may help prevent overweight and obesity.
ER  - 

BibTeX

@misc{02b37d15633a2185cb094bef2e294e0d8c817b55,
  author = {Adámková and J. A. Hubacek and Lánská and M. Vrablik and I. KralovaLesna and P. Suchánek and P. Zimmelová and M. Velemínský},
  title = {Association between duration of the sleep and body weight.},
  journal = {Physiological research},
  year = {2009},
  volume = {58 Suppl 1},
  pages = {
          S27-31
        },
  abstract = {Some studies have suggested that there could be an association between the duration of sleep in humans and development of the obesity. We have analyzed the group of the probands (n = 3970, 2038 males and 1932 females, aged 18-65 years), with permanent address in the Central or South Bohemia. We ascertained the relationship between the duration of their sleep (obtained per questionnaire) and body mass index, weight, height, the value of systolic and diastolic blood pressure, heart rate, waist and hip circumference, the values of total-, high density- and low density- cholesterol, thyroid hormone and body exercise performed. The optimal values of the body mass index (and optimal body weight) were associated with the duration of sleep 7 hours per night (P < 0.001). This association was found both in males and females and in both districts. Other anthropometrical and biochemical parameters were not associated with the sleep duration.}
}

RIS

TY  - JOUR
AU  - Adámková
AU  - J. A. Hubacek
AU  - Lánská
AU  - M. Vrablik
AU  - I. KralovaLesna
AU  - P. Suchánek
AU  - P. Zimmelová
AU  - M. Velemínský
T1  - Association between duration of the sleep and body weight.
JO  - Physiological research
PY  - 2009
VL  - 58 Suppl 1
SP  - 
          S27-31
        
EP  - 
          S27-31
        
AB  - Some studies have suggested that there could be an association between the duration of sleep in humans and development of the obesity. We have analyzed the group of the probands (n = 3970, 2038 males and 1932 females, aged 18-65 years), with permanent address in the Central or South Bohemia. We ascertained the relationship between the duration of their sleep (obtained per questionnaire) and body mass index, weight, height, the value of systolic and diastolic blood pressure, heart rate, waist and hip circumference, the values of total-, high density- and low density- cholesterol, thyroid hormone and body exercise performed. The optimal values of the body mass index (and optimal body weight) were associated with the duration of sleep 7 hours per night (P < 0.001). This association was found both in males and females and in both districts. Other anthropometrical and biochemical parameters were not associated with the sleep duration.
ER  - 

BibTeX

@misc{ca0c7be5884b7f600ed041bd6d739c5a7a607b69,
  author = {J. Borja and L. Adair},
  title = {Sleep duration and risk of obesity among Filipino women},
  journal = {The FASEB Journal},
  year = {2010},
  volume = {24},
  doi = {10.1096/FASEBJ.24.1_SUPPLEMENT.213.1},
  abstract = {Recent studies have shown a negative association between sleep duration and obesity. We examined this relationship using data on 2002 non‐pregnant women aged 35–68 from the Cebu Longitudinal Health and Nutrition Survey, a community‐based prospective study in Metro Cebu, Philippines. Sleep duration (total hours sleep) was calculated from the women's 24‐hr activity diaries (reported for a typical weekday). Obesity measures include body mass index (BMI; in kg/m2)) and waist circumference (in cm). Linear regression models adjusted for age, energy intake, parity, activity levels, and other confounders show a negative association between sleep duration and BMI [beta (95% CI) −0.26 (−0.38 to −0.14)] and waist circumference[beta (95% CI) −0.57 (−0.88 to −0.27)]. Getting more than 6 hours of sleep protected against the risk of overweight (BMI=>25) [Odds ratio (95% CI) −0.68 (0.55 to 0.84)]. These findings are consistent with those of other studies, and provide valuable insights particularly for older women given that 39% of these women are aged >=50. Among older adults, this sleep‐obesity association gains relevance as sleep duration gets shorter and losing weight becomes more difficult with age.}
}

RIS

TY  - JOUR
AU  - J. Borja
AU  - L. Adair
T1  - Sleep duration and risk of obesity among Filipino women
JO  - The FASEB Journal
PY  - 2010
VL  - 24
DO  - 10.1096/FASEBJ.24.1_SUPPLEMENT.213.1
AB  - Recent studies have shown a negative association between sleep duration and obesity. We examined this relationship using data on 2002 non‐pregnant women aged 35–68 from the Cebu Longitudinal Health and Nutrition Survey, a community‐based prospective study in Metro Cebu, Philippines. Sleep duration (total hours sleep) was calculated from the women's 24‐hr activity diaries (reported for a typical weekday). Obesity measures include body mass index (BMI; in kg/m2)) and waist circumference (in cm). Linear regression models adjusted for age, energy intake, parity, activity levels, and other confounders show a negative association between sleep duration and BMI [beta (95% CI) −0.26 (−0.38 to −0.14)] and waist circumference[beta (95% CI) −0.57 (−0.88 to −0.27)]. Getting more than 6 hours of sleep protected against the risk of overweight (BMI=>25) [Odds ratio (95% CI) −0.68 (0.55 to 0.84)]. These findings are consistent with those of other studies, and provide valuable insights particularly for older women given that 39% of these women are aged >=50. Among older adults, this sleep‐obesity association gains relevance as sleep duration gets shorter and losing weight becomes more difficult with age.
ER  - 

BibTeX

@misc{ff731e89e09a9f98b800005e80ecc31df6b0ef6b,
  author = {O. Bayer and A. S. Rosario and M. Wabitsch and R. von Kries},
  title = {Sleep duration and obesity in children: is the association dependent on age and choice of the outcome parameter?},
  journal = {Sleep},
  year = {2009},
  volume = {32 9},
  pages = {
          1183-9
        },
  doi = {10.1093/SLEEP/32.9.1183},
  abstract = {STUDY OBJECTIVES
To assess the association between sleep duration in children and different markers of body fat by age and weight status.


DESIGN
Nation-wide health survey. Measurement of BMI and body fat percentage (KFA) calculated from weight, height, skin fold thickness, age, and sex. Sleep duration and potential confounding variables were assessed in a parent questionnaire.


SETTING
N/A.


PARTICIPANTS
7767 German resident children from 3 to 10 years of age.


INTERVENTIONS
N/A.


MEASUREMENTS AND RESULTS
Prolongation of sleep duration from the lowest to the highest percentile accounted for a similar mean decrease founding variables and did not show a systematic age dependency. The greatest effects of sleep duration were seen for the upper tails of the BMI and KFA distributions, which were about four as high as the lower tails.


CONCLUSIONS
The association between sleep duration and weight status is of similar size through ages 3 to 10 years. The sleep-associated changes in BMI are likely to be a consequence of higher body fat and primarily affect children whose BMI or KFA is already elevated. These findings favor hormonal pathways nurturing adipose tissue playing a key role in the underlying physiological mechanisms.}
}

RIS

TY  - JOUR
AU  - O. Bayer
AU  - A. S. Rosario
AU  - M. Wabitsch
AU  - R. von Kries
T1  - Sleep duration and obesity in children: is the association dependent on age and choice of the outcome parameter?
JO  - Sleep
PY  - 2009
VL  - 32 9
SP  - 
          1183-9
        
EP  - 
          1183-9
        
DO  - 10.1093/SLEEP/32.9.1183
AB  - STUDY OBJECTIVES
To assess the association between sleep duration in children and different markers of body fat by age and weight status.


DESIGN
Nation-wide health survey. Measurement of BMI and body fat percentage (KFA) calculated from weight, height, skin fold thickness, age, and sex. Sleep duration and potential confounding variables were assessed in a parent questionnaire.


SETTING
N/A.


PARTICIPANTS
7767 German resident children from 3 to 10 years of age.


INTERVENTIONS
N/A.


MEASUREMENTS AND RESULTS
Prolongation of sleep duration from the lowest to the highest percentile accounted for a similar mean decrease founding variables and did not show a systematic age dependency. The greatest effects of sleep duration were seen for the upper tails of the BMI and KFA distributions, which were about four as high as the lower tails.


CONCLUSIONS
The association between sleep duration and weight status is of similar size through ages 3 to 10 years. The sleep-associated changes in BMI are likely to be a consequence of higher body fat and primarily affect children whose BMI or KFA is already elevated. These findings favor hormonal pathways nurturing adipose tissue playing a key role in the underlying physiological mechanisms.
ER  - 

BibTeX

@misc{5ab9a763d84cb727ecd8b699d95f9b034d90c035,
  author = {A. Kwon and Sujin Kim and Youngha Choi and Ha Yan Kim and Myeongjee Lee and M. Lee and H. I. Lee and Kyungchul Song and Junghwan Suh and H. Chae and H. Kim},
  title = {Effects of Early Wake-Up Time on Obesity in Adolescents.},
  journal = {Childhood obesity},
  year = {2023},
  doi = {10.1089/chi.2023.0016},
  abstract = {Background: Although numerous studies have reported that obesity in adolescents is related to shorter sleep duration, few studies have reported the effect of sleep timing, particularly early wake-up time, on obesity. Objectives: To investigate the association between wake-up time and adolescent obesity. Methods: Using the Korean National Health and Nutrition Examination Survey VII data, 1301 middle school and high school students were selected and grouped according to BMI. Sleep timing and lifestyle factors were evaluated using self-reported questionnaires. Results: The mean bedtime and wake-up time were 00:09 am and 07:06 am, respectively. Despite similar bedtimes, the group with overweight/obesity woke up earlier than the group with underweight/normal weight. The BMI z-score and the overweight/obesity relative risk decreased as the wake-up time was delayed, even after adjustment for covariates. Participants who woke up before 06:50 am had a 1.82-fold higher risk of having overweight/obesity than those who woke up after 07:30 am. Participants who woke up late tended to sleep longer than those who woke up early. Conclusions: Waking up early is significantly associated with an increased BMI z-score in adolescents and may be a risk factor for overweight/obesity.}
}

RIS

TY  - JOUR
AU  - A. Kwon
AU  - Sujin Kim
AU  - Youngha Choi
AU  - Ha Yan Kim
AU  - Myeongjee Lee
AU  - M. Lee
AU  - H. I. Lee
AU  - Kyungchul Song
AU  - Junghwan Suh
AU  - H. Chae
AU  - H. Kim
T1  - Effects of Early Wake-Up Time on Obesity in Adolescents.
JO  - Childhood obesity
PY  - 2023
DO  - 10.1089/chi.2023.0016
AB  - Background: Although numerous studies have reported that obesity in adolescents is related to shorter sleep duration, few studies have reported the effect of sleep timing, particularly early wake-up time, on obesity. Objectives: To investigate the association between wake-up time and adolescent obesity. Methods: Using the Korean National Health and Nutrition Examination Survey VII data, 1301 middle school and high school students were selected and grouped according to BMI. Sleep timing and lifestyle factors were evaluated using self-reported questionnaires. Results: The mean bedtime and wake-up time were 00:09 am and 07:06 am, respectively. Despite similar bedtimes, the group with overweight/obesity woke up earlier than the group with underweight/normal weight. The BMI z-score and the overweight/obesity relative risk decreased as the wake-up time was delayed, even after adjustment for covariates. Participants who woke up before 06:50 am had a 1.82-fold higher risk of having overweight/obesity than those who woke up after 07:30 am. Participants who woke up late tended to sleep longer than those who woke up early. Conclusions: Waking up early is significantly associated with an increased BMI z-score in adolescents and may be a risk factor for overweight/obesity.
ER  - 

BibTeX

@misc{23d3795df0d141f15edbf798cf92148d3bd07a6d,
  author = {Sofía I Mercado-Gonzales and Antonella N Carpio-Rodríguez and R. Carrillo-Larco and A. Bernabé-Ortiz},
  title = {Sleep Duration and Risk of Obesity by Sex: Nine-Year Follow-Up of the Young Lives Study in Peru.},
  journal = {Childhood obesity},
  year = {2019},
  volume = {15 4},
  pages = {
          237-243
        },
  doi = {10.1089/chi.2018.0247},
  abstract = {Background: We aimed to evaluate if there is association between hours of sleep and the risk of obesity among children and whether this association differs by sex. Methods: A secondary data analysis, using information of the Young Lives study, was conducted. The outcome was obesity, based on the BMI for age z-score; the exposure was child's sleep duration (reported by parents) categorized using the National Sleep Foundation guidelines, and as a numerical variable. Baseline and three follow-ups information were used to evaluate association, reporting relative risks (RRs), and 95% confidence intervals (CIs), as well as coefficients and 95% CI. Results: Data from 1949 children, baseline mean age 4.3 (standard deviation: 0.3) and 962 (49.5%) females, were analyzed. Short sleep duration was present in 26.0% (95% CI: 24.0-28.0) at baseline. After 9.6 years of follow-up, the incidence of obesity was 0.83 (95% CI: 0.70-0.98) per 100 person-years at risk. In multivariable model (n = 1579), there was no association between short sleep duration and obesity in the whole sample (p = 0.13); but the risk of obesity was lower among girls (n = 816; RR = 0.45; 95% CI: 0.21-0.96; p = 0.03) compared with boys (n = 763; RR = 1.43; 95% CI: 0.95-2.14; p = 0.09). On the contrary, each additional hour of sleep was associated with an increase of boy's BMI mean (0.05; 95% CI: 0.02-0.08; p < 0.001), but not among girls (-0.02; 95% CI: -0.05 to 0.01; p = 0.11). Conclusions: Our results evidenced a lower risk of obesity due to short sleep duration in girls, but not in boys. Each additional hour of sleep was associated with an increase of BMI in boys, but not in girls. Strategies are needed to guarantee adequate sleep duration in Peruvian children.}
}

RIS

TY  - JOUR
AU  - Sofía I Mercado-Gonzales
AU  - Antonella N Carpio-Rodríguez
AU  - R. Carrillo-Larco
AU  - A. Bernabé-Ortiz
T1  - Sleep Duration and Risk of Obesity by Sex: Nine-Year Follow-Up of the Young Lives Study in Peru.
JO  - Childhood obesity
PY  - 2019
VL  - 15 4
SP  - 
          237-243
        
EP  - 
          237-243
        
DO  - 10.1089/chi.2018.0247
AB  - Background: We aimed to evaluate if there is association between hours of sleep and the risk of obesity among children and whether this association differs by sex. Methods: A secondary data analysis, using information of the Young Lives study, was conducted. The outcome was obesity, based on the BMI for age z-score; the exposure was child's sleep duration (reported by parents) categorized using the National Sleep Foundation guidelines, and as a numerical variable. Baseline and three follow-ups information were used to evaluate association, reporting relative risks (RRs), and 95% confidence intervals (CIs), as well as coefficients and 95% CI. Results: Data from 1949 children, baseline mean age 4.3 (standard deviation: 0.3) and 962 (49.5%) females, were analyzed. Short sleep duration was present in 26.0% (95% CI: 24.0-28.0) at baseline. After 9.6 years of follow-up, the incidence of obesity was 0.83 (95% CI: 0.70-0.98) per 100 person-years at risk. In multivariable model (n = 1579), there was no association between short sleep duration and obesity in the whole sample (p = 0.13); but the risk of obesity was lower among girls (n = 816; RR = 0.45; 95% CI: 0.21-0.96; p = 0.03) compared with boys (n = 763; RR = 1.43; 95% CI: 0.95-2.14; p = 0.09). On the contrary, each additional hour of sleep was associated with an increase of boy's BMI mean (0.05; 95% CI: 0.02-0.08; p < 0.001), but not among girls (-0.02; 95% CI: -0.05 to 0.01; p = 0.11). Conclusions: Our results evidenced a lower risk of obesity due to short sleep duration in girls, but not in boys. Each additional hour of sleep was associated with an increase of BMI in boys, but not in girls. Strategies are needed to guarantee adequate sleep duration in Peruvian children.
ER  - 

BibTeX

@misc{8692d9cf26926dcbbfd76d8c9be173890739a0a5,
  author = {Gregory D M Potter and J. Cade and L. Hardie},
  title = {Longer sleep is associated with lower BMI and favorable metabolic profiles in UK adults: Findings from the National Diet and Nutrition Survey},
  journal = {PLoS ONE},
  year = {2017},
  volume = {12},
  doi = {10.1371/journal.pone.0182195},
  abstract = {Ever more evidence associates short sleep with increased risk of metabolic diseases such as obesity, which may be related to a predisposition to non-homeostatic eating. Few studies have concurrently determined associations between sleep duration and objective measures of metabolic health as well as sleep duration and diet, however. We therefore analyzed associations between sleep duration, diet and metabolic health markers in UK adults, assessing associations between sleep duration and 1) adiposity, 2) selected metabolic health markers and 3) diet, using National Diet and Nutrition Survey data. Adults (n = 1,615, age 19–65 years, 57.1% female) completed questions about sleep duration and 3 to 4 days of food diaries. Blood pressure and waist circumference were recorded. Fasting blood lipids, glucose, glycated haemoglobin (HbA1c), thyroid hormones, and high-sensitivity C-reactive protein (CRP) were measured in a subset of participants. We used regression analyses to explore associations between sleep duration and outcomes. After adjustment for age, ethnicity, sex, smoking, and socioeconomic status, sleep duration was negatively associated with body mass index (-0.46 kg/m2 per hour, 95% CI -0.69 to -0.24 kg/m2, p < 0.001) and waist circumference (-0.9 cm per hour, 95% CI -1.5 to -0.3cm, p = 0.004), and positively associated with high-density lipoprotein cholesterol (0.03 mmol/L per hour, 95% CI 0.00 to 0.05, p = 0.03). Sleep duration tended to be positively associated with free thyroxine levels and negatively associated with HbA1c and CRP (p = 0.09 to 0.10). Contrary to our hypothesis, sleep duration was not associated with any dietary measures (p ≥ 0.14). Together, our findings show that short-sleeping UK adults are more likely to have obesity, a disease with many comorbidities.}
}

RIS

TY  - JOUR
AU  - Gregory D M Potter
AU  - J. Cade
AU  - L. Hardie
T1  - Longer sleep is associated with lower BMI and favorable metabolic profiles in UK adults: Findings from the National Diet and Nutrition Survey
JO  - PLoS ONE
PY  - 2017
VL  - 12
DO  - 10.1371/journal.pone.0182195
AB  - Ever more evidence associates short sleep with increased risk of metabolic diseases such as obesity, which may be related to a predisposition to non-homeostatic eating. Few studies have concurrently determined associations between sleep duration and objective measures of metabolic health as well as sleep duration and diet, however. We therefore analyzed associations between sleep duration, diet and metabolic health markers in UK adults, assessing associations between sleep duration and 1) adiposity, 2) selected metabolic health markers and 3) diet, using National Diet and Nutrition Survey data. Adults (n = 1,615, age 19–65 years, 57.1% female) completed questions about sleep duration and 3 to 4 days of food diaries. Blood pressure and waist circumference were recorded. Fasting blood lipids, glucose, glycated haemoglobin (HbA1c), thyroid hormones, and high-sensitivity C-reactive protein (CRP) were measured in a subset of participants. We used regression analyses to explore associations between sleep duration and outcomes. After adjustment for age, ethnicity, sex, smoking, and socioeconomic status, sleep duration was negatively associated with body mass index (-0.46 kg/m2 per hour, 95% CI -0.69 to -0.24 kg/m2, p < 0.001) and waist circumference (-0.9 cm per hour, 95% CI -1.5 to -0.3cm, p = 0.004), and positively associated with high-density lipoprotein cholesterol (0.03 mmol/L per hour, 95% CI 0.00 to 0.05, p = 0.03). Sleep duration tended to be positively associated with free thyroxine levels and negatively associated with HbA1c and CRP (p = 0.09 to 0.10). Contrary to our hypothesis, sleep duration was not associated with any dietary measures (p ≥ 0.14). Together, our findings show that short-sleeping UK adults are more likely to have obesity, a disease with many comorbidities.
ER  - 

BibTeX

@misc{ed6df6bea2b8ffb8f8900ec33592e0ab5ea3ff7a,
  author = {M. St-Onge and A. Shechter},
  title = {Sleep restriction in adolescents: forging the path towards obesity and diabetes?},
  journal = {Sleep},
  year = {2013},
  volume = {36 6},
  pages = {
          813-4
        },
  doi = {10.5665/sleep.2694},
  abstract = {813 Editorial—St-Onge and Shechter There is mounting epidemiological data linking short sleep duration and obesity in children and adults.1-4 Although not defining cause, these studies have raised questions that are beginning to be answered, albeit mostly in adults. Moderate (2 h per night) and severe (up to 4 h per night) sleep restriction in adults have been found to affect both food intake and appetite. Our laboratory5 and others6,7 have reported that restricting sleep in normal sleepers increases food intake, mostly by increasing intake of snacks7 and fat.5 However, despite clearer associations between short sleep duration and obesity in the pediatric than the adult epidemiological literature,4 there is little clinical evidence for a causal effect in children. To our knowledge, only one intervention study has assessed the impact of sleep restriction on energy balance in adolescents.8 That study, conducted in 15to 19-year-old boys, tested the effects of 3 nights of 4-h (i.e., restricted sleep) compared to 9-h bedtimes (i.e., longer sleep) on energy expenditure and energy intake. The results showed greater energy expenditure and lower energy intake at a test meal during restricted sleep relative to healthy sleep, contrary to the authors’ hypotheses. Although this was a well-designed study, it may have failed to provide accurate information on the impact of sleep restriction on energy balance due to the limited extent of the sleep restriction, lack of ad libitum access to food and physical activity data, and restriction of assessments to only adolescent males. In this issue of SLEEP, Beebe and colleagues9 are to be commended for providing much needed information on the impact of sleep restriction, to an extent similar to what happens on school nights, in both adolescent boys and girls. They conducted a study with an innovative design in an attempt to reproduce real-life situations: teens spent 6.5 or 10 h time in bed (TIB) Monday through Friday, with free sleep times on weekend nights, in a crossover design. During the 5 controlled sleep nights, wake-up times were held constant between sleep conditions. This resulted in 6.3 h and 8.9 h of sleep under restricted sleep and unrestricted sleep, respectively, similar to the self-reported sleep times of 9.3 h on non-school nights and 7.1 h on school nights. In the restricted sleep condition, food intake, assessed by a single 24-h recall, tended to be higher than in the 10-h TIB sleep condition, which we speculate was accounted for by a greater intake of sweets and desserts. EDITORIAL}
}

RIS

TY  - JOUR
AU  - M. St-Onge
AU  - A. Shechter
T1  - Sleep restriction in adolescents: forging the path towards obesity and diabetes?
JO  - Sleep
PY  - 2013
VL  - 36 6
SP  - 
          813-4
        
EP  - 
          813-4
        
DO  - 10.5665/sleep.2694
AB  - 813 Editorial—St-Onge and Shechter There is mounting epidemiological data linking short sleep duration and obesity in children and adults.1-4 Although not defining cause, these studies have raised questions that are beginning to be answered, albeit mostly in adults. Moderate (2 h per night) and severe (up to 4 h per night) sleep restriction in adults have been found to affect both food intake and appetite. Our laboratory5 and others6,7 have reported that restricting sleep in normal sleepers increases food intake, mostly by increasing intake of snacks7 and fat.5 However, despite clearer associations between short sleep duration and obesity in the pediatric than the adult epidemiological literature,4 there is little clinical evidence for a causal effect in children. To our knowledge, only one intervention study has assessed the impact of sleep restriction on energy balance in adolescents.8 That study, conducted in 15to 19-year-old boys, tested the effects of 3 nights of 4-h (i.e., restricted sleep) compared to 9-h bedtimes (i.e., longer sleep) on energy expenditure and energy intake. The results showed greater energy expenditure and lower energy intake at a test meal during restricted sleep relative to healthy sleep, contrary to the authors’ hypotheses. Although this was a well-designed study, it may have failed to provide accurate information on the impact of sleep restriction on energy balance due to the limited extent of the sleep restriction, lack of ad libitum access to food and physical activity data, and restriction of assessments to only adolescent males. In this issue of SLEEP, Beebe and colleagues9 are to be commended for providing much needed information on the impact of sleep restriction, to an extent similar to what happens on school nights, in both adolescent boys and girls. They conducted a study with an innovative design in an attempt to reproduce real-life situations: teens spent 6.5 or 10 h time in bed (TIB) Monday through Friday, with free sleep times on weekend nights, in a crossover design. During the 5 controlled sleep nights, wake-up times were held constant between sleep conditions. This resulted in 6.3 h and 8.9 h of sleep under restricted sleep and unrestricted sleep, respectively, similar to the self-reported sleep times of 9.3 h on non-school nights and 7.1 h on school nights. In the restricted sleep condition, food intake, assessed by a single 24-h recall, tended to be higher than in the 10-h TIB sleep condition, which we speculate was accounted for by a greater intake of sweets and desserts. EDITORIAL
ER  - 

BibTeX

@misc{1149f2e1bce6069ef9a3a53946fba50fa76b35be,
  author = {Xuejuan Ning and J. Lv and Yu Guo and Z. Bian and Yunlong Tan and P. Pei and Junshi Chen and Shichuan Yan and Huimei Li and Zhifang Fu and Yiping Chen and H. Du and Zhengming Chen and Canqing Yu and Liming Li},
  title = {Association of Sleep Duration with Weight Gain and General and Central Obesity Risk in Chinese Adults: A Prospective Study},
  journal = {Obesity (Silver Spring, Md.)},
  year = {2019},
  volume = {28},
  pages = {468 - 474},
  doi = {10.1002/oby.22713},
  abstract = {Evidence on the association between sleep duration and obesity among adults is inconsistent. Prospective studies investigating the association in Chinese adults have been limited. This study aims to prospectively evaluate sleep duration in relation to subsequent weight gain and general and central obesity risk among Chinese adults.}
}

RIS

TY  - JOUR
AU  - Xuejuan Ning
AU  - J. Lv
AU  - Yu Guo
AU  - Z. Bian
AU  - Yunlong Tan
AU  - P. Pei
AU  - Junshi Chen
AU  - Shichuan Yan
AU  - Huimei Li
AU  - Zhifang Fu
AU  - Yiping Chen
AU  - H. Du
AU  - Zhengming Chen
AU  - Canqing Yu
AU  - Liming Li
T1  - Association of Sleep Duration with Weight Gain and General and Central Obesity Risk in Chinese Adults: A Prospective Study
JO  - Obesity (Silver Spring, Md.)
PY  - 2019
VL  - 28
SP  - 468 - 474
EP  - 468 - 474
DO  - 10.1002/oby.22713
AB  - Evidence on the association between sleep duration and obesity among adults is inconsistent. Prospective studies investigating the association in Chinese adults have been limited. This study aims to prospectively evaluate sleep duration in relation to subsequent weight gain and general and central obesity risk among Chinese adults.
ER  - 

BibTeX

@misc{28d9d73c41bb4780c80dcb39e47eb6a0e57e5e34,
  author = {Sunhee Park},
  title = {Association Between Short Sleep Duration and Obesity Among South Korean Adolescents},
  journal = {Western Journal of Nursing Research},
  year = {2011},
  volume = {33},
  pages = {207 - 223},
  doi = {10.1177/0193945910371317},
  abstract = {Short sleep duration and obesity are common health concerns in youth. This study of South Korean adolescents explores the association between the two conditions analyzing secondary data from the 2007 Korea Youth Risk Behavior Web-Based Survey. The sample is representative of the South Korean adolescent population (N = 73,836). For data analysis, analysis of variance, chi-square test, and logistic regression were used. Findings indicated that (a) sleep duration is inversely associated with levels of body mass index, F(4, 72654) = 240.07, p < .0001, and risks for overweight and obesity, χ2(4, 72659) = 27.41, p < .0001; and (b) after controlling for obesity-related factors, reduced sleep is strongly associated with a greater risk for overweight and obesity, OR = 0.94, p < .0001. Given the important link between sleep and obesity, health professionals should consider sleep habits as a significant factor in obesity-related problems of youth.}
}

RIS

TY  - JOUR
AU  - Sunhee Park
T1  - Association Between Short Sleep Duration and Obesity Among South Korean Adolescents
JO  - Western Journal of Nursing Research
PY  - 2011
VL  - 33
SP  - 207 - 223
EP  - 207 - 223
DO  - 10.1177/0193945910371317
AB  - Short sleep duration and obesity are common health concerns in youth. This study of South Korean adolescents explores the association between the two conditions analyzing secondary data from the 2007 Korea Youth Risk Behavior Web-Based Survey. The sample is representative of the South Korean adolescent population (N = 73,836). For data analysis, analysis of variance, chi-square test, and logistic regression were used. Findings indicated that (a) sleep duration is inversely associated with levels of body mass index, F(4, 72654) = 240.07, p < .0001, and risks for overweight and obesity, χ2(4, 72659) = 27.41, p < .0001; and (b) after controlling for obesity-related factors, reduced sleep is strongly associated with a greater risk for overweight and obesity, OR = 0.94, p < .0001. Given the important link between sleep and obesity, health professionals should consider sleep habits as a significant factor in obesity-related problems of youth.
ER  - 

BibTeX

@misc{2acef0a6ab1fda63071211cf7b0effb81da03d2f,
  author = {Qian Xiao and H. Arem and S. Moore and A. Hollenbeck and C. Matthews},
  title = {A large prospective investigation of sleep duration, weight change, and obesity in the NIH-AARP Diet and Health Study cohort.},
  journal = {American journal of epidemiology},
  year = {2013},
  volume = {178 11},
  pages = {
          1600-10
        },
  doi = {10.1093/aje/kwt180},
  abstract = {The relationship between sleep and obesity or weight gain in adults, particularly older populations, remains unclear. In a cohort of 83,377 US men and women aged 51-72 years, we prospectively investigated the association between self-reported sleep duration and weight change over an average of 7.5 years of follow-up (1995-2004). Participants were free of cancer, heart disease, and stroke at baseline and throughout the follow-up. We observed an inverse association between sleep duration per night and weight gain in both men (P for trend = 0.02) and women (P for trend < 0.001). Compared with 7-8 hours of sleep, shorter sleep (<5 hours or 5-6 hours) was associated with more weight gain (in kilograms; men: for <5 hours, β = 0.66, 95% confidence interval (CI): 0.19, 1.13, and for 5-6 hours, β = 0.12, 95% CI: -0.02, 0.26; women: for <5 hours, β = 0.43, 95% CI: 0.00, 0.86, and for 5-6 hours, β = 0.23, 95% CI: 0.08, 0.37). Among men and women who were not obese at baseline, participants who reported less than 5 hours of sleep per night had an approximately 40% higher risk of developing obesity than did those who reported 7-8 hours of sleep (for men, odds ratio = 1.45, 95% CI: 1.06, 1.99; for women, odds ratio = 1.37, 95% CI: 1.04, 1.79). The association between short sleep and excess weight gain was generally consistent across different categories of age, educational level, smoking status, baseline body mass index, and physical activity level.}
}

RIS

TY  - JOUR
AU  - Qian Xiao
AU  - H. Arem
AU  - S. Moore
AU  - A. Hollenbeck
AU  - C. Matthews
T1  - A large prospective investigation of sleep duration, weight change, and obesity in the NIH-AARP Diet and Health Study cohort.
JO  - American journal of epidemiology
PY  - 2013
VL  - 178 11
SP  - 
          1600-10
        
EP  - 
          1600-10
        
DO  - 10.1093/aje/kwt180
AB  - The relationship between sleep and obesity or weight gain in adults, particularly older populations, remains unclear. In a cohort of 83,377 US men and women aged 51-72 years, we prospectively investigated the association between self-reported sleep duration and weight change over an average of 7.5 years of follow-up (1995-2004). Participants were free of cancer, heart disease, and stroke at baseline and throughout the follow-up. We observed an inverse association between sleep duration per night and weight gain in both men (P for trend = 0.02) and women (P for trend < 0.001). Compared with 7-8 hours of sleep, shorter sleep (<5 hours or 5-6 hours) was associated with more weight gain (in kilograms; men: for <5 hours, β = 0.66, 95% confidence interval (CI): 0.19, 1.13, and for 5-6 hours, β = 0.12, 95% CI: -0.02, 0.26; women: for <5 hours, β = 0.43, 95% CI: 0.00, 0.86, and for 5-6 hours, β = 0.23, 95% CI: 0.08, 0.37). Among men and women who were not obese at baseline, participants who reported less than 5 hours of sleep per night had an approximately 40% higher risk of developing obesity than did those who reported 7-8 hours of sleep (for men, odds ratio = 1.45, 95% CI: 1.06, 1.99; for women, odds ratio = 1.37, 95% CI: 1.04, 1.79). The association between short sleep and excess weight gain was generally consistent across different categories of age, educational level, smoking status, baseline body mass index, and physical activity level.
ER  - 

BibTeX

@misc{6cfe7f3ff731af6ace59d7f11ee903b07c516b8d,
  author = {C. Nishiura and H. Hashimoto},
  title = {Sleep Duration and Weight Gain: Reconsideration by Panel Data Analysis},
  journal = {Journal of Epidemiology},
  year = {2014},
  volume = {24},
  pages = {404-409},
  doi = {10.2188/JEA.JE20140012},
  abstract = {BACKGROUND Inconsistent findings in previous studies of the association between sleep duration and changes in body mass index (BMI) may be attributed to misclassification of sleep duration fluctuations over time and unmeasured confounders such as genetic factors. The aim of the present study was to overcome these failings by using repeated measurements and panel data analysis to examine the sleep-BMI association. METHODS Panel data were derived by secondary use of the data from mandatory health checkups at a Japanese gas company. Male non-shift workers aged 19-39 years in 2007 were annually followed until 2010 (n = 1687, 6748 records). BMI was objectively measured, and sleep duration was self-reported. RESULTS Compared with 7-hour sleepers, panel analysis with the population-averaged model showed a significant increase in BMI among 5-hour (0.11 kg/m(2), P = 0.001), 6-hour (0.07 kg/m(2), P = 0.038), and ≥8-hour (0.19 kg/m(2), P = 0.009) sleepers. On the other hand, after adjustment for unobserved time-invariant confounders using the fixed-effects model, the magnitude of the association was considerably attenuated and no longer significant (5-hour, 0.07 kg/m(2), P = 0.168; 6-hour, 0.02 kg/m(2), P = 0.631; ≥8-hour sleepers, 0.06 kg/m(2), P = 0.460). CONCLUSIONS The longitudinal association between sleep duration and changes in BMI may be upwardly biased by unobserved time-invariant confounders rather than misclassified sleep duration. The net effect of sleep duration on weight gain may therefore be less than previously believed.}
}

RIS

TY  - JOUR
AU  - C. Nishiura
AU  - H. Hashimoto
T1  - Sleep Duration and Weight Gain: Reconsideration by Panel Data Analysis
JO  - Journal of Epidemiology
PY  - 2014
VL  - 24
SP  - 404-409
EP  - 404-409
DO  - 10.2188/JEA.JE20140012
AB  - BACKGROUND Inconsistent findings in previous studies of the association between sleep duration and changes in body mass index (BMI) may be attributed to misclassification of sleep duration fluctuations over time and unmeasured confounders such as genetic factors. The aim of the present study was to overcome these failings by using repeated measurements and panel data analysis to examine the sleep-BMI association. METHODS Panel data were derived by secondary use of the data from mandatory health checkups at a Japanese gas company. Male non-shift workers aged 19-39 years in 2007 were annually followed until 2010 (n = 1687, 6748 records). BMI was objectively measured, and sleep duration was self-reported. RESULTS Compared with 7-hour sleepers, panel analysis with the population-averaged model showed a significant increase in BMI among 5-hour (0.11 kg/m(2), P = 0.001), 6-hour (0.07 kg/m(2), P = 0.038), and ≥8-hour (0.19 kg/m(2), P = 0.009) sleepers. On the other hand, after adjustment for unobserved time-invariant confounders using the fixed-effects model, the magnitude of the association was considerably attenuated and no longer significant (5-hour, 0.07 kg/m(2), P = 0.168; 6-hour, 0.02 kg/m(2), P = 0.631; ≥8-hour sleepers, 0.06 kg/m(2), P = 0.460). CONCLUSIONS The longitudinal association between sleep duration and changes in BMI may be upwardly biased by unobserved time-invariant confounders rather than misclassified sleep duration. The net effect of sleep duration on weight gain may therefore be less than previously believed.
ER  - 

BibTeX

@misc{b231b76851c2847eb9b4766beb2be2fc8f7d0825,
  author = {Kyuwoong Kim and Doosup Shin and Go‐Un Jung and Donghoon Lee and Sang Min Park},
  title = {Association between sleep duration, fat mass, lean mass and obesity in Korean adults: the fourth and fifth Korea National Health and Nutrition Examination Surveys},
  journal = {Journal of Sleep Research},
  year = {2017},
  volume = {26},
  doi = {10.1111/jsr.12504},
  abstract = {This study investigated the association between sleep duration, fat mass, lean mass and obesity. Participants of this cross‐sectional study were 16 905 adults included into the 4th and 5th Korea National Health and Nutrition Examination Surveys. Sleep duration was assessed by self‐reported survey and categorized into ≤ 5, 6, 7, 8 and ≥ 9 h per day. The group reporting 7 h of sleep per day (comprised of those sleeping 7–8 h per day) was used as the reference group. Body composition was measured by dual X‐ray absorptiometry (DEXA). Obesity was defined based on the criteria from the Korean Society for the Study of Obesity. Least‐squares means of fat mass index (FMI) and lean mass index (LMI) adjusted for age, employment status, comorbidities and physical activity were used to assess the relation between sleep duration and body composition. Multivariable logistic regression was used to calculate the adjusted odds ratios (aOR) and 95% confidence intervals (95% CI) of obesity according to sleep duration after adjusting for sociodemographic and health‐related factors. After adjustment, FMI increased with fewer hours of sleep (P for trend: < 0.001) and LMI decreased with more hours of sleep (P for trend: 0.011). Compared to the reference group, sleep‐deprived individuals were 1.22 times more likely to have general obesity (aOR: 1.22; 95% CI: 1.03–1.45) and 1.32 times more likely to have abdominal obesity (aOR: 1.32; 95% CI: 1.10–1.58). Our findings suggest that sleep deprivation might be related to an increase of fat mass and obesity, while oversleeping could be linked to a reduction of lean mass.}
}

RIS

TY  - JOUR
AU  - Kyuwoong Kim
AU  - Doosup Shin
AU  - Go‐Un Jung
AU  - Donghoon Lee
AU  - Sang Min Park
T1  - Association between sleep duration, fat mass, lean mass and obesity in Korean adults: the fourth and fifth Korea National Health and Nutrition Examination Surveys
JO  - Journal of Sleep Research
PY  - 2017
VL  - 26
DO  - 10.1111/jsr.12504
AB  - This study investigated the association between sleep duration, fat mass, lean mass and obesity. Participants of this cross‐sectional study were 16 905 adults included into the 4th and 5th Korea National Health and Nutrition Examination Surveys. Sleep duration was assessed by self‐reported survey and categorized into ≤ 5, 6, 7, 8 and ≥ 9 h per day. The group reporting 7 h of sleep per day (comprised of those sleeping 7–8 h per day) was used as the reference group. Body composition was measured by dual X‐ray absorptiometry (DEXA). Obesity was defined based on the criteria from the Korean Society for the Study of Obesity. Least‐squares means of fat mass index (FMI) and lean mass index (LMI) adjusted for age, employment status, comorbidities and physical activity were used to assess the relation between sleep duration and body composition. Multivariable logistic regression was used to calculate the adjusted odds ratios (aOR) and 95% confidence intervals (95% CI) of obesity according to sleep duration after adjusting for sociodemographic and health‐related factors. After adjustment, FMI increased with fewer hours of sleep (P for trend: < 0.001) and LMI decreased with more hours of sleep (P for trend: 0.011). Compared to the reference group, sleep‐deprived individuals were 1.22 times more likely to have general obesity (aOR: 1.22; 95% CI: 1.03–1.45) and 1.32 times more likely to have abdominal obesity (aOR: 1.32; 95% CI: 1.10–1.58). Our findings suggest that sleep deprivation might be related to an increase of fat mass and obesity, while oversleeping could be linked to a reduction of lean mass.
ER  - 

BibTeX

@misc{0296edf5e291eadc96900da3da2849357553c027,
  author = {M. Atar and Ö. Pirgon and A. Buyukgebiz},
  title = {Sleep Disorders and Obesity in Childhood: A New Component in Solving Obesity.},
  journal = {Pediatric endocrinology reviews : PER},
  year = {2019},
  volume = {16 4},
  pages = {
          441-451
        },
  doi = {10.17458/per.vol16.2019.apb.sleepdisordersobesitychildhood},
  abstract = {Sleep disorders have been widely reported in obese individuals. Previous studies have shown that together with an increase in obesity prevalence, so does sleep duration in children and adolescents decrease. By contributing to energy imbalances, hormonal changes occurring with reduced sleep quality may cause weight gain and obesity. Current evidence shows that short sleep duration has effects on body weight and weight gain. Compared to individuals sleeping for a normal duration, insulin sensitivity is lower in those who sleep less. Lack of sleep increases the desire for food and has a direct effect on physical activity. Further studies are required to determine the contribution of sufficient sleep to obesity treatment.}
}

RIS

TY  - JOUR
AU  - M. Atar
AU  - Ö. Pirgon
AU  - A. Buyukgebiz
T1  - Sleep Disorders and Obesity in Childhood: A New Component in Solving Obesity.
JO  - Pediatric endocrinology reviews : PER
PY  - 2019
VL  - 16 4
SP  - 
          441-451
        
EP  - 
          441-451
        
DO  - 10.17458/per.vol16.2019.apb.sleepdisordersobesitychildhood
AB  - Sleep disorders have been widely reported in obese individuals. Previous studies have shown that together with an increase in obesity prevalence, so does sleep duration in children and adolescents decrease. By contributing to energy imbalances, hormonal changes occurring with reduced sleep quality may cause weight gain and obesity. Current evidence shows that short sleep duration has effects on body weight and weight gain. Compared to individuals sleeping for a normal duration, insulin sensitivity is lower in those who sleep less. Lack of sleep increases the desire for food and has a direct effect on physical activity. Further studies are required to determine the contribution of sufficient sleep to obesity treatment.
ER  - 

BibTeX

@misc{92c7d360b5e6100171ab643176b10a48934dcebd,
  author = {M. St-Onge},
  title = {Sleep–obesity relation: underlying mechanisms and consequences for treatment},
  journal = {Obesity Reviews},
  year = {2017},
  volume = {18},
  pages = {34 - 39},
  doi = {10.1111/obr.12499},
  abstract = {Short sleep duration has been associated with obesity in numerous epidemiological studies. However, such association studies cannot establish evidence of causality. Clinical intervention studies, on the other hand, can provide information on a causal effect of sleep duration on markers of weight gain: energy intake and energy expenditure. Herein is an overview of the science related to the impact of sleep restriction, in the context of clinical intervention studies, on energy intake, energy expenditure and body weight. Additionally, studies that evaluate the impact of sleep restriction on weight loss and the impact of sleep extension on appetite are discussed. Information to date suggests that weight management is hindered when attempted in the context of sleep restriction, and the public should be made aware of the negative consequences of sleep restriction for weight regulation.}
}

RIS

TY  - JOUR
AU  - M. St-Onge
T1  - Sleep–obesity relation: underlying mechanisms and consequences for treatment
JO  - Obesity Reviews
PY  - 2017
VL  - 18
SP  - 34 - 39
EP  - 34 - 39
DO  - 10.1111/obr.12499
AB  - Short sleep duration has been associated with obesity in numerous epidemiological studies. However, such association studies cannot establish evidence of causality. Clinical intervention studies, on the other hand, can provide information on a causal effect of sleep duration on markers of weight gain: energy intake and energy expenditure. Herein is an overview of the science related to the impact of sleep restriction, in the context of clinical intervention studies, on energy intake, energy expenditure and body weight. Additionally, studies that evaluate the impact of sleep restriction on weight loss and the impact of sleep extension on appetite are discussed. Information to date suggests that weight management is hindered when attempted in the context of sleep restriction, and the public should be made aware of the negative consequences of sleep restriction for weight regulation.
ER  - 

BibTeX

@misc{d0747a68047bf87594961dc45a74ebfa9ce88c93,
  author = {Michelle A. Miller and Marlot Kruisbrink and J. Wallace and Chen Ji and F. Cappuccio},
  title = {Sleep duration and incidence of obesity in infants, children, and adolescents: a systematic review and meta-analysis of prospective studies},
  journal = {SLEEP},
  year = {2018},
  volume = {41},
  pages = {&NA;},
  doi = {10.1093/sleep/zsy018},
  abstract = {Study Objectives
To assess the prospective relationship between sleep and obesity in a paediatric population.


Methods
We performed a systematic search using PubMed, Embase, Web of Science, and Cochrane (up to September 25, 2017). Included studies were prospective, had follow-up of ≥1 year, had duration of sleep at baseline, and measures of incidence of overweight or obesity and/or changes in body mass index (BMI) z-score and BMI during follow-up. We extracted relative risks or changes in BMI z-score or BMI and 95% confidence intervals (CI) and pooled them using a random effect model.


Results
Forty-two studies were included but, as there was significant heterogeneity, results are presented by age strata. Short sleep was associated with a greater risk of developing overweight or obesity in infancy (seven studies, 14738 participants, risk ratio [RR]: 1.40; 95% CI 1.19 to 1.65; p < .001), early childhood (eight studies, 31104 participants, RR: 1.57; 1.40 to 1.76; p < .001), middle childhood (three studies, 3005 participants, RR: 2.23; 2.18 to 2.27; p < .001), and adolescence (three studies, 26652 participants, RR: 1.30; 1.11 to 1.53; p < .002). Sleep duration was also associated with a significant change in BMI z-score (14 studies, 18 cohorts, 31665 participants; mean difference -0.03; -0.04 to -0.01 per hour sleep; p = .001) and in BMI (16 studies, 24 cohorts, 24894 participants; mean difference -0.03 kg/m2; -0.04 to -0.01 for every hour of increase in sleep; p = .001).


Conclusions
Short sleep duration is a risk factor or marker of the development of obesity in infants, children, and adolescents.}
}

RIS

TY  - JOUR
AU  - Michelle A. Miller
AU  - Marlot Kruisbrink
AU  - J. Wallace
AU  - Chen Ji
AU  - F. Cappuccio
T1  - Sleep duration and incidence of obesity in infants, children, and adolescents: a systematic review and meta-analysis of prospective studies
JO  - SLEEP
PY  - 2018
VL  - 41
SP  - &NA;
EP  - &NA;
DO  - 10.1093/sleep/zsy018
AB  - Study Objectives
To assess the prospective relationship between sleep and obesity in a paediatric population.


Methods
We performed a systematic search using PubMed, Embase, Web of Science, and Cochrane (up to September 25, 2017). Included studies were prospective, had follow-up of ≥1 year, had duration of sleep at baseline, and measures of incidence of overweight or obesity and/or changes in body mass index (BMI) z-score and BMI during follow-up. We extracted relative risks or changes in BMI z-score or BMI and 95% confidence intervals (CI) and pooled them using a random effect model.


Results
Forty-two studies were included but, as there was significant heterogeneity, results are presented by age strata. Short sleep was associated with a greater risk of developing overweight or obesity in infancy (seven studies, 14738 participants, risk ratio [RR]: 1.40; 95% CI 1.19 to 1.65; p < .001), early childhood (eight studies, 31104 participants, RR: 1.57; 1.40 to 1.76; p < .001), middle childhood (three studies, 3005 participants, RR: 2.23; 2.18 to 2.27; p < .001), and adolescence (three studies, 26652 participants, RR: 1.30; 1.11 to 1.53; p < .002). Sleep duration was also associated with a significant change in BMI z-score (14 studies, 18 cohorts, 31665 participants; mean difference -0.03; -0.04 to -0.01 per hour sleep; p = .001) and in BMI (16 studies, 24 cohorts, 24894 participants; mean difference -0.03 kg/m2; -0.04 to -0.01 for every hour of increase in sleep; p = .001).


Conclusions
Short sleep duration is a risk factor or marker of the development of obesity in infants, children, and adolescents.
ER  - 

BibTeX

@misc{59da1395b133db3a9f7c1373ad0883f4d6ab238c,
  author = {Mi Young Lee and J. Jeong and Eun-Hee Cho and Jung Min Kim and M. Shin},
  title = {Association of sleep duration and obesity according to sex and age in Korean adults: results from the Korea national health and nutrition examination survey 2007-2015},
  journal = {},
  year = {2018},
  volume = {},
  doi = {10.1530/ENDOABS.56.P543},
  abstract = {Background: This study aimed to investigate associations between self-reported sleep duration and general and abdominal obesity in Korean adults stratified according to gender and age. Methods: Data from 41,805 adults, 18–110 years of age, collected by the Korea National Health and Nutrition Examination Survey (KNHANES) in 2007 and 2015, were analyzed. Multivariable logistic regression was used to calculate adjusted odds ratios (AORs) and 95% confidence intervals (CIs) for obesity and abdominal obesity by sleep duration after controlling for sociodemographic and lifestyle variables. Results: Among individuals 30–49 years of age, there was an increased AOR for obesity only for sleep duration ≤ 5 hour/day compared with sleep duration 6 to 8 hour/day, both in men (OR, 1.25; 95% CI, 1.02–1.54) and women (OR, 1.56; 95% CI, 1.29–1.90), after controlling for covariates. Regarding women, there was increased AOR for abdominal obesity for sleep duration ≤ 5 hour/day (OR, 1.45; 95% CI, 1.18–1.78) and ≥ 9 hour/day (OR, 1.38; 95% CI, 1.09–1.76) compared with sleep duration 6 to 8 hour/day. However, for elderly individuals (≥ 65 years), there was a negative association between sleep duration ≤ 5 hour/day and obesity, but not with abdominal obesity, in both men and women. Conclusion: This study demonstrated a significant association between sleep duration and obesity, which varied according to gender and age.}
}

RIS

TY  - JOUR
AU  - Mi Young Lee
AU  - J. Jeong
AU  - Eun-Hee Cho
AU  - Jung Min Kim
AU  - M. Shin
T1  - Association of sleep duration and obesity according to sex and age in Korean adults: results from the Korea national health and nutrition examination survey 2007-2015
PY  - 2018
DO  - 10.1530/ENDOABS.56.P543
AB  - Background: This study aimed to investigate associations between self-reported sleep duration and general and abdominal obesity in Korean adults stratified according to gender and age. Methods: Data from 41,805 adults, 18–110 years of age, collected by the Korea National Health and Nutrition Examination Survey (KNHANES) in 2007 and 2015, were analyzed. Multivariable logistic regression was used to calculate adjusted odds ratios (AORs) and 95% confidence intervals (CIs) for obesity and abdominal obesity by sleep duration after controlling for sociodemographic and lifestyle variables. Results: Among individuals 30–49 years of age, there was an increased AOR for obesity only for sleep duration ≤ 5 hour/day compared with sleep duration 6 to 8 hour/day, both in men (OR, 1.25; 95% CI, 1.02–1.54) and women (OR, 1.56; 95% CI, 1.29–1.90), after controlling for covariates. Regarding women, there was increased AOR for abdominal obesity for sleep duration ≤ 5 hour/day (OR, 1.45; 95% CI, 1.18–1.78) and ≥ 9 hour/day (OR, 1.38; 95% CI, 1.09–1.76) compared with sleep duration 6 to 8 hour/day. However, for elderly individuals (≥ 65 years), there was a negative association between sleep duration ≤ 5 hour/day and obesity, but not with abdominal obesity, in both men and women. Conclusion: This study demonstrated a significant association between sleep duration and obesity, which varied according to gender and age.
ER  - 

BibTeX

@misc{485b39c33b8ad7ab486b77f0548adf4d1ee388ac,
  author = {Yavuz Furuncuoğlu and S. Emet and M. Kose and A. Sertkaya and Ş. Aras},
  title = {Is long sleep duratıon a new r isk factor for obesity ?},
  year = {2016},
  abstract = {Sleep is an important modulator for neuroendocrine functions and glucose metabolism. However the data about the relation between long sleep duration and obesity is scarce and unclear, so herein we evaluated both long and short sleep durations either with high or poor quality: within different age and obesity groups to define the interaction between obesity and sleep. Here in our group most of the patients (79.6%) sleeping longer than 8 hours -long sleeperswere either obese (49%) or morbid obese (30.6%) and long sleep duration was significantly common compared to control group (79.5% vs. 6%; and P < 0.001). We can conclude that as well as short sleep duration; long sleep duration has to be taken into concern as a cause or result of obesity.}
}

RIS

TY  - JOUR
AU  - Yavuz Furuncuoğlu
AU  - S. Emet
AU  - M. Kose
AU  - A. Sertkaya
AU  - Ş. Aras
T1  - Is long sleep duratıon a new r isk factor for obesity ?
PY  - 2016
AB  - Sleep is an important modulator for neuroendocrine functions and glucose metabolism. However the data about the relation between long sleep duration and obesity is scarce and unclear, so herein we evaluated both long and short sleep durations either with high or poor quality: within different age and obesity groups to define the interaction between obesity and sleep. Here in our group most of the patients (79.6%) sleeping longer than 8 hours -long sleeperswere either obese (49%) or morbid obese (30.6%) and long sleep duration was significantly common compared to control group (79.5% vs. 6%; and P < 0.001). We can conclude that as well as short sleep duration; long sleep duration has to be taken into concern as a cause or result of obesity.
ER  - 

BibTeX

@misc{f318efe3ac18f0c0828f95f6af0c18a8ec1a8283,
  author = {Guobo Li and Shi Wei and Fu-Hao Zheng and Xingyan Xu and Yingying Cai and Siying Wu},
  title = {Association between short sleep duration and overweight/obesity among middle-school students: a cross-sectional study in Fuzhou, China},
  journal = {},
  year = {2020},
  volume = {},
  doi = {10.21203/rs.3.rs-30129/v2},
  abstract = {
 Background: This study was designed to investigate the prevalence of short sleep duration and its association with overweight or obesity in adolescents from middle school in Fuzhou, China.Methods: A total of 13,063 students (male: 6,500; female: 6,553) from 18 different schools in Fuzhou city were included in this study. Questionnaires focusing on short sleep duration and overweight or obesity related factors were collected. The sleep duration was self-reported by the subjects. The body weight and height were measured by the qualified personnel. Logistic regression analysis was used to evaluate the effects of short sleep duration on overweight or obesity in school students.Results: The overall rates of overweight and obesity were 12.1% and 7.1%, respectively. The prevalence of short sleep duration among students was 82.8%. The majority of high school students (92.5%) showed a short sleep duration. Compared with male children with a sleep duration of 8-10 h per day, the odds ratios (ORs) of overweight/obesity for those with a sleep duration of less than 6 h or 6-8 h per day were 1.60 (1.22-2.09) and 1.07 (0.90-1.29) after adjusting social and demographic status, mental health and lifestyle factors. The homologous ORs of female children were 1.45 (1.04-2.04) and 1.11(0.85-1.45), respectively.Conclusions: A large number of adolescents suffered from short sleep duration. Short sleep duration is associated with a higher OR of overweight/obesity in middle-school children, especially in the male adolescences.}
}

RIS

TY  - JOUR
AU  - Guobo Li
AU  - Shi Wei
AU  - Fu-Hao Zheng
AU  - Xingyan Xu
AU  - Yingying Cai
AU  - Siying Wu
T1  - Association between short sleep duration and overweight/obesity among middle-school students: a cross-sectional study in Fuzhou, China
PY  - 2020
DO  - 10.21203/rs.3.rs-30129/v2
AB  - 
 Background: This study was designed to investigate the prevalence of short sleep duration and its association with overweight or obesity in adolescents from middle school in Fuzhou, China.Methods: A total of 13,063 students (male: 6,500; female: 6,553) from 18 different schools in Fuzhou city were included in this study. Questionnaires focusing on short sleep duration and overweight or obesity related factors were collected. The sleep duration was self-reported by the subjects. The body weight and height were measured by the qualified personnel. Logistic regression analysis was used to evaluate the effects of short sleep duration on overweight or obesity in school students.Results: The overall rates of overweight and obesity were 12.1% and 7.1%, respectively. The prevalence of short sleep duration among students was 82.8%. The majority of high school students (92.5%) showed a short sleep duration. Compared with male children with a sleep duration of 8-10 h per day, the odds ratios (ORs) of overweight/obesity for those with a sleep duration of less than 6 h or 6-8 h per day were 1.60 (1.22-2.09) and 1.07 (0.90-1.29) after adjusting social and demographic status, mental health and lifestyle factors. The homologous ORs of female children were 1.45 (1.04-2.04) and 1.11(0.85-1.45), respectively.Conclusions: A large number of adolescents suffered from short sleep duration. Short sleep duration is associated with a higher OR of overweight/obesity in middle-school children, especially in the male adolescences.
ER  - 

BibTeX

@misc{32ceb303f3a64119bee5dc23f9db8e177bffcbae,
  author = {Lu Wang and W. Jansen and M. Boere-Boonekamp and E. Vlasblom and M. L'Hoir and M. Beltman and A. van Grieken and H. Raat},
  title = {Sleep and body mass index in infancy and early childhood (6‐36 mo): a longitudinal study},
  journal = {Pediatric Obesity},
  year = {2019},
  volume = {14},
  doi = {10.1111/ijpo.12506},
  abstract = {Relatively, few longitudinal studies have evaluated the association between sleep and body mass index (BMI) among younger children. In addition, few studies have evaluated the bidirectional longitudinal association between sleep duration and child BMI.}
}

RIS

TY  - JOUR
AU  - Lu Wang
AU  - W. Jansen
AU  - M. Boere-Boonekamp
AU  - E. Vlasblom
AU  - M. L'Hoir
AU  - M. Beltman
AU  - A. van Grieken
AU  - H. Raat
T1  - Sleep and body mass index in infancy and early childhood (6‐36 mo): a longitudinal study
JO  - Pediatric Obesity
PY  - 2019
VL  - 14
DO  - 10.1111/ijpo.12506
AB  - Relatively, few longitudinal studies have evaluated the association between sleep and body mass index (BMI) among younger children. In addition, few studies have evaluated the bidirectional longitudinal association between sleep duration and child BMI.
ER  - 

BibTeX

@misc{c6fb5c683f0778233ee2eed7f632abbfb4c35680,
  author = {B. Morrissey and E. Taveras and S. Allender and Claudia Strugnell},
  title = {Sleep and obesity among children: A systematic review of multiple sleep dimensions},
  journal = {Pediatric Obesity},
  year = {2020},
  volume = {15},
  doi = {10.1111/ijpo.12619},
  abstract = {The objectives were to systematically investigate the multiple dimensions of sleep and their association with overweight or obesity among primary school‐aged children. CINHAL, PsycINFO, SPORTDiscus, Medline, Cochrane, Embase, and PubMed databases were searched for papers reporting on an association between children's sleep and weight status. Studies on clinical populations, published in languages other than English, without objectively measured weight status, or where weight status was reported outside the outlined age bracket (5‐13 years) were excluded. A total of 34  248 citations were extracted from our systematic search protocol, of which 112 were included for detailed review. Compared with sleep duration, of which 86/103 articles found a significant inverse association between sleep duration and measured weight status, few studies examined other dimensions of sleep, such as quality, efficiency and bed/wake times, and relationship with weight status. Where studies existed, variation in defining and measurement of these dimensions restricted comparison and potentially influenced discrepancies across results. Overall, the findings of this review warrant the need for further research of the outlined dimensions of sleep. Future research would benefit from clarity on definitions across the different dimensions, along with the use of valid and reliable tools.}
}

RIS

TY  - JOUR
AU  - B. Morrissey
AU  - E. Taveras
AU  - S. Allender
AU  - Claudia Strugnell
T1  - Sleep and obesity among children: A systematic review of multiple sleep dimensions
JO  - Pediatric Obesity
PY  - 2020
VL  - 15
DO  - 10.1111/ijpo.12619
AB  - The objectives were to systematically investigate the multiple dimensions of sleep and their association with overweight or obesity among primary school‐aged children. CINHAL, PsycINFO, SPORTDiscus, Medline, Cochrane, Embase, and PubMed databases were searched for papers reporting on an association between children's sleep and weight status. Studies on clinical populations, published in languages other than English, without objectively measured weight status, or where weight status was reported outside the outlined age bracket (5‐13 years) were excluded. A total of 34  248 citations were extracted from our systematic search protocol, of which 112 were included for detailed review. Compared with sleep duration, of which 86/103 articles found a significant inverse association between sleep duration and measured weight status, few studies examined other dimensions of sleep, such as quality, efficiency and bed/wake times, and relationship with weight status. Where studies existed, variation in defining and measurement of these dimensions restricted comparison and potentially influenced discrepancies across results. Overall, the findings of this review warrant the need for further research of the outlined dimensions of sleep. Future research would benefit from clarity on definitions across the different dimensions, along with the use of valid and reliable tools.
ER  - 

BibTeX

@misc{637d4dd846bb2162883640dd7ac674771c4de5a2,
  author = {A. Palm and C. Janson and E. Lindberg},
  title = {The impact of weight gain and obesity on development of sleep problems in a population-based sample},
  journal = {European Respiratory Journal},
  year = {2014},
  volume = {44},
  pages = {4665},
  abstract = {Objectives:To investigate the role of obesity and weight gain on development of sleep problems in a population-based cohort. Method: A population-based sample of men (n=1,896, aged 40-79 years) and women (n=5,116, age g20years) responded to questionnaires at baseline and follow-up after 10-13 years. Sleep problems were assessed by questions of difficulties in inducing sleep (DIS), difficulties maintaining sleep (DMS), excessive daytime sleepiness (EDS) and insomnia. Body mass index (BMI) was calculated from self-reported weight and height at both baseline and follow-up while confounding factors (physical activity, tobacco and alcohol use, somatic disease and snoring) were based on responses at baseline. Results: Subjects in the quartile with the highest rise in BMI with a weight gain exceeding 2.06 kg/m 2 , had a higher risk to develop DMS (Adj. OR, 95% CI) (1.57, 1.24-1.98), EDS (2.23, 1.64-3.02) and insomnia (2.51, 1.48-4.27). Weight gain was not associated with development of DIS. Overweight and obese subjects reported more DIS, DMS and EDS at baseline but there was no independent association between BMI-level at baseline and development of new sleep problems. Increasing age was independently associated with an increased risk of developing DIS (adj. OR 95% CI /10 years) (1.17, 1.07-1.29 but a reduced risk of developing DMS (0.92, 0.85-0.98), EDS (0.79, 0.72-0.87) and insomnia (0.80, 0.69-0.94). Conclusions: Weight gain is an independent risk factor for development of several sleep problems and daytime sleepiness. The presence of overweight and weight gain should be taken into account when treating patients with sleep problems.}
}

RIS

TY  - JOUR
AU  - A. Palm
AU  - C. Janson
AU  - E. Lindberg
T1  - The impact of weight gain and obesity on development of sleep problems in a population-based sample
JO  - European Respiratory Journal
PY  - 2014
VL  - 44
SP  - 4665
EP  - 4665
AB  - Objectives:To investigate the role of obesity and weight gain on development of sleep problems in a population-based cohort. Method: A population-based sample of men (n=1,896, aged 40-79 years) and women (n=5,116, age g20years) responded to questionnaires at baseline and follow-up after 10-13 years. Sleep problems were assessed by questions of difficulties in inducing sleep (DIS), difficulties maintaining sleep (DMS), excessive daytime sleepiness (EDS) and insomnia. Body mass index (BMI) was calculated from self-reported weight and height at both baseline and follow-up while confounding factors (physical activity, tobacco and alcohol use, somatic disease and snoring) were based on responses at baseline. Results: Subjects in the quartile with the highest rise in BMI with a weight gain exceeding 2.06 kg/m 2 , had a higher risk to develop DMS (Adj. OR, 95% CI) (1.57, 1.24-1.98), EDS (2.23, 1.64-3.02) and insomnia (2.51, 1.48-4.27). Weight gain was not associated with development of DIS. Overweight and obese subjects reported more DIS, DMS and EDS at baseline but there was no independent association between BMI-level at baseline and development of new sleep problems. Increasing age was independently associated with an increased risk of developing DIS (adj. OR 95% CI /10 years) (1.17, 1.07-1.29 but a reduced risk of developing DMS (0.92, 0.85-0.98), EDS (0.79, 0.72-0.87) and insomnia (0.80, 0.69-0.94). Conclusions: Weight gain is an independent risk factor for development of several sleep problems and daytime sleepiness. The presence of overweight and weight gain should be taken into account when treating patients with sleep problems.
ER  - 

BibTeX

@misc{51516d90f54d59b551f9c08863df059a3bbec43d,
  author = {C. Kline and M. Lambiase and Margaret B Conroy and M. Brooks and A. Kriska and E. Barinas-Mitchell},
  title = {0176 Short Sleep Duration and Poor Sleep Quality Predict Blunted Weight Loss in a Behavioral Weight Loss Intervention},
  journal = {Sleep},
  year = {2020},
  volume = {43},
  doi = {10.1093/sleep/zsaa056.174},
  abstract = {
 
 
 Short sleep duration and poor sleep quality have each been associated with obesity and weight gain. However, less is known regarding how sleep may impact attempted weight loss. The purpose of this study was to investigate the associations between sleep duration and sleep quality, both independently and in combination, with weight loss in a 12-month behavioral weight loss intervention.
 
 
 
 Young to middle-aged adults who were overweight or obese (N=296) completed a 12-month behavioral weight loss intervention, with weight assessed at baseline, 6 and 12 months. Sleep duration and quality were derived from the Pittsburgh Sleep Quality Index. Analyses examined the change in sleep over time and the association between baseline sleep and changes in sleep with 6- and 12-month weight loss following adjustment for relevant covariates including age, gender, race, education, baseline body mass index, and baseline risk for sleep apnea.
 
 
 
 Participants (with an average baseline weight of 97.0±1.0 kg) lost 6.6±1.1 kg (6.8%) and 6.7±1.2 kg (6.9%) at 6 and 12 months relative to baseline, respectively. Global sleep quality significantly improved over the 12-month intervention (P=.03), but average sleep duration and the prevalence of short sleep duration (<6 h) or poor sleep quality did not change significantly (each P≥.45). Adults with short sleep duration at baseline lost 3.3±0.9% less weight than those with ≥6 h sleep duration (P<.001). Adults with poor sleep quality at baseline lost 1.6±0.8% less weight than those with good sleep quality (P=.04). When considered together, adults with both short sleep duration and poor sleep quality lost at least 5.0% less weight compared with all other sleep duration/quality group combinations (P<.001).
 
 
 
 Our findings highlight the importance of both sleep duration and sleep quality as predictors of behavioral weight loss and suggest that screening for sleep disturbance may be useful to determine who may benefit from additional counseling and resources.
 
 
 
 R01HL077525, K23HL118318
}
}

RIS

TY  - JOUR
AU  - C. Kline
AU  - M. Lambiase
AU  - Margaret B Conroy
AU  - M. Brooks
AU  - A. Kriska
AU  - E. Barinas-Mitchell
T1  - 0176 Short Sleep Duration and Poor Sleep Quality Predict Blunted Weight Loss in a Behavioral Weight Loss Intervention
JO  - Sleep
PY  - 2020
VL  - 43
DO  - 10.1093/sleep/zsaa056.174
AB  - 
 
 
 Short sleep duration and poor sleep quality have each been associated with obesity and weight gain. However, less is known regarding how sleep may impact attempted weight loss. The purpose of this study was to investigate the associations between sleep duration and sleep quality, both independently and in combination, with weight loss in a 12-month behavioral weight loss intervention.
 
 
 
 Young to middle-aged adults who were overweight or obese (N=296) completed a 12-month behavioral weight loss intervention, with weight assessed at baseline, 6 and 12 months. Sleep duration and quality were derived from the Pittsburgh Sleep Quality Index. Analyses examined the change in sleep over time and the association between baseline sleep and changes in sleep with 6- and 12-month weight loss following adjustment for relevant covariates including age, gender, race, education, baseline body mass index, and baseline risk for sleep apnea.
 
 
 
 Participants (with an average baseline weight of 97.0±1.0 kg) lost 6.6±1.1 kg (6.8%) and 6.7±1.2 kg (6.9%) at 6 and 12 months relative to baseline, respectively. Global sleep quality significantly improved over the 12-month intervention (P=.03), but average sleep duration and the prevalence of short sleep duration (<6 h) or poor sleep quality did not change significantly (each P≥.45). Adults with short sleep duration at baseline lost 3.3±0.9% less weight than those with ≥6 h sleep duration (P<.001). Adults with poor sleep quality at baseline lost 1.6±0.8% less weight than those with good sleep quality (P=.04). When considered together, adults with both short sleep duration and poor sleep quality lost at least 5.0% less weight compared with all other sleep duration/quality group combinations (P<.001).
 
 
 
 Our findings highlight the importance of both sleep duration and sleep quality as predictors of behavioral weight loss and suggest that screening for sleep disturbance may be useful to determine who may benefit from additional counseling and resources.
 
 
 
 R01HL077525, K23HL118318

ER  - 

BibTeX

@misc{8f329e04c2b1552fec896c39bde732fc102f871d,
  author = {Lorrie Magee and L. Hale},
  title = {Longitudinal associations between sleep duration and subsequent weight gain: a systematic review.},
  journal = {Sleep medicine reviews},
  year = {2012},
  volume = {16 3},
  pages = {
          231-41
        },
  doi = {10.1016/j.smrv.2011.05.005}
}

RIS

TY  - JOUR
AU  - Lorrie Magee
AU  - L. Hale
T1  - Longitudinal associations between sleep duration and subsequent weight gain: a systematic review.
JO  - Sleep medicine reviews
PY  - 2012
VL  - 16 3
SP  - 
          231-41
        
EP  - 
          231-41
        
DO  - 10.1016/j.smrv.2011.05.005
ER  - 

BibTeX

@misc{d6f3a5cb7a3f02098acd0d2b1ce50d59b15c91fc,
  author = {Hye Kim and I. Kang and W. Choe and Kyung-Sik Yoon},
  title = {Sleep duration and risk of obesity: A genome and epidemiological study},
  journal = {},
  year = {2021},
  volume = {3},
  pages = {1-1},
  doi = {10.3892/WASJ.2021.91},
  abstract = {Obesity is one of the risk factors for metabolic and cardiovascular diseases. Although numerous epidemiological studies have reported a possible causal association between sleep duration and the risk of obesity, evidence for an associa‐ tion in adults remains unclear. Accordingly, the present study aimed to evaluate the association between sleep duration and the risk of obesity in an adult group. To this end, 5,719 adults (2,876 males and 2,843 females) who participated in a genome and epidemiology study, were analyzed. Data were obtained on sleep duration, body mass index (BMI) and other demographic data including age, sex, waist circumference, alcohol consumption and smoking status. The association between sleep duration and obesity was investigated using a Chi‐squared test or logistic regression analysis. Multivariate logistic regression analysis revealed that a short sleep duration (≤6 h) was significantly associated with obesity after adjus‐ ting for various confounding factors (OR, 1.446; 95% CI, 1.254‐1.667; P<0.0001). Strong associations between a short sleep duration (≤6 h) and obesity were  identified  in males (OR, 1.567; 95% CI, 1.258‐1.953; P<0.0001) and the early midlife group (OR, 1.584; 95% CI, 1.320‐1.902; P<0.0001). Furthermore, the risk of obesity significantly increased with a short sleep duration (≤6 h) in participants without hyperten‐ sion (OR, 1.534; 95% CI, 1.263‐1.863; P<0.0001) or diabetes (OR, 1.473; 95% CI, 1.272‐1.705; P<0.0001). A short sleep duration was positively related to a high prevalence of obesity, and this association varied with sex, age, and the presence of metabolic disease. On the whole, the findings of the present study provide evidence that sleep duration can affect obesity. Introduction Obesity is a major public health concern worldwide and is related to a number of health consequences, including hyper‐ tension, diabetes mellitus, dyslipidemia, cardiovascular disease and cancer (1,2). Obesity rates have markedly increased in recent years. The number of obese individuals worldwide was estimated at 500 million in 2005, and this number is expected to increase steadily to approximately 1.1 billion by 2030 (3). The prevalence of obesity among the Korean population has also been reported to have increased from 27% in 1998 to 32% in 2011 (4). Consequently, changing one's lifestyle factors is crucial to preventing or reducing the burden of obesity. Disorders of sleep, an essential component of lifestyle, are associated with metabolic dysfunctions, which can lead to the progression of obesity (5). To date, studies have largely suggested that several hormones are involved in the regulation that occurs between sleep and body mass index (BMI) (6‐8). Some researchers have also observed that sleep restriction increased food intake and that poor sleep could exert negative effects on maintaining nervous, endocrine and cardiovas‐ cular health (9). Moreover, recent epidemiological surveys have investigated the association between sleep time and the prevalence of obesity (10‐12) as the hours of sleep represent the most essential measure of sleep. However, the overall findings remain unclear, as studies have shown associations that presented as an inverted bell‐shaped (13), were negatively linear (14‐16), or exhibited no correlation (17). Furthermore, these previous studies were mostly conducted in the United States (18), Europe (19), China (11,20) and Japan (21,22), with few studies conducted in Korea (23). Therefore, using the Korean Genome and Epidemiology Study (KoGES) data, the present study examined the associa‐ tion between sleep duration and the probability of obesity in Korean adults and investigated age‐ and sex‐specific differ‐ ences. In addition, at the reference time point, the effects of sleep on the risk of obesity were investigated, depending upon whether the participants had hypertension or diabetes. Patients and methods Study participants and design. The study dataset was obtained from the KoGES. The KoGES is an ongoing prospective cohort study that was initiated in 2001 to assess the risk and Sleep duration and risk of obesity: A genome and epidemiological study HYE OK KIM1, INSUG KANG1, WONCHAE CHOE1 and KYUNG‐SIK YOON1,2 1Department of Biochemistry and Molecular Biology, School of Medicine, Kyung Hee University; 2Medical Science Research Institute, Kyung Hee University Medical Center, Seoul 02447, Republic of Korea Received November 5, 2020; Accepted February 4, 2021 DOI: 10.3892/wasj.2021.91 Correspondence to: Professor Kyung‐Sik Yoon, Department of Biochemistry and Molecular Biology, School of Medicine, Kyung Hee University, 26 Kyungheedae‐ro, Dongdaemun‐gu, Seoul 02447,}
}

RIS

TY  - JOUR
AU  - Hye Kim
AU  - I. Kang
AU  - W. Choe
AU  - Kyung-Sik Yoon
T1  - Sleep duration and risk of obesity: A genome and epidemiological study
PY  - 2021
VL  - 3
SP  - 1-1
EP  - 1-1
DO  - 10.3892/WASJ.2021.91
AB  - Obesity is one of the risk factors for metabolic and cardiovascular diseases. Although numerous epidemiological studies have reported a possible causal association between sleep duration and the risk of obesity, evidence for an associa‐ tion in adults remains unclear. Accordingly, the present study aimed to evaluate the association between sleep duration and the risk of obesity in an adult group. To this end, 5,719 adults (2,876 males and 2,843 females) who participated in a genome and epidemiology study, were analyzed. Data were obtained on sleep duration, body mass index (BMI) and other demographic data including age, sex, waist circumference, alcohol consumption and smoking status. The association between sleep duration and obesity was investigated using a Chi‐squared test or logistic regression analysis. Multivariate logistic regression analysis revealed that a short sleep duration (≤6 h) was significantly associated with obesity after adjus‐ ting for various confounding factors (OR, 1.446; 95% CI, 1.254‐1.667; P<0.0001). Strong associations between a short sleep duration (≤6 h) and obesity were  identified  in males (OR, 1.567; 95% CI, 1.258‐1.953; P<0.0001) and the early midlife group (OR, 1.584; 95% CI, 1.320‐1.902; P<0.0001). Furthermore, the risk of obesity significantly increased with a short sleep duration (≤6 h) in participants without hyperten‐ sion (OR, 1.534; 95% CI, 1.263‐1.863; P<0.0001) or diabetes (OR, 1.473; 95% CI, 1.272‐1.705; P<0.0001). A short sleep duration was positively related to a high prevalence of obesity, and this association varied with sex, age, and the presence of metabolic disease. On the whole, the findings of the present study provide evidence that sleep duration can affect obesity. Introduction Obesity is a major public health concern worldwide and is related to a number of health consequences, including hyper‐ tension, diabetes mellitus, dyslipidemia, cardiovascular disease and cancer (1,2). Obesity rates have markedly increased in recent years. The number of obese individuals worldwide was estimated at 500 million in 2005, and this number is expected to increase steadily to approximately 1.1 billion by 2030 (3). The prevalence of obesity among the Korean population has also been reported to have increased from 27% in 1998 to 32% in 2011 (4). Consequently, changing one's lifestyle factors is crucial to preventing or reducing the burden of obesity. Disorders of sleep, an essential component of lifestyle, are associated with metabolic dysfunctions, which can lead to the progression of obesity (5). To date, studies have largely suggested that several hormones are involved in the regulation that occurs between sleep and body mass index (BMI) (6‐8). Some researchers have also observed that sleep restriction increased food intake and that poor sleep could exert negative effects on maintaining nervous, endocrine and cardiovas‐ cular health (9). Moreover, recent epidemiological surveys have investigated the association between sleep time and the prevalence of obesity (10‐12) as the hours of sleep represent the most essential measure of sleep. However, the overall findings remain unclear, as studies have shown associations that presented as an inverted bell‐shaped (13), were negatively linear (14‐16), or exhibited no correlation (17). Furthermore, these previous studies were mostly conducted in the United States (18), Europe (19), China (11,20) and Japan (21,22), with few studies conducted in Korea (23). Therefore, using the Korean Genome and Epidemiology Study (KoGES) data, the present study examined the associa‐ tion between sleep duration and the probability of obesity in Korean adults and investigated age‐ and sex‐specific differ‐ ences. In addition, at the reference time point, the effects of sleep on the risk of obesity were investigated, depending upon whether the participants had hypertension or diabetes. Patients and methods Study participants and design. The study dataset was obtained from the KoGES. The KoGES is an ongoing prospective cohort study that was initiated in 2001 to assess the risk and Sleep duration and risk of obesity: A genome and epidemiological study HYE OK KIM1, INSUG KANG1, WONCHAE CHOE1 and KYUNG‐SIK YOON1,2 1Department of Biochemistry and Molecular Biology, School of Medicine, Kyung Hee University; 2Medical Science Research Institute, Kyung Hee University Medical Center, Seoul 02447, Republic of Korea Received November 5, 2020; Accepted February 4, 2021 DOI: 10.3892/wasj.2021.91 Correspondence to: Professor Kyung‐Sik Yoon, Department of Biochemistry and Molecular Biology, School of Medicine, Kyung Hee University, 26 Kyungheedae‐ro, Dongdaemun‐gu, Seoul 02447,
ER  - 

BibTeX

@misc{6ee263d63b9e04d95d5f7988bafae868775cf03d,
  author = {Fenglei Wang and Huijuan Liu and Y. Wan and Jing Li and Yu Chen and Ju-Sheng Zheng and Tao Huang and Duo Li},
  title = {Sleep Duration and Overweight/Obesity in Preschool-Aged Children: A Prospective Study of up to 48,922 Children of the Jiaxing Birth Cohort.},
  journal = {Sleep},
  year = {2016},
  volume = {39 11},
  pages = {
          2013-2019
        },
  doi = {10.5665/SLEEP.6234},
  abstract = {STUDY OBJECTIVES
To examine the association between sleep duration and overweight/obesity in preschool-aged children.


METHODS
A total of 48,922 3-year old children enrolled in the Jiaxing Birth Cohort, who provided sleep information and anthropometric data, were included in the present study as baseline and were followed up to 5 years of age. Sleep duration was categorized as ≤ 10 hours, 11-12 hours, and ≥ 13 hours. Overweight and obesity were defined according to the cut point criteria in China. Prevalence ratios and risk ratios were used to assess the association between sleep duration and risk of overweight/obesity.


RESULTS
In cross-sectional analyses at baseline, the adjusted prevalence ratios (95% confidence interval) of overweight (with 11-12 h of sleep being considered the reference group) for children sleeping ≤ 10 h and ≥ 13 h were 1.13 (1.06-1.20) and 1.16 (1.09-1.24), respectively, whereas the adjusted prevalence ratios (95% confidence interval) of obesity were 1.25 (1.11-1.40) and 1.25 (1.11-1.42). In longitudinal analyses, the adjusted risk ratios (95% confidence interval) of overweight for children sleeping ≤ 10 h and ≥ 13 h were 1.48 (1.26-1.74) and 1.13 (0.96-1.34), while adjusted risk ratios (95% confidence interval) of obesity were 1.77 (1.30-2.40) and 1.19 (0.85-1.66). Restricted cubic splines regression supported U-shaped curvilinear associations between sleep duration and overweight/obesity in both cross-sectional and longitudinal analyses.


CONCLUSIONS
Both short and overlong sleep duration are associated with a higher risk of overweight/obesity in preschool-aged children. Optimizing sleep duration may be an important modifiable intervention for overweight and obesity.}
}

RIS

TY  - JOUR
AU  - Fenglei Wang
AU  - Huijuan Liu
AU  - Y. Wan
AU  - Jing Li
AU  - Yu Chen
AU  - Ju-Sheng Zheng
AU  - Tao Huang
AU  - Duo Li
T1  - Sleep Duration and Overweight/Obesity in Preschool-Aged Children: A Prospective Study of up to 48,922 Children of the Jiaxing Birth Cohort.
JO  - Sleep
PY  - 2016
VL  - 39 11
SP  - 
          2013-2019
        
EP  - 
          2013-2019
        
DO  - 10.5665/SLEEP.6234
AB  - STUDY OBJECTIVES
To examine the association between sleep duration and overweight/obesity in preschool-aged children.


METHODS
A total of 48,922 3-year old children enrolled in the Jiaxing Birth Cohort, who provided sleep information and anthropometric data, were included in the present study as baseline and were followed up to 5 years of age. Sleep duration was categorized as ≤ 10 hours, 11-12 hours, and ≥ 13 hours. Overweight and obesity were defined according to the cut point criteria in China. Prevalence ratios and risk ratios were used to assess the association between sleep duration and risk of overweight/obesity.


RESULTS
In cross-sectional analyses at baseline, the adjusted prevalence ratios (95% confidence interval) of overweight (with 11-12 h of sleep being considered the reference group) for children sleeping ≤ 10 h and ≥ 13 h were 1.13 (1.06-1.20) and 1.16 (1.09-1.24), respectively, whereas the adjusted prevalence ratios (95% confidence interval) of obesity were 1.25 (1.11-1.40) and 1.25 (1.11-1.42). In longitudinal analyses, the adjusted risk ratios (95% confidence interval) of overweight for children sleeping ≤ 10 h and ≥ 13 h were 1.48 (1.26-1.74) and 1.13 (0.96-1.34), while adjusted risk ratios (95% confidence interval) of obesity were 1.77 (1.30-2.40) and 1.19 (0.85-1.66). Restricted cubic splines regression supported U-shaped curvilinear associations between sleep duration and overweight/obesity in both cross-sectional and longitudinal analyses.


CONCLUSIONS
Both short and overlong sleep duration are associated with a higher risk of overweight/obesity in preschool-aged children. Optimizing sleep duration may be an important modifiable intervention for overweight and obesity.
ER  - 

BibTeX

@misc{55eb9939abcb1c1ba311dbe507787f958fbb0b88,
  author = {Michelle A. Miller and Sarah Bates and Chen Ji and F. Cappuccio},
  title = {Systematic review and meta‐analyses of the relationship between short sleep and incidence of obesity and effectiveness of sleep interventions on weight gain in preschool children},
  journal = {Obesity Reviews},
  year = {2020},
  volume = {22},
  doi = {10.1111/obr.13113},
  abstract = {The aim of this study is to determine (a) whether short sleep is associated with the incidence of obesity and (b) whether interventions beneficial for sleep reduce weight gain in preschool children. We systematically searched PubMed, Embase, Web of Science and Cochrane up to 12/09/2019. (a) Studies that were included were prospective, had follow‐up ≥1 year, with sleep duration at baseline and required outcome measures. (b) Intervention trials with sleep intervention and measures of overweight or obesity were included. Data were extracted according to PRISMA guidelines. (a) The risk of developing overweight/obesity was greater in short sleeping children (13 studies, 42 878 participants, RR: 1.54; 95% CI, 1.33 to 1.77; p < 0.001). Sleep duration was associated with a significant change in BMI z‐score (10 studies, 11 cohorts and 29 553 participants) (mean difference: −0.02 unit per hour sleep; −0.03 to −0.01; p < 0.001). (b) Four of the five intervention studies reported improved outcomes: for BMI (−0.27 kg/m2; −0.50 to −0.03; p = 0.03); for BMI z‐score (−0.07 unit; −0.12 to −0.02; p = 0.006). Short sleep duration is a risk factor or marker of the development of obesity in preschool children. Intervention studies suggest that improved sleep may be beneficially associated with a reduced weight gain in these children.}
}

RIS

TY  - JOUR
AU  - Michelle A. Miller
AU  - Sarah Bates
AU  - Chen Ji
AU  - F. Cappuccio
T1  - Systematic review and meta‐analyses of the relationship between short sleep and incidence of obesity and effectiveness of sleep interventions on weight gain in preschool children
JO  - Obesity Reviews
PY  - 2020
VL  - 22
DO  - 10.1111/obr.13113
AB  - The aim of this study is to determine (a) whether short sleep is associated with the incidence of obesity and (b) whether interventions beneficial for sleep reduce weight gain in preschool children. We systematically searched PubMed, Embase, Web of Science and Cochrane up to 12/09/2019. (a) Studies that were included were prospective, had follow‐up ≥1 year, with sleep duration at baseline and required outcome measures. (b) Intervention trials with sleep intervention and measures of overweight or obesity were included. Data were extracted according to PRISMA guidelines. (a) The risk of developing overweight/obesity was greater in short sleeping children (13 studies, 42 878 participants, RR: 1.54; 95% CI, 1.33 to 1.77; p < 0.001). Sleep duration was associated with a significant change in BMI z‐score (10 studies, 11 cohorts and 29 553 participants) (mean difference: −0.02 unit per hour sleep; −0.03 to −0.01; p < 0.001). (b) Four of the five intervention studies reported improved outcomes: for BMI (−0.27 kg/m2; −0.50 to −0.03; p = 0.03); for BMI z‐score (−0.07 unit; −0.12 to −0.02; p = 0.006). Short sleep duration is a risk factor or marker of the development of obesity in preschool children. Intervention studies suggest that improved sleep may be beneficially associated with a reduced weight gain in these children.
ER  - 

BibTeX

@misc{23f69fe032ead3cf0cb269082ab22d8d7dbf561f,
  author = {C. Ievers-Landis and A. Storfer‐Isser and C. Rosen and N. Johnson and S. Redline},
  title = {Relationship of Sleep Parameters, Child Psychological Functioning, and Parenting Stress to Obesity Status Among Preadolescent Children},
  journal = {Journal of Developmental & Behavioral Pediatrics},
  year = {2008},
  volume = {29},
  pages = {243-252},
  doi = {10.1097/DBP.0b013e31816d923d},
  abstract = {Objective: Insufficient sleep may be a significant contributing factor to the increase in pediatric obesity and thus may also contribute to adult obesity and chronic illness. Previous research has been based on large survey studies with consideration of demographics and lifestyle factors (e.g., snacking and TV watching) but not of child psychological/behavioral functioning and parenting factors. Method: This study investigated the relationship of sleep duration to obesity status in 819 children ages 8 to 11 years old, with consideration of demographics, clinical elevations in child psychological/behavioral functioning, and parenting stress. Results: In unadjusted and adjusted analyses, parent-reported child sleep duration was significantly associated with the odds of obesity with an increase of 41% for each 1-hour reduction in sleep duration. In addition to sleep duration, only median neighborhood income was significantly related to obesity status. Indices of child psychological/behavioral functioning and parenting stress were associated with sleep duration but not with obesity, and adjusting for these behavioral and parenting characteristics did not appreciably alter the relationship between sleep duration and obesity status. Exploratory gender-specific analyses found that mean sleep duration was significantly associated with the odds of obesity for boys but not for girls. Conclusions: These results show that the relationship of shorter sleep duration to a greater likelihood of being obese persists even after adjusting for potential confounders of child psychological/behavioral functioning and parenting stress. Gender-specific associations are similar to findings reported in samples that include adolescents.}
}

RIS

TY  - JOUR
AU  - C. Ievers-Landis
AU  - A. Storfer‐Isser
AU  - C. Rosen
AU  - N. Johnson
AU  - S. Redline
T1  - Relationship of Sleep Parameters, Child Psychological Functioning, and Parenting Stress to Obesity Status Among Preadolescent Children
JO  - Journal of Developmental & Behavioral Pediatrics
PY  - 2008
VL  - 29
SP  - 243-252
EP  - 243-252
DO  - 10.1097/DBP.0b013e31816d923d
AB  - Objective: Insufficient sleep may be a significant contributing factor to the increase in pediatric obesity and thus may also contribute to adult obesity and chronic illness. Previous research has been based on large survey studies with consideration of demographics and lifestyle factors (e.g., snacking and TV watching) but not of child psychological/behavioral functioning and parenting factors. Method: This study investigated the relationship of sleep duration to obesity status in 819 children ages 8 to 11 years old, with consideration of demographics, clinical elevations in child psychological/behavioral functioning, and parenting stress. Results: In unadjusted and adjusted analyses, parent-reported child sleep duration was significantly associated with the odds of obesity with an increase of 41% for each 1-hour reduction in sleep duration. In addition to sleep duration, only median neighborhood income was significantly related to obesity status. Indices of child psychological/behavioral functioning and parenting stress were associated with sleep duration but not with obesity, and adjusting for these behavioral and parenting characteristics did not appreciably alter the relationship between sleep duration and obesity status. Exploratory gender-specific analyses found that mean sleep duration was significantly associated with the odds of obesity for boys but not for girls. Conclusions: These results show that the relationship of shorter sleep duration to a greater likelihood of being obese persists even after adjusting for potential confounders of child psychological/behavioral functioning and parenting stress. Gender-specific associations are similar to findings reported in samples that include adolescents.
ER  - 

BibTeX

@misc{caf26bffc40054205a31a17d32c82ebe9d90b8cc,
  author = {K. Spiegel and E. Tasali and P. Penev and E. Cauter},
  title = {Brief Communication: Sleep Curtailment in Healthy Young Men Is Associated with Decreased Leptin Levels, Elevated Ghrelin Levels, and Increased Hunger and Appetite},
  journal = {Annals of Internal Medicine},
  year = {2004},
  volume = {141},
  pages = {846-850},
  doi = {10.7326/0003-4819-141-11-200412070-00008},
  abstract = {Context Studies in animals and humans suggest that sleep duration is an important regulator of metabolism. Contribution In this study, 12 young, healthy, normal-weight men exhibited reductions in the satiety hormone leptin, increases in the hunger hormone ghrelin, and increases in hunger after 2 nights of only 4 hours of sleep compared with after 2 nights of 10 hours of sleep. Implications Inadequate sleep seems to influence the hormones that regulate satiety and hunger in a way that could promote excess eating. The Editors Sleep plays an important role in energy balance. In rodents, food shortage or starvation results in decreased sleep (1), and, conversely, total sleep deprivation leads to marked hyperphagia (2). Leptin and ghrelin are peripheral signals that contribute to the central regulation of food intake. Leptin, a hormone released by the adipocytes, provides information about energy status to hypothalamic regulatory centers (3). In humans, circulating leptin levels rapidly decrease or increase in response to acute caloric shortage or surplus, respectively (4). These changes in leptin levels have been associated with reciprocal changes in hunger (4). Ghrelin, a peptide produced predominantly by the stomach, is also involved in energy balance regulation, but, in contrast to the anorexigenic effects of leptin, ghrelin stimulates appetite (5). It has been proposed that leptin and ghrelin represent the yinyang of one regulatory system that has developed to inform the brain about the current energy balance state (6). Over the past 40 years, sleep duration in the U.S. population has decreased by 1 to 2 hours (7-10). The proportion of young adults sleeping fewer than 7 hours per night has more than doubled between 1960 and 20012002 (from 15.6% to 37.1%) (7-10). The effect of sleep curtailment on the control of appetite and food intake is not known. Because of the well-documented associations between sleep and food intake (1, 2), we sought to determine whether sleep duration influences the daytime profiles of leptin and ghrelin. Methods Participants Twelve healthy men(mean age [SD], 22 2 years]; mean body mass index [SD], 23.6 2.0 kg/m2) who did not smoke or take any medications participated in the study. All of the men were within 10% of ideal body weight and had regular nocturnal time in bed of 7 to 9 hours. We excluded persons who had traveled across time zones less than 4 weeks before the study. Experimental Protocol The Institutional Review Board of the University of Chicago approved the protocol, and we obtained written informed consent from all participants. During the week preceding each study, we asked participants not to deviate from a fixed time in bed (11:00 p.m. to 7:00 a.m.) by more than 30 minutes. Naps were not allowed. The men participated in 2 studies that were conducted in a randomized order, were spaced at least 6 weeks apart, and were performed in the Clinical Research Center at the University of Chicago, Chicago, Illinois. Six of the 12 men first performed the study with restricted time in bed, and the remaining 6 men first performed the study with extended time in bed. Average weight did not change over the time period separating the 2 study conditions (75.2 kg in the sleep restriction condition vs. 75.4 kg in the sleep extension condition; P> 0.2). We obtained blood samples at 20-minute intervals from 8:00 a.m. to 9:00 p.m. after 2 consecutive nights of 10 hours in bed (10:00 p.m. to 8:00 a.m.; sleep extension) and after 2 consecutive nights of 4 hours in bed (1:00 a.m. to 5:00 a.m.; sleep restriction). Sleep was recorded every night. For both extension and restriction conditions, each overnight stay began at 7:00 p.m. with a standard hospital dinner, and the first overnight stay ended after breakfast, which was served at 8:00 a.m. We instructed the participants not to deviate from their usual eating habits between breakfast and dinner, but caloric intake was not otherwise monitored. Participants were readmitted in the early evening and, after receiving a standard hospital dinner at 7:00 p.m., remained at bed rest. At 8:00 a.m. after the second night, the participants' caloric intake was kept constant to avoid meal-related fluctuations of hunger and satiety and consisted of an intravenous glucose infusion at a constant rate of 5 g/kg of body weight every 24 hours. There was no other source of calories. Every hour from 9:00 a.m. to 9:00 p.m., the men completed validated visual analogue scales (0 to 10 cm) for hunger (11) and appetite for various food categories (12). To assess hunger, we asked participants to mark their response to the question How hungry do you feel right now? on a 10-cm scale (with not at all hungry on the left and extremely hungry on the right). To assess appetite, we asked participants to mark their response to how much they would enjoy eating foods from 7 different food categories on a 10-cm scale (with not at all on the left and very much on the right). They were asked to provide a score based only on their appetite at the moment, without concern for calories, fat, or a healthy diet. The food categories were sweets (such as cake, candy, cookies, ice cream, and pastry); salty foods (such as chips, salted nuts, pickles, and olives); starchy foods (such as bread, pasta, cereal, and potatoes); fruits and fruit juices; vegetables; meat, poultry, fish, and eggs; and dairy products (such as milk, cheese, and yogurt). Assays We measured serum leptin levels in all samples by using a human leptin radioimmunoassay kit (Linco Research, St. Charles, Missouri) with a sensitivity of 0.5 ng/mL and an intra-assay coefficient of variation of 8.3%. In 9 of the 12 participants, we also measured total ghrelin levels at hourly intervals by radioimmunoassay (Linco Research) with a sensitivity of 0.5 ng/mL and an intra-assay coefficient of variation of 7.9%. Statistical Analysis We performed paired comparisons by using the Wilcoxon matched-pairs signed-rank test. We calculated correlations by using the Spearman coefficient. The mean relative changes in leptin, ghrelin, hunger, and appetite between extended sleep (the reference category) and restricted sleep were calculated by using the ratios of the corresponding individual data and then deriving the mean across all individuals. Role of the Funding Sources This work was partially supported by grants from the National Institutes of Health, the European Sleep Research Society, the Belgian Fonds de la Recherche Scientifique Mdicale, the University of Chicago Diabetes Research and Training Grant, and The University of Chicago General Clinical Research Center. The funding sources had no role in the design, conduct, and reporting of the study or in the decision to submit the manuscript for publication. Results Leptin levels were stable across the daytime period under both sleep conditions, which was consistent with the fact that calories were exclusively delivered in the form of a constant glucose infusion. Average total sleep time was 9 hours and 8 minutes when the men spent 10 hours in bed and 3 hours and 53 minutes when the men spent 4 hours in bed (P< 0.01). When spending 4 hours in bed, the participants had mean leptin levels that were 18% lower (2.1 ng/mL vs. 2.6 ng/mL; P= 0.04) (Figure 1, part A) and mean ghrelin levels that were 28% higher (3.3 ng/mL vs. 2.6 ng/mL; P= 0.04) (Figure 1, part B) than when the participants spent 10 hours in bed. The ratio of the concentrations of orexigenic ghrelin to anorexigenic leptin increased by 71% (CI, 7% to 135%) with 4 hours in bed compared with 10 hours in bed. Sleep restriction relative to sleep extension was associated with a 24% increase in hunger ratings on the 10-cm visual analogue scale (P< 0.01) and a 23% increase in appetite ratings for all food categories combined (P= 0.01) (Figure 1, parts C and D, and Table 1). The increase in appetite tended to be greatest for calorie-dense foods with high carbohydrate content (sweets, salty foods, and starchy foods: increase, 33% to 45%; P= 0.06) (Table 1). The increase in appetite for fruits and vegetables was less consistent and of lesser magnitude (increase, 17% to 21%) (Table 1). Appetite for protein-rich nutrients (meat, poultry, fish, eggs, and dairy foods) was not significantly affected by sleep duration (Table 1). When we considered the changes in ghrelin and leptin in an integrated fashion by calculating the ghrelin-to-leptin ratio, the increase in hunger was proportional to the increase in ghrelin-to-leptin ratio (r= 0.87) (Figure 2). Almost 70% of the variance in increased hunger could be accounted for by the increase in the ghrelin-to-leptin ratio. Figure 1. Effect of sleep duration on daytime leptin levels, ghrelin levels, hunger, and appetite. A. B. C D C D Table. Average Ratings of Appetite after 2 Days of Sleep Restriction or Sleep Extension Figure 2. Association between the change in hunger ratings and the change in ghrelin-to-leptin ratio during the 12:00 noon to 9:00 p.m. time period when sleep is restricted as compared with extended. P Discussion We observed that sleep duration may affect the circulating levels of neuroendocrine factors that regulate hunger and appetite. Two days with 4 hours of time in bed each night were associated with an 18% decrease in the levels of the anorexigenic hormone leptin. By comparison, 3 days of underfeeding by approximately 900 calories per day in healthy lean volunteers has been reported to result in a decrease of leptin levels averaging 22% (4). Sleep curtailment was also associated with an almost 28% increase in daytime levels of the orexigenic factor ghrelin. The reciprocal changes in leptin and ghrelin that we observed in response to sleep restriction were associated with a 24% increase in hunger and a 23% increase in appetite. Appetite for calorie-dense nutrients with high carbohydrate content, including sweets, salty snacks, and starchy foods, increased by 33% to 45%. In contrast, appetite }
}

RIS

TY  - JOUR
AU  - K. Spiegel
AU  - E. Tasali
AU  - P. Penev
AU  - E. Cauter
T1  - Brief Communication: Sleep Curtailment in Healthy Young Men Is Associated with Decreased Leptin Levels, Elevated Ghrelin Levels, and Increased Hunger and Appetite
JO  - Annals of Internal Medicine
PY  - 2004
VL  - 141
SP  - 846-850
EP  - 846-850
DO  - 10.7326/0003-4819-141-11-200412070-00008
AB  - Context Studies in animals and humans suggest that sleep duration is an important regulator of metabolism. Contribution In this study, 12 young, healthy, normal-weight men exhibited reductions in the satiety hormone leptin, increases in the hunger hormone ghrelin, and increases in hunger after 2 nights of only 4 hours of sleep compared with after 2 nights of 10 hours of sleep. Implications Inadequate sleep seems to influence the hormones that regulate satiety and hunger in a way that could promote excess eating. The Editors Sleep plays an important role in energy balance. In rodents, food shortage or starvation results in decreased sleep (1), and, conversely, total sleep deprivation leads to marked hyperphagia (2). Leptin and ghrelin are peripheral signals that contribute to the central regulation of food intake. Leptin, a hormone released by the adipocytes, provides information about energy status to hypothalamic regulatory centers (3). In humans, circulating leptin levels rapidly decrease or increase in response to acute caloric shortage or surplus, respectively (4). These changes in leptin levels have been associated with reciprocal changes in hunger (4). Ghrelin, a peptide produced predominantly by the stomach, is also involved in energy balance regulation, but, in contrast to the anorexigenic effects of leptin, ghrelin stimulates appetite (5). It has been proposed that leptin and ghrelin represent the yinyang of one regulatory system that has developed to inform the brain about the current energy balance state (6). Over the past 40 years, sleep duration in the U.S. population has decreased by 1 to 2 hours (7-10). The proportion of young adults sleeping fewer than 7 hours per night has more than doubled between 1960 and 20012002 (from 15.6% to 37.1%) (7-10). The effect of sleep curtailment on the control of appetite and food intake is not known. Because of the well-documented associations between sleep and food intake (1, 2), we sought to determine whether sleep duration influences the daytime profiles of leptin and ghrelin. Methods Participants Twelve healthy men(mean age [SD], 22 2 years]; mean body mass index [SD], 23.6 2.0 kg/m2) who did not smoke or take any medications participated in the study. All of the men were within 10% of ideal body weight and had regular nocturnal time in bed of 7 to 9 hours. We excluded persons who had traveled across time zones less than 4 weeks before the study. Experimental Protocol The Institutional Review Board of the University of Chicago approved the protocol, and we obtained written informed consent from all participants. During the week preceding each study, we asked participants not to deviate from a fixed time in bed (11:00 p.m. to 7:00 a.m.) by more than 30 minutes. Naps were not allowed. The men participated in 2 studies that were conducted in a randomized order, were spaced at least 6 weeks apart, and were performed in the Clinical Research Center at the University of Chicago, Chicago, Illinois. Six of the 12 men first performed the study with restricted time in bed, and the remaining 6 men first performed the study with extended time in bed. Average weight did not change over the time period separating the 2 study conditions (75.2 kg in the sleep restriction condition vs. 75.4 kg in the sleep extension condition; P> 0.2). We obtained blood samples at 20-minute intervals from 8:00 a.m. to 9:00 p.m. after 2 consecutive nights of 10 hours in bed (10:00 p.m. to 8:00 a.m.; sleep extension) and after 2 consecutive nights of 4 hours in bed (1:00 a.m. to 5:00 a.m.; sleep restriction). Sleep was recorded every night. For both extension and restriction conditions, each overnight stay began at 7:00 p.m. with a standard hospital dinner, and the first overnight stay ended after breakfast, which was served at 8:00 a.m. We instructed the participants not to deviate from their usual eating habits between breakfast and dinner, but caloric intake was not otherwise monitored. Participants were readmitted in the early evening and, after receiving a standard hospital dinner at 7:00 p.m., remained at bed rest. At 8:00 a.m. after the second night, the participants' caloric intake was kept constant to avoid meal-related fluctuations of hunger and satiety and consisted of an intravenous glucose infusion at a constant rate of 5 g/kg of body weight every 24 hours. There was no other source of calories. Every hour from 9:00 a.m. to 9:00 p.m., the men completed validated visual analogue scales (0 to 10 cm) for hunger (11) and appetite for various food categories (12). To assess hunger, we asked participants to mark their response to the question How hungry do you feel right now? on a 10-cm scale (with not at all hungry on the left and extremely hungry on the right). To assess appetite, we asked participants to mark their response to how much they would enjoy eating foods from 7 different food categories on a 10-cm scale (with not at all on the left and very much on the right). They were asked to provide a score based only on their appetite at the moment, without concern for calories, fat, or a healthy diet. The food categories were sweets (such as cake, candy, cookies, ice cream, and pastry); salty foods (such as chips, salted nuts, pickles, and olives); starchy foods (such as bread, pasta, cereal, and potatoes); fruits and fruit juices; vegetables; meat, poultry, fish, and eggs; and dairy products (such as milk, cheese, and yogurt). Assays We measured serum leptin levels in all samples by using a human leptin radioimmunoassay kit (Linco Research, St. Charles, Missouri) with a sensitivity of 0.5 ng/mL and an intra-assay coefficient of variation of 8.3%. In 9 of the 12 participants, we also measured total ghrelin levels at hourly intervals by radioimmunoassay (Linco Research) with a sensitivity of 0.5 ng/mL and an intra-assay coefficient of variation of 7.9%. Statistical Analysis We performed paired comparisons by using the Wilcoxon matched-pairs signed-rank test. We calculated correlations by using the Spearman coefficient. The mean relative changes in leptin, ghrelin, hunger, and appetite between extended sleep (the reference category) and restricted sleep were calculated by using the ratios of the corresponding individual data and then deriving the mean across all individuals. Role of the Funding Sources This work was partially supported by grants from the National Institutes of Health, the European Sleep Research Society, the Belgian Fonds de la Recherche Scientifique Mdicale, the University of Chicago Diabetes Research and Training Grant, and The University of Chicago General Clinical Research Center. The funding sources had no role in the design, conduct, and reporting of the study or in the decision to submit the manuscript for publication. Results Leptin levels were stable across the daytime period under both sleep conditions, which was consistent with the fact that calories were exclusively delivered in the form of a constant glucose infusion. Average total sleep time was 9 hours and 8 minutes when the men spent 10 hours in bed and 3 hours and 53 minutes when the men spent 4 hours in bed (P< 0.01). When spending 4 hours in bed, the participants had mean leptin levels that were 18% lower (2.1 ng/mL vs. 2.6 ng/mL; P= 0.04) (Figure 1, part A) and mean ghrelin levels that were 28% higher (3.3 ng/mL vs. 2.6 ng/mL; P= 0.04) (Figure 1, part B) than when the participants spent 10 hours in bed. The ratio of the concentrations of orexigenic ghrelin to anorexigenic leptin increased by 71% (CI, 7% to 135%) with 4 hours in bed compared with 10 hours in bed. Sleep restriction relative to sleep extension was associated with a 24% increase in hunger ratings on the 10-cm visual analogue scale (P< 0.01) and a 23% increase in appetite ratings for all food categories combined (P= 0.01) (Figure 1, parts C and D, and Table 1). The increase in appetite tended to be greatest for calorie-dense foods with high carbohydrate content (sweets, salty foods, and starchy foods: increase, 33% to 45%; P= 0.06) (Table 1). The increase in appetite for fruits and vegetables was less consistent and of lesser magnitude (increase, 17% to 21%) (Table 1). Appetite for protein-rich nutrients (meat, poultry, fish, eggs, and dairy foods) was not significantly affected by sleep duration (Table 1). When we considered the changes in ghrelin and leptin in an integrated fashion by calculating the ghrelin-to-leptin ratio, the increase in hunger was proportional to the increase in ghrelin-to-leptin ratio (r= 0.87) (Figure 2). Almost 70% of the variance in increased hunger could be accounted for by the increase in the ghrelin-to-leptin ratio. Figure 1. Effect of sleep duration on daytime leptin levels, ghrelin levels, hunger, and appetite. A. B. C D C D Table. Average Ratings of Appetite after 2 Days of Sleep Restriction or Sleep Extension Figure 2. Association between the change in hunger ratings and the change in ghrelin-to-leptin ratio during the 12:00 noon to 9:00 p.m. time period when sleep is restricted as compared with extended. P Discussion We observed that sleep duration may affect the circulating levels of neuroendocrine factors that regulate hunger and appetite. Two days with 4 hours of time in bed each night were associated with an 18% decrease in the levels of the anorexigenic hormone leptin. By comparison, 3 days of underfeeding by approximately 900 calories per day in healthy lean volunteers has been reported to result in a decrease of leptin levels averaging 22% (4). Sleep curtailment was also associated with an almost 28% increase in daytime levels of the orexigenic factor ghrelin. The reciprocal changes in leptin and ghrelin that we observed in response to sleep restriction were associated with a 24% increase in hunger and a 23% increase in appetite. Appetite for calorie-dense nutrients with high carbohydrate content, including sweets, salty snacks, and starchy foods, increased by 33% to 45%. In contrast, appetite 
ER  - 

BibTeX

@misc{7151a9a7523fba78023350f42ee983b8989d8f4b,
  author = {Zumin Shi and Anne W. Taylor and T. Gill and J. Tuckerman and R. Adams and James Martin},
  title = {Short sleep duration and obesity among},
  journal = {},
  year = {2010},
  volume = {},
  abstract = {Background: There is limited information on sleep duration and obesity among Australian children. The objective of the study is to cross-sectionally examine the relationship between sleep duration and obesity in Australian children aged 5 to 15 years. Methods: Data were collected using the South Australian Monitoring and Surveillance System between January 2004 and December 2008. Each month a representative random sample of South Australians are selected from the Electronic White Pages with interviews conducted using Computer Assisted Telephone Interviewing (CATI). Within each household, the person who was last to have a birthday was selected for interview. Parents reported the number of hours their children slept each day. Obesity was defined according to the International Obesity Task Force (IOTF) definition based on BMI calculated from reported body weight and height. Results: Overall, parents of 3495 children aged 5-15 years (mean 10.7 years, 50.3% boys) were interviewed. The prevalence of obesity was 7.7% (8.9% in boys, 6.6% in girls). In multivariate analysis after adjusting for sociodemographic variables, intake of fruit and vegetables, physical activity and inactivity, the odds ratio (OR) for obesity comparing sleeping <9 hours with ≥10 hours was 2.23 (95% CI 1.04-4.76) among boys, 1.70(0.78-3.73) among girls, and 1.97(1.15-3.38) in both genders. The association between short sleep (<9 hours) and obesity was stronger in the younger age group. No significant association between short sleep and obesity was found among children aged 13-15. There was also an additive interaction between short sleep and low level of physical activity. Conclusion: Short sleep duration is associated with increased obesity in children especially among younger age groups and boys.}
}

RIS

TY  - JOUR
AU  - Zumin Shi
AU  - Anne W. Taylor
AU  - T. Gill
AU  - J. Tuckerman
AU  - R. Adams
AU  - James Martin
T1  - Short sleep duration and obesity among
PY  - 2010
AB  - Background: There is limited information on sleep duration and obesity among Australian children. The objective of the study is to cross-sectionally examine the relationship between sleep duration and obesity in Australian children aged 5 to 15 years. Methods: Data were collected using the South Australian Monitoring and Surveillance System between January 2004 and December 2008. Each month a representative random sample of South Australians are selected from the Electronic White Pages with interviews conducted using Computer Assisted Telephone Interviewing (CATI). Within each household, the person who was last to have a birthday was selected for interview. Parents reported the number of hours their children slept each day. Obesity was defined according to the International Obesity Task Force (IOTF) definition based on BMI calculated from reported body weight and height. Results: Overall, parents of 3495 children aged 5-15 years (mean 10.7 years, 50.3% boys) were interviewed. The prevalence of obesity was 7.7% (8.9% in boys, 6.6% in girls). In multivariate analysis after adjusting for sociodemographic variables, intake of fruit and vegetables, physical activity and inactivity, the odds ratio (OR) for obesity comparing sleeping <9 hours with ≥10 hours was 2.23 (95% CI 1.04-4.76) among boys, 1.70(0.78-3.73) among girls, and 1.97(1.15-3.38) in both genders. The association between short sleep (<9 hours) and obesity was stronger in the younger age group. No significant association between short sleep and obesity was found among children aged 13-15. There was also an additive interaction between short sleep and low level of physical activity. Conclusion: Short sleep duration is associated with increased obesity in children especially among younger age groups and boys.
ER  - 

BibTeX

@misc{ccb3f73b67f62845c4f88c3f95ceb33e0ab180ae,
  author = {G. Beccuti and S. Pannain},
  title = {Sleep and obesity},
  journal = {Current Opinion in Clinical Nutrition and Metabolic Care},
  year = {2011},
  volume = {14},
  pages = {402–412},
  doi = {10.1097/MCO.0b013e3283479109},
  abstract = {Purpose of reviewThis review summarizes the most recent evidence linking decreased sleep duration and poor sleep quality to obesity, focusing upon studies in adults. Recent findingsPublished and unpublished health examination surveys and epidemiological studies suggest that the worldwide prevalence of obesity has doubled since 1980. In 2008, 1 in 10 adults was obese, with women more likely to be obese than men. This obesity epidemic has been paralleled by a trend of reduced sleep duration. Poor sleep quality, which leads to overall sleep loss has also become a frequent complaint. Growing evidence from both laboratory and epidemiological studies points to short sleep duration and poor sleep quality as new risk factors for the development of obesity. SummarySleep is an important modulator of neuroendocrine function and glucose metabolism and sleep loss has been shown to result in metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. Recent epidemiological and laboratory evidence confirm previous findings of an association between sleep loss and increased risk of obesity.}
}

RIS

TY  - JOUR
AU  - G. Beccuti
AU  - S. Pannain
T1  - Sleep and obesity
JO  - Current Opinion in Clinical Nutrition and Metabolic Care
PY  - 2011
VL  - 14
SP  - 402–412
EP  - 402–412
DO  - 10.1097/MCO.0b013e3283479109
AB  - Purpose of reviewThis review summarizes the most recent evidence linking decreased sleep duration and poor sleep quality to obesity, focusing upon studies in adults. Recent findingsPublished and unpublished health examination surveys and epidemiological studies suggest that the worldwide prevalence of obesity has doubled since 1980. In 2008, 1 in 10 adults was obese, with women more likely to be obese than men. This obesity epidemic has been paralleled by a trend of reduced sleep duration. Poor sleep quality, which leads to overall sleep loss has also become a frequent complaint. Growing evidence from both laboratory and epidemiological studies points to short sleep duration and poor sleep quality as new risk factors for the development of obesity. SummarySleep is an important modulator of neuroendocrine function and glucose metabolism and sleep loss has been shown to result in metabolic and endocrine alterations, including decreased glucose tolerance, decreased insulin sensitivity, increased evening concentrations of cortisol, increased levels of ghrelin, decreased levels of leptin, and increased hunger and appetite. Recent epidemiological and laboratory evidence confirm previous findings of an association between sleep loss and increased risk of obesity.
ER  - 

BibTeX

@misc{95898418c550a91bdb6cfbd03e72e8ea8c4866b7,
  author = {M. Grandner},
  title = {Abstract P344: Sleep Duration and Obesity: Impact of Demographics, Socioeconomic Status, Health Behaviors, and Health Status},
  journal = {Circulation},
  year = {2018},
  volume = {},
  abstract = {Introduction: Many studies have shown that sleep duration is associated with obesity. It is unclear, though, whether this relationship exists equally across groups and whether it depends on demogra...}
}

RIS

TY  - JOUR
AU  - M. Grandner
T1  - Abstract P344: Sleep Duration and Obesity: Impact of Demographics, Socioeconomic Status, Health Behaviors, and Health Status
JO  - Circulation
PY  - 2018
AB  - Introduction: Many studies have shown that sleep duration is associated with obesity. It is unclear, though, whether this relationship exists equally across groups and whether it depends on demogra...
ER  - 

BibTeX

@misc{93fd0383ee589552594e50de5768a797947d8464,
  author = {S. Taheri},
  title = {The Interactions Between Sleep , Metabolism , and Obesity},
  year = {2007},
  abstract = {20 The precise physiological functions of sleep remain to be determined, but it is increasingly being recognized that sleep plays a significant role in maintaining our physical and psychological well-being [1]. This is because several large epidemiological studies have identified important contributions of sleep to various health outcomes and mortality. Simultaneously, there has been a shift in sleep research from concentrating on the brain and neurocognitive effects of sleep loss to investigating the impact of sleep on other organs and on global physiology. This has been accompanied by more “real-life” experimental paradigms aiming to unravel the mechanisms involved, i.e. studies on the effects of chronic partial sleep loss instead of acute total sleep deprivation. In addition, there is now substantial interest in determining the characteristics of individuals who lie at the extremes of sleep duration: the “short” and “long” sleepers [2]. These simultaneous advances in sleep research have highlighted the possibility that alterations in sleep duration could result in metabolic changes that may contribute to the development of obesity, insulin resistance, and cardiovascular disease [3–24]. The objective of this review is to examine the evidence for an interaction between sleep duration and obesity and to discuss the potential metabolic mechanisms involved. The burden of obesity Obesity is a global public health problem [25]. A recent report from the World Health Organization estimated that, in 2005, >1 billion people worldwide were overweight and >300 million were obese [26]. The report forecasts that the number of overweight individuals will reach 1.5 billion by 2015. In the US and other western countries, obesity is expected to become the most common preventable cause of death [27]. Most alarming has been a dramatic rise in the number of children who fit the criteria necessary for the diagnosis of obesity, not least because childhood obesity tracks into adulthood [28]. Data from the Center for Disease Control and Prevention in the US show that the prevalence of children aged 6–19 years old who were considered to be overweight increased from 4–5% in 1963–1970 to 15% in 1999–2000 [29]. Major contributors to the morbidity and mortality associated with obesity include concomitant insulin resistance, type 2 diabetes mellitus, sleep-disordered breathing, and cardiovascular disease [25]. Although there is a strong genetic contribution to obesity, it is believed that the current obesity pandemic is largely driven by environmental factors that alter the balance between energy intake and energy expenditure. Unfortunately, current interventions aimed at altering food selection (with different diets encouraging alterations in different macronutrients) and calorie intake (e.g. smaller portions), and increasing physical activity, have not resulted in long-term weight loss and maintenance. This is because our understanding of factors that influence individuals to choose and over-consume The Interactions Between Sleep, Metabolism, and Obesity}
}

RIS

TY  - JOUR
AU  - S. Taheri
T1  - The Interactions Between Sleep , Metabolism , and Obesity
PY  - 2007
AB  - 20 The precise physiological functions of sleep remain to be determined, but it is increasingly being recognized that sleep plays a significant role in maintaining our physical and psychological well-being [1]. This is because several large epidemiological studies have identified important contributions of sleep to various health outcomes and mortality. Simultaneously, there has been a shift in sleep research from concentrating on the brain and neurocognitive effects of sleep loss to investigating the impact of sleep on other organs and on global physiology. This has been accompanied by more “real-life” experimental paradigms aiming to unravel the mechanisms involved, i.e. studies on the effects of chronic partial sleep loss instead of acute total sleep deprivation. In addition, there is now substantial interest in determining the characteristics of individuals who lie at the extremes of sleep duration: the “short” and “long” sleepers [2]. These simultaneous advances in sleep research have highlighted the possibility that alterations in sleep duration could result in metabolic changes that may contribute to the development of obesity, insulin resistance, and cardiovascular disease [3–24]. The objective of this review is to examine the evidence for an interaction between sleep duration and obesity and to discuss the potential metabolic mechanisms involved. The burden of obesity Obesity is a global public health problem [25]. A recent report from the World Health Organization estimated that, in 2005, >1 billion people worldwide were overweight and >300 million were obese [26]. The report forecasts that the number of overweight individuals will reach 1.5 billion by 2015. In the US and other western countries, obesity is expected to become the most common preventable cause of death [27]. Most alarming has been a dramatic rise in the number of children who fit the criteria necessary for the diagnosis of obesity, not least because childhood obesity tracks into adulthood [28]. Data from the Center for Disease Control and Prevention in the US show that the prevalence of children aged 6–19 years old who were considered to be overweight increased from 4–5% in 1963–1970 to 15% in 1999–2000 [29]. Major contributors to the morbidity and mortality associated with obesity include concomitant insulin resistance, type 2 diabetes mellitus, sleep-disordered breathing, and cardiovascular disease [25]. Although there is a strong genetic contribution to obesity, it is believed that the current obesity pandemic is largely driven by environmental factors that alter the balance between energy intake and energy expenditure. Unfortunately, current interventions aimed at altering food selection (with different diets encouraging alterations in different macronutrients) and calorie intake (e.g. smaller portions), and increasing physical activity, have not resulted in long-term weight loss and maintenance. This is because our understanding of factors that influence individuals to choose and over-consume The Interactions Between Sleep, Metabolism, and Obesity
ER  - 

BibTeX

@misc{34a064d990ae1cac9ef2410ef00642ac78c36817,
  author = {L. Hon},
  title = {Sleep duration and obesity in children and adolescents},
  journal = {Chinese Journal of School Health},
  year = {2015},
  volume = {},
  abstract = {Objective To investigate the relationship between sleep duration and obesity in children and adolescents and to provide scientific evidence for prevention and intervention strategies of obesity. Methods A cross-sectional study was conducted in Kunming,Dali,Honghe among 7 200 Han students aged 7 to 18 years by stratified random cluster sampling method. Weight and height were measured. Sleep duration was obtained by self-report. Results The overweight and obesity rates with children and adolescents aged 7 to 18 years was 11. 8% and 5. 7%,respectively. Overweight and obesity rate was significantly higher in boys versus girls,the first and second economic areas versus the third economic area,urban versus rural areas; overweight and obesity rates decreased with age. Overweight and obesity rate was significantly higher in exercise time more than 1 hour. Overweight rate was significantly higher in students with insufficient sleep. Univariate logistic regression analysis revealed that dynamic activity and sleep had effects on overweight or obesity. When demographic,static and dynamic factors were controlled,logistic regression analysis revealed insufficient sleep was risk factor for overweight( OR = 1. 954 in girls and 2. 429 in boys). Conclusion Insufficient sleep is an independent risk factor of overweight among children and adolescents. The prevalence of overweight and obesity should be prevented and controlled by improving the sleep status of children and adolescents.}
}

RIS

TY  - JOUR
AU  - L. Hon
T1  - Sleep duration and obesity in children and adolescents
JO  - Chinese Journal of School Health
PY  - 2015
AB  - Objective To investigate the relationship between sleep duration and obesity in children and adolescents and to provide scientific evidence for prevention and intervention strategies of obesity. Methods A cross-sectional study was conducted in Kunming,Dali,Honghe among 7 200 Han students aged 7 to 18 years by stratified random cluster sampling method. Weight and height were measured. Sleep duration was obtained by self-report. Results The overweight and obesity rates with children and adolescents aged 7 to 18 years was 11. 8% and 5. 7%,respectively. Overweight and obesity rate was significantly higher in boys versus girls,the first and second economic areas versus the third economic area,urban versus rural areas; overweight and obesity rates decreased with age. Overweight and obesity rate was significantly higher in exercise time more than 1 hour. Overweight rate was significantly higher in students with insufficient sleep. Univariate logistic regression analysis revealed that dynamic activity and sleep had effects on overweight or obesity. When demographic,static and dynamic factors were controlled,logistic regression analysis revealed insufficient sleep was risk factor for overweight( OR = 1. 954 in girls and 2. 429 in boys). Conclusion Insufficient sleep is an independent risk factor of overweight among children and adolescents. The prevalence of overweight and obesity should be prevented and controlled by improving the sleep status of children and adolescents.
ER  - 

BibTeX

@misc{33bf17f28732874dade86c1b0ab0e5dd5d80a1f8,
  author = {Patrick M. Krueger and E. Reither and P. Peppard and Andrew E. Burger and L. Hale},
  title = {Cumulative exposure to short sleep and body mass outcomes: a prospective study},
  journal = {Journal of Sleep Research},
  year = {2015},
  volume = {24},
  doi = {10.1111/jsr.12327},
  abstract = {Short sleep duration is associated with excess body mass among adolescents and young adults. The mechanisms theorized to drive that association suggest that persistent exposure to short sleep should be associated with greater accumulations of body mass. We use prospective cohort data from four waves of the National Longitudinal Study of Adolescent to Adult Health (1994–2009; n = 14 800) to examine associations between cumulative exposure to short sleep throughout adolescence and early adulthood and obesity and elevated waist circumference outcomes. We compare several clinical and distribution‐based standards of short sleep to assess which measures are associated most strongly with body mass. Cumulative exposure to short sleep exhibits dose–response associations with obesity and elevated waist circumference. Relative to respondents with no instances of short sleep, those who slept −0.50 standard deviations or less than the age and sex‐specific average sleep hours in all four waves had 1.45 [95% confidence interval (CI): 1.03, 2.04] times the odds of being obese and 1.45 (95% CI: 1.02, 2.06) times the odds of having an elevated waist circumference. Our findings suggest that cumulative exposure to short sleep during adolescence and young adulthood may play an important role in the etiology of obesity and elevated waist circumference during this important developmental period.}
}

RIS

TY  - JOUR
AU  - Patrick M. Krueger
AU  - E. Reither
AU  - P. Peppard
AU  - Andrew E. Burger
AU  - L. Hale
T1  - Cumulative exposure to short sleep and body mass outcomes: a prospective study
JO  - Journal of Sleep Research
PY  - 2015
VL  - 24
DO  - 10.1111/jsr.12327
AB  - Short sleep duration is associated with excess body mass among adolescents and young adults. The mechanisms theorized to drive that association suggest that persistent exposure to short sleep should be associated with greater accumulations of body mass. We use prospective cohort data from four waves of the National Longitudinal Study of Adolescent to Adult Health (1994–2009; n = 14 800) to examine associations between cumulative exposure to short sleep throughout adolescence and early adulthood and obesity and elevated waist circumference outcomes. We compare several clinical and distribution‐based standards of short sleep to assess which measures are associated most strongly with body mass. Cumulative exposure to short sleep exhibits dose–response associations with obesity and elevated waist circumference. Relative to respondents with no instances of short sleep, those who slept −0.50 standard deviations or less than the age and sex‐specific average sleep hours in all four waves had 1.45 [95% confidence interval (CI): 1.03, 2.04] times the odds of being obese and 1.45 (95% CI: 1.02, 2.06) times the odds of having an elevated waist circumference. Our findings suggest that cumulative exposure to short sleep during adolescence and young adulthood may play an important role in the etiology of obesity and elevated waist circumference during this important developmental period.
ER  - 

BibTeX

@misc{04f4ffc4b65f8eb23e09e6e91b239447e5fbf876,
  author = {S. Patel},
  title = {Invited Commentary Invited Commentary : Understanding the Role of Sleep},
  year = {2009},
  abstract = {Chronic sleep deprivation is increasingly entertained as a novel risk factor for obesity. However, the vast majority of studies on this topic have relied on unvalidated subjective measures of habitual sleep habits. The accompanying paper by Lauderdale et al. (Am J Epidemiol. 2009;170(7):805–813) presents the first longitudinal analysis of the relation between sleep duration and weight change by using an objective assessment of sleep. The lack of evidence for an association in this work suggests that the absolute time slept may not be important for weight regulation but raises questions as to what self-reported sleep duration is measuring. One intriguing possibility is that self-reported sleep may reflect the time spent in deeper stages of sleep, which physiologic studies suggest may be more relevant from a metabolic standpoint. Further research into the relation between sleep quantity and quality relative to obesity by use of more refined measures of sleep is needed to identify which, if any, aspects of sleep are important in weight homeostasis.}
}

RIS

TY  - JOUR
AU  - S. Patel
T1  - Invited Commentary Invited Commentary : Understanding the Role of Sleep
PY  - 2009
AB  - Chronic sleep deprivation is increasingly entertained as a novel risk factor for obesity. However, the vast majority of studies on this topic have relied on unvalidated subjective measures of habitual sleep habits. The accompanying paper by Lauderdale et al. (Am J Epidemiol. 2009;170(7):805–813) presents the first longitudinal analysis of the relation between sleep duration and weight change by using an objective assessment of sleep. The lack of evidence for an association in this work suggests that the absolute time slept may not be important for weight regulation but raises questions as to what self-reported sleep duration is measuring. One intriguing possibility is that self-reported sleep may reflect the time spent in deeper stages of sleep, which physiologic studies suggest may be more relevant from a metabolic standpoint. Further research into the relation between sleep quantity and quality relative to obesity by use of more refined measures of sleep is needed to identify which, if any, aspects of sleep are important in weight homeostasis.
ER  - 

BibTeX

@misc{d37239065b94fa99c7faf6e9fd6a6c92cf298f2d,
  author = {S. Patel},
  title = {Invited Commentary: understanding the role of sleep.},
  journal = {American journal of epidemiology},
  year = {2009},
  volume = {170 7},
  pages = {
          814-6; discussion 817-8
        },
  doi = {10.1093/aje/kwp228},
  abstract = {Chronic sleep deprivation is increasingly entertained as a novel risk factor for obesity. However, the vast majority of studies on this topic have relied on unvalidated subjective measures of habitual sleep habits. The accompanying paper by Lauderdale et al. (Am J Epidemiol. 2009;170(7):805-813) presents the first longitudinal analysis of the relation between sleep duration and weight change by using an objective assessment of sleep. The lack of evidence for an association in this work suggests that the absolute time slept may not be important for weight regulation but raises questions as to what self-reported sleep duration is measuring. One intriguing possibility is that self-reported sleep may reflect the time spent in deeper stages of sleep, which physiologic studies suggest may be more relevant from a metabolic standpoint. Further research into the relation between sleep quantity and quality relative to obesity by use of more refined measures of sleep is needed to identify which, if any, aspects of sleep are important in weight homeostasis.}
}

RIS

TY  - JOUR
AU  - S. Patel
T1  - Invited Commentary: understanding the role of sleep.
JO  - American journal of epidemiology
PY  - 2009
VL  - 170 7
SP  - 
          814-6; discussion 817-8
        
EP  - 
          814-6; discussion 817-8
        
DO  - 10.1093/aje/kwp228
AB  - Chronic sleep deprivation is increasingly entertained as a novel risk factor for obesity. However, the vast majority of studies on this topic have relied on unvalidated subjective measures of habitual sleep habits. The accompanying paper by Lauderdale et al. (Am J Epidemiol. 2009;170(7):805-813) presents the first longitudinal analysis of the relation between sleep duration and weight change by using an objective assessment of sleep. The lack of evidence for an association in this work suggests that the absolute time slept may not be important for weight regulation but raises questions as to what self-reported sleep duration is measuring. One intriguing possibility is that self-reported sleep may reflect the time spent in deeper stages of sleep, which physiologic studies suggest may be more relevant from a metabolic standpoint. Further research into the relation between sleep quantity and quality relative to obesity by use of more refined measures of sleep is needed to identify which, if any, aspects of sleep are important in weight homeostasis.
ER  - 

BibTeX

@misc{a21f1784dda1587b35dd926a63a151e4dca4bf0a,
  author = {Kisian C Guimarães and C. M. Silva and Carolina de Oliveira Cruz Latorraca and Ricardo de Ávila Oliveira and C. A. Crispim},
  title = {Is self-reported short sleep duration associated with obesity? A systematic review and meta-analysis of cohort studies.},
  journal = {Nutrition reviews},
  year = {2021},
  doi = {10.1093/nutrit/nuab064},
  abstract = {CONTEXT
A possible association between self-reported short sleep duration and risk of obesity has been studied.


OBJECTIVE
To analyze the association between sleep duration and obesity.


METHODS
The LILACS, Medline, Central, Embase, and OpenGrey databases were searched from inception until July 2020. Two authors screened the studies independently according to the PECO strategy, as follows: participants: > 18 years old; exposure: short sleep duration; control: regular sleep; outcome: obesity). Only cohort studies were included. A total of 3286 studies were retrieved with the search strategy, but only 36 were included. Disagreements were resolved by a third author. The quality of studies was assessed with Newcastle-Ottawa Quality Assessment Form for Cohort Studies. The certainty of the evidence was assessed using Grading of Recommendations Assessment, Development, and Evaluation.


RESULTS
Thirty-six studies were included, and 22 contributed quantitative data. Most of the studies (n = 27) assessed sleep by self-report. The meta-analysis showed a significant association between self-reported short sleep and development of obesity, and the chances of developing obesity increased when self-reported sleep duration decreased.


CONCLUSIONS
Self-reported short sleep was significantly associated with a higher incidence of obesity, with moderate quality of evidence.


SYSTEMATIC REVIEW REGISTRATION
PROSPERO registration no. CRD42019130143.}
}

RIS

TY  - JOUR
AU  - Kisian C Guimarães
AU  - C. M. Silva
AU  - Carolina de Oliveira Cruz Latorraca
AU  - Ricardo de Ávila Oliveira
AU  - C. A. Crispim
T1  - Is self-reported short sleep duration associated with obesity? A systematic review and meta-analysis of cohort studies.
JO  - Nutrition reviews
PY  - 2021
DO  - 10.1093/nutrit/nuab064
AB  - CONTEXT
A possible association between self-reported short sleep duration and risk of obesity has been studied.


OBJECTIVE
To analyze the association between sleep duration and obesity.


METHODS
The LILACS, Medline, Central, Embase, and OpenGrey databases were searched from inception until July 2020. Two authors screened the studies independently according to the PECO strategy, as follows: participants: > 18 years old; exposure: short sleep duration; control: regular sleep; outcome: obesity). Only cohort studies were included. A total of 3286 studies were retrieved with the search strategy, but only 36 were included. Disagreements were resolved by a third author. The quality of studies was assessed with Newcastle-Ottawa Quality Assessment Form for Cohort Studies. The certainty of the evidence was assessed using Grading of Recommendations Assessment, Development, and Evaluation.


RESULTS
Thirty-six studies were included, and 22 contributed quantitative data. Most of the studies (n = 27) assessed sleep by self-report. The meta-analysis showed a significant association between self-reported short sleep and development of obesity, and the chances of developing obesity increased when self-reported sleep duration decreased.


CONCLUSIONS
Self-reported short sleep was significantly associated with a higher incidence of obesity, with moderate quality of evidence.


SYSTEMATIC REVIEW REGISTRATION
PROSPERO registration no. CRD42019130143.
ER  - 

BibTeX

@misc{dd7b789454b209fd23c20ff2be6cd06864fa790b,
  author = {S. Shin},
  title = {The Association between Sleep Duration and Overweight in a School-Age Population in Seoul},
  journal = {Journal of Obesity & Metabolic Syndrome},
  year = {2017},
  volume = {26},
  pages = {45 - 51},
  doi = {10.7570/jomes.2017.26.1.45},
  abstract = {Background This study aimed to investigate the association between body mass index standard deviation scores and sleep duration in a school-age population in Seoul. Methods By using the secondary data of a sample survey (n=25,182) of the Student Health Examination in 2010, the proportions of those who were overweight (World Health Organization definition of body mass index >1.0 standard deviation score) were identified. The association of overweight with sleep duration was also explored after adjusting for age and for meat and vegetable intake, by using multiple logistic and linear regression analysis. Results Only 23.2% of participants slept 8 hours or more per day. The proportions of overweight participants were 34.2% of boys and 19.5% of girls. Significant odds ratios for overweight according to sleep duration (based on ≥8 sleeping hours) were 1.28 (for less than 6 hours), 1.31 (for 6–7 hours), and 1.28 (for 7–8 hours) in boys; and 1.19 (for 6–7 hours) and 1.17 (for 7–8 hours) in girls. Furthermore, significant regression coefficients for standard deviation scores (based on ≥8 sleeping hours) were 0.218 for less than 6 hours, 0.149 for 6–7 hours, and 0.099 for 7–8 hours in boys, and 0.137, 0.101, and 0.079 in girls, respectively. Conclusion To prevent and manage overweight, it is necessary to develop a school health policy for ensuring optimal sleep duration in school-age population.}
}

RIS

TY  - JOUR
AU  - S. Shin
T1  - The Association between Sleep Duration and Overweight in a School-Age Population in Seoul
JO  - Journal of Obesity & Metabolic Syndrome
PY  - 2017
VL  - 26
SP  - 45 - 51
EP  - 45 - 51
DO  - 10.7570/jomes.2017.26.1.45
AB  - Background This study aimed to investigate the association between body mass index standard deviation scores and sleep duration in a school-age population in Seoul. Methods By using the secondary data of a sample survey (n=25,182) of the Student Health Examination in 2010, the proportions of those who were overweight (World Health Organization definition of body mass index >1.0 standard deviation score) were identified. The association of overweight with sleep duration was also explored after adjusting for age and for meat and vegetable intake, by using multiple logistic and linear regression analysis. Results Only 23.2% of participants slept 8 hours or more per day. The proportions of overweight participants were 34.2% of boys and 19.5% of girls. Significant odds ratios for overweight according to sleep duration (based on ≥8 sleeping hours) were 1.28 (for less than 6 hours), 1.31 (for 6–7 hours), and 1.28 (for 7–8 hours) in boys; and 1.19 (for 6–7 hours) and 1.17 (for 7–8 hours) in girls. Furthermore, significant regression coefficients for standard deviation scores (based on ≥8 sleeping hours) were 0.218 for less than 6 hours, 0.149 for 6–7 hours, and 0.099 for 7–8 hours in boys, and 0.137, 0.101, and 0.079 in girls, respectively. Conclusion To prevent and manage overweight, it is necessary to develop a school health policy for ensuring optimal sleep duration in school-age population.
ER  - 

BibTeX

@misc{82f48ac6298565100e85c9a6ab99b9f8c02c1635,
  author = {S. Frank and K. Gonzalez and Lorraine Lee-Ang and Marielle C. Young and Martha Tamez and J. Mattei},
  title = {Diet and Sleep Physiology: Public Health and Clinical Implications},
  journal = {Frontiers in Neurology},
  year = {2017},
  volume = {8},
  doi = {10.3389/fneur.2017.00393},
  abstract = {This mini-review examines the complex relationship between diet and sleep and explores the clinical and public health implications of the current evidence. Dietary quality and intake of specific nutrients can impact regulatory hormonal pathways to alter sleep quantity and quality. Sleep, in turn, affects the intake of total energy, as well as of specific foods and nutrients, through biological and behavioral mechanisms. Initial research in this field focused primarily on the effects of short sleep duration on nutritional quality. However, more recent studies have explored the dynamic relationship between long sleep duration and diet. Current evidence suggests that extremes of sleep duration alter sleep patterns, hormonal levels, and circadian rhythms, which contribute to weight-related outcomes and obesity, and other risk factors for the development of chronic disease such as type 2 diabetes and cardiovascular disease. These patterns may begin as early as childhood and have impacts throughout the life course. Given that non-communicable diseases are among the leading causes of death globally, deeper understanding of the interactions between sleep and nutrition has implications for both public health and clinical practice.}
}

RIS

TY  - JOUR
AU  - S. Frank
AU  - K. Gonzalez
AU  - Lorraine Lee-Ang
AU  - Marielle C. Young
AU  - Martha Tamez
AU  - J. Mattei
T1  - Diet and Sleep Physiology: Public Health and Clinical Implications
JO  - Frontiers in Neurology
PY  - 2017
VL  - 8
DO  - 10.3389/fneur.2017.00393
AB  - This mini-review examines the complex relationship between diet and sleep and explores the clinical and public health implications of the current evidence. Dietary quality and intake of specific nutrients can impact regulatory hormonal pathways to alter sleep quantity and quality. Sleep, in turn, affects the intake of total energy, as well as of specific foods and nutrients, through biological and behavioral mechanisms. Initial research in this field focused primarily on the effects of short sleep duration on nutritional quality. However, more recent studies have explored the dynamic relationship between long sleep duration and diet. Current evidence suggests that extremes of sleep duration alter sleep patterns, hormonal levels, and circadian rhythms, which contribute to weight-related outcomes and obesity, and other risk factors for the development of chronic disease such as type 2 diabetes and cardiovascular disease. These patterns may begin as early as childhood and have impacts throughout the life course. Given that non-communicable diseases are among the leading causes of death globally, deeper understanding of the interactions between sleep and nutrition has implications for both public health and clinical practice.
ER  - 

BibTeX

@misc{c7e8ce3870334a5bddbfefa09a17e205ec08add1,
  author = {O. Bayer and A. S. Rosario and M. Wabitsch and R. Kries},
  title = {A GROWING BODY OF EVIDENCE THAT SLEEP DURA- TION AFFECTS WEIGHT STATUS IN CHILDREN HAS},
  year = {2009},
  abstract = {1183 A GROWING BODY OF EVIDENCE THAT SLEEP DURATION AFFECTS WEIGHT STATUS IN CHILDREN HAS BEEN SUMMARIZED IN 3 RECENT REVIEWS.1-3 A metaanalysis by Chen and colleagues calculated a 9% decrease in overweight/obesity risk for a one-hour increase in sleep duration in children up to 10 years of age. There is still considerable variability in the effects reported by different individual studies, which could be explained by different ages included. This issue has never been systematically assessed in publications the authors are aware of. The majority of studies use measures of overweight/obesity risk, while BMI is reported less frequently. The changes in BMI distributions have merely been addressed, although they might have implications for choosing the appropriate outcome measure and are interesting for physiological and epidemiological reasons. Furthermore, there is limited evidence on whether sleep duration is linked to body fat in children.4-6 A more detailed analysis of epidemiological data with respect to these issues may have implications for further research regarding underlying physiological mechanisms. The aims of this study were 1) to describe the sleep duration typical for children 3 to 10 years of age in a nationally representative sample 2) to assess whether the effects of sleep duration on body mass are age dependent 3) to investigate if these effects are also seen on body fat (KFA) as outcome 4) to analyze how the distribution of BMI and KFA is changed in relation to sleep duration}
}

RIS

TY  - JOUR
AU  - O. Bayer
AU  - A. S. Rosario
AU  - M. Wabitsch
AU  - R. Kries
T1  - A GROWING BODY OF EVIDENCE THAT SLEEP DURA- TION AFFECTS WEIGHT STATUS IN CHILDREN HAS
PY  - 2009
AB  - 1183 A GROWING BODY OF EVIDENCE THAT SLEEP DURATION AFFECTS WEIGHT STATUS IN CHILDREN HAS BEEN SUMMARIZED IN 3 RECENT REVIEWS.1-3 A metaanalysis by Chen and colleagues calculated a 9% decrease in overweight/obesity risk for a one-hour increase in sleep duration in children up to 10 years of age. There is still considerable variability in the effects reported by different individual studies, which could be explained by different ages included. This issue has never been systematically assessed in publications the authors are aware of. The majority of studies use measures of overweight/obesity risk, while BMI is reported less frequently. The changes in BMI distributions have merely been addressed, although they might have implications for choosing the appropriate outcome measure and are interesting for physiological and epidemiological reasons. Furthermore, there is limited evidence on whether sleep duration is linked to body fat in children.4-6 A more detailed analysis of epidemiological data with respect to these issues may have implications for further research regarding underlying physiological mechanisms. The aims of this study were 1) to describe the sleep duration typical for children 3 to 10 years of age in a nationally representative sample 2) to assess whether the effects of sleep duration on body mass are age dependent 3) to investigate if these effects are also seen on body fat (KFA) as outcome 4) to analyze how the distribution of BMI and KFA is changed in relation to sleep duration
ER  - 

BibTeX

@misc{0a189226079bf3c50cd0c8b93654b1bed08ab1d7,
  author = {Q. Gong and Sixuan Li and Hui Li and J. Cui and Guozhang Xu},
  title = {Insufficient Sleep Duration and Overweight/Obesity among Adolescents in a Chinese Population},
  journal = {International Journal of Environmental Research and Public Health},
  year = {2018},
  volume = {15},
  doi = {10.3390/ijerph15050997},
  abstract = {Objectives: The objective of this study was to investigate the relationship between sleep duration and overweight/obesity among Chinese adolescents. Methods: A school-based cross-sectional study was conducted among Chinese adolescents in 2016. In total, 2795 school-aged Chinese children aged 12 to 13 years participated in this study. Participants were asked to complete self-administered surveys during a 45-min class period in their classroom. Details of the questionnaire about health-related behaviors included sleep habits, physical activity, screen time, cigarette use, and alcohol use. Height, weight, waist circumference and hip circumference were directly measured. Results: The mean sleep duration was 8.7 h/day. In total, 43.0% of the participants had a sleep duration of less than 9 h/day. Sleep duration was significantly inversely related to BMI, waist circumference, hip circumference and WHtR in multiple linear regression analyses in both genders. Logistic regression models showed that insufficient sleep (<9 h/day) was associated with high odds of overweight/obesity among both young boys and girls. Conclusions: Insufficient sleep duration was associated with overweight/obesity in Chinese adolescents, and short sleep duration was probably associated with central adiposity, especially among boys.}
}

RIS

TY  - JOUR
AU  - Q. Gong
AU  - Sixuan Li
AU  - Hui Li
AU  - J. Cui
AU  - Guozhang Xu
T1  - Insufficient Sleep Duration and Overweight/Obesity among Adolescents in a Chinese Population
JO  - International Journal of Environmental Research and Public Health
PY  - 2018
VL  - 15
DO  - 10.3390/ijerph15050997
AB  - Objectives: The objective of this study was to investigate the relationship between sleep duration and overweight/obesity among Chinese adolescents. Methods: A school-based cross-sectional study was conducted among Chinese adolescents in 2016. In total, 2795 school-aged Chinese children aged 12 to 13 years participated in this study. Participants were asked to complete self-administered surveys during a 45-min class period in their classroom. Details of the questionnaire about health-related behaviors included sleep habits, physical activity, screen time, cigarette use, and alcohol use. Height, weight, waist circumference and hip circumference were directly measured. Results: The mean sleep duration was 8.7 h/day. In total, 43.0% of the participants had a sleep duration of less than 9 h/day. Sleep duration was significantly inversely related to BMI, waist circumference, hip circumference and WHtR in multiple linear regression analyses in both genders. Logistic regression models showed that insufficient sleep (<9 h/day) was associated with high odds of overweight/obesity among both young boys and girls. Conclusions: Insufficient sleep duration was associated with overweight/obesity in Chinese adolescents, and short sleep duration was probably associated with central adiposity, especially among boys.
ER  - 

BibTeX

@misc{b3acc99bcae075b6b26491d8a2ca3868988c0230,
  author = {A. Shechter and M. Grandner and M. St-Onge},
  title = {The Role of Sleep in the Control of Food Intake},
  journal = {American Journal of Lifestyle Medicine},
  year = {2014},
  volume = {8},
  pages = {371 - 374},
  doi = {10.1177/1559827614545315},
  abstract = {Short sleep duration is increasingly recognized as a risk factor for obesity. Sleep is now considered 1 of the 3 lifestyle behaviors, along with diet and exercise, which are closely associated with health. If sleep duration is a causal factor in the etiology of obesity, it must affect energy intake and/or energy expenditure to create a positive energy balance. The preponderance of evidence to date points to an effect of sleep restriction on energy intake that exceeds the added energy costs of maintaining longer wakefulness. Observational studies describe greater intakes of energy, fat, and possibly carbohydrates in short sleepers and this is corroborated by clinical interventions. These intervention studies further provide mechanistic explanations, via alterations in hormonal and neuronal controls of food intake, for an association between short sleep and obesity.}
}

RIS

TY  - JOUR
AU  - A. Shechter
AU  - M. Grandner
AU  - M. St-Onge
T1  - The Role of Sleep in the Control of Food Intake
JO  - American Journal of Lifestyle Medicine
PY  - 2014
VL  - 8
SP  - 371 - 374
EP  - 371 - 374
DO  - 10.1177/1559827614545315
AB  - Short sleep duration is increasingly recognized as a risk factor for obesity. Sleep is now considered 1 of the 3 lifestyle behaviors, along with diet and exercise, which are closely associated with health. If sleep duration is a causal factor in the etiology of obesity, it must affect energy intake and/or energy expenditure to create a positive energy balance. The preponderance of evidence to date points to an effect of sleep restriction on energy intake that exceeds the added energy costs of maintaining longer wakefulness. Observational studies describe greater intakes of energy, fat, and possibly carbohydrates in short sleepers and this is corroborated by clinical interventions. These intervention studies further provide mechanistic explanations, via alterations in hormonal and neuronal controls of food intake, for an association between short sleep and obesity.
ER  - 

BibTeX

@misc{6383a5daa1933b4f292d70f1b761a1d5d9e83b61,
  author = {Adam P Knowlden and Megan Ottati and Meaghan McCallum and J. Allegrante},
  title = {The relationship between sleep quantity, sleep quality and weight loss in adults: A scoping review.},
  journal = {Clinical obesity},
  year = {2023},
  pages = {
          e12634
        },
  doi = {10.1111/cob.12634},
  abstract = {Sleep is hypothesized to interact with weight gain and loss; however, modelling this relationship remains elusive. Poor sleep perpetuates a cascade of cardiovascular and metabolic consequences that may not only increase risk of adiposity, but also confound weight loss efforts. We conducted a scoping review to assess the research on sleep and weight loss interventions. We searched six databases for studies of behavioural weight loss interventions that included assessments of sleep in the general, non-clinical adult human population. Our synthesis focused on dimensions of Population, Intervention, Control, and Outcomes (PICO) to identify research and knowledge gaps. We identified 35 studies that fell into one of four categories: (a) sleep at baseline as a predictor of subsequent weight loss during an intervention, (b) sleep assessments after a history of successful weight loss, (c) concomitant changes in sleep associated with weight loss and (d) experimental manipulation of sleep and resulting weight loss. There was some evidence of improvements in sleep in response to weight-loss interventions; however, randomized controlled trials of weight loss interventions tended not to report improvements in sleep when compared to controls. We conclude that baseline sleep characteristics may predict weight loss in studies of dietary interventions and that sleep does not improve because of weight loss alone. Future studies should enrol large and diverse, normal, overweight and obese short sleepers in trials to assess the efficacy of sleep as a behavioural weight loss treatment.}
}

RIS

TY  - JOUR
AU  - Adam P Knowlden
AU  - Megan Ottati
AU  - Meaghan McCallum
AU  - J. Allegrante
T1  - The relationship between sleep quantity, sleep quality and weight loss in adults: A scoping review.
JO  - Clinical obesity
PY  - 2023
SP  - 
          e12634
        
EP  - 
          e12634
        
DO  - 10.1111/cob.12634
AB  - Sleep is hypothesized to interact with weight gain and loss; however, modelling this relationship remains elusive. Poor sleep perpetuates a cascade of cardiovascular and metabolic consequences that may not only increase risk of adiposity, but also confound weight loss efforts. We conducted a scoping review to assess the research on sleep and weight loss interventions. We searched six databases for studies of behavioural weight loss interventions that included assessments of sleep in the general, non-clinical adult human population. Our synthesis focused on dimensions of Population, Intervention, Control, and Outcomes (PICO) to identify research and knowledge gaps. We identified 35 studies that fell into one of four categories: (a) sleep at baseline as a predictor of subsequent weight loss during an intervention, (b) sleep assessments after a history of successful weight loss, (c) concomitant changes in sleep associated with weight loss and (d) experimental manipulation of sleep and resulting weight loss. There was some evidence of improvements in sleep in response to weight-loss interventions; however, randomized controlled trials of weight loss interventions tended not to report improvements in sleep when compared to controls. We conclude that baseline sleep characteristics may predict weight loss in studies of dietary interventions and that sleep does not improve because of weight loss alone. Future studies should enrol large and diverse, normal, overweight and obese short sleepers in trials to assess the efficacy of sleep as a behavioural weight loss treatment.
ER  - 

BibTeX

@misc{ca272f9cd89f3cff501cdc9d02a7c1e67db4a338,
  author = {Chaput Jp},
  title = {Short sleep duration as a cause of obesity: myth or reality?},
  journal = {Obesity Reviews},
  year = {2011},
  volume = {12},
  doi = {10.1111/j.1467-789X.2010.00848.x},
  abstract = {The evidence that short sleep duration is a determinant of obesity is accumulating (1–3). Prospective cohort studies have repeatedly shown that short sleep duration is associated with weight gain and an increased incidence of obesity in children and adults. Intervention studies have provided a mechanistic explanation, i.e. that sleep restriction impacts hormonal regulation of appetite. Ongoing research is assessing whether sleep extension in sleep deprived obese individuals can influence appetite control and/or reduce body fat (4). Given that chronic sleep restriction is highly tied to the modern way of living, studies aiming to investigate the possible causal association between short sleep duration and obesity are of particular importance from a public health standpoint. In this issue of Obesity Reviews, Professor Jim Horne critically addressed the link between habitual short sleep duration and obesity from a sleep perspective (5). The author argued that the clinical significance of short sleep as a risk factor for obesity is very little, with weight gains attributed to short sleep of <2 kg over a year. Additionally, the author mentioned that the growing interest in short sleep as a determinant of obesity has the potential to focus attention on sleep rather than to exercise and diet as behaviours to promote weight loss and healthy body weight. Globally, the review paper is interesting and provides a different view point that can generate new studies on the field. However, some aspects need to be put into perspective. First, many people fail to realize that obesity has a complex causation. The underlying causes and paths to obesity are manifold and one should thus expect the effect of any single factor to be relatively small. In this context, I would rather argue that a weight gain of as little as 1 kg over a year attributed to short sleep is clinically important on a population level. Second, it is important to realize that the main issue is not to determine whether or not short sleep is a cause of obesity (likely impossible to prove), but to understand the implications of chronic short sleep duration on energy balance. Time pressure is a reality of the modern way of living and many people prefer to reduce the time allocated to sleep in order to improve their quality of life. Such a strategy is understandable, but few realize that there might be a price to pay in the long run. Any influence of short sleep duration on food intake and physical activity participation is of importance in our understanding of the different pathways to obesity. Additionally, recent results have shown that sleep might be an important factor in successful weight loss, emphasizing that a narrow focus on diet and exercise might not be enough (6). Third, the objective in studying the association between short sleep duration and obesity should not be to neglect diet and physical activity in promoting healthy body weights, but rather to understand the interactions between short sleep duration and energy balance. In particular, we have recently realized that many factors (e.g. vitamins and minerals, chemical pollutants, sleep duration, etc.) can influence energy balance despite the fact that they do not have any caloric value per se (7,8). Finally, the author elegantly indicated in its review that ‘sleep-related obesity might even be protective’. This argument is interesting and agrees with the physiological perception of obesity that considers fat gain as a biological adaptation that ultimately permits the person gaining weight to reach a new homeostatic state (9). For the short sleeper, adipose tissue may at least provide an increased leptinemia. It is obvious that the causal relation between short sleep duration and obesity will be difficult to obtain, if at all possible. Experimental evidence that sleep restriction induces obesity is not possible in humans for both ethical and logistic reasons. The best evidence would be to conduct a randomized-controlled trial in which we would have to restrict sleep duration in a group of lean people in order to be able to compare them with a control group. However, the slow development of obesity implies that such a study would have to run for years in a large sample of individuals. This means that we need to rely on short-term intervention studies and longitudinal studies. As recently addressed (2), short-term experiments may elucidate biological mechanisms supposedly inducing continuous weight gain, but whether they do produce obesity in the long run is unknown. Likewise, observations of obesity development in non-obese population groups over time after exposure to short sleep duration can be addressed with the use of various epidemiological methods, each of which have pros and cons. As mentioned by Professor Horne in his article, ‘the case for short sleep as a cause rather than just a correlate of obesity has yet to be established’. This is perhaps true, but the main message is that sleep obesity reviews doi: 10.1111/j.1467-789X.2010.00848.x}
}

RIS

TY  - JOUR
AU  - Chaput Jp
T1  - Short sleep duration as a cause of obesity: myth or reality?
JO  - Obesity Reviews
PY  - 2011
VL  - 12
DO  - 10.1111/j.1467-789X.2010.00848.x
AB  - The evidence that short sleep duration is a determinant of obesity is accumulating (1–3). Prospective cohort studies have repeatedly shown that short sleep duration is associated with weight gain and an increased incidence of obesity in children and adults. Intervention studies have provided a mechanistic explanation, i.e. that sleep restriction impacts hormonal regulation of appetite. Ongoing research is assessing whether sleep extension in sleep deprived obese individuals can influence appetite control and/or reduce body fat (4). Given that chronic sleep restriction is highly tied to the modern way of living, studies aiming to investigate the possible causal association between short sleep duration and obesity are of particular importance from a public health standpoint. In this issue of Obesity Reviews, Professor Jim Horne critically addressed the link between habitual short sleep duration and obesity from a sleep perspective (5). The author argued that the clinical significance of short sleep as a risk factor for obesity is very little, with weight gains attributed to short sleep of <2 kg over a year. Additionally, the author mentioned that the growing interest in short sleep as a determinant of obesity has the potential to focus attention on sleep rather than to exercise and diet as behaviours to promote weight loss and healthy body weight. Globally, the review paper is interesting and provides a different view point that can generate new studies on the field. However, some aspects need to be put into perspective. First, many people fail to realize that obesity has a complex causation. The underlying causes and paths to obesity are manifold and one should thus expect the effect of any single factor to be relatively small. In this context, I would rather argue that a weight gain of as little as 1 kg over a year attributed to short sleep is clinically important on a population level. Second, it is important to realize that the main issue is not to determine whether or not short sleep is a cause of obesity (likely impossible to prove), but to understand the implications of chronic short sleep duration on energy balance. Time pressure is a reality of the modern way of living and many people prefer to reduce the time allocated to sleep in order to improve their quality of life. Such a strategy is understandable, but few realize that there might be a price to pay in the long run. Any influence of short sleep duration on food intake and physical activity participation is of importance in our understanding of the different pathways to obesity. Additionally, recent results have shown that sleep might be an important factor in successful weight loss, emphasizing that a narrow focus on diet and exercise might not be enough (6). Third, the objective in studying the association between short sleep duration and obesity should not be to neglect diet and physical activity in promoting healthy body weights, but rather to understand the interactions between short sleep duration and energy balance. In particular, we have recently realized that many factors (e.g. vitamins and minerals, chemical pollutants, sleep duration, etc.) can influence energy balance despite the fact that they do not have any caloric value per se (7,8). Finally, the author elegantly indicated in its review that ‘sleep-related obesity might even be protective’. This argument is interesting and agrees with the physiological perception of obesity that considers fat gain as a biological adaptation that ultimately permits the person gaining weight to reach a new homeostatic state (9). For the short sleeper, adipose tissue may at least provide an increased leptinemia. It is obvious that the causal relation between short sleep duration and obesity will be difficult to obtain, if at all possible. Experimental evidence that sleep restriction induces obesity is not possible in humans for both ethical and logistic reasons. The best evidence would be to conduct a randomized-controlled trial in which we would have to restrict sleep duration in a group of lean people in order to be able to compare them with a control group. However, the slow development of obesity implies that such a study would have to run for years in a large sample of individuals. This means that we need to rely on short-term intervention studies and longitudinal studies. As recently addressed (2), short-term experiments may elucidate biological mechanisms supposedly inducing continuous weight gain, but whether they do produce obesity in the long run is unknown. Likewise, observations of obesity development in non-obese population groups over time after exposure to short sleep duration can be addressed with the use of various epidemiological methods, each of which have pros and cons. As mentioned by Professor Horne in his article, ‘the case for short sleep as a cause rather than just a correlate of obesity has yet to be established’. This is perhaps true, but the main message is that sleep obesity reviews doi: 10.1111/j.1467-789X.2010.00848.x
ER  - 

BibTeX

@misc{99b0389154ff05cf624a976f59fef9a622c58f6c,
  author = {Weijia Liu and Yajun Chen and K. Cheng and M. Pallan},
  title = {Prevalence of adiposity and its association with sleep duration, quality, and timing among 9-12-year-old children in Guangzhou, China},
  year = {2017},
  abstract = {Background: Short sleep duration has been reported to be associated with obesity in children, but findings are not consistent. Since few studies have examined the relationship between more complex sleep characteristics and obesity, we examined the association between adiposity and self-reported sleep duration, bedtime, and sleep quality in 9e12-year-old Chinese children using multilevel mixed models. Methods: 5518 children aged 9e12 years were recruited from 29 randomly selected primary schools in Guangzhou, China in 2014. Standardized questionnaires were used to obtain data to estimate sleep duration on typical weekdays and weekends. Sleep quality data were collected using the Children's Sleep Habits Questionnaire (CSHQ). Trained researchers undertook measurements of weight, height, and waist circumference (WC) for all participating children. Body mass index (BMI) z-scores were derived using the World Health Organization (WHO) child growth reference, and children were classified as overweight or obese using þ1 and þ2 SD as cut-offs, respectively. Percentage body fat (BF%) was calculated using bioelectrical impedance. Results: Longer sleep duration was inversely associated with BMI z-score (b 1⁄4 0.16, p < 0.05), WC (b 1⁄4 1.11, p < 0.05) and later bedtime was associated with higher BMI z-score (b 1⁄4 0.03, p < 0.05), WC (b 1⁄4 1.72, p < 0.001), and BF% (b 1⁄4 0.15, p < 0.05) in multivariable multilevel mixed models, after adjustment for age, gender, physical activity, parental education level, and average monthly income. No association was seen between sleep quality and adiposity. Conclusion: Shorter sleep duration and later bedtime are associated with higher adiposity indices in early adolescents from southern China. © 2017 The Authors. Publishing services by Elsevier B.V. on behalf of The Japan Epidemiological Association. This is an open access article under the CC BY license (http://creativecommons.org/licenses/ by/4.0/).}
}

RIS

TY  - JOUR
AU  - Weijia Liu
AU  - Yajun Chen
AU  - K. Cheng
AU  - M. Pallan
T1  - Prevalence of adiposity and its association with sleep duration, quality, and timing among 9-12-year-old children in Guangzhou, China
PY  - 2017
AB  - Background: Short sleep duration has been reported to be associated with obesity in children, but findings are not consistent. Since few studies have examined the relationship between more complex sleep characteristics and obesity, we examined the association between adiposity and self-reported sleep duration, bedtime, and sleep quality in 9e12-year-old Chinese children using multilevel mixed models. Methods: 5518 children aged 9e12 years were recruited from 29 randomly selected primary schools in Guangzhou, China in 2014. Standardized questionnaires were used to obtain data to estimate sleep duration on typical weekdays and weekends. Sleep quality data were collected using the Children's Sleep Habits Questionnaire (CSHQ). Trained researchers undertook measurements of weight, height, and waist circumference (WC) for all participating children. Body mass index (BMI) z-scores were derived using the World Health Organization (WHO) child growth reference, and children were classified as overweight or obese using þ1 and þ2 SD as cut-offs, respectively. Percentage body fat (BF%) was calculated using bioelectrical impedance. Results: Longer sleep duration was inversely associated with BMI z-score (b 1⁄4 0.16, p < 0.05), WC (b 1⁄4 1.11, p < 0.05) and later bedtime was associated with higher BMI z-score (b 1⁄4 0.03, p < 0.05), WC (b 1⁄4 1.72, p < 0.001), and BF% (b 1⁄4 0.15, p < 0.05) in multivariable multilevel mixed models, after adjustment for age, gender, physical activity, parental education level, and average monthly income. No association was seen between sleep quality and adiposity. Conclusion: Shorter sleep duration and later bedtime are associated with higher adiposity indices in early adolescents from southern China. © 2017 The Authors. Publishing services by Elsevier B.V. on behalf of The Japan Epidemiological Association. This is an open access article under the CC BY license (http://creativecommons.org/licenses/ by/4.0/).
ER  - 

BibTeX

@misc{bcefad3faed975c90b98a331e4c1695ac436443c,
  author = {E. Suliga and D. Kozieł and E. Cieśla and D. Rębak and S. Głuszek},
  title = {Sleep duration and the risk of obesity – a cross-sectional study},
  journal = {},
  year = {2017},
  volume = {33},
  pages = {176-183},
  doi = {10.5114/MS.2017.70343},
  abstract = {Introduction: So far, the association between a longer sleep duration, state of health, and the risk of obesity and the influence of gender on the association between sleep duration and the risk of obesity has not been fully explained. Aim of the research: To examine the relationships between self-reported sleep duration, body mass index (BMI), and body fat percentage (%BF), and also to determine whether such associations are the same in men and in women. Material and methods: This study included 10,367 participants aged 37 to 66 years. Logistic regression was applied for risk assessment of the prevalence of abnormal BMI values and %BF in groups of sleep duration. Sleep of 7–8 h per night was adopted as a reference level. Results: In men, the risk of obesity was significantly greater only in the group sleeping ≤ 6 h (OR = 1.18, 95% CI: 1.08–1.28; p < 0.05); however, in women, only among those sleeping ≥ 9 h (OR = 1.14, 95% CI: 1.02–1.26; p < 0.05). The risk of obesity, determined on the basis of %BF, was higher only in individuals sleeping ≥ 9 h. In the adjusted model, it turned out to be significant in the general study population (OR = 1.28, 95% CI: 1.07–1.53; p < 0.05) and in women (OR = 1.14, 95% CI: 1.03–1.27; p < 0.05). Conclusions: In women, a greater risk of obesity was related to a longer sleep duration (≥ 9 h), whereas in men, the tendency of obesity occurrence along with shorter sleep (≤ 6 h). Thus, the physiological consequences of sleep duration may be different in women than in men.}
}

RIS

TY  - JOUR
AU  - E. Suliga
AU  - D. Kozieł
AU  - E. Cieśla
AU  - D. Rębak
AU  - S. Głuszek
T1  - Sleep duration and the risk of obesity – a cross-sectional study
PY  - 2017
VL  - 33
SP  - 176-183
EP  - 176-183
DO  - 10.5114/MS.2017.70343
AB  - Introduction: So far, the association between a longer sleep duration, state of health, and the risk of obesity and the influence of gender on the association between sleep duration and the risk of obesity has not been fully explained. Aim of the research: To examine the relationships between self-reported sleep duration, body mass index (BMI), and body fat percentage (%BF), and also to determine whether such associations are the same in men and in women. Material and methods: This study included 10,367 participants aged 37 to 66 years. Logistic regression was applied for risk assessment of the prevalence of abnormal BMI values and %BF in groups of sleep duration. Sleep of 7–8 h per night was adopted as a reference level. Results: In men, the risk of obesity was significantly greater only in the group sleeping ≤ 6 h (OR = 1.18, 95% CI: 1.08–1.28; p < 0.05); however, in women, only among those sleeping ≥ 9 h (OR = 1.14, 95% CI: 1.02–1.26; p < 0.05). The risk of obesity, determined on the basis of %BF, was higher only in individuals sleeping ≥ 9 h. In the adjusted model, it turned out to be significant in the general study population (OR = 1.28, 95% CI: 1.07–1.53; p < 0.05) and in women (OR = 1.14, 95% CI: 1.03–1.27; p < 0.05). Conclusions: In women, a greater risk of obesity was related to a longer sleep duration (≥ 9 h), whereas in men, the tendency of obesity occurrence along with shorter sleep (≤ 6 h). Thus, the physiological consequences of sleep duration may be different in women than in men.
ER  - 

BibTeX

@misc{1aed520565e04a694e5320cf30f59dc294868e84,
  author = {Qi Sun and Yinglong Bai and Lingling Zhai and Wei Wei and Lihong Jia},
  title = {Association between Sleep Duration and Overweight/Obesity at Age 7–18 in Shenyang, China in 2010 and 2014},
  journal = {International Journal of Environmental Research and Public Health},
  year = {2018},
  volume = {15},
  doi = {10.3390/ijerph15050854},
  abstract = {This study was designed to examine the association between sleep duration and being overweight/obese in primary, middle, and high school students. This was a multiple cross-sectional study using data from the 2010 and 2014 National Survey on Students’ Constitution and Health (CNSSCH). A total of 23,602 students aged 7–18 years were enrolled in this study. The prevalence of being overweight and obese—stratified by age, gender, and sleep duration—in 2010 and 2014 were compared. Sleep duration was categorized as <7 h, ≥7 to 8 h, ≥8 to 9 h, and ≥9 h. Overweight and obesity were defined according to the cut-point criteria in China. Multivariable logistic regression results in 2010 and 2014 revealed that students sleeping <7 h and aged 7–12 years had an increased risk of becoming overweight/obese. In 2010, the adjusted prevalence ratios of overweight for 7–12-year-old students sleeping <9 h was 1.196 (95%CI: 1.004–1.424) and 13–15-year-old students sleeping <8 h was 1.265 (95%CI: 1.023–1.565). In 2014, the adjusted prevalence ratios of overweight and obesity for 7–12-year-old students sleeping <9 h were 1.295 (95%CI: 1.091–1.537) and 1.231 (95%CI: 1.045–1.449); 16–18-year-old students sleeping <7 h were 1.530 (95%CI: 1.239–1.888) and 1.585 (95%CI: 1.270–2.081). Our study revealed that different levels of sleep curtailment increased the risk of becoming overweight/obesity in different age groups of students.}
}

RIS

TY  - JOUR
AU  - Qi Sun
AU  - Yinglong Bai
AU  - Lingling Zhai
AU  - Wei Wei
AU  - Lihong Jia
T1  - Association between Sleep Duration and Overweight/Obesity at Age 7–18 in Shenyang, China in 2010 and 2014
JO  - International Journal of Environmental Research and Public Health
PY  - 2018
VL  - 15
DO  - 10.3390/ijerph15050854
AB  - This study was designed to examine the association between sleep duration and being overweight/obese in primary, middle, and high school students. This was a multiple cross-sectional study using data from the 2010 and 2014 National Survey on Students’ Constitution and Health (CNSSCH). A total of 23,602 students aged 7–18 years were enrolled in this study. The prevalence of being overweight and obese—stratified by age, gender, and sleep duration—in 2010 and 2014 were compared. Sleep duration was categorized as <7 h, ≥7 to 8 h, ≥8 to 9 h, and ≥9 h. Overweight and obesity were defined according to the cut-point criteria in China. Multivariable logistic regression results in 2010 and 2014 revealed that students sleeping <7 h and aged 7–12 years had an increased risk of becoming overweight/obese. In 2010, the adjusted prevalence ratios of overweight for 7–12-year-old students sleeping <9 h was 1.196 (95%CI: 1.004–1.424) and 13–15-year-old students sleeping <8 h was 1.265 (95%CI: 1.023–1.565). In 2014, the adjusted prevalence ratios of overweight and obesity for 7–12-year-old students sleeping <9 h were 1.295 (95%CI: 1.091–1.537) and 1.231 (95%CI: 1.045–1.449); 16–18-year-old students sleeping <7 h were 1.530 (95%CI: 1.239–1.888) and 1.585 (95%CI: 1.270–2.081). Our study revealed that different levels of sleep curtailment increased the risk of becoming overweight/obesity in different age groups of students.
ER  - 

BibTeX

@misc{0729a4406b7e5dec736fe09cc5a6b8eb6e738eef,
  author = {H. Dashti and P. Jacques and S. Lamon-Fava and J. Ordovás},
  title = {Short Sleep Duration and Dietary Intake: Epidemiologic Evidence, Mechanisms,},
  journal = {},
  year = {2015},
  volume = {},
  abstract = {Links between short sleep duration and obesity, type 2 diabetes, hypertension, and cardiovascular disease may be mediated through changes in dietary intake. This review provides an overview of recent epidemiologic studies on the relations between habitual short sleep duration and dietary intake in adults from 16 cross-sectional studies. The studies have observed consistent associations between short sleep duration and higher total energy intake and higher total fat intake, and limited evidence for lower fruit intake, and lower quality diets. Evidence also suggests that short sleepers may have irregular eating behavior deviating from the traditional 3 meals/d to fewer main meals and more frequent, smaller, energy-dense, and highly palatable snacks at night. Although the impact of short sleep duration on dietary intake tends to be small, if chronic, it may contribute to an increased risk of obesity and related chronic disease. Mechanisms mediating the associations between sleep duration and dietary intake are likely to be multifactorial and include differences in the appetite-related hormones leptin and ghrelin, hedonic pathways, extended hours for intake, and altered time of intake. Taking into account these epidemiologic relations and the evidence for causal relations between sleep loss and metabolism and cardiovascular function, health promotion strategies should emphasize improved sleep as an additional factor in health and weight management. Moreover, future sleep interventions in controlled studies and sleep extension trials in chronic short sleepers are imperative for establishing whether there is a causal relation between short sleep duration and changes in dietary intake. Adv Nutr 2015;6:648–59.}
}

RIS

TY  - JOUR
AU  - H. Dashti
AU  - P. Jacques
AU  - S. Lamon-Fava
AU  - J. Ordovás
T1  - Short Sleep Duration and Dietary Intake: Epidemiologic Evidence, Mechanisms,
PY  - 2015
AB  - Links between short sleep duration and obesity, type 2 diabetes, hypertension, and cardiovascular disease may be mediated through changes in dietary intake. This review provides an overview of recent epidemiologic studies on the relations between habitual short sleep duration and dietary intake in adults from 16 cross-sectional studies. The studies have observed consistent associations between short sleep duration and higher total energy intake and higher total fat intake, and limited evidence for lower fruit intake, and lower quality diets. Evidence also suggests that short sleepers may have irregular eating behavior deviating from the traditional 3 meals/d to fewer main meals and more frequent, smaller, energy-dense, and highly palatable snacks at night. Although the impact of short sleep duration on dietary intake tends to be small, if chronic, it may contribute to an increased risk of obesity and related chronic disease. Mechanisms mediating the associations between sleep duration and dietary intake are likely to be multifactorial and include differences in the appetite-related hormones leptin and ghrelin, hedonic pathways, extended hours for intake, and altered time of intake. Taking into account these epidemiologic relations and the evidence for causal relations between sleep loss and metabolism and cardiovascular function, health promotion strategies should emphasize improved sleep as an additional factor in health and weight management. Moreover, future sleep interventions in controlled studies and sleep extension trials in chronic short sleepers are imperative for establishing whether there is a causal relation between short sleep duration and changes in dietary intake. Adv Nutr 2015;6:648–59.
ER  - 

BibTeX

@misc{97875be703d14febe73c0a574160aedb7ece3a93,
  author = {B. Borowska and A. Suder and K. Kliś and I. Wronka},
  title = {Associations between Sleep Duration and Anthropometric Indices of Adiposity in Female University Students},
  journal = {International Journal of Environmental Research and Public Health},
  year = {2022},
  volume = {19},
  doi = {10.3390/ijerph191811681},
  abstract = {Objectives: To examine associations between sleep duration as well as time of going to sleep and anthropometric indices related to the amount and distribution of adiposity. Material: A total of 969 female university students, aged 19–24 years. Methods: Participants self-reported their sleep duration. Body weight, height, and waist circumference were measured. BMI, WHR and WHtR were calculated. Statistical analyses of results involved logistic regression models. Socioeconomic status and level of stress were added as covariates. Results: In 15% of the sample, sleep was too short (<6 h), and 10% slept too long (>8 h). Compared to women who followed the recommended sleep duration, among short sleepers, both underweight and overweight were more frequent, while long sleepers were more likely to be overweight. A higher prevalence of abdominal obesity and increased risk of metabolic syndrome were observed in both short and long sleepers than in recommended sleepers. Irregular sleep times were connected with higher OR, both for BMI < 18.5 and BMI > 25, for WC > 80, and WHtR below 0.4 and above 0.5. Irregular sleep times also led to an increased risk of metabolic diseases prevalence. Conclusions: Both too long and too short sleep increases the risk of overweight, obesity and abdominal obesity and, as a consequence, the risk of metabolic syndrome in young women.}
}

RIS

TY  - JOUR
AU  - B. Borowska
AU  - A. Suder
AU  - K. Kliś
AU  - I. Wronka
T1  - Associations between Sleep Duration and Anthropometric Indices of Adiposity in Female University Students
JO  - International Journal of Environmental Research and Public Health
PY  - 2022
VL  - 19
DO  - 10.3390/ijerph191811681
AB  - Objectives: To examine associations between sleep duration as well as time of going to sleep and anthropometric indices related to the amount and distribution of adiposity. Material: A total of 969 female university students, aged 19–24 years. Methods: Participants self-reported their sleep duration. Body weight, height, and waist circumference were measured. BMI, WHR and WHtR were calculated. Statistical analyses of results involved logistic regression models. Socioeconomic status and level of stress were added as covariates. Results: In 15% of the sample, sleep was too short (<6 h), and 10% slept too long (>8 h). Compared to women who followed the recommended sleep duration, among short sleepers, both underweight and overweight were more frequent, while long sleepers were more likely to be overweight. A higher prevalence of abdominal obesity and increased risk of metabolic syndrome were observed in both short and long sleepers than in recommended sleepers. Irregular sleep times were connected with higher OR, both for BMI < 18.5 and BMI > 25, for WC > 80, and WHtR below 0.4 and above 0.5. Irregular sleep times also led to an increased risk of metabolic diseases prevalence. Conclusions: Both too long and too short sleep increases the risk of overweight, obesity and abdominal obesity and, as a consequence, the risk of metabolic syndrome in young women.
ER  - 

BibTeX

@misc{3b9b286018f036a41b6fd87e6a8a35ecd02ca60f,
  author = {Y. Fatima and S. Doi and Abdullah Al Mamun},
  title = {Longitudinal impact of sleep on overweight and obesity in children and adolescents: a systematic review and bias‐adjusted meta‐analysis},
  journal = {Obesity Reviews},
  year = {2015},
  volume = {16},
  doi = {10.1111/obr.12245},
  abstract = {Short sleep duration is considered a potential risk for overweight/obesity in childhood and adolescence. However, most of the evidence on this topic is obtained from cross‐sectional studies; therefore, the nature and extent of the longitudinal associations are unclear. This study explores the prospective association between short sleep and overweight/obesity in young subjects. The MEDLINE, EMBASE, Pubmed, and CINAHL databases were searched for English‐language articles, published until May 2014, reporting longitudinal association between sleep and body mass index (BMI) in children and adolescents. Recommendations of the Sleep Health Foundation were used to standardize reference sleep duration. Sleep category, with sleep duration less than the reference sleep, was considered as the short sleep category. Meta‐analysis was conducted to explore the association between short sleep and overweight/obesity. A review of 22 longitudinal studies, with subjects from diverse backgrounds, suggested an inverse association between sleep duration and BMI. Meta‐analysis of 11 longitudinal studies, comprising 24,821 participants, revealed that subjects sleeping for short duration had twice the risk of being overweight/obese, compared with subjects sleeping for long duration (odds ratio 2.15; 95% confidence interval: 1.64–2.81). This study provides evidence that short sleep duration in young subjects is significantly associated with future overweight/obesity.}
}

RIS

TY  - JOUR
AU  - Y. Fatima
AU  - S. Doi
AU  - Abdullah Al Mamun
T1  - Longitudinal impact of sleep on overweight and obesity in children and adolescents: a systematic review and bias‐adjusted meta‐analysis
JO  - Obesity Reviews
PY  - 2015
VL  - 16
DO  - 10.1111/obr.12245
AB  - Short sleep duration is considered a potential risk for overweight/obesity in childhood and adolescence. However, most of the evidence on this topic is obtained from cross‐sectional studies; therefore, the nature and extent of the longitudinal associations are unclear. This study explores the prospective association between short sleep and overweight/obesity in young subjects. The MEDLINE, EMBASE, Pubmed, and CINAHL databases were searched for English‐language articles, published until May 2014, reporting longitudinal association between sleep and body mass index (BMI) in children and adolescents. Recommendations of the Sleep Health Foundation were used to standardize reference sleep duration. Sleep category, with sleep duration less than the reference sleep, was considered as the short sleep category. Meta‐analysis was conducted to explore the association between short sleep and overweight/obesity. A review of 22 longitudinal studies, with subjects from diverse backgrounds, suggested an inverse association between sleep duration and BMI. Meta‐analysis of 11 longitudinal studies, comprising 24,821 participants, revealed that subjects sleeping for short duration had twice the risk of being overweight/obese, compared with subjects sleeping for long duration (odds ratio 2.15; 95% confidence interval: 1.64–2.81). This study provides evidence that short sleep duration in young subjects is significantly associated with future overweight/obesity.
ER  - 

BibTeX

@misc{e641eef30d190c2aec5547713c0903877ecb12a8,
  author = {C. Cooper and Eric V. Neufeld and B. Dolezal and Jennifer L Martin},
  title = {Sleep deprivation and obesity in adults: a brief narrative review},
  journal = {BMJ Open Sport — Exercise Medicine},
  year = {2018},
  volume = {4},
  doi = {10.1136/bmjsem-2018-000392},
  abstract = {Background/aims Obesity and sleep deprivation are two epidemics that pervade developed nations. Their rates have been steadily rising worldwide, especially in the USA. This short communication will explore the link between the two conditions and outline the proposed mechanisms behind their relationship. Methods Studies on the topic of sleep and obesity were reviewed, and findings were used to develop a theoretical model for the biological link between short sleep duration and obesity. Results Individuals who regularly slept less than 7  hours per night were more likely to have higher average body mass indexes and develop obesity than those who slept more. Studies showed that experimental sleep restriction was associated with increased levels of ghrelin, salt retention and inflammatory markers as well as decreased levels of leptin and insulin sensitivity. Conclusions There may be a link between obesity and sleep deprivation. We recommend further investigations are to elucidate the potential mechanisms.}
}

RIS

TY  - JOUR
AU  - C. Cooper
AU  - Eric V. Neufeld
AU  - B. Dolezal
AU  - Jennifer L Martin
T1  - Sleep deprivation and obesity in adults: a brief narrative review
JO  - BMJ Open Sport — Exercise Medicine
PY  - 2018
VL  - 4
DO  - 10.1136/bmjsem-2018-000392
AB  - Background/aims Obesity and sleep deprivation are two epidemics that pervade developed nations. Their rates have been steadily rising worldwide, especially in the USA. This short communication will explore the link between the two conditions and outline the proposed mechanisms behind their relationship. Methods Studies on the topic of sleep and obesity were reviewed, and findings were used to develop a theoretical model for the biological link between short sleep duration and obesity. Results Individuals who regularly slept less than 7  hours per night were more likely to have higher average body mass indexes and develop obesity than those who slept more. Studies showed that experimental sleep restriction was associated with increased levels of ghrelin, salt retention and inflammatory markers as well as decreased levels of leptin and insulin sensitivity. Conclusions There may be a link between obesity and sleep deprivation. We recommend further investigations are to elucidate the potential mechanisms.
ER  - 

BibTeX

@misc{949b97fd2a92f3cac7ced7ea8cafec83cd8ae4d5,
  author = {F. Scheer},
  title = {Hungry for Sleep: A Role for Endocannabinoids?},
  journal = {Sleep},
  year = {2016},
  volume = {39 3},
  pages = {
          495-6
        },
  doi = {10.5665/sleep.5510},
  abstract = {Inadequate sleep, both in quantity and quality, has been associated with increased body weight and risk for obesity.1 While this link has been established convincingly in epidemiologic studies, such studies cannot determine causality or the direction of effect between sleep and weight. Indeed, both directions appear to play a role. On the one hand, e.g., an increased body mass index increases the risk for obstructive sleep apnea, and thus for disturbed sleep.2 On the other hand, experimental sleep restriction influences processes involved in energy balance and body weight regulation. Seminal studies under controlled laboratory conditions and controlled caloric intake have shown that experimental sleep restriction leads to decreases in the satiety hormone leptin, increases in the appetite hormone ghrelin, and increases in hunger and appetite.3 While this may be in part due to the increased energy expenditure required by the increased time awake,4 and thus a normal homeostatic response to a lower energy balance, this doesn’t appear to be the full story. Indeed, recent experimental studies have demonstrated that restricted sleep results in overeating, thus eating in excess of the slight increase in energy expenditure, and results in weight gain.5,6 This raises the question why people would overeat when sleep deprived. In this issue of SLEEP, Hanlon and colleagues7 provide insights into the possible involvement of food reward mechanisms to underlie increased (unhealthy) food intake following sleep restriction. Using a randomized, crossover design, in fourteen healthy non-obese study participants on a fixed diet (three identical meals per day), they investigated the influence of restricting the sleep opportunity to 4.5 h per night (4 h 11 min of actual sleep as determined by PSG) as compared to 8.5 h per night (7 h 33 min) for 3 nights on the 24-h daily profile of hourly-sampled circulating endocannabinoids. The midpoint of the sleep opportunity was kept the same for the two conditions to limit any changes in circadian phase. Sleep restriction resulted in an increase in the peak and amplitude of the 24-h daily rhythm in circulating concentrations of a key endocannabinoid, 2-arachidonoylglycerol (2-AG) and its structural analogue 2-oleoylglycerol (2-OG), without changes in the 24-h mean. Their increase was most apparent between 2:00 pm and 9:00 pm, and the timing of their peaks was significantly delayed. Serum leptin, ghrelin, and cortisol were assessed simultaneously with the endocannabinoid rhythms. While the 24-h mean didn’t differ for any of the three hormones, sleep restriction reduced the amplitude of the 24-h leptin rhythm, advanced the timing of the first peak in ghrelin (close to bedtime), delayed the cortisol trough (close to bedtime) and advanced the cortisol peak (at scheduled awakening). The changes in the pii: sp-00041-16 ht tp://dx.doi.org/10.5665/sleep.5510}
}

RIS

TY  - JOUR
AU  - F. Scheer
T1  - Hungry for Sleep: A Role for Endocannabinoids?
JO  - Sleep
PY  - 2016
VL  - 39 3
SP  - 
          495-6
        
EP  - 
          495-6
        
DO  - 10.5665/sleep.5510
AB  - Inadequate sleep, both in quantity and quality, has been associated with increased body weight and risk for obesity.1 While this link has been established convincingly in epidemiologic studies, such studies cannot determine causality or the direction of effect between sleep and weight. Indeed, both directions appear to play a role. On the one hand, e.g., an increased body mass index increases the risk for obstructive sleep apnea, and thus for disturbed sleep.2 On the other hand, experimental sleep restriction influences processes involved in energy balance and body weight regulation. Seminal studies under controlled laboratory conditions and controlled caloric intake have shown that experimental sleep restriction leads to decreases in the satiety hormone leptin, increases in the appetite hormone ghrelin, and increases in hunger and appetite.3 While this may be in part due to the increased energy expenditure required by the increased time awake,4 and thus a normal homeostatic response to a lower energy balance, this doesn’t appear to be the full story. Indeed, recent experimental studies have demonstrated that restricted sleep results in overeating, thus eating in excess of the slight increase in energy expenditure, and results in weight gain.5,6 This raises the question why people would overeat when sleep deprived. In this issue of SLEEP, Hanlon and colleagues7 provide insights into the possible involvement of food reward mechanisms to underlie increased (unhealthy) food intake following sleep restriction. Using a randomized, crossover design, in fourteen healthy non-obese study participants on a fixed diet (three identical meals per day), they investigated the influence of restricting the sleep opportunity to 4.5 h per night (4 h 11 min of actual sleep as determined by PSG) as compared to 8.5 h per night (7 h 33 min) for 3 nights on the 24-h daily profile of hourly-sampled circulating endocannabinoids. The midpoint of the sleep opportunity was kept the same for the two conditions to limit any changes in circadian phase. Sleep restriction resulted in an increase in the peak and amplitude of the 24-h daily rhythm in circulating concentrations of a key endocannabinoid, 2-arachidonoylglycerol (2-AG) and its structural analogue 2-oleoylglycerol (2-OG), without changes in the 24-h mean. Their increase was most apparent between 2:00 pm and 9:00 pm, and the timing of their peaks was significantly delayed. Serum leptin, ghrelin, and cortisol were assessed simultaneously with the endocannabinoid rhythms. While the 24-h mean didn’t differ for any of the three hormones, sleep restriction reduced the amplitude of the 24-h leptin rhythm, advanced the timing of the first peak in ghrelin (close to bedtime), delayed the cortisol trough (close to bedtime) and advanced the cortisol peak (at scheduled awakening). The changes in the pii: sp-00041-16 ht tp://dx.doi.org/10.5665/sleep.5510
ER  - 

BibTeX

@misc{e418ea25defd38df5bdf39cd202d9b028e3be4a2,
  author = {J. Chaput and M. St-Onge},
  title = {Increased Food Intake by Insufficient Sleep in Humans: Are We Jumping the Gun on the Hormonal Explanation?},
  journal = {Frontiers in Endocrinology},
  year = {2014},
  volume = {5},
  doi = {10.3389/fendo.2014.00116},
  abstract = {Concurrent with the increase in obesity prevalence, a decrease in sleep duration has been observed over the past decades (1, 2). Scientists have been intrigued by this inverse trend between sleep and body weight, and studies published in the past 10 years have been helpful in determining if the lack of sleep is a possible cause of obesity (3). Epidemiologic evidence has shown that short sleep duration is associated with obesity and weight gain (4–6), while short-term experimental studies have provided important mechanistic explanations (7–9). More recently, intervention studies have also been able to demonstrate that sleep restriction causes weight gain (10, 11). Collectively, the evidence suggests that insufficient sleep plays a role in the risk of gaining weight, and sleep hygiene should be considered in the prevention as well as in the treatment of obesity (12–14).}
}

RIS

TY  - JOUR
AU  - J. Chaput
AU  - M. St-Onge
T1  - Increased Food Intake by Insufficient Sleep in Humans: Are We Jumping the Gun on the Hormonal Explanation?
JO  - Frontiers in Endocrinology
PY  - 2014
VL  - 5
DO  - 10.3389/fendo.2014.00116
AB  - Concurrent with the increase in obesity prevalence, a decrease in sleep duration has been observed over the past decades (1, 2). Scientists have been intrigued by this inverse trend between sleep and body weight, and studies published in the past 10 years have been helpful in determining if the lack of sleep is a possible cause of obesity (3). Epidemiologic evidence has shown that short sleep duration is associated with obesity and weight gain (4–6), while short-term experimental studies have provided important mechanistic explanations (7–9). More recently, intervention studies have also been able to demonstrate that sleep restriction causes weight gain (10, 11). Collectively, the evidence suggests that insufficient sleep plays a role in the risk of gaining weight, and sleep hygiene should be considered in the prevention as well as in the treatment of obesity (12–14).
ER  - 

BibTeX

@misc{95230c73258787d04876a26b6751ce6697c31a71,
  author = {C. Antza and G. Kostopoulos and S. Mostafa and K. Nirantharakumar and A. Tahrani},
  title = {The links between sleep duration, obesity and type 2 diabetes mellitus},
  journal = {The Journal of Endocrinology},
  year = {2021},
  volume = {252},
  pages = {125 - 141},
  doi = {10.1530/JOE-21-0155},
  abstract = {Global rates of obesity and type 2 diabetes mellitus (T2DM) are increasing globally concomitant with a rising prevalence of sleep deprivation and sleep disorders. Understanding the links between sleep, obesity and T2DM might offer an opportunity to develop better prevention and treatment strategies for these epidemics. Experimental studies have shown that sleep restriction is associated with changes in energy homeostasis, insulin resistance and β-cell function. Epidemiological cohort studies established short sleep duration as a risk factor for developing obesity and T2DM. In addition, small studies suggested that short sleep duration was associated with less weight loss following lifestyle interventions or bariatric surgery. In this article, we review the epidemiological evidence linking sleep duration to obesity and T2DM and plausible mechanisms. In addition, we review the impact of changes in sleep duration on obesity and T2DM.}
}

RIS

TY  - JOUR
AU  - C. Antza
AU  - G. Kostopoulos
AU  - S. Mostafa
AU  - K. Nirantharakumar
AU  - A. Tahrani
T1  - The links between sleep duration, obesity and type 2 diabetes mellitus
JO  - The Journal of Endocrinology
PY  - 2021
VL  - 252
SP  - 125 - 141
EP  - 125 - 141
DO  - 10.1530/JOE-21-0155
AB  - Global rates of obesity and type 2 diabetes mellitus (T2DM) are increasing globally concomitant with a rising prevalence of sleep deprivation and sleep disorders. Understanding the links between sleep, obesity and T2DM might offer an opportunity to develop better prevention and treatment strategies for these epidemics. Experimental studies have shown that sleep restriction is associated with changes in energy homeostasis, insulin resistance and β-cell function. Epidemiological cohort studies established short sleep duration as a risk factor for developing obesity and T2DM. In addition, small studies suggested that short sleep duration was associated with less weight loss following lifestyle interventions or bariatric surgery. In this article, we review the epidemiological evidence linking sleep duration to obesity and T2DM and plausible mechanisms. In addition, we review the impact of changes in sleep duration on obesity and T2DM.
ER  - 

BibTeX

@misc{45e786d813c29201976734b8bcd8b1141899349f,
  author = {A. S. Gharamaleki and A. Zamani and M. Hosseini and S. Abedymanesh and K. Jaafarian},
  title = {1671 The Relationship Between Sleep Pattern and Obesity in Children’s of Tehran City},
  journal = {Archives of Disease in Childhood},
  year = {2012},
  volume = {97},
  pages = {A472 - A473},
  doi = {10.1136/archdischild-2012-302724.1671},
  abstract = {Objective To determine the relationship of sleep duration and efficiency with obesity in Tehran city children’s. Methods This study was conducted in Tehran city with 270 children aged 6 to 9 years. Heights, Weight, were measured and Body mass index (BMI), was calculated. Directly measured height and weight data were used to define overweight and obesity according to the centers for disease Control and Prevention’s (CDC) 2000 Growth Charts. Nocturnal sleep duration and sleep efficiency were recorded by the Actigraphy method. Results Sleep duration showed a significant inverse association with weight (p<0.05) and height (p<0.005) during 7 day and weekdays. A significant relationship was found between Sleep efficiency and weight, BMI, weight for age (p<0.05) during weekdays and weekend days. Conclusion In the current study, we found Sleep pattern may be an important environmental factor for obesity in school age children’s. These findings are important because sleep duration is a potentially modifiable risk factor that could be important to consider in the prevention and treatment of childhood obesity.}
}

RIS

TY  - JOUR
AU  - A. S. Gharamaleki
AU  - A. Zamani
AU  - M. Hosseini
AU  - S. Abedymanesh
AU  - K. Jaafarian
T1  - 1671 The Relationship Between Sleep Pattern and Obesity in Children’s of Tehran City
JO  - Archives of Disease in Childhood
PY  - 2012
VL  - 97
SP  - A472 - A473
EP  - A472 - A473
DO  - 10.1136/archdischild-2012-302724.1671
AB  - Objective To determine the relationship of sleep duration and efficiency with obesity in Tehran city children’s. Methods This study was conducted in Tehran city with 270 children aged 6 to 9 years. Heights, Weight, were measured and Body mass index (BMI), was calculated. Directly measured height and weight data were used to define overweight and obesity according to the centers for disease Control and Prevention’s (CDC) 2000 Growth Charts. Nocturnal sleep duration and sleep efficiency were recorded by the Actigraphy method. Results Sleep duration showed a significant inverse association with weight (p<0.05) and height (p<0.005) during 7 day and weekdays. A significant relationship was found between Sleep efficiency and weight, BMI, weight for age (p<0.05) during weekdays and weekend days. Conclusion In the current study, we found Sleep pattern may be an important environmental factor for obesity in school age children’s. These findings are important because sleep duration is a potentially modifiable risk factor that could be important to consider in the prevention and treatment of childhood obesity.
ER  - 

BibTeX

@misc{9b886aec11ff1f02f15d856a583f78c04b216b1e,
  author = {M. St-Onge},
  title = {The role of sleep duration in the regulation of energy balance: effects on energy intakes and expenditure.},
  journal = {Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine},
  year = {2013},
  volume = {9 1},
  pages = {
          73-80
        },
  doi = {10.5664/jcsm.2348},
  abstract = {Short sleep duration and obesity are common occurrence in today's society. An extensive literature from cross-sectional and longitudinal epidemiological studies shows a relationship between short sleep and prevalence of obesity and weight gain. However, causality cannot be inferred from such studies. Clinical intervention studies have examined whether reducing sleep in normal sleepers, typically sleeping 7-9 h/night, can affect energy intake, energy expenditure, and endocrine regulators of energy balance. The aim of this review is to evaluate studies that have assessed food intake, energy expenditure, and leptin and ghrelin levels after periods of restricted and normal sleep. Most studies support the notion that restricting sleep increases food intake, but the effects on energy expenditure are mixed. Differences in methodology and component of energy expenditure analyzed may account for the discrepancies. Studies examining the effects of sleep on leptin and ghrelin have provided conflicting results with increased, reduced, or unchanged leptin and ghrelin levels after restricted sleep compared to normal sleep. Energy balance of study participants and potential sex differences may account for the varied results. Studies should strive for constant energy balance and feeding schedules when assessing the role of sleep on hormonal profile. Although studies suggest that restricting sleep may lead to weight gain via increased food intake, research is needed to examine the impact on energy expenditure and endocrine controls. Also, studies have been of short duration, and there is little knowledge on the reverse question: does increasing sleep duration in short sleepers lead to negative energy balance?}
}

RIS

TY  - JOUR
AU  - M. St-Onge
T1  - The role of sleep duration in the regulation of energy balance: effects on energy intakes and expenditure.
JO  - Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine
PY  - 2013
VL  - 9 1
SP  - 
          73-80
        
EP  - 
          73-80
        
DO  - 10.5664/jcsm.2348
AB  - Short sleep duration and obesity are common occurrence in today's society. An extensive literature from cross-sectional and longitudinal epidemiological studies shows a relationship between short sleep and prevalence of obesity and weight gain. However, causality cannot be inferred from such studies. Clinical intervention studies have examined whether reducing sleep in normal sleepers, typically sleeping 7-9 h/night, can affect energy intake, energy expenditure, and endocrine regulators of energy balance. The aim of this review is to evaluate studies that have assessed food intake, energy expenditure, and leptin and ghrelin levels after periods of restricted and normal sleep. Most studies support the notion that restricting sleep increases food intake, but the effects on energy expenditure are mixed. Differences in methodology and component of energy expenditure analyzed may account for the discrepancies. Studies examining the effects of sleep on leptin and ghrelin have provided conflicting results with increased, reduced, or unchanged leptin and ghrelin levels after restricted sleep compared to normal sleep. Energy balance of study participants and potential sex differences may account for the varied results. Studies should strive for constant energy balance and feeding schedules when assessing the role of sleep on hormonal profile. Although studies suggest that restricting sleep may lead to weight gain via increased food intake, research is needed to examine the impact on energy expenditure and endocrine controls. Also, studies have been of short duration, and there is little knowledge on the reverse question: does increasing sleep duration in short sleepers lead to negative energy balance?
ER  - 

BibTeX

@misc{65e7aa72fcb937fac76edffb859bdf9927d21d09,
  author = {Yin Liu and M. Palta and J. Barnet and Max T. Roberts and E. Hagen and P. Peppard and E. Reither},
  title = {Habitual Sleep, Sleep Duration Differential, and Weight Change Among Adults},
  journal = {Innovation in Aging},
  year = {2021},
  volume = {5},
  pages = {35 - 35},
  doi = {10.1093/geroni/igab046.129},
  abstract = {Abstract We assessed longitudinal associations between diary-measured sleep duration and clinically assessed body mass index (BMI) among 784 men and women enrolled in the Wisconsin Sleep Cohort Study (mean [SD] age = 51.1 [8.0] years at baseline). The outcome was BMI (kg/m2). Key predictors were habitual sleep duration (defined as average weekday nighttime sleep duration) and sleep duration differential (defined as the difference between average weekday and average weekend nighttime sleep duration) at each data collection wave. Men with shorter habitual sleep duration on weekdays had higher BMI than men with longer habitual sleep duration on weekdays. Participants with larger differentials between weekday and weekend sleep duration experienced more rapid BMI gain over time for both men and women. Inadequate sleep, characterized as shorter habitual sleep during weekdays and larger weekday-weekend sleep differential, is positively associated with BMI levels and trajectories among men and women in mid-to-late life.}
}

RIS

TY  - JOUR
AU  - Yin Liu
AU  - M. Palta
AU  - J. Barnet
AU  - Max T. Roberts
AU  - E. Hagen
AU  - P. Peppard
AU  - E. Reither
T1  - Habitual Sleep, Sleep Duration Differential, and Weight Change Among Adults
JO  - Innovation in Aging
PY  - 2021
VL  - 5
SP  - 35 - 35
EP  - 35 - 35
DO  - 10.1093/geroni/igab046.129
AB  - Abstract We assessed longitudinal associations between diary-measured sleep duration and clinically assessed body mass index (BMI) among 784 men and women enrolled in the Wisconsin Sleep Cohort Study (mean [SD] age = 51.1 [8.0] years at baseline). The outcome was BMI (kg/m2). Key predictors were habitual sleep duration (defined as average weekday nighttime sleep duration) and sleep duration differential (defined as the difference between average weekday and average weekend nighttime sleep duration) at each data collection wave. Men with shorter habitual sleep duration on weekdays had higher BMI than men with longer habitual sleep duration on weekdays. Participants with larger differentials between weekday and weekend sleep duration experienced more rapid BMI gain over time for both men and women. Inadequate sleep, characterized as shorter habitual sleep during weekdays and larger weekday-weekend sleep differential, is positively associated with BMI levels and trajectories among men and women in mid-to-late life.
ER  - 

BibTeX

@misc{309b5d608fd7190a3d68071328f2adaa3d3c95e7,
  author = {D. Lauderdale and K. Knutson and P. Rathouz and Lijing L. Yan and S. Hulley and Kiang Liu},
  title = {American Journal of Epidemiology Original Contribution Cross-sectional and Longitudinal Associations between Objectively Measured Sleep Duration and Body Mass Index the Cardia Sleep Study},
  abstract = {Numerous studies have found an association between shorter sleep duration and higher body mass index (BMI) in adults. Most previous studies have been cross-sectional and relied on self-reported sleep duration, which may not be very accurate. In the Coronary Artery Risk Development in Young Adults (CARDIA) Sleep Study (2000– 2006), the authors examine whether objectively measured sleep is associated with BMI and change in BMI. They use several nights of wrist actigraphy to measure sleep among participants in an ongoing cohort of middle-aged adults. By use of linear regression, the authors examine whether average sleep duration or fragmentation is associated with BMI and 5-year change in BMI, adjusting for confounders. Among 612 participants, sleep duration averaged 6.1 hours and was grouped into 4 categories. Both shorter sleep and greater fragmentation were strongly associated with higher BMI in unadjusted cross-sectional analysis. After adjustment, BMI decreased by 0.78 kg/m 2 (95% confidence interval: À1.6, À0.002) for each increasing sleep category. The association was very strong in persons who reported snoring and weak in those who did not. There were no longitudinal associations between sleep measurements and change in BMI. The authors confirmed a cross-sectional association between sleep duration and BMI using objective sleep measures, but they did not find that sleep predicted change in BMI. The mechanism underlying the cross-sectional association is not clear. Numerous studies have found a cross-sectional association between sleep duration and body mass index (BMI) in adults: Those reporting shorter sleep are more likely to have higher BMI (1–3). The possibility that this is a causal association and that declining sleep hours could be contributing to the obesity epidemic has heightened interest among researchers and the public (4), but there are critical gaps in the epidemiologic evidence (5). Cross-sectional studies present ambiguous evidence concerning the causal direction. In this case, both directions are plausible. Persons who weigh more may sleep less, perhaps because of comorbidities or reduced sleep quality, or persons who sleep less may gain weight, perhaps because of increased hunger or late-night eating. A third possibility is that the association is not causal in either direction but due to as yet unidentified confounding. Some studies find a U-shaped association with increased risk of obesity for both short and long sleepers (6–8). Several established cohorts have sleep information and have examined this association longitudinally, with mixed}
}

RIS

TY  - JOUR
AU  - D. Lauderdale
AU  - K. Knutson
AU  - P. Rathouz
AU  - Lijing L. Yan
AU  - S. Hulley
AU  - Kiang Liu
T1  - American Journal of Epidemiology Original Contribution Cross-sectional and Longitudinal Associations between Objectively Measured Sleep Duration and Body Mass Index the Cardia Sleep Study
AB  - Numerous studies have found an association between shorter sleep duration and higher body mass index (BMI) in adults. Most previous studies have been cross-sectional and relied on self-reported sleep duration, which may not be very accurate. In the Coronary Artery Risk Development in Young Adults (CARDIA) Sleep Study (2000– 2006), the authors examine whether objectively measured sleep is associated with BMI and change in BMI. They use several nights of wrist actigraphy to measure sleep among participants in an ongoing cohort of middle-aged adults. By use of linear regression, the authors examine whether average sleep duration or fragmentation is associated with BMI and 5-year change in BMI, adjusting for confounders. Among 612 participants, sleep duration averaged 6.1 hours and was grouped into 4 categories. Both shorter sleep and greater fragmentation were strongly associated with higher BMI in unadjusted cross-sectional analysis. After adjustment, BMI decreased by 0.78 kg/m 2 (95% confidence interval: À1.6, À0.002) for each increasing sleep category. The association was very strong in persons who reported snoring and weak in those who did not. There were no longitudinal associations between sleep measurements and change in BMI. The authors confirmed a cross-sectional association between sleep duration and BMI using objective sleep measures, but they did not find that sleep predicted change in BMI. The mechanism underlying the cross-sectional association is not clear. Numerous studies have found a cross-sectional association between sleep duration and body mass index (BMI) in adults: Those reporting shorter sleep are more likely to have higher BMI (1–3). The possibility that this is a causal association and that declining sleep hours could be contributing to the obesity epidemic has heightened interest among researchers and the public (4), but there are critical gaps in the epidemiologic evidence (5). Cross-sectional studies present ambiguous evidence concerning the causal direction. In this case, both directions are plausible. Persons who weigh more may sleep less, perhaps because of comorbidities or reduced sleep quality, or persons who sleep less may gain weight, perhaps because of increased hunger or late-night eating. A third possibility is that the association is not causal in either direction but due to as yet unidentified confounding. Some studies find a U-shaped association with increased risk of obesity for both short and long sleepers (6–8). Several established cohorts have sleep information and have examined this association longitudinally, with mixed
ER  - 

BibTeX

@misc{5964ce088b359221e2fefceb45d0866d66c2c4bb,
  author = {Annika Janson},
  title = {Sleeping it off? The value of sleep in children treated for obesity},
  journal = {Acta Paediatrica},
  year = {2022},
  volume = {111},
  pages = {1296 - 1297},
  doi = {10.1111/apa.16366},
  abstract = {There is something counterintuitive about sleep and obesity. We use less energy while sleeping, yet lack of sleep may be one of the reasons why people gain weight. If so, sleep would be one of the modifiable factors for preventing overweight and obesity. We sleep less than we used to. Relatively recent changes, such as electric light, and very recent changes, such as mobile phones, have dissociated our sleeping habits from daylight which dictated the patterns for millennia. So, are we more likely to gain surplus weight if we have poor sleeping habits? Or are we looking at a reverse causality, where people with overweight or obesity are more likely to have disturbed sleep? Or are we tricked by confounders, factors common to obesity and sleep disturbances? There are plenty of candidates, such as sedentary behaviour, alcohol use, stress and psychiatric illnesses. For children, the American Association of Pediatrics has endorsed the consensus recommendation by their colleagues in the American Academy of Sleep Medicine. Children aged 3– 5 years should sleep 10– 13 h, including naps, children aged 6– 12 years should sleep 9– 12 h, and teenagers should sleep 8– 10 h per 24 h.1 Data on children do not look too bad, although reports are conflicting. For girls and boys aged 6– 9 years in 25 countries in the WHO European Childhood Obesity Surveillance Initiative (COSI), most children achieved 9– 11 h of sleep. A fair fraction of those who did not were actually sleeping more.2 For adolescents, we know less and fear worse. Many studies have confirmed the association between lack of sleep and overweight.3 A systematic review of 42 prospective studies could demonstrate an effect size with small reductions in BMI for every additional hour of sleep.4 Some researchers have argued that the relationship is Ushaped; both children who sleep very much and those who sleep little are at increased risk of obesity.5 Others described a linear relationship between the amount of sleep and the risk of obesity.6 A lack of sleep affects metabolism and the appetiteregulating hormones, the desire to eat and food selection. Some of the landmark studies were carried out a few decades ago. In one of these studies, eleven young men were studied for 16 nights. They were allowed to sleep for 4 h per night for 6 nights and then they were able to catch up. During sleep deprivation, the insulin sensitivity decreased, and the metabolism of carbohydrates was impaired.7 In another study with a similar design, eleven adult men and women were studied for two 14 days periods and had free access to food. During the days of sleep deprivation, the participants ate more snacks.8 Researchers can keep their study participants awake and send them to bed, but they cannot make them sleep unless they use medications. The groundbreaking allinclusive studies were probably both expensive and cumbersome. Instead, most studies rely on selfreported data on the time, duration, and quality of sleep. In the case of children, parents are often the reporters, which adds to the complexity. Important confounders in children would be neuropsychiatric illnesses and psychosocial wellbeing. In a Swedish study, twothirds of adolescents seeking surgical weight loss presented with substantial mental health problems as reported by themselves or their parents.9 The everyday struggle for some of these adolescents may evolve into vicious circles of tiredness, sedentary behaviour and disturbed eating and sleeping patterns. And for the reverse, good parenting skills may affect both meal structure and sleeping habits positively. Clearly, the mechanisms, causality and confounders that may explain the association between obesity and sleep are still not fully understood. In this issue of Acta Paediatrica, Kjetså et al. make an important contribution to the field by reporting how sleeping habits at baseline affected the outcome for children who participated in obesity treatment, in addition to age and family income.10 It sounds like an obvious approach, but the effect of sleeping habits on the results of obesity treatment is much less studied than the associations between sleep and body weight for children in general. Kjetså ́s study is a followup of 97 children aged 6– 12 years with a body mass index (BMI) just under the cutoff for obesity and above, corresponding to adult BMI 27.5 kg/m2 and above. The children had been included in a 2 years randomised study, the Finnmark Activity School study in Norway, with typical lifestyle components. The authors gathered data on 62/97 (64%) at three years, one year after the end of the intervention, and regardless of randomisation.10 For a longterm evaluation of obesity treatment, 64% is a decent retention rate. At baseline, most children slept 9– 11 h per night and had more than 2 h of screen time. The authors showed that the children who reported longer sleep duration at baseline had lost more weight at three years.10}
}

RIS

TY  - JOUR
AU  - Annika Janson
T1  - Sleeping it off? The value of sleep in children treated for obesity
JO  - Acta Paediatrica
PY  - 2022
VL  - 111
SP  - 1296 - 1297
EP  - 1296 - 1297
DO  - 10.1111/apa.16366
AB  - There is something counterintuitive about sleep and obesity. We use less energy while sleeping, yet lack of sleep may be one of the reasons why people gain weight. If so, sleep would be one of the modifiable factors for preventing overweight and obesity. We sleep less than we used to. Relatively recent changes, such as electric light, and very recent changes, such as mobile phones, have dissociated our sleeping habits from daylight which dictated the patterns for millennia. So, are we more likely to gain surplus weight if we have poor sleeping habits? Or are we looking at a reverse causality, where people with overweight or obesity are more likely to have disturbed sleep? Or are we tricked by confounders, factors common to obesity and sleep disturbances? There are plenty of candidates, such as sedentary behaviour, alcohol use, stress and psychiatric illnesses. For children, the American Association of Pediatrics has endorsed the consensus recommendation by their colleagues in the American Academy of Sleep Medicine. Children aged 3– 5 years should sleep 10– 13 h, including naps, children aged 6– 12 years should sleep 9– 12 h, and teenagers should sleep 8– 10 h per 24 h.1 Data on children do not look too bad, although reports are conflicting. For girls and boys aged 6– 9 years in 25 countries in the WHO European Childhood Obesity Surveillance Initiative (COSI), most children achieved 9– 11 h of sleep. A fair fraction of those who did not were actually sleeping more.2 For adolescents, we know less and fear worse. Many studies have confirmed the association between lack of sleep and overweight.3 A systematic review of 42 prospective studies could demonstrate an effect size with small reductions in BMI for every additional hour of sleep.4 Some researchers have argued that the relationship is Ushaped; both children who sleep very much and those who sleep little are at increased risk of obesity.5 Others described a linear relationship between the amount of sleep and the risk of obesity.6 A lack of sleep affects metabolism and the appetiteregulating hormones, the desire to eat and food selection. Some of the landmark studies were carried out a few decades ago. In one of these studies, eleven young men were studied for 16 nights. They were allowed to sleep for 4 h per night for 6 nights and then they were able to catch up. During sleep deprivation, the insulin sensitivity decreased, and the metabolism of carbohydrates was impaired.7 In another study with a similar design, eleven adult men and women were studied for two 14 days periods and had free access to food. During the days of sleep deprivation, the participants ate more snacks.8 Researchers can keep their study participants awake and send them to bed, but they cannot make them sleep unless they use medications. The groundbreaking allinclusive studies were probably both expensive and cumbersome. Instead, most studies rely on selfreported data on the time, duration, and quality of sleep. In the case of children, parents are often the reporters, which adds to the complexity. Important confounders in children would be neuropsychiatric illnesses and psychosocial wellbeing. In a Swedish study, twothirds of adolescents seeking surgical weight loss presented with substantial mental health problems as reported by themselves or their parents.9 The everyday struggle for some of these adolescents may evolve into vicious circles of tiredness, sedentary behaviour and disturbed eating and sleeping patterns. And for the reverse, good parenting skills may affect both meal structure and sleeping habits positively. Clearly, the mechanisms, causality and confounders that may explain the association between obesity and sleep are still not fully understood. In this issue of Acta Paediatrica, Kjetså et al. make an important contribution to the field by reporting how sleeping habits at baseline affected the outcome for children who participated in obesity treatment, in addition to age and family income.10 It sounds like an obvious approach, but the effect of sleeping habits on the results of obesity treatment is much less studied than the associations between sleep and body weight for children in general. Kjetså ́s study is a followup of 97 children aged 6– 12 years with a body mass index (BMI) just under the cutoff for obesity and above, corresponding to adult BMI 27.5 kg/m2 and above. The children had been included in a 2 years randomised study, the Finnmark Activity School study in Norway, with typical lifestyle components. The authors gathered data on 62/97 (64%) at three years, one year after the end of the intervention, and regardless of randomisation.10 For a longterm evaluation of obesity treatment, 64% is a decent retention rate. At baseline, most children slept 9– 11 h per night and had more than 2 h of screen time. The authors showed that the children who reported longer sleep duration at baseline had lost more weight at three years.10
ER  - 

BibTeX

@misc{3c67e72a0c72dc3f0c2e37a8bde5a08108ac54a8,
  author = {L. Meng and Ai-ling Liu and Xiaoqi Hu and Qian Zhang and S. Du and H. Fang and Jun Ma and Guifa Xu and Ying Li and H. Guo and L. Du and G. Ma},
  title = {Report on childhood obesity in China (10): association of sleep duration with obesity.},
  journal = {Biomedical and environmental sciences : BES},
  year = {2012},
  volume = {25 2},
  pages = {
          133-40
        },
  doi = {10.3967/0895-3988.2012.02.002},
  abstract = {OBJECTIVES
To explore the association of sleep duration with obesity among children in urban areas of China.


METHODS
A total of 6 576 children (3 293 boys and 3 283 girls) aged 7-11 years were randomly selected from 36 primary schools in 6 metropolitan cities in China. A 7-day Physical Activity Recall was used to assess the sleep duration and physical activity level. The height, weight, waist circumference (WC) and percentage of body fat (%BF, as determined by bioelectrical impedance analysis technique) were measured by following the standardized operation procedures. The information on demography, lifestyle and eating habits was collected with a self-administered questionnaire from participants and their parents.


RESULTS
The average sleep duration per night in the children was 9.7 h with the decreasing trends along with the increase of age (P < 0.05). The sleep duration was negatively associated with body mass index (BMI) and WC in both boys and girls after adjustment for confounders (beta value -0.23 and -0.82 for boys, -0.24 and -0.91 for girls, respectively, P < 0.01). However, no significant association of sleep duration with %BF was found. Children who slept less than 9.0 h per night had a higher risk for overweight and obesity (OR = 1.29, 95% CI: 1.01, 1.64) and abdominal obesity (OR=1.38, 95% CI: 1.04, 1.83) as compared with those who slept for 10.0-10.9 h.


CONCLUSIONS
Short sleep duration is associated with obesity. It is important to ensure adequate sleep duration of children and foster their healthy lifestyle at an early stage of life.}
}

RIS

TY  - JOUR
AU  - L. Meng
AU  - Ai-ling Liu
AU  - Xiaoqi Hu
AU  - Qian Zhang
AU  - S. Du
AU  - H. Fang
AU  - Jun Ma
AU  - Guifa Xu
AU  - Ying Li
AU  - H. Guo
AU  - L. Du
AU  - G. Ma
T1  - Report on childhood obesity in China (10): association of sleep duration with obesity.
JO  - Biomedical and environmental sciences : BES
PY  - 2012
VL  - 25 2
SP  - 
          133-40
        
EP  - 
          133-40
        
DO  - 10.3967/0895-3988.2012.02.002
AB  - OBJECTIVES
To explore the association of sleep duration with obesity among children in urban areas of China.


METHODS
A total of 6 576 children (3 293 boys and 3 283 girls) aged 7-11 years were randomly selected from 36 primary schools in 6 metropolitan cities in China. A 7-day Physical Activity Recall was used to assess the sleep duration and physical activity level. The height, weight, waist circumference (WC) and percentage of body fat (%BF, as determined by bioelectrical impedance analysis technique) were measured by following the standardized operation procedures. The information on demography, lifestyle and eating habits was collected with a self-administered questionnaire from participants and their parents.


RESULTS
The average sleep duration per night in the children was 9.7 h with the decreasing trends along with the increase of age (P < 0.05). The sleep duration was negatively associated with body mass index (BMI) and WC in both boys and girls after adjustment for confounders (beta value -0.23 and -0.82 for boys, -0.24 and -0.91 for girls, respectively, P < 0.01). However, no significant association of sleep duration with %BF was found. Children who slept less than 9.0 h per night had a higher risk for overweight and obesity (OR = 1.29, 95% CI: 1.01, 1.64) and abdominal obesity (OR=1.38, 95% CI: 1.04, 1.83) as compared with those who slept for 10.0-10.9 h.


CONCLUSIONS
Short sleep duration is associated with obesity. It is important to ensure adequate sleep duration of children and foster their healthy lifestyle at an early stage of life.
ER  - 

BibTeX

@misc{14607b66a16265444f5bd938ad07b8509be42dfb,
  author = {H. Al-Hazzaa and A. Musaiger and Nada A. Abahussain and H. Al-Sobayel and Dina M Qahwaji},
  title = {Prevalence of short sleep duration and its association with obesity among adolescents 15- to 19-year olds: A cross-sectional study from three major cities in Saudi Arabia},
  journal = {Annals of Thoracic Medicine},
  year = {2012},
  volume = {7},
  pages = {133 - 139},
  doi = {10.4103/1817-1737.98845},
  abstract = {BACKGROUND: Adequate sleep has been considered important for the adolescent's health and well being. On the other hand, self-imposed sleep curtailment is now recognized as a potentially important and novel risk factor for obesity. The present study aimed to assess the prevalence of short sleep duration and its association with obesity among Saudi adolescents. METHODS: This is a school-based cross-sectional study with self-reported sleep questionnaires. It was conducted during the years 2009/2010 in three cities in Saudi Arabia; Al-Khobar, Jeddah, and Riyadh. Participants were 2868 secondary-school males (1379) and females (1389) aged 15 to 19 years, randomly selected using a multistage stratified sampling technique. Measurements included weight, height, waist circumference, BMI, and sleeping duration. Logistic regression analysis while adjusted for age, gender, and location was used to examine the associations between sleep duration and obesity measures. RESULTS: The mean (SD) of sleep duration was 7.2 (1.6) hours/day with no significant differences between males and females. About 31% of the participants obtain less than 7 hours of sleep per day, while approximately 50% of the sample gets less than 8 hours of daily sleep. Two-way ANCOVA results while controlling for the effect of age revealed a significant gender by school-type interaction (P<0.001). In addition, adequate sleep duration increased the odds of having normal weight (adjusted odds ratios = 1.28, 95% CI = 1.08-1.50, P = 0.003). CONCLUSION: The present study observed a high prevalence of short sleep duration among Saudi adolescents 15- to 19-year olds and that short sleep duration was significantly associated with increased risk of overweight and obesity. Future interventions should investigate whether adopting a healthy lifestyle by adolescents with short sleep duration would improve their sleeping habits or not.}
}

RIS

TY  - JOUR
AU  - H. Al-Hazzaa
AU  - A. Musaiger
AU  - Nada A. Abahussain
AU  - H. Al-Sobayel
AU  - Dina M Qahwaji
T1  - Prevalence of short sleep duration and its association with obesity among adolescents 15- to 19-year olds: A cross-sectional study from three major cities in Saudi Arabia
JO  - Annals of Thoracic Medicine
PY  - 2012
VL  - 7
SP  - 133 - 139
EP  - 133 - 139
DO  - 10.4103/1817-1737.98845
AB  - BACKGROUND: Adequate sleep has been considered important for the adolescent's health and well being. On the other hand, self-imposed sleep curtailment is now recognized as a potentially important and novel risk factor for obesity. The present study aimed to assess the prevalence of short sleep duration and its association with obesity among Saudi adolescents. METHODS: This is a school-based cross-sectional study with self-reported sleep questionnaires. It was conducted during the years 2009/2010 in three cities in Saudi Arabia; Al-Khobar, Jeddah, and Riyadh. Participants were 2868 secondary-school males (1379) and females (1389) aged 15 to 19 years, randomly selected using a multistage stratified sampling technique. Measurements included weight, height, waist circumference, BMI, and sleeping duration. Logistic regression analysis while adjusted for age, gender, and location was used to examine the associations between sleep duration and obesity measures. RESULTS: The mean (SD) of sleep duration was 7.2 (1.6) hours/day with no significant differences between males and females. About 31% of the participants obtain less than 7 hours of sleep per day, while approximately 50% of the sample gets less than 8 hours of daily sleep. Two-way ANCOVA results while controlling for the effect of age revealed a significant gender by school-type interaction (P<0.001). In addition, adequate sleep duration increased the odds of having normal weight (adjusted odds ratios = 1.28, 95% CI = 1.08-1.50, P = 0.003). CONCLUSION: The present study observed a high prevalence of short sleep duration among Saudi adolescents 15- to 19-year olds and that short sleep duration was significantly associated with increased risk of overweight and obesity. Future interventions should investigate whether adopting a healthy lifestyle by adolescents with short sleep duration would improve their sleeping habits or not.
ER  - 

BibTeX

@misc{6bb9411f609f501ee2c411000e27f027b4224b83,
  author = {L. Mamluk and L. J. Murray and M. Cantwell},
  title = {Sleep duration and body composition in adolescents},
  journal = {Proceedings of the Nutrition Society},
  year = {2011},
  volume = {70},
  doi = {10.1017/S0029665111001194},
  abstract = {The complexity of obesity pathogenesis requires the identification of novel risk factors. Increasing research suggests a relationship between sleep duration and obesity in adults and children; however, evidence in adolescents is scarce. Adolescence may be a crucial period for the modification of obesity risk factors and therefore in the prevention of its detrimental outcomes in adulthood. This study aims to examine the relationship between sleep duration and weight-related parameters in adolescents. Data from the Young Hearts 2000, a cross-sectional cohort study carried out in 1999–2001, were examined. The 2017 participants from Northern Ireland were aged 12 and 15 years. The relationship between BMI z-scores, waist circumference and skin-fold thickness and self-reported sleep variables was investigated using linear regression, gender stratified analyses after adjusting for age, energy intake, physical activity, smoking, pubertal status, social class and T.V. viewing. The prevalence of obesity and overweight was 4.4 and 15.8%, respectively. Mean sleep duration on school days was 8.9(0.95) h and 10.2(1.3) h during weekends (P<0.0001). Average sleep duration per day (school days and weekends) was 9.24(0.81) h. Duration of average night-time sleep per day was negatively associated with BMI z-score and waist circumference after adjusting for confounders, as shown in the table. For every hour increase in sleep duration, an 0.1 reduction in BMI z-scores and 6.8 mm in waist circumference was shown. The same relationship was not observed for % body fat. When stratified by gender, sleep duration was not associated with measures of obesity in females, however, remained significant in males for BMI z-score and waist circumference. An hour increase in sleep duration was associated with a 0.13 lower BMI z-score and a 12 mm reduction in waist circumference.}
}

RIS

TY  - JOUR
AU  - L. Mamluk
AU  - L. J. Murray
AU  - M. Cantwell
T1  - Sleep duration and body composition in adolescents
JO  - Proceedings of the Nutrition Society
PY  - 2011
VL  - 70
DO  - 10.1017/S0029665111001194
AB  - The complexity of obesity pathogenesis requires the identification of novel risk factors. Increasing research suggests a relationship between sleep duration and obesity in adults and children; however, evidence in adolescents is scarce. Adolescence may be a crucial period for the modification of obesity risk factors and therefore in the prevention of its detrimental outcomes in adulthood. This study aims to examine the relationship between sleep duration and weight-related parameters in adolescents. Data from the Young Hearts 2000, a cross-sectional cohort study carried out in 1999–2001, were examined. The 2017 participants from Northern Ireland were aged 12 and 15 years. The relationship between BMI z-scores, waist circumference and skin-fold thickness and self-reported sleep variables was investigated using linear regression, gender stratified analyses after adjusting for age, energy intake, physical activity, smoking, pubertal status, social class and T.V. viewing. The prevalence of obesity and overweight was 4.4 and 15.8%, respectively. Mean sleep duration on school days was 8.9(0.95) h and 10.2(1.3) h during weekends (P<0.0001). Average sleep duration per day (school days and weekends) was 9.24(0.81) h. Duration of average night-time sleep per day was negatively associated with BMI z-score and waist circumference after adjusting for confounders, as shown in the table. For every hour increase in sleep duration, an 0.1 reduction in BMI z-scores and 6.8 mm in waist circumference was shown. The same relationship was not observed for % body fat. When stratified by gender, sleep duration was not associated with measures of obesity in females, however, remained significant in males for BMI z-score and waist circumference. An hour increase in sleep duration was associated with a 0.13 lower BMI z-score and a 12 mm reduction in waist circumference.
ER  - 

BibTeX

@misc{f677a1be3dadb7c2f9428b136b34d7eacffa755c,
  author = {Y. Rhie},
  title = {The Association between Sleep Duration and Overweight in a School-Age Population in Seoul (J Obes Metab Syndr 2017;26:45–51)},
  journal = {Journal of Obesity & Metabolic Syndrome},
  year = {2017},
  volume = {26},
  pages = {231 - 232},
  doi = {10.7570/jomes.2017.26.3.231},
  abstract = {Sufficient sleep is an important factor for a healthy lifestyle. In addition to restoring body functions and recharging energy, sleep plays many important roles in various physiological functions. Sleep affects growth and pubertal development in children and adolescents. Kwon et al.1 showed that sleep duration was related to age at menarche in Korean elementary school girls. Sleep duration is inversely related to the prevalence of chronic diseases, including metabolic syndrome, type 2 diabetes mellitus, dyslipidemia, and cardiovascular diseases.2 Individuals with an insufficient sleep duration showed an increased risk of obesity.3 Pacheco et al.4 suggested a link between overweight and obesity and altered sleep quality and also a link between slow-wave sleep duration and insulin resistance. Insufficient sleep results in the dysregulation of hormones that control appetite and energy balance and subsequently increases the risk of obesity.5 Shin6 investigated the association between sleep duration and overweight in a school-age population in Seoul using the secondary data of a sample survey (n = 25,182) of the Student Health Examination in 2010. Shin6 identified the proportions of those who were overweight [World Health Organization (WHO) definition of body mass index (BMI) > 1.0 standard deviation score (SDS)] and explored the association of overweight with sleep duration after adjusting for age and for meat and vegetable intake using multiple logistic and linear regression analyses. Shin6 found that only 23.2% of participants slept 8 hours or more per day, and the proportions of overweight participants were 34.2% of boys and 19.5% of girls. Shin6 also showed that significant odds ratios for overweight according to sleep duration (based on ≥ 8 sleeping hours) were 1.28 (for less than 6 hours), 1.31 (for 6-7 hours), and 1.28 (for 7-8 hours) in boys and 1.19 (for 6-7 hours) and 1.17 (for 7-8 hours) in girls. These findings are very interesting and significant in terms of sleep as a causal variable of obesity in the Korean school-aged population. They also call attention to the activities that use sleep time to meet health needs. The fact that most students do not obtain an optimal sleep duration ( ≥ 8 hours) seems to be related to the educational environment in Korea. This study suggests that a school health policy for ensuring optimal sleep duration in the school-aged population should be developed for prevention of obesity. The quality of sleep also has a significant relationship with overweight and obesity.4 However, the association between the quality of sleep and overweight could not be investigated in this study. As Shin6 mentioned, this study defined overweight according to BMI SDS using growth reference values for 5-19-year-olds, as re-}
}

RIS

TY  - JOUR
AU  - Y. Rhie
T1  - The Association between Sleep Duration and Overweight in a School-Age Population in Seoul (J Obes Metab Syndr 2017;26:45–51)
JO  - Journal of Obesity & Metabolic Syndrome
PY  - 2017
VL  - 26
SP  - 231 - 232
EP  - 231 - 232
DO  - 10.7570/jomes.2017.26.3.231
AB  - Sufficient sleep is an important factor for a healthy lifestyle. In addition to restoring body functions and recharging energy, sleep plays many important roles in various physiological functions. Sleep affects growth and pubertal development in children and adolescents. Kwon et al.1 showed that sleep duration was related to age at menarche in Korean elementary school girls. Sleep duration is inversely related to the prevalence of chronic diseases, including metabolic syndrome, type 2 diabetes mellitus, dyslipidemia, and cardiovascular diseases.2 Individuals with an insufficient sleep duration showed an increased risk of obesity.3 Pacheco et al.4 suggested a link between overweight and obesity and altered sleep quality and also a link between slow-wave sleep duration and insulin resistance. Insufficient sleep results in the dysregulation of hormones that control appetite and energy balance and subsequently increases the risk of obesity.5 Shin6 investigated the association between sleep duration and overweight in a school-age population in Seoul using the secondary data of a sample survey (n = 25,182) of the Student Health Examination in 2010. Shin6 identified the proportions of those who were overweight [World Health Organization (WHO) definition of body mass index (BMI) > 1.0 standard deviation score (SDS)] and explored the association of overweight with sleep duration after adjusting for age and for meat and vegetable intake using multiple logistic and linear regression analyses. Shin6 found that only 23.2% of participants slept 8 hours or more per day, and the proportions of overweight participants were 34.2% of boys and 19.5% of girls. Shin6 also showed that significant odds ratios for overweight according to sleep duration (based on ≥ 8 sleeping hours) were 1.28 (for less than 6 hours), 1.31 (for 6-7 hours), and 1.28 (for 7-8 hours) in boys and 1.19 (for 6-7 hours) and 1.17 (for 7-8 hours) in girls. These findings are very interesting and significant in terms of sleep as a causal variable of obesity in the Korean school-aged population. They also call attention to the activities that use sleep time to meet health needs. The fact that most students do not obtain an optimal sleep duration ( ≥ 8 hours) seems to be related to the educational environment in Korea. This study suggests that a school health policy for ensuring optimal sleep duration in the school-aged population should be developed for prevention of obesity. The quality of sleep also has a significant relationship with overweight and obesity.4 However, the association between the quality of sleep and overweight could not be investigated in this study. As Shin6 mentioned, this study defined overweight according to BMI SDS using growth reference values for 5-19-year-olds, as re-
ER  - 

BibTeX

@misc{06a0b721e132c3a6020142f2cfd583da079ef2fe,
  author = {S. Taheri},
  title = {The role of sleep in metabolism and obesity},
  journal = {},
  year = {2008},
  volume = {},
  abstract = {Sleep is a complex behaviour whose precise physiological functions are unknown. Traditionally, it was believed that sleep is only for the brain, but increasingly, the role of sleep in other organs has been recognized. Recently the role of sleep duration and sleep disorders in the regulation of metabolism is increasingly appreciated. The hypothalamus, which is a major regulator for homeostatic regulatory mechanisms, is also a major regulator of sleep and wakefulness allowing integration of sleep, appetite, and metabolism. In particular, the lateral hypothalamic orexin (hypocretin) neurons have been shown to play a key role in wakefulness, appetite regulation, glucose sensing, locomotor activity and energy expenditure. The importance of hypothalamic integration of sleep and metabolism has been shown in both animals and also humans who suffer from the sleep disorder, narcolepsy. Narcolepsy is a profound neurological sleep disorder that results in excessive daytime sleepiness, but is also associated with obesity and insulin resistance. Patients with narcolepsy have undetectable orexin (hypocretin) neuropeptide levels in their cerebrospinal fluid and postmortem studies have shown that orexin (hypocretin) mRNA expression is absent in hypothalami from patients with narcolepsy. Data from large population studies show that both long and short sleep duration are associated with obesity, the metabolic syndrome, diabetes, cardiovascular disease and mortality. These studies have been carried out across all age groups, and in several countries and ethnic groups. In a study of over 1000 individuals, short sleep duration was shown to be associated with greater body mass index but lower levels of the adipocytokine hormone leptin and higher levels of the stomach-derived hormone ghrelin. Low leptin and high ghrelin levels are a powerful appetite stimulatory signal. Data from other population studies suggest a relationship between sleep duration and physical activity. Human sleep laboratory studies have shown that both short sleep duration and sleep disruption are associated with metabolic derangements that are associated with the metabolic syndrome and diabetes. These studies also report associations between shorter sleep and energy expenditure. The objective of the presentation will be to discuss data available regarding the relation between sleep, metabolism and obesity including potential neurohormonal mechanisms. Taheri S, Lin L, Austin D, Young T, Mignot E (2004). PLoS Med 1(3):e62.}
}

RIS

TY  - JOUR
AU  - S. Taheri
T1  - The role of sleep in metabolism and obesity
PY  - 2008
AB  - Sleep is a complex behaviour whose precise physiological functions are unknown. Traditionally, it was believed that sleep is only for the brain, but increasingly, the role of sleep in other organs has been recognized. Recently the role of sleep duration and sleep disorders in the regulation of metabolism is increasingly appreciated. The hypothalamus, which is a major regulator for homeostatic regulatory mechanisms, is also a major regulator of sleep and wakefulness allowing integration of sleep, appetite, and metabolism. In particular, the lateral hypothalamic orexin (hypocretin) neurons have been shown to play a key role in wakefulness, appetite regulation, glucose sensing, locomotor activity and energy expenditure. The importance of hypothalamic integration of sleep and metabolism has been shown in both animals and also humans who suffer from the sleep disorder, narcolepsy. Narcolepsy is a profound neurological sleep disorder that results in excessive daytime sleepiness, but is also associated with obesity and insulin resistance. Patients with narcolepsy have undetectable orexin (hypocretin) neuropeptide levels in their cerebrospinal fluid and postmortem studies have shown that orexin (hypocretin) mRNA expression is absent in hypothalami from patients with narcolepsy. Data from large population studies show that both long and short sleep duration are associated with obesity, the metabolic syndrome, diabetes, cardiovascular disease and mortality. These studies have been carried out across all age groups, and in several countries and ethnic groups. In a study of over 1000 individuals, short sleep duration was shown to be associated with greater body mass index but lower levels of the adipocytokine hormone leptin and higher levels of the stomach-derived hormone ghrelin. Low leptin and high ghrelin levels are a powerful appetite stimulatory signal. Data from other population studies suggest a relationship between sleep duration and physical activity. Human sleep laboratory studies have shown that both short sleep duration and sleep disruption are associated with metabolic derangements that are associated with the metabolic syndrome and diabetes. These studies also report associations between shorter sleep and energy expenditure. The objective of the presentation will be to discuss data available regarding the relation between sleep, metabolism and obesity including potential neurohormonal mechanisms. Taheri S, Lin L, Austin D, Young T, Mignot E (2004). PLoS Med 1(3):e62.
ER  - 

BibTeX

@misc{b6b5832a74f40c041ebbbf2d09fc999f00f18a64,
  author = {Xiao-fang Zou and Chunyan Li},
  title = {PO-108 The relationship between Obesity and Sleep behavior in adolescents},
  journal = {Exercise Biochemistry Review},
  year = {2018},
  doi = {10.14428/EBR.V1I4.9033},
  abstract = {Objective With the increasing detection rate of overweight and obesity in adolescents, many kinds of fat-related hazards affect the quality of life. This paper aims to study the relationship between overweight and obesity and sleep behavior in adolescents. To provide theoretical basis for adolescent obesity intervention policy according to the relevant links. 
Methods A questionnaire survey was conducted on 156 (84 males, 72 females) aged 12 to 18 years old to collect information on their basic physical condition and sleep related behaviors, and the questionnaire was collected on the spot. Subjects were divided into three groups according to BMI: normal, overweight and obese. The gender and BMI groups were analyzed and compared. 
Results  (1) Obesity The rate of obesity in boys was higher than that in girls , (P < 0. 05). (2). The rate of staying up late during workday (71.7%) and early rising (83.0%) in obese group was higher than that in girls (83.0%). In obese group, the sleeping time was longer (25.16 ±6.3min) and had no siesta behavior (30.8%). In normal group, 15-30min (38.6%), (P < 0.05). (3) BMI (29.86 ±7.53) in boys was significantly higher than that in girls (26.85 ±5.50), (), while in normal group (38.6%), (P < 0.05). (3), BMI in boys (29.86 ±7.53) was significantly higher than that in girls (26.85 ±5.50), ( P < 0.05). Male students with no siesta behavior and nap time in 15-30 minutes accounted for more (25. 5%), while female students had more lunch break time between 30 and 60 minutes (36.9%). (4). Male students' nap time and sleep duration were negatively correlated with BMI , (P < 0. 05). BMI was proportional to nap time and sleep time, but was inversely proportional to sleep time , (P < 0. 05). 
Conclusions Sleep behavior of adolescent boys and girls may have an effect on body weight status. Staying up late, lack of sleep and lack of lunch time will aggravate obesity and reduce sleep barrier. In addition, shortening sleep time can improve overweight and obesity status.}
}

RIS

TY  - JOUR
AU  - Xiao-fang Zou
AU  - Chunyan Li
T1  - PO-108 The relationship between Obesity and Sleep behavior in adolescents
JO  - Exercise Biochemistry Review
PY  - 2018
DO  - 10.14428/EBR.V1I4.9033
AB  - Objective With the increasing detection rate of overweight and obesity in adolescents, many kinds of fat-related hazards affect the quality of life. This paper aims to study the relationship between overweight and obesity and sleep behavior in adolescents. To provide theoretical basis for adolescent obesity intervention policy according to the relevant links. 
Methods A questionnaire survey was conducted on 156 (84 males, 72 females) aged 12 to 18 years old to collect information on their basic physical condition and sleep related behaviors, and the questionnaire was collected on the spot. Subjects were divided into three groups according to BMI: normal, overweight and obese. The gender and BMI groups were analyzed and compared. 
Results  (1) Obesity The rate of obesity in boys was higher than that in girls , (P < 0. 05). (2). The rate of staying up late during workday (71.7%) and early rising (83.0%) in obese group was higher than that in girls (83.0%). In obese group, the sleeping time was longer (25.16 ±6.3min) and had no siesta behavior (30.8%). In normal group, 15-30min (38.6%), (P < 0.05). (3) BMI (29.86 ±7.53) in boys was significantly higher than that in girls (26.85 ±5.50), (), while in normal group (38.6%), (P < 0.05). (3), BMI in boys (29.86 ±7.53) was significantly higher than that in girls (26.85 ±5.50), ( P < 0.05). Male students with no siesta behavior and nap time in 15-30 minutes accounted for more (25. 5%), while female students had more lunch break time between 30 and 60 minutes (36.9%). (4). Male students' nap time and sleep duration were negatively correlated with BMI , (P < 0. 05). BMI was proportional to nap time and sleep time, but was inversely proportional to sleep time , (P < 0. 05). 
Conclusions Sleep behavior of adolescent boys and girls may have an effect on body weight status. Staying up late, lack of sleep and lack of lunch time will aggravate obesity and reduce sleep barrier. In addition, shortening sleep time can improve overweight and obesity status.
ER  - 

BibTeX

@misc{458d87846c929d3a1a35e249142f955839c99ea3,
  author = {D. Dijk},
  title = {Sleep and metabolism: you sleep what you eat?},
  journal = {Journal of Sleep Research},
  year = {2014},
  volume = {23},
  doi = {10.1111/jsr.12129},
  abstract = {Sleep is generated by the brain, but sleep certainly does not only impact brain function. Several epidemiological studies and interventional studies have provided evidence for an association between sleep parameters and sleep duration, and outcomes such as body mass index and cardiovascular risk factors. However, how strong and clinically significant are these associations, and what happens to sleep if we change energy balance? Are there associations between what we eat and how we sleep? These questions are addressed in several papers concerning the interactions of sleep and metabolism in the first issue of the Journal of Sleep Research in 2014.}
}

RIS

TY  - JOUR
AU  - D. Dijk
T1  - Sleep and metabolism: you sleep what you eat?
JO  - Journal of Sleep Research
PY  - 2014
VL  - 23
DO  - 10.1111/jsr.12129
AB  - Sleep is generated by the brain, but sleep certainly does not only impact brain function. Several epidemiological studies and interventional studies have provided evidence for an association between sleep parameters and sleep duration, and outcomes such as body mass index and cardiovascular risk factors. However, how strong and clinically significant are these associations, and what happens to sleep if we change energy balance? Are there associations between what we eat and how we sleep? These questions are addressed in several papers concerning the interactions of sleep and metabolism in the first issue of the Journal of Sleep Research in 2014.
ER  - 

BibTeX

@misc{f81a8e766dbdef3e3f1c97c6bcd9b2b11475501c,
  author = {V. Dadaeva and A. Aleksandrov and A. Orlova and O. Drapkina},
  title = {Sleep and Obesity: Mechanisms of Association},
  journal = {Rational Pharmacotherapy in Cardiology},
  year = {2020},
  doi = {10.20996/1819-6446-2020-08-10},
  abstract = {Epidemiologic studies demonstrate that both prolonged and reduced sleep duration is associated with increased risk of excessive weight and obesity. The aim of the review was to analyze probable mechanisms of association of sleep duration and obesity elucidated in current scientific literature. Several proposed mechanisms of such an association exist: an imbalance of appetite regulating hormones resulting from decreased sleep duration; fatigue or decreased activity during the daytime, leading to sedentary behavior with decreased energy expenditure; changes in eating behavior with increased daily calorie intake. The article gives a comprehensive review of factors, mediating the association of sleep duration and obesity (age, gender, ethnic origin), studies of neurohormonal regulation of sleep in association with obesity (the influence of sleep duration on thermoregulation, appetite center – increased grelin-to-leptin ratio); the relationship of sleep with growth hormone and insulin-like growth factor-1, with hypothalamic-pituitary-adrenal axis, and non-hormonal factors, stimulating food intake. Data indicating that increased sleep duration is often associated with decreased sleep quality are presented. Besides traditional risk factors – unhealthy diet and decreased physical exercise, specific attention should be given to the problems, associated with sleep disorders to increase the efficacy of treatment and prevention of obesity.}
}

RIS

TY  - JOUR
AU  - V. Dadaeva
AU  - A. Aleksandrov
AU  - A. Orlova
AU  - O. Drapkina
T1  - Sleep and Obesity: Mechanisms of Association
JO  - Rational Pharmacotherapy in Cardiology
PY  - 2020
DO  - 10.20996/1819-6446-2020-08-10
AB  - Epidemiologic studies demonstrate that both prolonged and reduced sleep duration is associated with increased risk of excessive weight and obesity. The aim of the review was to analyze probable mechanisms of association of sleep duration and obesity elucidated in current scientific literature. Several proposed mechanisms of such an association exist: an imbalance of appetite regulating hormones resulting from decreased sleep duration; fatigue or decreased activity during the daytime, leading to sedentary behavior with decreased energy expenditure; changes in eating behavior with increased daily calorie intake. The article gives a comprehensive review of factors, mediating the association of sleep duration and obesity (age, gender, ethnic origin), studies of neurohormonal regulation of sleep in association with obesity (the influence of sleep duration on thermoregulation, appetite center – increased grelin-to-leptin ratio); the relationship of sleep with growth hormone and insulin-like growth factor-1, with hypothalamic-pituitary-adrenal axis, and non-hormonal factors, stimulating food intake. Data indicating that increased sleep duration is often associated with decreased sleep quality are presented. Besides traditional risk factors – unhealthy diet and decreased physical exercise, specific attention should be given to the problems, associated with sleep disorders to increase the efficacy of treatment and prevention of obesity.
ER  - 

BibTeX

@misc{99c5b68b88c9a0c59fb49019fa6709fe0a8f8c34,
  author = {Jiangshan He and Yaqi Fan and Li Zhang and Chun-jiang Li and Fenghua Guo and Jiahui Zhu and Pei Guo and Binbin Zhang and Mianzhi Zhang and Minying Zhang},
  title = {Habitual night sleep duration is associated with general obesity and visceral obesity among Chinese women, independent of sleep quality},
  journal = {Frontiers in Public Health},
  year = {2023},
  volume = {11},
  doi = {10.3389/fpubh.2023.1053421},
  abstract = {Purpose Research on the relationship between sleep duration and obesity defined using multiple anthropometric and bioelectrical indices in women remains scarce. We aimed to explore the association between sleep duration and body mass index (BMI), waist-hip ratio (WHR), body fat percentage (PBF) and visceral fat area (VFA) among females. Methods We recruited women for medical examination using multistage cluster sampling. Sleep was assessed using Pittsburgh Sleep Quality Index (PSQI) and sleep duration was categorized into short (<7 h), optimal (7 <9 h) and long sleep (≥ 9 h). Weight and height were measured using a calibrated stadiometer. Waist circumference was manually measured. PBF, and VFA were estimated by bioelectrical impedance analysis. Data on sociodemographic characteristics and lifestyle factors were also collected and included in the logistic regression models to explore the independent association between sleep duration and obesity defined by different indices. Results A total of 7,763 women with a mean age of 42.6 ± 13.5 years were included. The percentage of women reporting short and long sleep was 10.3 and 13.4% respectively. The mean BMI, WHR, PBF and VFA were 23.07 ± 3.30 kg/m2, 0.78 ± 0.06, 32.23 ± 6.08% and 91.64 ± 35.97cm2, respectively. Short sleep was independently associated with 35% (95% CI: 1.05–1.75) increased odds of general obesity (BMI ≥ 28 kg/cm2), and long sleep was associated with 18% (95% CI: 1.01–1.37) increased odds of visceral obesity (VFA > 100 cm2). No association was observed between sleep deprivation or excessive sleep and high WHR or high PBF. Conclusion In women, short sleep was associated with an increased odds of general obesity, whereas long sleep was associated with an increased odds of visceral obesity. Longitudinal observations are needed to confirm this cross-sectional relationship.}
}